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1 Chronic inflammatory demyelinating polyradiculoneuropath
2 t contactin-1, identified from a cohort with chronic inflammatory demyelinating polyradiculoneuropath
3 in acquired demyelinating disorders, such as chronic inflammatory demyelinating polyradiculoneuropath
4 harcot-Marie-Tooth disease type 1A (n = 32), chronic inflammatory demyelinating polyradiculoneuropath
5 group, but identifiable in all patients with chronic inflammatory demyelinating polyradiculoneuropath
6 noglobulin might reduce disability caused by chronic inflammatory demyelinating polyradiculoneuropath
7 s human diseases Guillain-Barre syndrome and chronic inflammatory demyelinating polyradiculoneuropath
8 A limited number of reports have described chronic inflammatory demyelinating polyradiculoneuropath
9 rom patients with Guillain-Barre syndrome or chronic inflammatory demyelinating polyradiculoneuropath
10 ome; and (iv) LSRPN should be set apart from chronic inflammatory demyelinating polyradiculoneuropath
11 s immunoglobulin in Guillain-Barre syndrome, chronic inflammatory demyelinating polyradiculoneuropath
12 n-1 by ELISA was found in four patients with chronic inflammatory demyelinating polyradiculoneuropath
13 that a subgroup of patients with aggressive chronic inflammatory demyelinating polyradiculoneuropath
14 s and relapsing polyneuropathy presenting as chronic inflammatory demyelinating polyradiculoneuropath
15 Chronic inflammatory demyelinating polyradiculoneuropath
16 rted, but also occurred in 58% of those with chronic inflammatory demyelinating polyradiculoneuropath
17 ayed with intervening incorrect diagnoses of chronic inflammatory demyelinating polyradiculoneuropath
18 pheral neuropathy that is often mistaken for chronic inflammatory demyelinating polyradiculoneuropath
19 nating peripheral neuropathies, particularly chronic inflammatory demyelinating polyradiculoneuropath
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