ライフサイエンスコーパス: chromosome 8

1 isomic segregation relatively more common in chromosome 8.

2 deleterious mutations in the RECQL4 gene on chromosome 8.

3 ne is localized in the C3-D1 region of mouse chromosome 8.

4 (HPC), using 24 markers on the short arm of chromosome 8.

5 ined in exon 1, and is located at band E1 on chromosome 8.

6 confirmed the synteny between LG24 and human chromosome 8.

7 ive, demyelinating peripheral neuropathy, to chromosome 8.

8 at or kat(2J), that map to the same locus on chromosome 8.

9 allele on chromosome 19 and an A/J allele on chromosome 8.

10 ene and the mapping of its location to mouse chromosome 8.

11 For radiation, abnormalities were mostly in chromosome 8.

12 The gene is located on mouse chromosome 8.

13 ML1 gene on chromosome 21 is fused to ETO on chromosome 8.

14 ndromes map or have been suggested to map to chromosome 8.

15 proto-oncogene at 67.0 centimorgans (cM) on chromosome 8.

16 Arg polymorphism) spanning a 57-cM region of chromosome 8.

17 1 (CBFA2) on chromosome 21 and ETO (MTG8) on chromosome 8.

18 ue to independent allelic mutations on mouse chromosome 8.

19 hereas the murine homolog gene is located on chromosome 8.

20 lepsy (IGE), for linkage to markers spanning chromosome 8.

21 ty (LOH) for 11 microsatellite loci on mouse chromosome 8.

22 to a noncoding region at 61.0 cM on the same chromosome 8.

23 to correspond to a SRPK2 pseudogene on human chromosome 8.

24 -STAR mapped to the syntenic region on human chromosome 8.

25 hroid ankyrin is encoded by the Ank1 gene on Chromosome 8.

26 translocations involving the gene for MYC on chromosome 8.

27 alized to a highly syntenic region of distal Chromosome 8.

28 t 15 microsatellite loci on the short arm of chromosome 8.

29 owed us to map the CK2 alpha' gene to murine Chromosome 8.

30 ogy to other CC chemokines and maps to mouse chromosome 8.

31 homology region is likely to reside on human chromosome 8.

32 vical ITD in two large Mennonite families to chromosome 8.

33 to TrnR1, and is located on the short arm of chromosome 8.

34 on chromosome 21 and the ETO (MTG8) gene on chromosome 8.

35 lele loss with RFLP probes from both arms of chromosome 8.

36 , GRINA, has been previously mapped to human chromosome 8.

37 , was mapped to the proximal region of mouse Chromosome 8.

38 by an interspecies backcross panel to mouse chromosome 8.

39 der using 30 microsatellite polymorphisms on chromosome 8.

40 nvulsions (BFNC), has also been localized to chromosome 8.

41 ng located at the same cytogenetic region of chromosome 8.

42 ster hybrids localized both HD-5 and HD-6 to chromosome 8.

43 e NODULIN3 (N3) gene family, located on rice chromosome 8.

44 cell line haploid for all chromosomes except chromosome 8.

45 and TG, not fully explained by the locus on chromosome 8.

46 icated an association with rearrangements of chromosome 8.

47 esolution over 8p12 and 1 Mb resolution over chromosome 8.

48 As part of this project, we have focused on chromosome 8.

49 The lop11 locus was mapped to mouse chromosome 8.

50 1) on chromosome 15 and mALT2 gene (gpt2) on chromosome 8.

51 (eight-twenty one, also called MTG8) gene on chromosome 8.

52 f chromosome 16q, which is syntenic to mouse chromosome 8.

53 nd are therefore present on both the X and Y chromosomes [8].

54 th significant increases in hyperdiploidy of chromosomes 8 (1.2, 1.5, and 2.4 per 100 metaphases; P <

55 d rice by resequencing 630 gene fragments on chromosomes 8, 10, and 12 from a diverse set of wild and

56 some ploidy of each cell, the centromeres of chromosomes 8, 10, and 17.

57 ere probes and locus-specific arm probes for chromosomes 8, 11, 17, and 18, we demonstrate that chrom

58 they are both associated with polysomies for chromosomes 8, 11, 17, and 20.

59 Related sequences were mapped on Chromosomes 8, 11, and 2 unlinked loci on Chromosome 2 u

60 the extent of wound closure were detected on chromosomes 8, 12, and 15 and at two separate locations

61 Detailed analysis of aneuploidy for chromosomes 8, 12, and 17 on intact paraffin sections re

62 The shape of the expression profiles for chromosomes 8, 12, and X correlated well with the numeri

63 However, there was random loss of chromosomes 8, 13, X, Y, and alteration in chromosome 4q

64 uction of Separase resulted in trisomies for chromosomes 8, 15, and 17; monosomy for chromosome 10; a

65 The Cat4 locus has been mapped to chromosome 8, 31 cM from the centromere.

66 o identify the translocation partner gene on chromosome 8, 5' and 3' RACE polymerase chain reactions

67 s of chromosome 4 (87%), chromosome 7 (80%), chromosome 8 (53%), chromosome 10 (33%), and chromosome

68 es coelicolor A3(2) possesses a large linear chromosome (8.67 Mb) with a centrally located origin of

69 Allelic losses on the short arm of chromosome 8 (8p) have been reported as frequent events

70 lular carcinoma (HCC) is of the short arm of chromosome 8 (8p).

71 The short arm of chromosome 8, 8p, is often rearranged in carcinomas, typ

72 ocus and a segment of the short arm of human chromosome 8 (8p21-p11.2).

73 Gains in the long arm of chromosome 8 (8q) are believed to be associated with poo

74 possible reciprocal translocation involving chromosomes 8, 9, and 12.

75 tein arginine methyltransferase 7 (Prmt7) on chromosome 8, a protein previously implicated in cellula

76 Only two cases (13%) lacked a chromosome 8 abnormality.

77 could be coamplified with a control gene on chromosome 8 (Acta1) so that the yield of each PCR produ

78 ntify a candidate region, which was found on chromosome 8; all genes within this interval were sequen

79 To investigate the chromosome 8 allele status in Chinese HCCs, a panel of 3

80 st tumors examined, such as RIPK2 and MYC on chromosome 8, also parallel these findings.

81 ots with putative sesquiterpene synthases on chromosome 8, an indication that the genes in Sst1 and S

82 higher pathological stage (P = 0.021) and on chromosome 8 and 16 with a higher Gleason score (P = 0.0

83 tic heterogeneity in PS and mapped a gene to chromosome 8 and a second to chromosome 10.

84 COP locus, Mcs4, has also been identified on chromosome 8 and acts in contrast to increase the number

85 Our FISH results indicate that: (a) gain of chromosome 8 and amplification of c-myc are potential ma

86 ated on human chromosome 16q22-q23 and mouse chromosome 8 and are alternatively spliced.

87 es, such as large copy number aberrations in chromosome 8 and complex rearrangement chains.

88 The mouse Nod2 locus is located at chromosome 8 and composed of 12 exons, 11 of which encod

89 The murine flt4-L gene was localized to chromosome 8 and demonstrated to be widely expressed.

90 mapping the gene (Arhgap7) to 20 cM on mouse chromosome 8 and for allelotyping of mouse liver tumor D

91 loss of heterozygosity for the short arm of chromosome 8 and has a complex karyotype.

92 didate genes to validate an eQTL hot spot in chromosome 8 and identified the gene 2310061C15Rik as a

93 Myo9b has been previously mapped to mouse chromosome 8 and is a candidate for the mouse mutations

94 tained pedigrees implicated two loci, one on chromosome 8 and the other on chromosome 15, that influe

95 ene duplicates: two alpha-globin paralogs on chromosome 8 and two beta-globin paralogs on chromosome

96 DNA amplification, particularly of chromosomes 8 and 11, occurs frequently in breast cancer

97 0; and amplification of the distal region of chromosomes 8 and 11.

98 Moreover, gains of sequences from chromosomes 8 and 13 occurred in most tumors, indicating

99 we evaluated microsatellite markers spanning chromosomes 8 and 13, for linkage to schizophrenia, in 2

100 ovided initial evidence for modifier loci on chromosomes 8 and 15 and suggestive evidence for a locus

101 nd saturation mapping purported intervals on chromosomes 8 and 15, two additional regions were identi

102 Rfx1 and Rfx2 to homologous regions of mouse chromosomes 8 and 17, respectively.

103 tors for PERV-A, encoded by genes located on chromosomes 8 and 17.

104 BIG2 genes were localized, respectively, to chromosomes 8 and 20.

105 -embedded tissue with centromeric probes for chromosomes 8 and 20.

106 Numerical and structural aberrations in chromosomes 8 and 21 are commonly observed in AML.

107 In the present study, we painted chromosomes 8 and 21 in lymphocyte metaphases from 43 he

108 Translocations between chromosomes 8 and 21 were increased up to 15-fold in hig

109 cation of the amplified Myc and HPV genes on chromosomes 8 and 21.

110 und on chromosomes 2 and 16 for liver and on chromosomes 8 and 9 for spleen.

111 nt1 and Ant2) and assigned the loci to mouse chromosomes 8 and X, respectively.

112 ents with t(8;14)(q11;q32) involved CEBPD on chromosome 8, and 9 patients with t(14;19)(q32;q13) invo

113 lines, aberrant gene expression, trisomy of chromosome 8, and abnormal H2A.X deposition were disting

114 to an extra isochromosome of the long arm of chromosome 8, and the near-diploid karyotype appears to

115 PHS1 and NDPS1 map to chromosome 8, and the presence of a segment of chromosom

116 t anomaly in PIN and carcinoma was a gain of chromosome 8, and the presence of this anomaly strongly

117 fied AML1 on chromosome 21 and ETO (MTG8) on chromosome 8, and the resultant chimeric gene product, A

118 The toppler mutation was mapped to mouse chromosome 8, and to assess whether it was novel or a re

119 A male-specific locus on proximal chromosome 8 (Ap8q) affected 3% ethanol preference, but

120 Genomic aberrations on chromosome 8 are common in colon cancer, and are associa

121 mor samples of high-grade chondrosarcoma for chromosome 8 are presented.

122 Deletions on the short arm of chromosome 8 are recurrent in liver, breast, lung, and p

123 Sequences previously localized to chromosome 8 are shown to be a pseudogene, and an additi

124 rder between CFA29 and the long arm of human chromosome 8 argued for homology between the cd locus an

125 romosome 8 polysomy had up to five copies of chromosome 8 as an isolated cytogenetic finding in an ot

126 g whether GRINA mapped to the same region of chromosome 8 as BFNC.

127 of evidence have implicated the short arm of chromosome 8 as harboring genes important in prostate ca

128 ous studies have implicated the short arm of chromosome 8 as the site of one or more tumor suppressor

129 gene encoding cortexin, Ctxn, which maps to chromosome 8, as a candidate for the mouse neurological

130 sruption of a cluster of six genes on murine chromosome 8, as exemplified by the Fused Toes (Ft) muta

131 Os) is a radiation-induced mutation on mouse chromosome 8 associated with early postimplantation leth

132 e we characterized the structural changes of chromosome 8 associated with this mutation.

133 hm of odds [LOD]=4.93, permuted P=0.002) and chromosome 8 at 33 cM (LOD=3.27, permuted P=0.058).

134 ific QTL, which we name Fsia1, is located on chromosome 8 at 52-84 cM from the centromere and account

135 The myd mutation maps to mouse chromosome 8 at approximately 33 centimorgans (cM).

136 zed by fluorescence in situ hybridization on chromosome 8 at bands p21.3-22.

137 ive HPV18 integration sites: three on normal chromosomes 8 at 8q24 and two on derivative chromosomes,

138 syntenic; the hTRF1 gene localized to human chromosome 8 band q13 while the mTRF1 gene localized to

139 ed that the murine FATP gene is localized to chromosome 8, band 8B3.3.

140 ST7 is located on human chromosome 8, band q22.2-23.1, the same locus as the gen

141 itu hybridization localized the ERK8 gene to chromosome 8, band q24.3.

142 for insulin response to glucose was found on chromosome 8 between D8S1130 and D8S1106, near the lipop

143 We mapped this new gene to chromosome 8, between markers D8S256 and D8S284 on a rad

144 gene is expressed in leaf tissue and maps to chromosome 8 (bin 8.05), with a duplicate locus on chrom

145 At the chromosome 8 breakpoint we identify a novel gene, MOZ, w

146 bridization (FISH) analysis to show that the chromosome 8 breakpoints fell within YAC 899e2 and that

147 s further localized to the central region of chromosome 8 by interspecific backcross mapping; a relat

148 ditional Southern and Northern analysis, and chromosome 8 cDNA microarray expression profiling.

149 The centromere of rice Chromosome 8 (Cen8) has an unusually low abundance of hi

150 creases in c-myc copy number relative to the chromosome 8 centromere), which was detected in 0, 8, an

151 ncrease in c-myc copy number relative to the chromosome 8 centromere), which was identified in 42, 25

152 ncrease in c-myc copy number relative to the chromosome 8 centromere, which was found in 8, 11, and 2

153 , perhaps as a result of a deletion near the chromosome 8 centromere.

154 alterations including chromosome 8p loss and chromosome 8 centromeric gain.

155 to have 8p LOH using a chromosome 8 paint, a chromosome 8 centromeric probe, and a series of single-c

156 two QTLs with significant linkage for TC: on chromosome 8, chr8-60.4 Mbp (LOD 3.8), and chromosome 2,

157 s 88% +/- 3%; Plog rank = .0008) and gain of chromosome 8 (CIR/NR, 16% +/- 7% vs 3% +/- 2%, PGray = .

158 The yellowbeak mutation mapped to chromosome 8, close to a cluster of cytochrome P450 loci

159 Slug maps to the long arm of chromosome 8, closely linked to D8S2090 between D8S519 a

160 F344(Cia4) (chromosome 7) and DA.F344(Cia6) (chromosome 8) congenics had significantly lower exudate

161 for relative water content were detected on chromosome 8, congruent with an osmotic adjustment QTL i

162 ithin mouse chromosome 8 was confirmed using chromosome 8 consomic mice.

163 etastasis suppressor gene, a truncated human chromosome 8 containing this region was transferred into

164 Interestingly, this region of chromosome 8 contains a cluster of three other genes imp

165 tients with MYC:CEP8 ratio < 1.3 with normal chromosome 8 copy number (n = 141; 18%) and >/= 1.3 or <

166 tential associations between alternative MYC/chromosome 8 copy number alterations and differential be

167 A single marker on chromosome 8 (D8S593) and two adjacent markers on chromo

168 Mutations of the MD-2 associated gene on chromosome 8 degrade a human's innate responses.

169 s of DNA from cell lines that contain either chromosome 8 deletions or duplications further localized

170 romosome 8, and the presence of a segment of chromosome 8 derived from Solanum pennellii LA0716 cause

171 Here we report that substitution of the chromosome 8-derived ETO protein for the multifunctional

172 ed, including three new loci in intervals on chromosomes 8 (eae14), 10 (eae17), and 18 (eae18).

173 significant QTL found was for CD4 levels on chromosome 8 (empirical P=.00005).

174 show that AML+8 blasts overexpress genes on chromosome 8, estimated at 32% on average, suggesting ge

175 Unique loci were identified on chromosome 8 for orchitis, chromosome 16 for epididymiti

176 neity within IGE and provide no evidence, on chromosome 8, for a gene common to all IGEs.

177 L1 gene on chromosome 21, to the ETO gene on chromosome 8, forming the AML1/ETO fusion gene.

178 ML1 gene on chromosome 21 to the ETO gene on chromosome 8 fuses the NH2-terminal portion of AML1 to n

179 ency, distribution, proliferative state, and chromosome 8 gain of benign prostate, SA, PAH, HGPIN, an

180 tify TRC8 (translocation in renal carcinoma, chromosome 8 gene), an ER-resident E3 ligase previously

181 on chromosome 21 and the ETO (MTG8) gene on chromosome 8, generates AML1-ETO fusion proteins.

182 L1 gene on chromosome 21 and the ETO gene on chromosome 8, generates an AML1-ETO fusion transcription

183 osome 21 and the ETO (MTG8, RUNX1T1) gene on chromosome 8 generating the AML1-ETO fusion proteins.

184 ene is juxtaposed to the ETO gene located on chromosome 8, generating an AML1/ETO fusion protein.

185 er variation in beta-defensin genes on human chromosome 8 has been proposed to underlie susceptibilit

186 We demonstrate that the Os chromosome 8 has suffered two breaks that are 5 cM ( app

187 orphism of the beta-defensin region on human chromosome 8 in 179 Dutch individuals with psoriasis and

188 high-resolution map of genomic imbalances of chromosome 8 in 51 primary colon carcinomas using a cust

189 have investigated interstitial deletions of chromosome 8 in 70 colorectal carcinomas and 11 colonic

190 how that its murine homologue maps to murine chromosome 8 in a region syntenic with the human WRN gen

191 We found evidence for linkage to chromosome 8 in adolescent-onset IGE families in which J

192 separate, two-exon genes that are located on chromosome 8 in both mice and humans.

193 ore was associated with the SNP rs2645424 on chromosome 8 in farnesyl diphosphate farnesyl transferas

194 onfirmed loss of CDKN2A and amplification of chromosome 8 in IM but not in a nearby NGM biopsy.

195 exon genes that map to homologous regions of chromosome 8 in mice and humans, and the differential ex

196 involvement of a tumor suppressor gene(s) on chromosome 8 in prostatic neoplasms, we performed a comp

197 Another three lipoblastomas had polysomy for chromosome 8 in the absence of PLAG1 rearrangement.

198 lines, we previously identified a region of chromosome 8 in the Solanum pennellii LA0716 genome (IL8

199 translocation breakpoint on the short arm of chromosome 8 in this patient.

200 ternal meiosis, we investigated the maternal chromosomes 8 in eight mothers of subjects with inv dup(

201 ce, Lmp10 is a single-copy gene localized to chromosome 8, in a region of conserved synteny with huma

202 The Zfp319 gene maps to chromosome 8, in a region of conserved synteny with the

203 me 6 for JME (lod score 2.5/4.2), a locus on chromosome 8 influencing non-JME forms of IGE (lod score

204 ersion (p31q12), and 3,168 were analyzed for chromosome 8 inversion (p23q22).

205 ecombination frequency of 13.1% recorded for chromosome 8 inversion was similar to the frequency of 1

206 This region of chromosome 8 is a gene desert, far from any recognized g

207 e have discovered that the Ft locus on mouse chromosome 8 is associated with cell cycle deficits with

208 henotype linked to the pericentric region of chromosome 8 is associated with mutations in a gene desi

209 he region of linkage to DTNBP1 expression on chromosome 8 is contiguous with linkage findings based u

210 content and fraction segmentally duplicated, chromosome 8 is distinctly typical in character, being v

211 The short arm of chromosome 8 is frequently lost in epithelial cancers, a

212 The CP on chromosome 8 is predicted to have a CPA-like specificity

213 The gene on chromosome 8 is the fibroblast growth factor receptor 1

214 Gain of chromosome 8 is the most common chromosomal gain in huma

215 The beta3 adrenergic receptor, located on chromosome 8, is a regulator of energy expenditure and l

216 which is a 278 amino acid protein encoded on chromosome 8, known to be synthesized in the liver.

217 Abnormalities in chromosome 8, leading to rearrangements of the PLAG1 gen

218 clone spanning the OVE459 insertion locus on chromosome 8 led to the identification of two novel cand

219 of CPP32 beta to the central region of mouse chromosome 8, linked to Scvr, Lpl, Jund1 and Mlr.

220 hese analyses suggest that the action of the chromosome 8 locus is specific to unesterified cholester

221 Linkage to the chromosome 8 locus was excluded in this family.

222 OD = 1.54) for moderate airflow obstruction, chromosomes 8 (LOD = 1.36) and 19 (LOD = 1.09) for mild

223 d non-drug-induced locomotor activity (e.g., chromosome 8; LOD = 18.9).

224 Chromosome 8 loss of heterozygosity (LOH) in cancer of t

225 nd a cytological marker on the centromere of chromosome 8 made it possible to discriminate between ea

226 No loss of heterozygosity of p53 or for chromosome 8 markers was found.

227 tations, loss of heterozygosity for TP53 and chromosome 8 markers, and ras mutations.

228 r Ets-1, located within one of these loci on chromosome 8, mediates glomerular injury in SS hypertens

229 Mf1 was assigned to mouse Chromosome 8, Mf2 to Chromosome 4, Mf3 to Chromosome 9,

230 el or a recurrence of a previously described chromosome 8 mouse mutant, toppler mice were crossed wit

231 d mapping localized this gene to a region of chromosome 8 near several other defensins, MBD-2, MBD-3,

232 s a single copy and located at the middle of Chromosome 8 near the mutations for myodystrophy (myd) a

233 The SNPs were rs4355801, on chromosome 8, near to the TNFRSF11B (osteoprotegerin) ge

234 roups: (a) those with simple gain of a whole chromosome 8 (no increase in c-myc copy number relative

235 Both genes lie on chromosome 8, not previously suspected to contain imprin

236 ing peptide receptor (GRPR) gene and that on chromosome 8 occurred approximately 30 kb distal to the

237 otein lipase (LPL) locus on the short arm of chromosome 8 (P = 0.002, n = 125 sib-pairs, for the hapl

238 t evidence for association was at a locus on chromosome 8 (P=1.5 x 10(-15)) spanning 4 Mb and contain

239 unts (P = .007), and more often trisomies of chromosomes 8 (P = .01) and 21 (P < .001) and less often

240 Loss of the p arms of chromosome 8 (p10>pter) and chromosome 10 (p10>pter) and

241 sma fatty acids identified a 20-cM region on chromosome 8 (p12-p21) with a quantitative trait locus f

242 cell lines predicted to have 8p LOH using a chromosome 8 paint, a chromosome 8 centromeric probe, an

243 Notably, the lipoblastomas with chromosome 8 polysomy had up to five copies of chromosom

244 nhardtii, which we mapped to the long arm of chromosome 8, provides a good experimental system for th

245 osome 10 (p10>pter) and gain of the q arm of chromosome 8 (q10>qter) were only observed in the tumor

246 ing 26 microsatellite markers, we mapped the chromosome 8 regions frequently deleted in lung cancer i

247 Two chromosome 8 regions were associated: p = 4.45 x 10(-8)

248 The region of human chromosome 8 retained in each microcell hybrid was deter

249 significant association was revealed for the chromosome 8 rs6996198 with HIV-1 acquisition and was re

250 Two YAC probes targeted chromosome 8 short arm regions known to be deleted frequ

251 several chromosome regions, particularly the chromosome 8 short arm.

252 The data from chromosome 8 should allow a better understanding of both

253 Alterations in chromosome 8 show unique correlation with melanocytosis.

254 on chromosomes 2, 4, and 8, with the QTL on chromosome 8 showing the highest significance, as well a

255 contain the GPT sequence were derived from a chromosome 8-specific library.

256 an integrated map for a 35-cM area of human chromosome 8 surrounding the Langer-Giedion syndrome del

257 herin-11, was mapped to a specific region of chromosome 8 that also includes cadherins-1, -3, and -5.

258 ited Htn1 to a 131.7-kb physical interval on chromosome 8 that contained three candidate genes encodi

259 to detect a modifier locus, Modpkdr1, on rat chromosome 8 that controls PKD severity, kidney mass and

260 CKbeta4GT-II maps to a region of chicken chromosome 8 that exhibits conserved synteny with human

261 ial tumors show deletion of the short arm of chromosome 8 that is related to aggressive disease or ad

262 lar dystrophy and maps to a segment of mouse chromosome 8 that is syntenic with human 4q31-4q35.

263 is part of a defensin beta (DEFB) cluster on chromosome 8 that is variable in copy number.

264 a high-resolution map of a segment of mouse chromosome 8 that places the nr locus in a genomic segme

265 ned within a "half-YAC" from the long arm of chromosome 8, that is, a YAC containing the 8q telomere.

266 At a second locus, Sst2, on chromosome 8, the L. hirsutum allele specified accumulat

267 gene is localized at the proximal portion of chromosome 8, the site where genes for mouse alpha- and

268 ene was localized at the proximal portion of chromosome 8, the site where mouse alpha-defensins are f

269 ine chromosome 16 and mXCP4 is positioned on chromosome 8; the hXCP1 and hXCP2 genes are located at h

270 ntains six exons and is located on zebrafish chromosome 8; the structure of the gene is highly homolo

271 que to delete in mice the syntenic region on chromosome 8 to create a Dnajb1-Prkaca fusion and monito

272 Allele loss from the long arm of chromosome 8 was also observed with 30.0% (6/20) and 33.

273 A locus within mouse chromosome 8 was confirmed using chromosome 8 consomic m

274 tandem 12 kb repeats which spanned a gap on chromosome 8 was found to be 600 kb to 1.7 Mbp in size,

275 The EXT1 gene on chromosome 8 was recently identified and characterized.

276 Maize chromosome 8 was recovered as a monosomic addition (2n =

277 ingle-nucleotide polymorphism (rs4445257) on chromosome 8 was strongly associated with the normal gro

278 In addition, a QTL on chromosome 8 was suggestive for linkage.

279 ies varied between the chromosomes examined; chromosome 8 was the least affected, and 17p loss was fo

280 The mouse Aprt locus on chromosome 8 was used as the selectable target for the s

281 tion to the previously identified linkage to chromosome 8, we find that a locus on chromosome 17, whi

282 e short arm and one locus on the long arm of chromosome 8 were used for PCR-based LOH analysis on mat

283 y at a pseudo attP sequence located on human chromosome 8, when measured in the native genomic enviro

284 e mapped this mutation to the same region of chromosome 8 where Os has been assigned.

285 ng evidence for selection was found on maize chromosome 8, where several putatively selected loci col

286 4 as well as at distinctly different loci on chromosome 8 which were undetected through CIM.

287 murine mTll gene (Tll) is mapped to central chromosome 8, which is a different chromosomal location

288 t except for the quantitative trait locus on chromosome 8, which is derived from C57BL/6.

289 mnd maps to the centromeric region of mouse chromosome 8, which likely corresponds to portions of hu

290 slocation, which fuses the ETO gene on human chromosome 8 with the AML1 gene on chromosome 21 (AML1-E

291 Results link the JDCM locus to canine chromosome 8 with two-point and multipoint lod scores of

292 ere not found by the microsatellite markers: chromosome 8, with a maximum model-free LOD score of 2.2

293 hromosome 6, and seven contiguous markers on chromosome 8, with empirical P values of .00001.

294 arkers, the rocker locus was mapped to mouse chromosome 8 within 2 centimorgans of the calcium channe

295 On pericentromeric chromosome 8 within our previously reported linkage peak