ライフサイエンスコーパス: chrysotile

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1 of rhizobacteria on the dissolution rate of chrysotile.

2 their lengths and were prepared from natural chrysotile.

3 nt and pulmonary fibrosis in mice exposed to chrysotile.

4 ontributing to ER stress in cells exposed to chrysotile.

5 Chrysotile accounts for more than 90% of the asbestos us

6 Groups of rats were exposed to chrysotile aerosol (10 mg/m3) for 3- to 5-h periods over

7 We found that chrysotile and crocidolite exposures have similar effect

8 nuclear translocation, were induced by both chrysotile and crocidolite.

9 nitrite formation, was evident in lungs from chrysotile- and crocidolite-exposed rats at 1 and 6 wk.

10 he UPR was present in macrophages exposed to chrysotile asbestos and if ER stress in macrophages was

11 Chrysotile asbestos exposure was measured in fiber-years

12 ulated by bronchiolar epithelial cells after chrysotile asbestos exposures.

13 nique for fluorescent tagging and imaging of chrysotile asbestos fibers and prepared samples with a d

14 Here, we show that inhaled chrysotile asbestos fibers cause increased myeloperoxida

15 at lung fibroblasts (RLFs) after exposure to chrysotile asbestos fibers in vitro, which results in as

16 ion was examined in mice after inhalation of chrysotile asbestos for 3, 9, and 40 days.

17 ties and differences between crocidolite and chrysotile asbestos in terms of their transcriptional ef

18 whether the present findings are specific to chrysotile asbestos or would be observed after inhalatio

19 Sham and chrysotile asbestos-exposed CC10-IkappaBalphasr Tg(+) an

20 medium-duty vehicle brake linings contained chrysotile asbestos.

21 eration and inflammation after inhalation of chrysotile asbestos.

22 The three exposures to chrysotile caused a large increase in DNA synthesis in t

23 Results showed that L. emarginata increased chrysotile dissolution by more than 2-fold .

24 f L. emarginata had no significant effect on chrysotile dissolution or plant accumulation of Ni in th

25 increased significantly within minutes after chrysotile exposure and remained elevated for a prolonge

26 ed to background levels within 8 weeks after chrysotile exposure.

27 riments also confirmed that highly elongated chrysotile fibers exhibit anisotropic diffusion at short

28 did not show any increase in sensitivity to chrysotile fibers in short-term (4-h) treatment when com

29 (2D) trajectories of thousands of diffusing chrysotile fibers.

30 Macrophages exposed to chrysotile had elevated transcript levels of several ER

31 Crocidolite and chrysotile induced differential expression of 438 out of

32 ced gene alterations were sustained, whereas chrysotile-induced gene alterations returned to backgrou

33 Chrysotile-induced increases in cytosolic Ca(2+) were pa

34 Inhalation models of crocidolite- and chrysotile-induced inflammation and asbestosis were used

35 ither filtered room air (sham-exposed) or to chrysotile or crocidolite asbestos fibers.

36 xposed to asbestos (crocidolite, amosite, or chrysotile) or control particles at moderate doses (1-10

37 bers, crocidolite is considered the most and chrysotile the least oncogenic.

38 Continuous administration of chrysotile was required for sustained high serum levels

39 The studied mineral was chrysotile which was characterized by a low Ni solubilit

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