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   1 ave recently been associated with asthma and chronic rhinosinusitis.                                 
     2 llergy, in particular those of patients with chronic rhinosinusitis.                                 
     3 airway diseases, such as cystic fibrosis and chronic rhinosinusitis.                                 
  
     5 cated in nasal polyps (NPs) of patients with chronic rhinosinusitis and might play a significant role
     6 cated in nasal polyps (NPs) of patients with chronic rhinosinusitis and might play a significant role
  
     8 ver, the exact role of microbial biofilms in chronic rhinosinusitis and orbital cellulitis were not e
     9 roducing ability of the clinical isolates in chronic rhinosinusitis and orbital cellulitis, and to lo
  
  
  
    13 atory tract diseases including otitis media, chronic rhinosinusitis, and exacerbations of both cystic
    14  mucosal tissue homogenates in patients with chronic rhinosinusitis, and this effect was most promine
    15    These features mimic essential aspects of chronic rhinosinusitis-associated olfactory loss, and il
  
    17 widespread prevalence of allergic, viral and chronic rhinosinusitis, but how the brain encodes and ma
    18 urgical specimens derived from patients with chronic rhinosinusitis compared to control patients.    
  
  
    21 chanisms and immune pathways associated with chronic rhinosinusitis (CRS) are not fully understood.  
  
    23 of antibiotic therapy is often initiated for chronic rhinosinusitis (CRS) based on symptomatology.   
  
  
    26 lines provide composite criteria to evaluate chronic rhinosinusitis (CRS) control, taking into consid
  
  
  
  
    31 LP protein and its function in patients with chronic rhinosinusitis (CRS) have not been fully explore
    32 revalence of asthma and its association with chronic rhinosinusitis (CRS) have not been widely studie
  
    34 s (EPOS) criteria to study the prevalence of chronic rhinosinusitis (CRS) in a general-population sam
    35 host-microbial interactions in patients with chronic rhinosinusitis (CRS) in hopes of elucidating mec
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
    51 Studies of the underlying cause or causes of chronic rhinosinusitis (CRS) over the past 20 or more ye
  
  
  
    55 ecretions, were collected from patients with chronic rhinosinusitis (CRS) with nasal polyps (CRSwNP) 
  
    57 luding eosinophilia, which is in contrast to chronic rhinosinusitis (CRS) without nasal polyps (NPs).
    58 en postbronchodilator lung function, asthma, chronic rhinosinusitis (CRS), and atopy with age using a
    59  in many mucosal diseases, including asthma, chronic rhinosinusitis (CRS), and eosinophilic esophagit
    60 eased in nasal polyps (NPs) of patients with chronic rhinosinusitis (CRS), as well as in bronchoalveo
    61 Despite the high prevalence and morbidity of chronic rhinosinusitis (CRS), little is known about the 
    62 nnaire items, we identified respondents with chronic rhinosinusitis (CRS), migraine headache, and fat
    63 t-reported outcomes during the management of chronic rhinosinusitis (CRS), PROMs will play an essenti
  
  
  
  
  
  
  
    71 oke (CS) plays a role in the exacerbation of chronic rhinosinusitis (CRS); however, the mechanism for
    72 lated comorbidities are discussed: rhinitis, chronic rhinosinusitis, gastroesophageal reflux, obstruc
  
  
    75 e relationship between allergic rhinitis and chronic rhinosinusitis has been assessed in a number of 
  
    77 lly transmitted infections, cystic fibrosis, chronic rhinosinusitis, inflammatory bowel disease, and 
  
  
    80 es of use included acute inflammation (n=6), chronic rhinosinusitis (n=2), and allergic rhinitis (n=2
    81 ory secretions in pathologic states, such as chronic rhinosinusitis or hyperglycemia, promotes tonic 
    82  underlying disease (asthma, nasal polyps or chronic rhinosinusitis, or both), as well as on the meth
  
    84 ce of symptoms of asthma, allergic rhinitis, chronic rhinosinusitis, smoking status, and history of N
    85 ns to NSAIDs was higher in participants with chronic rhinosinusitis symptoms (Odds Ratio 2.12; 95%CI 
  
    87  polyp specimens from patients with AERD and chronic rhinosinusitis were analyzed by using quantitati
    88 rty (240) patients with clinical features of chronic rhinosinusitis were examined; patients with firs
  
  
    91 d with uncinate tissue (UT) of patients with chronic rhinosinusitis with nasal polyps (CRSwNP) and th
  
  
  
  
  
    97 n of PD-1 and its ligands PD-L1 and PD-L2 in chronic rhinosinusitis with nasal polyps (CRSwNP) is poo
    98 osinusitis without nasal polyps (CRSsNP) and chronic rhinosinusitis with nasal polyps (CRSwNP) using 
    99 hinosinusitis without nasal polyps (CRSsNP), chronic rhinosinusitis with nasal polyps (CRSwNP), and a
  
  
   102 hronic airway inflammatory diseases, such as chronic rhinosinusitis with nasal polyps and asthma, sho
   103 ated respiratory disease is a severe form of chronic rhinosinusitis with nasal polyps in which nearly
  
  
  
   107  a range of premorbid medical conditions for chronic rhinosinusitis without nasal polyps (CRSsNP) and
   108  of IL-19, at lower extent, in patients with chronic rhinosinusitis without nasal polyps (CRSsNP) in 
   109 luids (NLFs) from controls and patients with chronic rhinosinusitis without nasal polyps (CRSsNP), ch
   110 is with nasal polyps (CRSwNP) and those with chronic rhinosinusitis without nasal polyps (CRSsNP; P <
   111 surgery from control subjects, patients with chronic rhinosinusitis without nasal polyps, and patient
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