ライフサイエンスコーパス: chromosome 5

1 31.1 (a region of conserved synteny to mouse Chromosome 5).

2 approximately 422 kbp, which is one third of chromosome 5.

3 n was mapped to 68.4 +/- 6.0 centimorgans on chromosome 5.

4 e X chromosome, the TAG genes are located on chromosome 5.

5 of H19 and at the alpha-fetoprotein locus on chromosome 5.

6 y to E2-induced mammary cancer on distal rat chromosome 5.

7 ative markers spaced over a 60-cM segment of chromosome 5.

8 y, the human protocadherin alpha cluster, on chromosome 5.

9 -10 gene is located at the 5p12-p13 locus of chromosome 5.

10 a single recessive mutation at the bottom of chromosome 5.

11 d was mapped previously to a region of mouse chromosome 5.

12 omologous with those on the domestic horse's chromosome 5.

13 f orthologous synteny lies on proximal mouse chromosome 5.

14 receptor 2 (TfR2) were cloned and mapped to chromosome 5.

15 ele because of an interstitial deletion from chromosome 5.

16 xe1-1 to axe1-5) mapped to the same locus on chromosome 5.

17 Both mSteap and mPsca map to chromosome 5.

18 dopsis mutant, scarface (sfc), which maps to chromosome 5.

19 ished sequence of a 204-kb region from mouse chromosome 5.

20 y span approximately 40 cM of proximal mouse chromosome 5.

21 ization, we show that Lungkine maps to mouse chromosome 5.

22 east artificial chromosome clone, CIC9E2, on chromosome 5.

23 The MOCO1 locus resides on human chromosome 5.

24 11 and ARC6 map approximately 60 cM apart on chromosome 5.

25 se BACs hybridized to a single site on mouse chromosome 5.

26 compassing the corresponding region on mouse chromosome 5.

27 nant myeloid disorders with abnormalities of chromosome 5.

28 ed to the conserved syntenic region on mouse chromosome 5.

29 terstitial deletion of the long arm of human chromosome 5.

30 K2 probes identified a single locus on mouse Chromosome 5.

31 he conserved syntenic region on distal mouse chromosome 5.

32 ta1 (Gabrb1), and gamma(1) (Gabrg1) on mouse chromosome 5.

33 nning 30 cM of the proximal portion of mouse chromosome 5.

34 4p16.1, the human region syntenic with mouse chromosome 5.

35 fragment length polymorphism marker m291 on chromosome 5.

36 sive D. americana alleles at a single QTL on chromosome 5.

37 n rice chromosomes 11 and 12 and arabidopsis chromosome 5.

38 rated to construct a comprehensive RH map of chromosome 5.

39 of AML/MDS characterized by abnormalities of chromosome 5.

40 ion of reduced recombination on B. decumbens chromosome 5.

41 e to map the disease gene in two families to chromosome 5.

42 a screen for ENU-induced mutations on mouse chromosome 5.

43 at the telomere to a full-length homolog of chromosome 5.

44 Quantitative trait loci on chromosomes 5, 10, 17, 18, and X were found to control a

45 mosome 18, with weaker effects detectable on chromosomes 5, 12, and 14.

46 We report in detail on baboon chromosomes 5, 12, and 18 for which the linkage maps are

47 this disease are located on the short arm of chromosome 5(1H).

48 ive or proliferative retinopathy occurred on chromosomes 5 (2.53 at 11.2 cM), 6 (2.28 at 30.6), and 1

49 islands associated with fiber initiation on chromosome 5, 3 islands for the early to middle elongati

50 with the predicted number of genes on wheat chromosome 5D (4286).

51 hromosome 5 or a deletion of the long arm of chromosome 5, -5/del(5q), is a recurring abnormality in

52 transcription factor located in a region of chromosome 5 (5p15.3) that has been proposed to contain

53 kage analyses revealed suggestive linkage on chromosome 5 (5q22) with a logarithm of odds (LOD) score

54 ic (heterozygous) markers were identified on chromosome 5, 6 to the left and 12 to the right of the M

55 of BXD strain survival identified linkage on chromosomes 5, 6, 9, 11, and 14.

56 lusion, we determined regions of interest on chromosomes 5, 6, and 10 where imprinted genes might be

57 Three other regions, on chromosomes 5, 6, and 15, that were nominally significan

58 Abnormalities of chromosome 5, 7, or both accounted for 76% of all cases

59 Chromosomes 5, 7, 10, and 14 were found to contain signi

60 ed linkage disequilibrium (LD) maps of human chromosomes 5, 7, 17, and X.

61 iking were the increases in abnormalities of chromosomes 5, 7, and 17 among the elderly.

62 andom interstitial deletions and monosomy of chromosomes 5, 7, and 17 in refractory myelodysplasia (M

63 enetic abnormalities, including deletions of chromosomes 5, 7, or both.

64 inkage to autism-spectrum disorders (ASD) on chromosomes 5, 8, 16, 19, and X and showing nominal evid

65 , and population-specific GWS for markers on chromosomes 5, 9 and 19.

66 nificant and suggestive evidence for loci on chromosomes 5, 9, 11, 15, 16 and 19, which influence hyp

67 We observed clustered loss of chromosomes 5, 9, and 16, orthologous to a similar patte

68 somes 1, 3, 11, 14, 17, and 18 and losses on chromosomes 5, 9, and 16.

69 3 open reading frames (ORFs) on one circular chromosome (5,928,787 bp) and two plasmids (131,950 bp a

70 e epiregulin gene (Ereg) is located on mouse chromosome 5 adjacent to three other epidermal growth fa

71 Although partially fertile, these chromosome 5 and 10 addition plants have not yet transmi

72 ause cytogenetic studies frequently identify chromosome 5 and 17 deletions within a single clone, we

73 Unlike in primary MDS, aberrancies of chromosome 5 and 20 were infrequent.

74 a marker of adverse prognosis independent of chromosome 5 and 7 deletions.

75 ealed that a single copy of the mdr1 gene on chromosome 5 and a novel amplification of the plasmepsin

76 lenalidomide for patients with alteration of chromosome 5 and anemia.

77 alization of genes on the short arm of human chromosome 5 and as a framework for both generating and

78 etween wheat consensus chromosome 1 and rice chromosome 5 and between the proximal portion of the lon

79 entation of four contigs of Candida albicans chromosome 5 and determined the sizes of three gaps betw

80 ional candidate genes in a linkage region on chromosome 5 and genotyped selected variants in several

81 e is enhanced by a second locus that maps to chromosome 5 and heightened by additional genetic modifi

82 the conservation of synteny between macaque chromosome 5 and human chromosome 4.

83 mposed of 31 exons spanning >100 kb on mouse chromosome 5 and is located between Cyln2 and Gtf2i.

84 hat are trisomic for 90% of the short arm of chromosome 5 and monosomic for a small distal portion of

85 d in close proximity to gp130 genes on human chromosome 5 and mouse chromosome 13.

86 scoring identified QTL on a unique region of chromosome 5 and on chromosome 11.

87 7/Q7Z4C4 on chromosome 5, rs1445898/CAPSL on chromosome 5 and rs2302188/NM_033543 on chromosome 19),

88 es showed significant evidence of linkage to chromosome 5 and suggestive evidence of linkage to chrom

89 k2beta that are encoded by distinct genes on chromosome 5 and that are ubiquitously expressed in matu

90 between genes linked to the targeted CD38 on Chromosome 5 and the ART2 complex on Chromosome 7.

91 nalysis of the GABA(A) gene cluster on mouse chromosome 5 and the corresponding region on human chrom

92 been identified from the cytokine cluster on chromosome 5 and the MHC on chromosome 6.

93 The UVH1 locus was mapped to chromosome 5 and the position of the UVH1 gene was furth

94 apped two additional mutations to the distal chromosome 5 and the proximal chromosome 10 in a second

95 ies]: HR, 2; P < .001), and abnormalities of chromosome 5 and/or 7 (HR, 1.89; P < .001).

96 bnormality associated with secondary MDS/AL (chromosome 5 and/or 7 abnormality).

97 The gene was mapped to the short arm of chromosome 5D and mediated recognition of the effector S

98 minor effect was mapped on the short arm of chromosome 5S and was designated QPh.ucd-5S.

99 ric duplicated segments shared between mouse chromosomes 5 and 1.

100 Monosomic additions for maize chromosomes 5 and 10 to a haploid complement of oat (n =

101 Chromosomes 5 and 10 yielded lod scores for linkage to b

102 Two of these loci, Hpi1 and Hpi2 on mouse chromosomes 5 and 13, were mapped to the significant and

103 ifiers of L. monocytogenes susceptibility to chromosomes 5 and 13.

104 ethod to detect interactions between SNPs on chromosomes 5 and 15 on lung cancer data.

105 Cosegregating deletions of chromosomes 5 and 17 were found to specifically include

106 Survival mapped to chromosomes 5 and 17, anti-chromatin Ab to chromosomes 4

107 een ancestral chromosomes, syntenic to human chromosomes 5 and 17, respectively.

108 L contributing to variation in gut length on chromosomes 5 and 18.

109 18, and 20, and secondary loci were found on chromosomes 5 and 19.

110 cloned and assigned to conserved regions of chromosomes 5 and 2, respectively.

111 alized HBECs to have duplication of parts of chromosomes 5 and 20.

112 acid identity) are encoded by genes on mouse chromosomes 5 and 6 and, together with the Tmhs-encoded

113 that the duplications associated with mouse chromosomes 5 and 6 are recent and that the resulting du

114 Twenty microsatellite markers spanning chromosomes 5 and 6 at an average distance of 20 cM were

115 In this study, we determined allelic loss on chromosomes 5 and 6 in 29 primary early-stage epithelial

116 pping and sequencing of the regions of mouse chromosomes 5 and 6 involved in this chromosome-fission

117 enes are located on five distinct regions on chromosomes 5 and 6, i.e., 5p15.2, 5q13-21, 6p24-25, 6q2

118 ts with a poor-prognosis karyotype involving chromosomes 5 and 7 (CR, 71%) and secondary MDSs (CR, 72

119 hromosome 5 (n = 63), chromosome 7 (n = 85), chromosomes 5 and 7 (n = 66), recurring balanced rearran

120 ells showed that only one of the two HORs on chromosomes 5 and 7 incorporate CENP-A, an organization

121 in offspring and identify loci on offspring chromosomes 5 and 7 that modify maternal behaviour.

122 ultiple cytogenetic aberrations, commonly on chromosomes 5 and 7, suggesting an association with prev

123 omplex, with abnormalities in either or both chromosomes 5 and 7.

124 togenetics, especially abnormalities in both chromosomes 5 and 7.

125 pattern was principally in abnormalities in chromosomes 5 and 7.

126 cant genomewide P values <.05 for markers on chromosomes 5 and 8 and with suggestive linkage evidence

127 inkage studies that have reported linkage to chromosomes 5 and X.

128 mia patients who had clonal abnormalities of chromosomes 5 and/or 7, 7 (16%) were homozygous for the

129 h radiotherapy, and in patients with loss of chromosomes 5 and/or 7, acquired abnormalities associate

130 , in particular, complete or partial loss of chromosomes 5 and/or 7.

131 among those with t-AML or an abnormality of chromosomes 5 and/or 7.

132 ave a rough map position on the upper arm of chromosome 5, and deep sequencing of DNA from these 13 l

133 complex etiology with significant linkage to chromosome 5, and marginal linkage was observed to five

134 NRH1 maps on chromosome 5, and NRH2 and NRH3 are less than 48kb apart

135 s, 3 SNPs (rs2540317 in MFSD9, rs10515353 on chromosome 5, and rs2242400 in BCAT1 were associated wit

136 tly localized to 1.0, 7.5, and 4.5 Mb of rat chromosome 5, and the orthologous regions are on human c

137 Nonrandom, complete and partial deletions of chromosome 5 are common anomalies in AML.

138 Complete loss or interstitial deletions of chromosome 5 are the most common karyotypic abnormality

139 (GENDEP) identified an intergenic region on chromosome 5 associated with early improvement after 2 w

140 .27), chromosome 4 at 135.3 cM (LOD = 2.63), chromosome 5 at 139.3 cM (LOD = 0.84), chromosome 6 at 1

141 n situ hybridization to the proximal half of chromosome 5 at bands q2.2-q2.3.

142 ion analysis localized the FYB gene to human chromosome 5 at position p13.1.

143 e carbamoyl phosphate synthetase I locus, on chromosome 5 at the alanine-glyoxylate aminotransferase

144 The KLHL3 gene is mapped to human chromosome 5, band q31, contains 17 exons, and spans app

145 h-resolution map of a 6-Mb interval of human chromosome 5, band q31, incorporating 175 sequence tagge

146 The RAB6KIFL gene is mapped to human chromosome 5, band q31, spans approximately 8.5kb of gen

147 tag clones within a genomic contig on human chromosome 5, band q31.

148 et identified a novel variant (rs7726475) on chromosome 5 between the SUB1 and NPR3 genes, as being a

149 zed to a 0.8-cM interval on the short arm of chromosome 5, between the polymorphic microsatellite mar

150 struct a map of 54 markers located on bovine chromosome 5 (BTA5).

151 elphia chromosome (Ph) positive (4 cases: 44 chromosomes; 5 cases: 40-43 chromosomes) and were not co

152 genomic orientation of this cluster on mouse chromosome 5: cen-D5Mit151-Gabrg1-Gabra2-Gabrb1-D5Mit58-

153 rce L-sorbose, by the reversible loss of one chromosome 5 (Ch5).

154 We examined CNV in a region of chromosome 5 (chr5) in haploid and diploid strains of Sa

155 LDL with significant linkage is detected on chromosome 5, chr5-104 Mbp (LOD 3.7).

156 f murine I-TAC (gene symbol Scyb11) to mouse chromosome 5 close to the genes for monokine induced by

157 In reciprocal chromosome 5 congenic mice, introgressed D2 alleles incr

158 t clones, suggesting that certain regions of chromosome 5 contain haploinsufficient developmental gen

159 ongenic strain, in which a smaller region of chromosome 5 containing the CYP4A genes from a Lewis rat

160 Transfer of chromosome 5 containing the CYP4A genes responsible for

161 The short arm of human chromosome 5 contains approximately 48 Mb of DNA and com

162 ximately 209-kbp portion of the right arm of chromosome 5 contains at least five spatially separated,

163 aucoma and provides additional evidence that chromosome 5 contains susceptibility loci for glaucoma i

164 features, which shows a promising signal on chromosome 5 corresponding to SERINC5, a protein involve

165 A heterozygous deletions of the long arm of chromosome 5 [del(5q)], observed in 40% of patients, is

166 Deletions of the long (q) arm of chromosome 5 [del(5q)]occur in patients with myelodyspla

167 Interstitial deletion or loss of chromosome 5, del(5q) or -5, is a frequent finding in my

168 itial loss of all or part of the long arm of chromosome 5, del(5q), is a hallmark of myelodysplastic

169 Partial deletion of the long arm of chromosome 5, del(5q), is the cytogenetic hallmark of th

170 An interstitial deletion of chromosome 5, del(5q), is the most common structural abn

171 e 'CNA-devoid' subgroup and a basal-specific chromosome 5 deletion-associated mitotic network.

172 The frequency of unbalanced chromosome 5 deletions has led to the idea that 5q harbo

173 The human MACROH2A1 gene, on chromosome 5, encodes two MACROH2A subtypes, MACROH2A1.1

174 me (BAC)-based map of a 5-Mb region on mouse Chromosome 5, encompassing three gene families: receptor

175 ukemic cell line, ML3, is diploid for all of chromosome 5, except for an inversion-coupled translocat

176 ultiple redundant regulators scattered along chromosome 5 explain, in a simple, elegant way, why the

177 QTLs were discovered near the centromere of chromosome 5 (Eye1: genomewide P < 0.005) and on proxima

178 d delineation of a 1.1-Mb critical region on chromosome 5 for the gene mutated in CdLS.

179 lines identified a dominant genetic locus on chromosome 5 from the wild relative that affected total

180 largest factor (0.30 variance) included the chromosome 5 gene cluster that encodes the most common G

181 ned the position of tlt relative to 17 mouse chromosome 5 genes with orthologous loci in the human 4p

182 Although chromosome 5 has been implicated in previous linkage stu

183 eterogeneity lod = 2.36, dominant model) and chromosome 5 (heterogeneity lod = 2.23, recessive model)

184 ide p < 0.005, between 65 and 100 cM) and to chromosome 5 (Hipp5a, p < 0.05, between 15 and 40 cM).

185 or homozygosis in one offspring, loss of one chromosome 5 homolog followed by duplication of the reta

186 combination, gene conversion, or loss of one chromosome 5 homolog, followed by duplication of the ret

187 spring, which arise primarily by loss of one chromosome 5 homologue followed by duplication of the re

188 of the heterozygosity of non-MTL genes along chromosome 5, however, results in larger decreases in vi

189 dered gene maps for equine homologs of human chromosome 5 (HSA5), viz., horse chromosomes 14 and 21 (

190 Crohn's disease have now been identified on chromosomes 5 (IBD5/5q31 risk haplotype) and 16 (IBD1 lo

191 This reduction in chromosome 5-imprinted Mkrn1-p1 transcripts was proposed

192 mouse, cappuccino (cno), that maps to mouse chromosome 5 in a region conserved with human 4p15-p16.

193 ingle locus, Gtf2i, which we mapped to mouse chromosome 5 in a region of conserved mouse-human synten

194 In mice, the homologs of these genes map to chromosome 5 in a region of conserved synteny with human

195 ly due to a gene(s) closely linked to Opn on chromosome 5 in conjunction with other randomly assortin

196 Preb maps to the proximal end of chromosome 5 in mouse, near the Hmx1 homeobox gene.

197 association (prs58502974 = 2.11 x 10(-8)) on chromosome 5 in the ADCY2 gene.

198 redicts the presence of a gene at the top of chromosome 5 in this subset of patients that is importan

199 Three QTL were identified--a major one on chromosome 5 in which the Col parent contributed the sup

200 The region of chromosome 5 including the osteopontin (OPN) gene (Opn)

201 ent cells had unique abnormalities involving chromosome 5, including amplicons centered on the target

202 e, which was mapped to the central region of chromosome 5, indicated that prosysA and prosysB transcr

203 A deletion of the long arm of chromosome 5 is a recurring abnormality in malignant mye

204 Interstitial deletion or loss of chromosome 5 is frequent in malignant myeloid disorders,

205 Complete loss or deletion of the long arm of chromosome 5 is frequent in myelodysplastic syndrome (MD

206 es in the conserved syntenic region of mouse chromosome 5 is inverted relative to the human map.

207 The distal portion of the long arm of chromosome 5 is linked to hypertension and contains func

208 Chromosome 5 is one of the largest human chromosomes and

209 nation frequencies across a large portion of chromosome 5 is the first large-scale analysis of mitoti

210 he discovery that deletion of all or part of chromosome 5 is the most common genetic aberration in my

211 ing of 3 dominant NZB contributions (Nba4 on chromosome 5, Lbw4 on chromosome 6, and Nba5 on chromoso

212 The strong signal on chromosome 5 lies in the region in which a novel gene, W

213 NCMD (MCDR1) and 2 individuals affected with chromosome 5-linked NCMD (MCDR3).

214 showed evidence for interactions between the chromosome 5 locus and region on chromosome 12 containin

215 (+/-), including a region upstream of Eln on chromosome 5 (LOD 4.5).

216 76 cM of chromosome 4 (lod = 2.7), 143 cM of chromosome 5 (lod = 2.0), 7 cM of chromosome 9 (lod = 2.

217 ewly discovered loci for absence seizures on chromosome 5 (lod scores 3.8/2.8 and 3.4/1.9).

218 I to maternally derived alleles was found on chromosome 5 (LOD(MO) = 1.7) and to paternally derived a

219 TLs influencing urine arsenic metabolites on chromosomes 5 (LOD = 2.03 for %iAs), 9 (LOD = 2.05 for %

220 diabetes with maternally derived alleles on chromosomes 5 (LOD(MO) = 1.5) and 14 (LOD(MO) = 1.6).

221 6 cM on chromosome 4; lod = 1.7 at 146 cM on chromosome 5; lod = 3.5 at 160 cM on chromosome 9; lod =

222 ve evidence for linkage was found for BMI on chromosome 5 (logarithm of odds [LOD] score = 1.9) and P

223 ritical region of loss in chromosome 5q31.1 (chromosome 5, long arm, region 3, band 1, subband 1) in

224 uman chromosome 13q12-13 and telomeric mouse chromosome 5 (MMU5).

225 282 (92%) had clonal abnormalities involving chromosome 5 (n = 63), chromosome 7 (n = 85), chromosome

226 d to hypergammaglobulinemia; an NZB locus on chromosome 5 (Nba4; peak at 15 cM), linked to IgG anti-s

227 Of particular interest was the region on chromosome 5 near D5S1480, where a reasonable candidate

228 locus of significant effect was detected on chromosome 5 (near D5Mit398).

229 D1S3462; chromosome 4, near marker D4S2361; chromosome 5, near marker D5S1505; and chromosome 17, ne

230 omain protein encoded by a gene that maps to chromosome 5, near the chromosomal location of the cop8

231 RPS4 is a disease-resistance locus on chromosome 5 of Arabidopsis thaliana specifying resistan

232 ng-time regulator FLC, located at the top of chromosome 5 of Arabidopsis thaliana.

233 to the Frost resistance-2 (Fr-Am2) locus on chromosome 5 of diploid wheat (Triticum monococcum) usin

234 Integration of EnPV into chromosome 5 of rats was confirmed by PCR cloning and se

235 a second form of the TFIIA small subunit on chromosome 5 of rice.

236 FC lyase is encoded by a gene on chromosome 5 of the Arabidopsis genome (FCLY, At5g63910)

237 d with the allelic state of the major QTL on chromosome 5, oligonucleotide array expression patterns

238 variant rs26232, in the first intron of the chromosome 5 open reading frame 30 (C5orf30) locus, has

239 introduced by microcell-mediated transfer of chromosome 5 or 2, respectively.

240 Losses of chromosome 18 or 18q and chromosome 5 or 5q were common in cancer and dysplasia a

241 unfavorable abnormalities (rearrangement of chromosome 5 or 7, or > or = 3 abnormalities) had a poor

242 g patients with partial/complete deletion of chromosome 5 or 7, or abnormalities of chromosome 3.

243 or gene lesions, and 18 had abnormalities of chromosome 5 or 7.

244 Loss of a whole chromosome 5 or a deletion of the long arm of chromosome

245 Loss of a whole chromosome 5 or a deletion of the long arm, del(5q), is

246 SNPs in the subset of patients with loss of chromosomes 5 or 7 or both, acquired abnormalities assoc

247 Abnormalities involving chromosomes 5 or 7 were found in 10 cases, which suggest

248 ncidence of TP53 mutations, abnormalities of chromosomes 5 or 7, complex cytogenetics and a reduced r

249 Loss of a whole chromosome 5, or a del(5q), are recurring abnormalities

250 Loss of a whole chromosome 5, or a del(5q), is a recurring abnormality i

251 arise by multiple mitotic cross-overs along chromosome 5 outside of the MTL region.

252 Tgfbm1 (chromosome 5, P = 8 x 10(-5)) and Tgfbm3 (chromosome 12,

253 ed the protocadherin gene family clusters on chromosome 5 (PCDHA, PCDHB, and PCDHG).

254 d strong evidence for susceptibility loci on chromosome 5 (per allele OR = 1.37 (95% CI = 1.19-1.58),

255 y, including a cluster of genes localized on chromosome 5 (PHT1;1, PHT1;2, and PHT1;3).

256 The gene located on chromosome 5 (PIMT2) produces two proteins differing by

257 ng-type (MTL) locus, not associated genes on chromosome 5, provides a competitive advantage.

258 a conventional backcross but also resolved a chromosome 5 QTL identified in the backcross into two QT

259 The identification of the chromosome 5 quantitative trait locus through linkage to

260 al chromosome clones from the syntenic mouse chromosome 5 region that contains Gtf2ird1 and Gtf2i as

261 A recessive mutation in the sim locus on chromosome 5 results in clusters of adjacent trichomes t

262 /NM_144715 on chromosome 3, rs9127/Q7Z4C4 on chromosome 5, rs1445898/CAPSL on chromosome 5 and rs2302

263 de significant locus was found in ANKRD55 on chromosome 5 (rs159572; odds ratio [OR], 1.62; 95% CI, 1

264 bon source, l-sorbose, by reversible loss of chromosome 5, serves as a model system.

265 y are related to rice genes on syntenic rice chromosome 5 short arm (5S).

266 on lines have regular transmission; however, chromosome 5 showed diminished paternal transmission, an

267 on chromosome 1 and a recessive CBA locus on chromosome 5; significantly, there was an epistatic inte

268 ide significant signal (p=1.12 x 10(-10)) on chromosome 5 spanning three genes: MSH3, DHFR, and MTRNR

269 panning approximately 5.8 cM) and another on chromosome 5 (spanning approximately 25.6 cM) resulted i

270 e report that CYP3a13 gene, located on mouse chromosome 5, spans 27.5 Kb and contains 13 exons.

271 in situ hybridization (FISH) studies using a chromosome 5-specific whole chromosome painting probe an

272 o completely sequenced versions of the large chromosome-5-specific internal duplications.

273 l-sorbose, was determined by copy number of chromosome 5, such that monosomic strains utilized l-sor

274 n site was localized to the region of distal chromosome 5 syntenic to the region on human chromosome

275 eport the finding of another gene, Slit2, on chromosome 5 that also accounts for variation in HSC num

276 y the analysis of a 180 kb interval on human chromosome 5 that encodes for the kinesin family member

277 region on Arabidopsis (Arabidopsis thaliana) chromosome 5 that includes 106 genes.

278 on chromosome 18 and suggest a new locus on chromosome 5 that was not identified using traditional l

279 CDH)-alpha, -beta and -gamma gene cluster on chromosome 5 that were associated with nicotine withdraw

280 re constructed by the loss of one homolog of chromosome 5, the site of the MTL loci, MTLa and MTLalph

281 ations from the ancestral homolog of macaque chromosome 5 to macaque chromosomes 1 and 6.

282 of a major female heterogametic ZW locus on chromosome 5, two separate male heterogametic XY loci on

283 ed and characterized a gene, Mospd3 on mouse chromosome 5 using gene trapping in ES cells.

284 Suggestive evidence of linkage to chromosome 5 was also found for rMBP, to chromosome 16 f

285 sition disease, a region on the short arm of chromosome 5 was found to be genetically linked to the p

286 knox10, which is located on the short arm of chromosome 5, was shown to be ectopically expressed in d

287 To identify a leukemia-related gene on chromosome 5, we previously delineated a 970-kb segment

288 High frequencies of loss on chromosome 5 were identified at loci D5S428 (48%), D5S42

289 imately 80 kb and was used for sequencing of chromosome 5, were arbitrarily selected for analysis.

290 ochromosome composed of the two left arms of chromosome 5, were associated with azole resistance.

291 ent signals within the TERT-CLPTM1L locus on chromosome 5, which has previously been associated with

292 evidence that this formation of monosomy of chromosome 5, which is apparently a result of nondisjunc

293 e gene within the homologous region on human Chromosome 5, which is linked to systolic and diastolic

294 ossesses a single mating-type (MTL) locus on chromosome 5, which is normally heterozygous (a/alpha).

295 s to isolate the effects of the major QTL on chromosome 5 while holding the minor QTL constant.

296 o families with 46,XY DSD to the long arm of chromosome 5 with a combined, multipoint parametric LOD

297 rovided "suggestive" evidence of a region on chromosome 5 with pleiotropic effects on both traits (ML

298 ized the disease interval to the long arm of chromosome 5, with a maximum two-point parametric LOD sc

299 sociations of ABR thresholds with markers on chromosome 5, with a peak lod score of 5.5 for D5Mit309.

300 accepted threshold for suggestive linkage on chromosomes 5, X, and 19.