ライフサイエンスコーパス: chromosome 10

1 , GPLD1 on chromosome 6, and JMJD1C-REEP3 on chromosome 10).

2 utions (Swrl-1 on chromosome 1 and Swrl-4 on chromosome 10).

3 (phosphatase and tensin homologue deleted on chromosome 10).

4 N (phosphatase and tensin homolog deleted on chromosome 10).

5 of phosphatase and tensin homolog deleted on chromosome 10.

6 nner and the disease gene localizes to mouse chromosome 10.

7 ation and SNPs): 6 on chromosome 18 and 1 on chromosome 10.

8 carcinoma of the lung to a 15.7 Mb region on chromosome 10.

9 on chromosome 1, and rs1509957 near EGR2 on chromosome 10.

10 n SDF-1 gene spans over 88 kilobase-pairs on chromosome 10.

11 arm of wheat consensus chromosome 1 and rice chromosome 10.

12 only 2 of 12 ungrouped markers hybridized to chromosome 10.

13 or phosphatase and tensin homolog deleted on chromosome 10.

14 lications have impacted on the gene count on chromosome 10.

15 le T-DNA insertion in the mutant, located on chromosome 10.

16 titative trait locus (QTL) located on murine chromosome 10.

17 lotype that shows partial homology to normal chromosome 10.

18 ains on 8q and 17q, and deletions on 17p and chromosome 10.

19 QTLs for thalamus volume were identified on chromosomes 10, 11 and 16.

20 Four regions on chromosomes 10, 11, 16, and 17 contained stable fruit As

21 The rs1619661 variant is on chromosome 10, 132 kilobase (kb) downstream from <em>CXC

22 Four additional regions on chromosomes 10, 15 and 16 showed suggestive association

23 enically conserved in three regions on mouse chromosomes 10, 16 and 17.

24 for the early to middle elongation stage on chromosome 10, 3 islands for the middle to late elongati

25 (phosphatase and tensin homologue deleted on chromosome 10), a known tumour suppressor, across tumour

26 N (phosphatase and tensin homolog deleted on chromosome 10), a recently discovered tumor suppressor g

27 N (phosphatase and tenson homolog deleted on chromosome 10), a tumor suppressor that antagonizes the

28 Horn type maps to chromosome 10, a location similar to that previously ide

29 it was possible to map and identify Hrml1 on chromosome 10, a locus responsible for modulating the HR

30 Loss of chromosome 10, a predominant genetic change, was associa

31 phosphatase and tensin homologue deleted on chromosome 10, a tumor suppressor protein that antagoniz

32 phosphatase and tensin homolog deleted from chromosome 10], a phosphatase and critical tumor suppres

33 Maize Abnormal chromosome 10 (Ab10) contains a classic meiotic drive sy

34 Maize contains the abnormal chromosome 10 (Ab10) drive system that transforms typica

35 cluded the structurally polymorphic abnormal chromosome 10 (Ab10) in our analysis.

36 The meiotic drive system on maize abnormal chromosome 10 (Ab10) is contained within a terminal doma

37 f the meiotic drive system on maize abnormal chromosome 10 (Ab10) that causes preferential segregatio

38 nd PTEN (phosphatase and tensin homologue on chromosome 10) activities.

39 phosphatase and tensin homologue deleted on chromosome 10 activity.

40 N (phosphatase and tensin homolog deleted on chromosome 10) activity and transcription by palmitic ac

41 N (phosphatase and tensin homolog deleted on chromosome 10) activity was significantly elevated and R

42 The whole chromosome 10 addition is available only in haploid oat

43 hoblastic leukemia-1 (ALL-1)-fused gene from chromosome 10 (AF10), ZFP-1, or tumor suppressor Rb, to

44 se-1/phosphate and tensin homolog deleted on chromosome 10/Akt) and p53 pathways, which converge on t

45 ametric linkage score of 4.27 at 107.2 cM on chromosome 10, although it did not attain genome-wide si

46 a previously unnoticed gene cluster in mouse chromosome 10 and a number of new genes, including mamma

47 ent of single base changes between the human chromosome 10 and chimpanzee sequence revealed nonsense

48 H1, 1p/19q loss, EGFR amplification, loss of chromosome 10 and chromosome arm 10q, gain of chromosome

49 ich lies in close proximity to pten on human chromosome 10 and encodes a 20-kDa nuclear protein.

50 al brain tumors--often have both monosomy of chromosome 10 and gains of the epidermal growth factor r

51 -aminotransferase (ALT) (CPN1-ERLIN1-CHUK on chromosome 10 and PNPLA3-SAMM50 on chromosome 22), one l

52 The l2 locus was mapped to the long arm of chromosome 10 and positional cloning revealed the existe

53 Since loss of chromosome 10 and PTEN are common events in cancer, this

54 ling-microarray analysis of the 20 OsWAKs on chromosome 10 and reverse transcription-PCR analysis for

55 the promoter region of the IFN-gamma gene on chromosome 10 and the regulatory regions of the T(H)2 cy

56 The SNPs on chromosomes 10 and 13 lie in or near genes involved in p

57 he mechanism behind it; however, two SNPs on chromosomes 10 and 13 may be useful markers of delayed p

58 an interaction was observed between loci on chromosomes 10 and 19.

59 N (phosphatase and tensin homolog deleted on chromosome 10) and PP1alpha phosphatases.

60 N (phosphatase and tensin homolog deleted on chromosome 10) and SHIP-1 (Src homology [SH2] domain-con

61 N (phosphatase and tensin homolog deleted on chromosome 10) and the androgen receptor (AR) play impor

62 chromosome 11, PHYB gene is in A-sub-genome chromosome 10, and HY5 gene is in D-sub-genome chromosom

63 Loss of 1p36 and 19q13, 17p13, chromosome 10, and p53 mutation were significantly assoc

64 10509852), 400 kb upstream of SORCS1 gene on chromosome 10, and the global trait of abnormal WM micro

65 atellites to genes on the short arm of human chromosome 10, and used this to generate 1.4 Mb of finis

66 for chromosomes 8, 15, and 17; monosomy for chromosome 10; and amplification of the distal region of

67 ent genes impacted by broad regional loss of chromosome 10 are tumor suppressors capable of affecting

68 N (phosphatase and tensin homolog deleted on chromosome 10) are associated with the multihamartomatou

69 N (phosphatase and tensin homolog deleted on chromosome 10) are found in sporadic cancers and Cowden

70 ions of individual maize chromosomes 1-9 and chromosome 10 as a short-arm telosome.

71 al phosphatase and tensin homolog deleted on chromosome 10 as well as increased phosphorylation/inact

72 Novel regions were identified on chromosome 10 associated with LA (rs10740118; P=8.1x10(-

73 howed a lod score of 3.56 at theta = 0.00 on chromosome 10 at area q25.

74 We also obtained weak evidence of linkage to chromosome 10 at the same location as a previous report

75 Chromosome 10 breakpoints were mapped by fluorescence in

76 hanges in the expression of genes located on chromosome 10 but also with genome-wide differences in g

77 N (phosphatase and tensin homolog deleted on chromosome 10) by cell surface receptors has not been de

78 describe detailed studies of a large knob on chromosome 10 called K10L2.

79 (phosphatase and tensin homology deleted on chromosome 10) cause Cowden and Bannayan-Riley-Ruvalcaba

80 A significant QTL was mapped to chromosome 10 (Chr 10) and confirmed using B6.A<10> mice

81 pes (B6D2, D2B6) with the proximal region of chromosome 10 (Chr10) introgressed into opposing backgro

82 The finished sequence of human chromosome 10 comprises a total of 131,666,441 base pair

83 Of particular interest is a locus on chromosome 10 containing the autophagy-related protein 1

84 reviously mapped PPCD to a region (PPCD3) on chromosome 10 containing the gene that encodes the two-h

85 pe Tsc2 allele while retaining two copies of chromosome 10 containing the mutant Tsc2 allele along wi

86 gh-density mapping and annotation studies of chromosome 10, contig 395, showed that the De18 locus wa

87 using progeny testing confirmed that sov1 on chromosome 10 controlled obovoid and elongated shape, wh

88 hosphatase and tensin homolog gene (PTEN) on chromosome 10 controls the first step in the phosphatidy

89 KL-40 was significantly associated with both chromosome 10 copy number loss and poorer survival.

90 earance of a clone containing trisomy 12 and chromosome 10 copy-neutral loss of heterogeneity that ma

91 tumor suppressor mechanisms associated with chromosome 10 deletions.

92 ely, PTEN (phosphatase and tensin homolog on chromosome 10) dephosphorylates PtdIns(3,4,5)P3 and nega

93 Moreover, phosphate tensin homolog on chromosome 10 depletion in human kidney-2 cells resulted

94 mors is correlated with the frequent loss of chromosome 10 during progression of gliomas.

95 N (phosphatase and tensin homolog deleted on chromosome 10) during neurite outgrowth of human embryon

96 omplement factor H (CFH) gene and a locus on chromosome 10 encompassing the HTRA1/LOC387715/ARMS2 gen

97 (phosphatase and tensin homologue deleted on chromosome 10) enhance cell migration, yet the underlyin

98 tumor (Ept)1], chromosome 3 (Ept2 and Ept6), chromosome 10 (Ept9), and chromosome 1 (Ept10 and Ept13)

99 nergy function using the Hi-C contact map of chromosome 10 from human GM12878 lymphoblastoid cells.

100 of phosphatase and tensin homolog deleted on chromosome 10 function.

101 king PTEN (phosphatase and tensin homolog on chromosome 10) function were relatively resistant to dru

102 N (phosphatase and tensin homolog deleted on chromosome 10) function, increased growth factor signali

103 by phosphatase and tensin homolog deleted on chromosome 10 functional status.

104 hosphatase and tensin homologue deleted from chromosome 10) functions as a tumour suppressor by oppos

105 tified a common good prognostic signature of chromosome 10 genes from two gene expression datasets (T

106 re (BP) quantitative trait loci (QTL) on rat chromosome 10 has been clearly demonstrated by linkage a

107 (phosphatase and tensin homologue deleted on chromosome 10) has been shown to be inactivated in a wid

108 and allelic association in the IDE region of chromosome 10 have been reported in families with late-o

109 s with several biological candidate genes on chromosome 10 have been reported, these findings have no

110 bular inflammation included SNP rs1227756 on chromosome 10 in COL13A1 (P = 2.0 x 10(-7)), rs6591182 o

111 ichment of differentially regulated genes on chromosome 10 in the course of validating RNA-Seq result

112 tion between type 2 diabetes and variants on chromosome 10 in the Framingham SHARe data.

113 N (phosphatase and tensin homolog located on chromosome 10) in cancer has surpassed all predictions a

114 Loci on chromosome 10 include MSMB, which encodes beta-microsemi

115 PTEN (phosphatase/tensin homolog on chromosome 10)-induced putative kinase 1 (PINK1), a puta

116 we report the discovery of a large region on chromosome 10 involved in adaptation or domestication th

117 Thus, phosphate tensin homolog on chromosome 10 is an upstream regulator of renal PPM1A de

118 mutation in a second gene, Slc35d3, on mouse chromosome 10 is the basis of this discrepancy.

119 showed diminished paternal transmission, and chromosome 10 is transmitted to offspring only as a shor

120 N (phosphatase and tensin homolog deleted on chromosome 10) is a potent tumor suppressor gene frequen

121 N (phosphatase and tensin homolog deleted on chromosome 10) is a proven critical tumor suppressor.

122 (phosphatase and tensin homologue deleted on chromosome 10) is a tumor suppressor that is mutated or

123 PTEN (phosphatase and tensin homolog on chromosome 10) is a tumor suppressor whose cellular regu

124 phosphatase and tensin homologue, deleted on chromosome 10) is a tumor suppressor with dual phosphata

125 (phosphatase and tensin homologue deleted on chromosome 10) is frequently mutated or deleted in vario

126 N (phosphatase and tensin homolog deleted on chromosome 10) is the underlying cause of PTEN hamartoma

127 n (phosphatase and tensin homolog deleted on chromosome 10) is thought to mediate the majority of pro

128 N (phosphatase and tensin homolog deleted on chromosome 10) is well characterized for its role in ant

129 Furthermore, LRRTM3 maps to a region of chromosome 10 linked to both LOAD and elevated plasma Ab

130 hat the genetic linkage of AD in this set of chromosome 10-linked AD families may be the result of sy

131 12 affected and unaffected members of three chromosome 10-linked AD pedigrees in the National Instit

132 therto unknown association between SNPs at a chromosome 10 locus and the severity of NASH fibrosis.

133 The chromosome 10 locus is particularly promising given that

134 investigations on the ATG18 and genes at the chromosome 10 locus may provide an important lead for a

135 s (Ho) to a approximately 7.4 cM interval on chromosome 10 (LOD=8.78).

136 On chromosome 10 maximal logarithm of odds (LOD) scores of

137 onserved with three regions located on mouse chromosome 10 (Mmu10), Mmu16 and Mmu17.

138 egions in the mouse genome, located on mouse chromosome 10 (Mmu10), Mmu16, and Mmu17.

139 phosphatase and tension homologue deleted on chromosome 10), modestly enhancing basal Akt activity, d

140 phosphatase and tensin homologue deleted on chromosome 10 mutations or deletions were also resistant

141 pe I (Ab10-I) does not recombine with normal chromosome 10 (N10) over an approximately 32-cM terminal

142 Three SNPs on chromosome 10 near the CHUK (conserved helix-loop-helix

143 me 16 (the top meta-analysis SNP) and one on chromosome 10 (not reported by RHM or in the meta-analys

144 Four SNPs on chromosomes 10 (one), 13 (two), and 14 (one) were signif

145 (phosphatase and tensin homologue deleted on chromosome 10), one of the most important tumor suppress

146 ites was observed on chromosome 9 but not on chromosome 10 or 19.

147 Starting from a single donor locus on chromosome 10, over 1500 elements were distributed throu

148 N (phosphatase and tensin homolog deleted on chromosome 10) phosphatase represents a novel switch bet

149 Loss of tumor suppressor genes on chromosome 10 plays an important role in the development

150 N (phosphatase and tensin homolog deleted on chromosome 10) plays important roles in tumor developmen

151 A single SNP, rs10824026 (chromosome 10: position 73661450), was found to signific

152 phosphatase and tensin homologue deleted on chromosome 10 protein, whereas the mass of phosphorylate

153 he tumor suppressor ANXA7 due to monosomy of chromosome 10 provides a clinically relevant mechanism t

154 ncRNA gene lincRNA-Tnfaip3, located at mouse chromosome 10 proximal to the tumor necrosis factor alph

155 phosphatase and tensin homolog deleted from chromosome 10 (Pten) (either as heterozygotes or by cond

156 1 and/or ptase and tensin homolog deleted on chromosome 10 (PTEN) (such as Jurkat, CEM, Molt) and, co

157 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) abnormal HNSCC cell line FaDu, inhi

158 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) activity.

159 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) and activation of the downstream pr

160 es phosphatase and tensin homolog deleted on chromosome 10 (Pten) and androgen upregulated gene 19 (U

161 or phosphatase and tensin homolog deleted on chromosome 10 (PTEN) and decreases its phosphorylation.

162 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) and glycogen synthase kinase beta (

163 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) and p53 tumor suppressors.

164 ted phosphatase and tensin homolog delete on chromosome 10 (PTEN) and promoted the phosphoinositide 3

165 ne phosphatase and tensin homolog deleted on chromosome 10 (Pten) are associated with multiple cancer

166 in phosphatase and tensin homolog deleted on chromosome 10 (PTEN) are implicated in neuropsychiatric

167 ty phosphatase and tensin homolog deleted on chromosome 10 (PTEN) blocks PI3K signaling by dephosphor

168 nd phosphatase and tensin homolog deleted on chromosome 10 (PTEN) by all 3 HexAbs and the notable dif

169 of phosphatase and tensin homolog deleted on chromosome 10 (PTEN) confers sensitivity to N-methyl-N'-

170 in phosphatase and tensin homolog deleted in chromosome 10 (PTEN) contributes to renal cell hypertrop

171 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) down-regulates phosphorylation of p

172 or phosphatase and tensin homolog deleted on chromosome 10 (Pten) elicits a senescence response that

173 hosphatase and tensin homologue deleted from chromosome 10 (Pten) encodes a lipid phosphatase control

174 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN) encodes a tumor-suppressor phosphat

175 in phosphatase and tensin homolog deleted on chromosome 10 (PTEN) experience autoimmunity and lymphoi

176 nd phosphatase and tensin homolog deleted on chromosome 10 (PTEN) function to block growth factor-ind

177 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) gene in a p38-dependent manner, but

178 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) gene is often altered in prostate c

179 sphatase and tensin homologue deleted on the chromosome 10 (PTEN) gene was significantly up-regulated

180 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) has inhibitory effects on the PI3K/

181 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) in AD pathogenesis have not been ex

182 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) in AMs, we attenuated the inhibitor

183 -phosphatase and tensin homologue deleted on chromosome 10 (PTEN) in Fc gamma R-induced macrophage fu

184 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) in repressing the protumorigenic ef

185 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) in the regulation of PDGF expressio

186 nd phosphatase and tensin homolog deleted on chromosome 10 (PTEN) induces activation of the phospho-5

187 ic phosphatase and tensin homolog deleted on chromosome 10 (PTEN) inhibitor SF1670.

188 Phosphatase and tensin homologue deleted on chromosome 10 (PTEN) is a dual-function phosphatase with

189 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN) is a lipid phosphatase implicated i

190 Phosphatase and tensin homologue on chromosome 10 (PTEN) is a lipid phosphatase that, by ant

191 Phosphatase and tensin homologue deleted on chromosome 10 (PTEN) is a lipid phosphatase.

192 Phosphatase and tensin homologue deleted on chromosome 10 (PTEN) is a phosphatidylinositol phosphate

193 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN) is a tumor suppressor that antagoni

194 Phosphatase and tensin-homolog deleted on chromosome 10 (PTEN) is a tumor-suppressor protein that

195 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN) is an important negative regulator

196 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN) is an important negative regulator

197 hosphatase and tensin homologue deleted from chromosome 10 (Pten) is expressed aberrantly in non-smal

198 f phosphatase and tensin homology deleted on chromosome 10 (PTEN) is linked to increased PI3K-AKT sig

199 Phosphatase and tensin homologue deleted on chromosome 10 (PTEN) is mutated or lost in 60% to 70% of

200 or phosphatase and tensin homolog deleted on chromosome 10 (PTEN) is strongly correlated with human l

201 on phosphatase and tensin homolog deleted on chromosome 10 (PTEN) is the second most frequently mutat

202 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) lacking lipid (G129E) or lipid and

203 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) levels, or the downregulation of PI

204 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) localization in the nucleus and cyt

205 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) loss regulated protein kinase B (AK

206 nd phosphatase and tensin homolog deleted on chromosome 10 (PTEN) negatively regulate this insulin si

207 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN) negatively regulates phosphatidylin

208 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) or have reduced PTEN expression thr

209 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) overexpression down-regulated PH do

210 Phosphatase with TENsin homology deleted in chromosome 10 (PTEN) phosphatase and a PI3K activating p

211 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) plays a critical role in the mainte

212 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) plays a pivotal role in various cel

213 re phosphatase and tensin homolog deleted on chromosome 10 (PTEN) protein when compared with CD56(dim

214 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) regulates innate immune responses i

215 he Phosphatase And Tensin Homolog Deleted On Chromosome 10 (PTEN) regulates the phosphoinositol-3-kin

216 sor gene phosphatase and tensin homologue on chromosome 10 (PTEN) result in elevated levels of phosph

217 or phosphatase and tensin homolog deleted on chromosome 10 (PTEN) through its interaction with a hist

218 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) tumor suppressor gene, one of the m

219 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) tumor suppressor is a phosphatase t

220 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) tumor suppressor is frequently phos

221 phosphatase and tensin homolog deleted on chromosome 10 (PTEN) tumor suppressor is the major brake

222 he phosphatase and tensin homolog mutated on chromosome 10 (PTEN) tumor-suppressor gene.

223 of phosphatase and tensin homolog deleted on chromosome 10 (PTEN) was observed both in fluorescence a

224 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) were coordinately expressed, with c

225 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) were detected in ULTH cells.

226 Phosphatase and tensin homologue deleted on chromosome 10 (PTEN) with shRNA in three estrogen recept

227 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN), a multifunctional tumor suppressor

228 hosphatase and a tensin homolog deleted from chromosome 10 (PTEN), a negative regulator of the phosph

229 phosphatase and tension homologue deleted on chromosome 10 (PTEN), a negative regulator of the phosph

230 of phosphatase and tensin homolog deleted on chromosome 10 (PTEN), a negative regulator of the PI3K/A

231 phosphatase and tensin homologue deleted on chromosome 10 (PTEN), a phosphatase that dampens phospha

232 of phosphatase and tensin homolog deleted on chromosome 10 (PTEN), a phosphatase that inactivates Akt

233 of phosphatase and tensin homolog deleted on chromosome 10 (PTEN), a phosphatidylinositol 3-phosphata

234 Phosphatase and tensin homologue deleted on chromosome 10 (PTEN), a tumor suppressor phosphatase tha

235 of phosphatase and tensin homolog deleted on chromosome 10 (Pten), activation of AKT, fast proliferat

236 r, phosphatase and tensin homolog deleted on chromosome 10 (PTEN), alters the invasive potential of m

237 regulates phosphatase and tensin deleted on chromosome 10 (PTEN), AMP-activated protein kinase (AMPK

238 hosphatase and tensin homologue deleted from chromosome 10 (PTEN), and this action of oncogenic Ras i

239 phosphatase and tensin homologue deleted on chromosome 10 (PTEN), but also targets Fas ligand (FasL)

240 atase, phosphatase tensin homolog deleted on chromosome 10 (PTEN), has been reported to block insulin

241 ll phosphatase and tensin homolog deleted on chromosome 10 (PTEN), increased cell survival with down-

242 phosphatase and tensin homologue deleted on chromosome 10 (PTEN), inhibits cell growth and survival

243 y, phosphatase and tensin homolog deleted on chromosome 10 (PTEN), is primarily localized in nonraft

244 phosphatase and tensin homologue deleted on chromosome 10 (PTEN), leading to activation of the epide

245 Phosphatase and tensin homologue, deleted on chromosome 10 (PTEN), the major tumor suppressor and key

246 r, phosphatase and tensin homolog deleted on chromosome 10 (PTEN), was identified as a bona fide targ

247 by phosphatase and tensin homolog deleted on chromosome 10 (PTEN)-dependent activation of PI 3-kinase

248 1 (phosphatase and tensin homolog deleted on chromosome 10 (PTEN)-induced kinase 1), a Parkinson's di

249 phosphatase and tensin homologue deleted on chromosome 10 (PTEN).

250 phosphatase and tensin homologue deleted on chromosome 10 (PTEN).

251 hosphatase and tensin homologue deleted from chromosome 10 (PTEN).

252 by phosphatase and tensin homolog deleted on chromosome 10 (PTEN).

253 of phosphatase and tensin homolog deleted on chromosome 10 (PTEN).

254 osphatase, phosphatase and tensin homolog on chromosome 10 (Pten).

255 phosphatase and tensin homologue deleted on chromosome 10 (PTEN)/MMAC1/TEP gene in human cancer cell

256 phosphatase and tensin homologue detected on chromosome 10 (PTEN)/phosphatidylinositol 3-kinase/AKT/T

257 phosphatase and tensin homologue deleted on chromosome 10 (PTEN); assessed apoptosis; and determined

258 phosphatase and tensin homologue deleted on chromosome 10 (PTEN; eg, phosphatidylinositol-3-kinase [

259 et phosphatase-and-tensin-homolog-deleted-on-chromosome-10 (PTEN).

260 s "phosphatase and tensin homolog deleted on chromosome 10" (PTEN), a key regulator of Akt activation

261 phosphatase and tensin homologue deleted on chromosome 10, PTEN.

262 (phosphatase and tensin homologue deleted on chromosome 10) reduces PIP3 levels and leads to fatty li

263 constructed and characterized to fine-map a chromosome 10 region (QTL1) linked to blood pressure.

264 putative familial late-onset AD gene in this chromosome 10 region.

265 N (phosphatase and tensin homolog deleted on chromosome 10) regulates the peripheral homeostasis of T

266 lated retroelements surrounding the locus on Chromosome 10 responsible for double minute chromosome f

267 The Mcs7 region on rat chromosome 10 (RNO10) is orthologous to human 17q, a com

268 to mtDNA levels: one within the TFAM gene on chromosome 10 (rs11006126, p value = 8.73 x 10(-28), var

269 e maternal genotype for an intergenic SNP on chromosome 10 (rs11008222, combined P = 6.3 x 10(-7); re

270 leotide polymorphism in the CYP2C cluster on chromosome 10 (rs12777823) and warfarin dose requirement

271 dence interval=1.05-1.11; P=2.86x10(-8)) and chromosome 10 (rs7920721; closest gene, ECHDC3; odds rat

272 A locus carried on SWR/J chromosome 10 seems to be particularly important in AR f

273 A secondary structure formation on the human chromosome 10 sequence, and utilize this analysis to com

274 (phosphatase and tension homolog deleted on chromosome 10), Ser(380)/Thr(382/383) PTEN phosphorylati

275 We performed a chromosome 10-specific association study with 1,412 gene

276 ocus (QTL) mapping identified a QTL on mouse chromosome 10 that accounts for greater than 50% of the

277 are segments homologous to regions on human chromosome 10 that have been linked to late onset Alzhei

278 on at 16p12, and a novel 1.2-Mb inversion on chromosome 10 that is supported by the presence of two d

279 variant in a kelch domain-containing gene on chromosome 10 that may epistatically modulate artemisini

280 (phosphatase and tensin homolog deleted from chromosome 10), the antagonist of the phosphosphoinosito

281 s identified one locus near the NEK9 gene on chromosome 10 to have a significant effect on crossover

282 N (phosphatase and tensin homolog deleted on chromosome 10) tumor suppressor gene functions that elud

283 N (phosphatase and tensin homolog deleted on chromosome 10) tumor suppressor protein and phosphate-de

284 N (phosphatase and tensin homolog deleted on chromosome 10) tumor suppressor regulates a variety of c

285 (phosphatase and tensin homologue deleted on chromosome 10) tumor suppressor, and Ybr077cp (Nir1p), a

286 (phosphatase and tensin homologue deleted in chromosome 10) tumour suppressor has been associated wit

287 verse strand of the last 11.2 Mb sequence of chromosome 10 was analyzed in detail based on a mathemat

288 de polymorphisms (SNPs) in a novel region on chromosome 10 was genome-wide significant (lowest P=1.3E

289 2 near 60 cM, proximal chromosome 6, and mid-chromosome 10 were distinct from BW, lean tissue, and bo

290 gene for type 2 diabetes to the long arm of chromosome 10, where we have previously identified a qua

291 gments containing noncontiguous sequences of chromosome 10, whereas, in the other two families, the b

292 allele of the gene Columnar (Co) located on chromosome 10 which can appear in a heterozygous (Co/co)

293 Cia5 is a locus on rat chromosome 10 which regulates the severity of collagen-

294 pitulating the fact that loss of one copy of chromosome 10 (which harbors the tumor suppressor PTEN)

295 de phosphatase and tensin homolog deleted on chromosome 10, which has been suggested to mediate S1P(2

296 lzheimer disease (LOAD) has been observed on chromosome 10, which implicates a wide region and at lea

297 these, one major QTL was mapped to bin 10 of chromosome 10, which is 29.7 kb in size and harbors thre

298 hosphatase and tensin homologue deleted from chromosome 10), whose inactivation would shift the equil

299 netic mapping associated a large deletion on chromosome 10 with a quantitative change in seed composi

300 A peak for %DMA on chromosome 10 within 2 Mb of AS3MT had an LOD of 1.80.