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1 ave recently been associated with asthma and chronic rhinosinusitis.
2 llergy, in particular those of patients with chronic rhinosinusitis.
3 airway diseases, such as cystic fibrosis and chronic rhinosinusitis.
5 cated in nasal polyps (NPs) of patients with chronic rhinosinusitis and might play a significant role
6 cated in nasal polyps (NPs) of patients with chronic rhinosinusitis and might play a significant role
8 ver, the exact role of microbial biofilms in chronic rhinosinusitis and orbital cellulitis were not e
9 roducing ability of the clinical isolates in chronic rhinosinusitis and orbital cellulitis, and to lo
13 atory tract diseases including otitis media, chronic rhinosinusitis, and exacerbations of both cystic
14 mucosal tissue homogenates in patients with chronic rhinosinusitis, and this effect was most promine
15 These features mimic essential aspects of chronic rhinosinusitis-associated olfactory loss, and il
17 widespread prevalence of allergic, viral and chronic rhinosinusitis, but how the brain encodes and ma
18 urgical specimens derived from patients with chronic rhinosinusitis compared to control patients.
21 chanisms and immune pathways associated with chronic rhinosinusitis (CRS) are not fully understood.
23 of antibiotic therapy is often initiated for chronic rhinosinusitis (CRS) based on symptomatology.
26 lines provide composite criteria to evaluate chronic rhinosinusitis (CRS) control, taking into consid
31 LP protein and its function in patients with chronic rhinosinusitis (CRS) have not been fully explore
32 revalence of asthma and its association with chronic rhinosinusitis (CRS) have not been widely studie
34 s (EPOS) criteria to study the prevalence of chronic rhinosinusitis (CRS) in a general-population sam
35 host-microbial interactions in patients with chronic rhinosinusitis (CRS) in hopes of elucidating mec
51 Studies of the underlying cause or causes of chronic rhinosinusitis (CRS) over the past 20 or more ye
55 ecretions, were collected from patients with chronic rhinosinusitis (CRS) with nasal polyps (CRSwNP)
57 luding eosinophilia, which is in contrast to chronic rhinosinusitis (CRS) without nasal polyps (NPs).
58 en postbronchodilator lung function, asthma, chronic rhinosinusitis (CRS), and atopy with age using a
59 in many mucosal diseases, including asthma, chronic rhinosinusitis (CRS), and eosinophilic esophagit
60 eased in nasal polyps (NPs) of patients with chronic rhinosinusitis (CRS), as well as in bronchoalveo
61 Despite the high prevalence and morbidity of chronic rhinosinusitis (CRS), little is known about the
62 nnaire items, we identified respondents with chronic rhinosinusitis (CRS), migraine headache, and fat
63 t-reported outcomes during the management of chronic rhinosinusitis (CRS), PROMs will play an essenti
71 oke (CS) plays a role in the exacerbation of chronic rhinosinusitis (CRS); however, the mechanism for
72 lated comorbidities are discussed: rhinitis, chronic rhinosinusitis, gastroesophageal reflux, obstruc
75 e relationship between allergic rhinitis and chronic rhinosinusitis has been assessed in a number of
77 lly transmitted infections, cystic fibrosis, chronic rhinosinusitis, inflammatory bowel disease, and
80 es of use included acute inflammation (n=6), chronic rhinosinusitis (n=2), and allergic rhinitis (n=2
81 ory secretions in pathologic states, such as chronic rhinosinusitis or hyperglycemia, promotes tonic
82 underlying disease (asthma, nasal polyps or chronic rhinosinusitis, or both), as well as on the meth
84 ce of symptoms of asthma, allergic rhinitis, chronic rhinosinusitis, smoking status, and history of N
85 ns to NSAIDs was higher in participants with chronic rhinosinusitis symptoms (Odds Ratio 2.12; 95%CI
87 polyp specimens from patients with AERD and chronic rhinosinusitis were analyzed by using quantitati
88 rty (240) patients with clinical features of chronic rhinosinusitis were examined; patients with firs
91 d with uncinate tissue (UT) of patients with chronic rhinosinusitis with nasal polyps (CRSwNP) and th
97 n of PD-1 and its ligands PD-L1 and PD-L2 in chronic rhinosinusitis with nasal polyps (CRSwNP) is poo
98 osinusitis without nasal polyps (CRSsNP) and chronic rhinosinusitis with nasal polyps (CRSwNP) using
99 hinosinusitis without nasal polyps (CRSsNP), chronic rhinosinusitis with nasal polyps (CRSwNP), and a
102 hronic airway inflammatory diseases, such as chronic rhinosinusitis with nasal polyps and asthma, sho
103 ated respiratory disease is a severe form of chronic rhinosinusitis with nasal polyps in which nearly
107 a range of premorbid medical conditions for chronic rhinosinusitis without nasal polyps (CRSsNP) and
108 of IL-19, at lower extent, in patients with chronic rhinosinusitis without nasal polyps (CRSsNP) in
109 luids (NLFs) from controls and patients with chronic rhinosinusitis without nasal polyps (CRSsNP), ch
110 is with nasal polyps (CRSwNP) and those with chronic rhinosinusitis without nasal polyps (CRSsNP; P <
111 surgery from control subjects, patients with chronic rhinosinusitis without nasal polyps, and patient
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