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1 dy state erythropoiesis in a murine model of chronic anemia.
2 solely related to hemodynamic adaptations to chronic anemia.
3 a common adaptive response in patients with chronic anemia.
4 ry artery disease, chronic lung disease, and chronic anemia.
5 p that presents as pure red cell aplasia and chronic anemia.
6 sors and is widely used for the treatment of chronic anemia.
10 major causes ineffective erythropoiesis and chronic anemia and is associated with iron overload due
12 ; for example, EE individuals exhibit a mild chronic anemia, and HbE/beta-thalassemia individuals sho
14 for adequate oxygen delivery in the face of chronic anemia) are mediated by non-NO vasodilators (i.e
16 symptomatic until they developed B19-induced chronic anemia at the ages of 8, 14, and 17 years, respe
17 m acute anemia, analysis of a mouse model of chronic anemia demonstrated that activation of the BMP4-
19 reased red cell turnover in SCD and in other chronic anemia (eg, thalassemia) and may contribute to i
22 because of the cardiovascular adaptation to chronic anemia in non-cardiac-loaded TM patients, which
23 s process has led to strategies for treating chronic anemia in persistently infected patients and may
25 rticoids and other factors that occur during chronic anemia, inflammation, and other conditions of st
26 during any 12-month period, with SCD pain or chronic anemia interfering with daily activities, or wit
28 in vivo and alleviated anemic symptoms in a chronic anemia mouse model by regulating erythroid gene
30 binant epoetin therapy and correction of the chronic anemia of renal failure have greatly reduced the
32 embolism; (2) do not routinely transfuse for chronic anemia or uncomplicated pain crises in patients
34 ystic fibrosis disease is characterized by a chronic anemia that is inadequately compensated by the c
36 he hypoxic response and not other effects of chronic anemia, we compared expression variation in peri
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