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1 valent to a similar stay-green locus on rice chromosome 9.
2 Submergence 1 (Sub1) near the centromere of chromosome 9.
3 hort (p) arm and part of the long (q) arm of chromosome 9.
4 road, gene-rich centromeric region of canine chromosome 9.
5 at had been translocated to the short arm of chromosome 9.
6 ransferrin-like gene map close to the QTL on chromosome 9.
7 of late-onset Alzheimer's disease (LOAD) to chromosome 9.
8 y of Plasmodium falciparum was identified on chromosome 9.
9 Genetic mapping placed mhl1 within bin 4 on chromosome 9.
10 nkage group marker BNL4053 was reassigned to chromosome 9.
11 ignment of marker BNL4053 to the long arm of chromosome 9.
12 nge of 1.6-2.1 previously reported for maize chromosome 9.
13 ce of a BP QTL in a large 34.2-cM segment of chromosome 9.
14 motion susceptibility locus, Psl1, to distal chromosome 9.
15 d 17, and for triglyceride concentrations on chromosome 9.
16 ked to either the bz or the wx donor loci on chromosome 9.
17 n in neonatal brain and was located on mouse chromosome 9.
18 mapped to human chromosome 15q22.1 and mouse chromosome 9.
19 A0020, a gene of unknown function located on chromosome 9.
20 polymorphism placing the pccb locus on mouse chromosome 9.
21 22.3-q23 and to the syntenic region on mouse chromosome 9.
22 of regulatory domains identified a locus on chromosome 9.
23 , and NY-BR-1.1 was tentatively localized to chromosome 9.
24 ysis, GBDR1 cDNA identified a single gene on chromosome 9.
25 y to rat type II collagen LOD 5.2) mapped to chromosome 9.
26 ructure, and assigned its location to distal chromosome 9.
27 d all three genes in close vicinity on mouse chromosome 9.
28 ied due to its proximity to the Punc gene on chromosome 9.
29 e MMP gene cluster at the centromeric end of chromosome 9.
30 TB-9Sb is a translocation between the B and chromosome 9.
31 e families is linked to markers near TIE2 on chromosome 9.
32 spans approximately 9 kb pericentromeric on chromosome 9.
33 apolipoprotein (apo) AI-CIII gene cluster on chromosome 9.
34 gene, which maps close to the Waxy locus of chromosome 9.
35 ects were detected across a large portion of chromosome 9.
36 Hnf6 gene is located in the middle of mouse chromosome 9.
37 ll adhesion molecule (Ncam) locus located on chromosome 9.
38 the very terminal region of the short arm of chromosome 9.
39 e major histocompatibility complex (MHC) and chromosome 9.
40 ansion recently found within the gene X25 on chromosome 9.
41 a putative pseudogene of MMAC1 localized on chromosome 9.
42 with other CC chemokines, MIP-3beta maps to chromosome 9.
43 beta is a single-copy gene and is located on chromosome 9.
44 d for localized 9p21 deletion or monosomy of chromosome 9.
45 Nktr gene located on the distal end of mouse chromosome 9.
46 spots coinciding with GLS resistance QTLs on chromosome 9.
47 osome 7 and a deletion of at least 232 kb on chromosome 9.
48 ern leaf blight-has been identified on maize chromosome 9.
49 ing cases previously identified as linked to chromosome 9.
50 gene, harlequin (H), previously localized to chromosome 9.
51 was mapped to a position near the bottom of chromosome 9.
52 keratinocyte stem cell locus (Ksc1) on mouse chromosome 9.
53 ion of the chromosome 7 locus and a locus on chromosome 9.
54 pedigrees, we mapped the EIC locus to canine chromosome 9.
55 ) was a frequent event, especially affecting chromosome 9.
56 that result in the fusion of NUT to BRD3 on chromosome 9.
57 The rc locus was previously mapped to Chromosome 9.
58 tic crosses that brought together two broken chromosomes 9.
59 identified five new susceptibility loci, on chromosomes 9, 10 and 11 (P = 4.6 x 10(-7) to P = 3.2 x
60 n FPC, the scenario for building the maps of chromosomes 9, 10 and 13, and the results from the simul
62 cardiac modifiers of nmd were identified on chromosomes 9, 10 and 16, which account for over 26% of
63 e loss strategy to individually delete mouse chromosomes 9, 10, 12, or 14 in tetraploid immortalized
64 s (Cia15, Cia16*, Cia17, Cia18* and Cia19 on chromosomes 9, 10, 18 and two on the X chromosome, respe
65 genes among the three cell lines, notably on chromosomes 9, 11, 12, and 14, suggesting their involvem
66 n chromosome 10q22 and suggestive linkage on chromosomes 9, 11, and 13 for major genetic determinants
67 al quantitative trait loci (QTLs) located on chromosomes 9, 12, 14, and 19, whereas maximum disease s
69 age to loci on proximal chromosome 14 and on chromosome 9; 129S6 alleles protect against the lethal e
73 The suggestive QTL near the proximal end of chromosome 9 (2.4 cM, LOD: 3.12) was replicated at both
74 .1], on chromosome 1 and a suggestive QTL on chromosome 9 (38 cM, LOD 2.3) that influenced plasma glu
75 icant QTL for aortic root lesion size was on chromosome 9 (61 Mb, LOD=6.9), but it was distinct from
79 B subunit gene, localized to the long arm of chromosome 9 (9q34) by fluorescence in situ hybridizatio
82 at 30, 40, and 50 krad) of a monosomic maize chromosome 9 addition line produced maize chromosome 9 r
83 lated from a genomic library of an oat-maize chromosome 9 addition line with the help of the 180-bp k
85 erferon-kappa is located on the short arm of chromosome 9 adjacent to the type I interferon gene clus
86 ve transcription factor ALL1-fused gene from chromosome 9 (Af9) as well as the release of this repres
90 ficant QTL, named Ath22 (42 cM, LOD 4.1), on chromosome 9 and a suggestive QTL near D11mit236 (20 cM,
91 and two distinct interacting QTLs (AS-f1 on chromosome 9 and AS-f2 on chromosome11, LOD 8.9) in fema
92 , we have now localized the B10.Q/J locus to chromosome 9 and established its identity as Tyk2, a Jan
93 ed the gene encoding alpha-tectorin to mouse chromosome 9 and human chromosome 11 in a known region o
95 ic single nucleotide polymorphisms (SNPs) on chromosome 9 and identified two SNPs in the death-associ
98 with this translocation, one on the abnormal chromosome 9 and one on the Philadelphia chromosome (Ph
99 atient's cells enabled screening of the MSSE chromosome 9 and showed no CDC14B deletion or mutation t
100 ly been localized to a small region of mouse chromosome 9 and we have now identified the gene and the
102 copy number changes indicate that losses of chromosomes 9 and 10 occur early on in melanoma progress
103 plications of the X chromosome, deletions of chromosomes 9 and 10, and translocations involving chrom
104 ed for genes with trans-eQTLs in hotspots on chromosomes 9 and 10, which also coincided with phenotyp
109 ere unique to each population with blocks on chromosomes 9 and 12 in DRH and 3, 4, 6 and 8 in D84.
111 3q, and 22q are syntenic with loci on mouse chromosomes 9 and 16 that are implicated in a murine tra
112 fically, the frequency of losing portions of chromosomes 9 and 16 was significantly modulated by gene
115 heterozygosity and microsatellite shifts on chromosomes 9 and 17, sequence mutations in CDKN2A/MTS1/
118 arises by a reciprocal translocation between chromosomes 9 and 22 and harbors the BCR-ABL fusion onco
119 esults from a balanced translocation between chromosomes 9 and 22 and is found in patients with chron
120 formed by a reciprocal translocation between chromosomes 9 and 22 that fuses BCR-encoded sequences up
122 formed by a reciprocal translocation between chromosomes 9 and 22 that results in the fusion of BCR a
124 sult from a reciprocal translocation between chromosomes 9 and 22; this translocation leads to a shor
126 rophic lateral sclerosis have been linked to chromosome 9, and an expansion of the GGGGCC hexanucleot
127 tailed physical mapping of markers from rice chromosome 9, and from syntenous (at the genetic level)
128 74, PDCD1LG2, and the centromeric portion of chromosome 9, and immunohistochemistry (IHC) using an an
129 distal mouse chromosome 1 and proximal mouse chromosome 9, and the human regions with conserved synte
134 romising pair of genes located 8 MB apart on chromosome 9 are identified based on as demonstrating st
136 ification of terpene synthase21 (ZmTps21) on chromosome 9 as a beta-costic acid pathway candidate gen
137 paralogous to the pericentromeric regions of chromosome 9 as well as to 2q13, the site of an ancestra
138 s panel, we place the Adamts8 locus on mouse chromosome 9 at a consensus position of 11 cM and its hu
139 related antimicrobial peptide, was mapped to chromosome 9 at a region of conserved synteny to which g
140 uman LHX3 maps to the subtelomeric region of chromosome 9 at band 9q34.3, within a region noted for c
144 fluorescence in situ hybridization to mouse chromosome 9 at region 9E3-F1 and to human chromosome 3
145 uman type I interferon (IFN) gene complex on chromosome 9, band p21-22, to examine the overall struct
146 entified by linkage analysis to distal mouse chromosome 9 between D9Mit154 and D9Mit330, closely link
147 etween D3S3053 and D3S2427 (LOD = 1.36), and chromosome 9 between D9S1120 and D9S910 (LOD = 1.46).
148 .2 gene and genetically mapped the marker on chromosome 9 between markers D9S1825 and D9S290 with odd
151 enes, and localizes to the central region of chromosome 9 by single-strand polymorphism analysis.
153 al that genetic information, such as loss of chromosome 9, can be obtained from widely available tech
154 patient had segmental uniparental disomy of chromosome 9, carrying 2 copies of the maternal CARD9 mu
155 erations to the locus of E-NTPDase2 on human chromosome 9 cause severe head and eye defects, includin
156 etained heterozygosity of the TSC1 region on chromosome 9 caused an apparently TSC2- and mTOR-indepen
157 gest that genetic variation at this locus on chromosome 9 causes sporadic ALS and familial ALS-fronto
159 nomic copies of CNTNAP3 on both sides of the chromosome 9 centromere flanking the polymorphic heteroc
160 prcd close to the centromeric end of canine chromosome 9 (CFA9), and excludes RARA as a candidate ge
161 pid valve malformation (CTVM) maps to canine chromosome 9 (CFA9), in a region syntenic with gene-dens
167 have recently identified a portion of mouse chromosome 9 containing a paternally methylated region a
170 l genome-wide significant locus with FEV1 on chromosome 9 [DBH] in a meta-analysis of three study pop
171 ogene highly homologous to ESO3 was found on chromosome 9, designated psiESO3.A search of the rodent
172 u, which is encoded by a cluster of genes on chromosome 9, differs between the sexes in cattle, as as
175 ocus, in PTPN22, and in a SNP (rs3761847) on chromosome 9 for all samples tested, the latter with an
177 associations (P values to ) with a region on chromosome 9 harboring cinnamoyl-CoA reductase, a key en
178 e also identified a novel significant QTL on chromosome 9 (Hfhl3) with moderate effects on 2f1-f2 emi
179 n mapped to the centromeric region of canine chromosome 9, homologous to human 17q, which contains th
180 panel, mouse Treh was shown to be located on Chromosome 9 in a broad region that is orthologous with
181 titative trait loci with additive effects on chromosome 9 in a region likely to have been inherited f
182 ouse lysyl oxidase-like gene (Loxl) to mouse Chromosome 9 in a region that shares linkage conservatio
183 acterized the RNAs encoded by this region of chromosome 9 in cell lines derived from individuals homo
185 ntified deletions near the Atm gene on mouse chromosome 9 in radiation-induced lymphomas from p53 het
186 r tumor suppressor genes on the short arm of chromosome 9 in SCLC, we tested 46 primary SCLCs by micr
188 e for chagun is recessive and maps to distal chromosome 9, in a region homologous to human chromosome
189 ines possessing different fragments of maize chromosome 9 including intergenomic translocations and m
191 of linkage to renal disease, and a locus on chromosome 9 influenced serum cholesterol but not nephro
194 mon genetic variant at the TRAF1-C5 locus on chromosome 9 is associated with an increased risk of ant
196 rs indicating that loss of heterozygosity on chromosome 9 is not driven by Atm, but by an alternative
197 OS case, WGS revealed paternal isodisomy for chromosome 9, leading to identification of the causal ho
198 eat expansion in C9orf72 was responsible for chromosome 9-linked amyotrophic lateral sclerosis and fr
199 xpansion in the C9ORF72 gene as the cause of chromosome 9-linked frontotemporal dementia and motor ne
200 mice, we validate that both the MHC and the chromosome 9 loci influence the proportion and absolute
202 both persistent and recovered stuttering) on chromosome 9 (LOD = 2.3 at 60 cM) and of the narrower di
203 143 cM of chromosome 5 (lod = 2.0), 7 cM of chromosome 9 (lod = 2.8), 12 cM of chromosome 11 (lod =
205 6 cM on chromosome 5; lod = 3.5 at 160 cM on chromosome 9; lod = 2.5 at 7 cM on chromosome 12; and lo
206 A suggestive linkage was also found on mouse Chromosome 9 (logarithm of odds ratio score 2.0) around
207 bility to whipworm infection, one located on chromosome 9 (logarithm of the odds ratio [LOD] score, 3
208 sity (LOH) and reduced genome copy number on chromosomes 9 (LOH9) and 16 (LOH16), aberrations which w
209 ified another trichome-expressed ASH gene on chromosome 9 (M82, Solyc09g075710; LA0716, Sopen09g03052
212 stributions for 12 markers spanning 20 cM on chromosome 9 narrowed the possible location of an DAT su
213 n the discovery meta-analysis, rs10757269 on chromosome 9 near CDKN2B had the strongest association w
214 th the strongest evidence of linkage were on chromosome 9 near D9S301 and on 10 near D10S1230, with p
218 3, near D3S3053 (multipoint LOD = 1.48); on chromosome 9, near D9S910 (multipoint LOD = 1.12) and D9
224 CC) expansion in the noncoding region of the chromosome 9 open reading frame 72 (C9orf72) gene is the
225 Hexanucleotide repeat expansions in the chromosome 9 open reading frame 72 (C9orf72) gene underl
227 w is to describe disease mechanisms by which chromosome 9 open reading frame 72 (C9ORF72) repeat expa
231 ucleotide repeat in the non-coding region of chromosome 9 open reading frame 72 has recently been ide
232 es including the recently described c9orf72 (chromosome 9 open reading frame 72) gene, suggesting an
233 otide repeat in the non-coding region of the Chromosome 9 open-reading frame 72 (C9orf72) gene is the
234 G4C2) hexanucleotide repeat expansion in the chromosome 9 ORF 72 (C9ORF72) gene is a common cause of
236 ne (P = 4.19 x 10(-8) for rs6445055), two on chromosome 9 (P = 2.80 x 10(-11) for rs2472493 near ABCA
237 Fluorescence in situ hybridization with a chromosome 9 paint revealed that some of the Ncam deleti
238 ng 21 pairs of oat chromosomes and one maize chromosome 9 pair was used to construct a cosmid library
239 it was associated with concomitant increased chromosome 9 polysomy (P = .002); and with a younger age
240 or CD1 genotype, CD1 protein expression, and chromosome 9 polysomy (representing genomic instability)
241 e evidence for linkage conditional on either chromosome 9 (positive) or chromosome 7 (negative); this
242 ysical map with the cytological structure of chromosome 9 provides a comprehensive cross-referenced c
244 ze chromosome 9 addition line produced maize chromosome 9 radiation hybrids (M9RHs)-oat lines possess
245 hermore, we mapped an overlapping homozygous chromosome 9 region containing PSAT1 in four consanguine
247 mia patients harboring a partial deletion of chromosome 9 revealed a significant decrease in HNRNPK e
251 o be 9S.79 (at position 79% of the length of chromosome 9 short arm) for sCBM9.1, 9S.65 for sCBM9.2,
253 ction effects of chromosome 7 SNP rs6960920, chromosome 9 SNP rs4744411, and NLRP1 SNP rs6502867 on b
254 umors selected for loss of heterozygosity on chromosome 9, some of which were present in the germ lin
256 entify a homozygous loss on the short arm of chromosome 9 suggesting the presence of a novel TSG locu
257 nterestingly, mapping of the mouse Cox7al to chromosome 9 suggests a new syntenic region between the
259 e identified a human genomic DNA sequence on chromosome 9 that encodes human inositol 1,3,4,5,6-penta
260 d genome-wide search, identifying a locus on chromosome 9 that influences the severity and progressio
262 ping, Folbp3 was mapped to a region on mouse chromosome 9 that is syntenic to human chromosome 19p13.
263 Apc(Min/+) mice has identified a modifier on chromosome 9 that significantly affects tumor multiplici
264 tified two exon-trapping products from human chromosome 9 that were highly homologous to hamster LMX1
265 polymorphic sites covering a 5-kb region of chromosome 9 (the msp1 gene) have been typed in 547 isol
267 In two large unrelated kindreds mapping to chromosome 9, the identical R849W missense mutation was
268 kage to markers in a single region of vervet chromosome 9; this observation suggests the possibility
269 rthologs map to a homologous region on mouse chromosome 9; TMOD4 maps to the telomeric end of 1q12 an
272 of nondisjunction causing the translocation chromosome 9 to be differentially distributed to the two
273 is in a region of conserved synteny with pig chromosome 9, to which the porcine gene was subsequently
275 ive trait locus was found (LOD = 5.0) on rat chromosome 9 using a large F2 population (N = 233) deriv
276 rait locus (QTL) was previously found on rat chromosome 9 using Dahl salt-sensitive (S) and Dahl salt
277 e estimate (24.2 Mb) of the disease locus on chromosome 9 using exon array capture identified indepen
279 29 affected sib pairs, the linkage signal on chromosome 9 vanished, while the chromosome 10 signal de
282 ed from the B chromosome to the short arm of chromosome 9 was recently found to be epigenetically sil
285 g of the ky locus to a small region of mouse chromosome 9, we have now undertaken a positional clonin
286 n a novel autoreactivity-associated locus on chromosome 9, we identify a putative modifier, TLR9.
287 us localization of the taiep mutation to rat chromosome 9, we tested whether the mutation resided wit
289 cohol preference quantitative trait locus on chromosome 9 were identified: Arhgef12, Carm1, Cryab, Co
291 k) x B10.BR) mice and reveal that a locus on chromosome 9, which coincides with Idd2, is linked to th
292 a GAA repeat in intron 1 of the FRDA gene on chromosome 9, which encodes a 210 amino acid protein cal
295 previously unannotated 693-bp transcript on chromosome 9 with a potential functional role in melanom
296 everal flanking markers confirmed linkage to chromosome 9 with maximum LOD scores of 3.4 (theta = 0.0
297 related to metabolic activities, a region of chromosome 9 with several growth factor proteins, and re
298 on of two single nucleotide polymorphisms on chromosome 9 with sporadic ALS, in line with findings fr
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