ライフサイエンスコーパス: chylomicron

1 ysis of triglyceride-rich lipoproteins (e.g. chylomicrons).

2 the unique lipoprotein of the intestine, the chylomicron.

3 ocalization of apolipoprotein B, and loss of chylomicrons.

4 to 112 nm, 87% exceeding 80 nm, the size of chylomicrons.

5 injected human VLDL and intestinally derived chylomicrons.

6 rs on cells the ability to bind both LPL and chylomicrons.

7 nated the ability of GPIHBP1 to bind LPL and chylomicrons.

8 ys a key role in the lipolytic processing of chylomicrons.

9 ted into a lipid emulsion as a surrogate for chylomicrons.

10 sensing to promote assembly and secretion of chylomicrons.

11 sulting from decreased clearance of VLDL and chylomicrons.

12 n of duodenal vagal afferents in response to chylomicrons.

13 proteins nor exclusively secreted as part of chylomicrons.

14 fed a high fat diet, or for the synthesis of chylomicrons.

15 igestion, absorption, and incorporation into chylomicrons.

16 ical properties and metabolic fate of plasma chylomicrons.

17 to be circulating dietary lipid packaged as chylomicrons.

18 is incorporated into the surface of nascent chylomicrons.

19 orm mature, very low-density lipoproteins or chylomicrons.

20 B (apoB): very low density lipoproteins and chylomicrons.

21 ins, i.e., very low density lipoproteins and chylomicrons.

22 maintain a range necessary for formation of chylomicrons.

23 (LDL) and very-low-density lipoproteins and chylomicrons.

24 cess Cu or Cu chelators impaired assembly of chylomicrons.

25 that enterocytes assembled and secreted more chylomicrons.

26 accommodate 300-400 nm procollagen fibres or chylomicrons.

27 In addition to chylomicrons, absorption of phospholipids, free choleste

28 PCTV-chylomicrons acquire apolipoprotein-AI (apoAI) only afte

29 ipoproteins in much the same way as VLDL and chylomicrons acquire most of their apoCs from HDL.

30 n and were considered fused when their cargo chylomicrons acquired apoAI but docked when they did not

31 Rather, the formation of chylomicrons acts as a signal for the induction of intes

32 tify the appearance of carotenoids in plasma chylomicrons after subjects ingested fresh vegetable sal

33 The rapid entry of chylomicrons after the ingestion of a second meal 5 h af

34 hich could be due to the formation of larger chylomicrons after the MUFA-rich meal.

35 Although chylomicrons also contain retinyl esters (REs), a role o

36 d fat resulted in earlier (>1 h) and sharper chylomicron and [(13)C]fatty acid peaks in plasma than i

37 sulin-sensitive men had similar postprandial chylomicron and chylomicron remnant TG concentrations, b

38 Secretion of lipids by the chylomicron and HDL pathways are dependent on microsomal

39 arifies the functions of key proteins of the chylomicron and the HDL pathways.

40 Lipoprotein lipase (LpL) hydrolyzes chylomicron and very low density lipoprotein triglycerid

41 ced postprandial lipemia via effects on both chylomicron and VLDL metabolism.

42 In a subgroup of the women (n = 7), plasma chylomicrons and 3 subfractions of VLDLs were separated

43 s in plasma including lipid-bound in HDL and chylomicrons and as monomeric and dimeric lipid-free/poo

44 s involved in the intracellular transport of chylomicrons and chylomicron-independent secretion pathw

45 asmic reticulum and are either secreted with chylomicrons and HDLs or stored as cytoplasmic lipid dro

46 effects on lipoprotein metabolism, including chylomicrons and HDLs.

47 The appearance of beta-carotene in chylomicrons and in each VLDL subfraction was lower afte

48 ed delayed clearance of intestinally derived chylomicrons and injected human very low density lipopro

49 the question of the direct atherogenicity of chylomicrons and large VLDL.

50 Cells transfected with GPIHBP1 bind both chylomicrons and lipoprotein lipase avidly.

51 Chylomicrons and lipoproteins were removed from the plas

52 We measured the production of chylomicrons and localized SR-B1 by immunohistochemistry

53 d cTAGE5 gene products, in the export of pre-chylomicrons and pre-VLDLs from the ER.

54 increase was limited to the lowest density (chylomicrons and Sf 100-400) subfractions.

55 ake of dietary retinyl ester incorporated in chylomicrons and their remnants and its transfer to the

56 iglycerides are exclusively transported with chylomicrons and this process is critically dependent on

57 sm for the transfer of surface components of chylomicrons and very low density lipoprotein to high de

58 glyceride (TAG)-rich lipoproteins, including chylomicrons and very low density lipoprotein, which is

59 s the CEL(+/+) mice produced primarily large chylomicrons and very low density lipoprotein.

60 accumulation of apoB-48-carrying remnants of chylomicrons and very low density lipoproteins in the pl

61 polipoprotein that dictates the synthesis of chylomicrons and very low density lipoproteins.

62 ipid metabolism, hydrolyzing triglyceride in chylomicrons and very low density lipoproteins.

63 is the essential nonexchangeable protein in chylomicrons and very low-density lipoprotein-derived li

64 TALI, in the endoplasmic reticulum export of chylomicrons and very low-density lipoproteins, but not

65 poprotein B (apoB) dictates the formation of chylomicrons and very low-density lipoproteins, two majo

66 ated with reduced fasting triacylglycerol in chylomicrons and very-low-density lipoproteins (VLDLs) (

67 conversion products were observed mainly in chylomicrons and very-low-density lipoproteins, indicati

68 d that hydrolysis of triglycerides from both chylomicrons and VLDL was significantly reduced in the a

69 intracellular transport of cargo, including chylomicrons and VLDL, may suggest new drug targets for

70 cerol-rich lipoprotein fraction, composed of chylomicrons and VLDLs.

71 Both dietary (chylomicron) and VLDL-triglyceride were cleared across a

72 porated into triglyceride-rich lipoproteins (chylomicrons), and transported in the circulation to var

73 d the appearance of canthaxanthin in plasma, chylomicrons, and each VLDL subfraction (P < 0.05), but

74 Procollagens, pre-chylomicrons, and pre-very low-density lipoproteins (pre

75 Remnants of lipoproteins, intestinal chylomicrons, and very low density lipoprotein (VLDL), a

76 n B-48, showed that HCV(VLDF) is composed of chylomicron- and VLDL-associated HCV particles; peaking

77 Because chylomicrons are 250 nm in average diameter and lipid ab

78 Chylomicrons are transported from the endoplasmic reticu

79 These observations were seen using chylomicrons as well as the synthetic lipid emulsion Int

80 deficiency in the formation and secretion of chylomicrons, as supported by the significantly less apo

81 features for MTTP involvement in intestinal chylomicron assembly and secretion and suggest that hepa

82 Chylomicron assembly and secretion is increased by the e

83 It is involved in chylomicron assembly and secretion, protection from athe

84 Chylomicron assembly begins with the formation of primor

85 blation studies showed that MTP function and chylomicron assembly is essential for the absorption of

86 In addition to chylomicron assembly, intestinal cells have been shown t

87 portant roles in lipid metabolism, including chylomicron assembly, reverse cholesterol transport, and

88 ys a role in intestinal lipid absorption and chylomicron assembly.

89 le source of cholesterol esters for VLDL and chylomicron assembly.

90 poprotein lipase hydrolyses triglycerides in chylomicrons at the luminal surface of the capillaries i

91 nge, soybean oil was linearly related to the chylomicron AUC and Cmax values for alpha-carotene, lyco

92 bean oil, there was a linear increase in the chylomicron AUC and Cmax values for beta-carotene.

93 il, there were minor linear increases in the chylomicron AUC for lutein and alpha- and total tocopher

94 g oil, the interindividual rank order of the chylomicron AUCs was consistent across the carotenoids a

95 sing cells bind lipoprotein lipase (LPL) and chylomicrons avidly.

96 the acidic domain of GPIHBP1 blocked LPL and chylomicron binding to GPIHBP1-expressing cells.

97 yaspartate and polyglutamate blocked LPL and chylomicron binding to GPIHBP1.

98 ntestine-specific inhibitors of MTP decrease chylomicron biogenesis and improve insulin sensitivity i

99 activation of LXR reduces the production of chylomicrons by a mechanism dependent on the apical loca

100 ry low density lipoproteins by the liver and chylomicrons by the intestine.

101 " LPL can mediate the acquisition of nascent chylomicrons by the placenta, although less efficiently.

102 Triglyceride-rich lipoproteins, primarily chylomicrons, can contribute to plasma free fatty acid (

103 The outcome variables were the chylomicron carotenoid and fat-soluble vitamin area unde

104 LCn3s reduced postprandial chylomicron cholesterol and VLDL apolipoprotein B-48.

105 cholesterol of up to 25 and 60% reduction in chylomicron cholesterol content are seen with a 10-mg do

106 re is an increasing tendency for large VLDL, chylomicrons, chylomicron remnants and small, dense LDL

107 uimolar amounts but have opposite effects on chylomicron (CM) production, with GLP-1 significantly re

108 he aim was to determine whether fed VLDL and chylomicron (CM) triacylglycerol (TAG) production rates

109 sm, we investigated uptake and hydrolysis of chylomicron (CM)-retinyl esters (RE) by rat hepatoma (Mc

110 Chylomicrons (CM) can bind endotoxin (lipopolysaccharide

111 n of very-low-density lipoprotein (VLDL) and chylomicrons (CM) was investigated in the ZDF rat model

112 culation for transfer to other lipoproteins (chylomicron [CM], CMr) to facilitate hepatic cholesterol

113 carried in low density lipoprotein (LDL) and chylomicrons (CMs) taken up into the tissues through the

114 Very-low-density lipoprotein (VLDL) and chylomicrons (CMs) transport triacylglycerol (TAG) to pe

115 There is evidence that chylomicron components are involved in this lipid transd

116 Chylomicrons containing newly absorbed retinyl esters ar

117 loped fatty liver disease, because of direct chylomicron deposition via misconnected portal vein and

118 erived fatty acid and a non-CD36 process for chylomicron-derived fatty acid uptake.

119 This was especially so for chylomicron-derived fatty acids, representing the direct

120 omicron production, mechanisms that underlie chylomicron dysregulation, and potential avenues for fut

121 in rodent models of obesity and postprandial chylomicron excursion to validate DGAT-1 inhibition as a

122 ved from the lipid emulsion (a surrogate for chylomicrons; extraction fraction 31 +/- 4%, P < 0.005 v

123 oscopy results and confirmed by the use of a chylomicron flow blocker, cycloheximide, that prevented

124 ified and packaged into triacylglycerol-rich chylomicrons for bodily distribution.

125 ly synthesized triacylglycerols into nascent chylomicrons for secretion.

126 CT (peanut oil), or LCT plus an inhibitor of chylomicron formation (Pluronic L81).

127 The key steps appear to be related to chylomicron formation and secretion and are closely coup

128 CD36 may play an important role in chylomicron formation and secretion and may also facilit

129 fatty acids (FA) and cholesterol uptake, and chylomicron formation and secretion.

130 iacylglycerol resynthesis in enterocytes for chylomicron formation and secretion.

131 ed in triglyceride synthesis and storage and chylomicron formation have altered expression, and large

132 etary fatty acid and cholesterol for optimal chylomicron formation, whereas CD36-independent mechanis

133 lpha activation in the enterocyte on HDL and chylomicron formation.

134 urve (AUC) and maximum content in the plasma chylomicron fraction (Cmax).

135 ow density lipoprotein (VLDL) and negligible chylomicron fraction in a subset of patients with marked

136 , retinyl esters are also present within the chylomicron fraction obtained from Lrat-/- mice.

137 om labeled [(13)C(1)]palmitate in the plasma chylomicron fraction, and [(13)C(1)]palmitate oxidation

138 ene supplements, measurement of postprandial chylomicron fractions after consumption of a beta-carote

139 acid complex, lipoprotein, protein-free, and chylomicron fractions with no need of salt or sugar dens

140 icrons through the enterocyte is the exit of chylomicrons from the endoplasmic reticulum in prechylom

141 iers that are essential for the transport of chylomicrons from the endoplasmic reticulum to the Golgi

142 that facilitates the lipolytic processing of chylomicrons has never been clearly defined.

143 dietary retinol primarily as free retinol in chylomicrons; however, retinyl esters are also present w

144 s after their ingestion, and release them in chylomicrons in response to oral glucose, sham feeding,

145 There is a rapid appearance of chylomicrons in the circulation carrying fat ingested wi

146 After injection of these labeled chylomicrons in the different mice, chylomicron TG uptak

147 the appearance of the carotenoids in plasma chylomicrons increased relative to that after ingestion

148 intracellular transport of chylomicrons and chylomicron-independent secretion pathways are expected

149 he retinyl esters are then incorporated into chylomicrons, intestinal lipoproteins containing other d

150 a rapid transfer of dietary fatty acids from chylomicrons into human milk.

151 ipoprotein lipase-generated fatty acids from chylomicrons into the plasma free fatty acid (FFA) pool

152 lutein concentration was measured in plasma chylomicrons isolated at regular time intervals over 8 h

153 4-RA method, which controls for variation in chylomicron kinetics in vivo and RE recovery during anal

154 n hepatocyte cell line HepG2, incubated with chylomicrons, led to increased accumulation of REs in en

155 treatment inhibited the secretion of larger chylomicron-like lipoproteins without affecting total ch

156 ease and Anderson disease selectively retain chylomicron-like particles within membrane-bound compart

157 rporates and rapidly secretes dietary fat as chylomicrons (lipoprotein particles comprising triglycer

158 fferences in postprandial serum LPS, LBP, or chylomicron LPS concentrations between acute RW, DRW, or

159 LPS and LBP concentrations and postprandial chylomicron LPS concentrations were measured.

160 ion of SNPs associated with the postprandial chylomicron lutein response (0-8-h area under the curve)

161 ned most of the variance in the postprandial chylomicron lutein response.

162 Postprandial chylomicron lutein responses measured after the 2 meals

163 Postprandial chylomicron lutein responses to meals were very variable

164 of the dietary vitamin A is absorbed via the chylomicron/lymphatic route, it is also clear that under

165 By analogy with procollagen, chylomicrons may drive the formation of endoplasmic reti

166 osed roles in vascular disease, satiety, and chylomicron metabolism, there is no known structural bas

167 ion was used as a surrogate for the study of chylomicron metabolism.

168 SNPs in 15 genes related to both lutein and chylomicron metabolism.

169 the membrane, and their conversion to large chylomicrons occurs in the lumen of the smooth endoplasm

170 ol, apolipoprotein B-48 (apoB-48; reflecting chylomicrons of intestinal origin), free fatty acids (FF

171 n meal would be transferred rapidly from the chylomicrons of the blood into human milk.

172 data suggest that in the intestinal mucosa, chylomicrons or their products release endogenous CCK wh

173 er characterized by the inability to produce chylomicrons or very low-density lipoproteins, with the

174 could be explained by changes in exogenous (chylomicron) or endogenous (VLDL) lipid metabolism and w

175 may play an inhibitory or modulatory role in chylomicron packaging in humans.

176 /L; P=0.03) very low-density lipoprotein and chylomicron particle number in addition to triglyceride

177 IV (apoA-IV) is a major component of HDL and chylomicron particles and is involved in reverse cholest

178 Blood sampling occurred intermittently, and chylomicron particles S(f) >400 TAGs were analyzed by ga

179 ing the packaging of additional core TG into chylomicron particles.

180 erol, and vitamin E are absorbed through the chylomicron pathway, a significant amount of these lipid

181 to LCTs, and in vitro assays indicated that chylomicrons prevent basophil activation.

182 role for CEL, namely the promotion of large chylomicron production in the intestine.

183 Because chylomicron production is most efficient proximally we e

184 in-depth understanding of the regulation of chylomicron production may provide leads for the develop

185 Elevated chylomicron production rate contributes to the dyslipide

186 In this review we discuss the regulation of chylomicron production, mechanisms that underlie chylomi

187 6-alpha-tocopherol AUC0- t final in both the chylomicron (r = -0.46 to -0.52) and VLDL (r = -0.49 to

188 The complexity of VLDL and chylomicron remnant clearance was exemplified by the stu

189 orm and then incorporated into reconstituted chylomicron remnant particles ( approximately 100 nm in

190 site of origin of apo E, endocytosis by the chylomicron remnant receptor (LRP) is absolutely depende

191 In the liver, LRP1 serves as a chylomicron remnant receptor and also participates in th

192 ated protein-1 (LRP1) is known to serve as a chylomicron remnant receptor in the liver responsible fo

193 men had similar postprandial chylomicron and chylomicron remnant TG concentrations, but insulin-resis

194 ation in macrophages were investigated using chylomicron remnant-like particles (CRLPs) containing th

195 It has been proposed that in the liver, chylomicron remnants (lipoproteins carrying dietary lipi

196 For chylomicron remnants an additional molecule or molecules

197 andial lipemia and the biological effects of chylomicron remnants and lipolysis products will be revi

198 asing tendency for large VLDL, chylomicrons, chylomicron remnants and small, dense LDL to fall on tre

199 nding and internalization of (125)I-labelled chylomicron remnants derived from palm, olive, corn, or

200 results suggest that antioxidants carried in chylomicron remnants enhance lipid accumulation in macro

201 The effects of protection of chylomicron remnants from oxidation on their uptake and

202 ted by diet composition; (c) the presence of chylomicron remnants in the fasting state on LF/HC diets

203 dings demonstrate that the hepatic uptake of chylomicron remnants is influenced both by the fatty aci

204 To study this, chylomicron remnants labeled with a fluorescent dye were

205 rom normal mice, there was clustering of the chylomicron remnants on the cell surface in the space of

206 Antioxidants carried in chylomicron remnants therefore may promote the developme

207 Clearance of VLDL and chylomicron remnants was hampered, leading to accumulati

208 e. nonesterified unsaturated fatty acids and chylomicron remnants) that induced FGF21 gene expression

209 ns, i.e. low density lipoproteins (LDLs) and chylomicron remnants, are atherogenic.

210 low-density lipoprotein, lipoprotein (a), or chylomicron remnants.

211 ensity lipoprotein (IDL) comprising VLDL and chylomicron remnants.

212 n of LpL activity, and impaired clearance of chylomicron remnants.

213 osed cholesteryl ester and triglyceride-rich chylomicron remnants.

214 mic very low density lipoproteins as well as chylomicron remnants.

215 monstrate any enhanced binding or removal of chylomicron remnants.

216 rotein supplement decreased the postprandial chylomicron response compared with casein in persons wit

217 Patients with chylomicron retention disease and Anderson disease selec

218 Sar1b is defective in chylomicron retention disease and Anderson disease, two

219 The identification of genetic mutations in chylomicron retention disease indicates that Sar1b may p

220 ects, cranio-lenticulo-sutural dysplasia, or chylomicron retention disease, but mechanisms to enlarge

221 ever, it is not known why some patients with chylomicron retention disorder develop hepatic steatosis

222 1B component of this machinery is mutated in chylomicron retention disorder, indicating that this Sar

223 The fractional and absolute transfer of chylomicron retinyl esters (CM-REs), retinol bound to re

224 role in the hydrolysis of newly-endocytosed, chylomicron retinyl esters in both neutral and acidic me

225 The fatty acyl composition of these (chylomicron) retinyl esters suggests that they are synth

226 dial triacylglycerol content and size of the chylomicron-rich fraction, plasma kinetics of [(13)C]fat

227 Cd36 null (Cd36(-/-)) mice exhibit impaired chylomicron secretion but no overall lipid absorption de

228 stinal absorption of lipids, but its role in chylomicron secretion in human beings is unknown.

229 Chylomicron secretion is subject to regulation by variou

230 Chylomicron secretion was reduced dramatically in vivo,

231 control of dietary fatty acid absorption and chylomicron secretion.

232 anes to intracellular organelles and reduced chylomicron secretion.

233 emulsified fat resulted in a 3-fold greater chylomicron size (218 +/- 24 nm) compared with the sprea

234 There were no significant differences in chylomicron size between the 2 groups for either meal, b

235 mbined there was a significant difference in chylomicron size between the SFA- and MUFA-rich meals (P

236 ot appear necessary for initial formation of chylomicron-sized lipid particles in the endoplasmic ret

237 In contrast, clearance of chylomicron-sized n-3 TG emulsions relies on LPL to a ve

238 Removal of chylomicron-sized n-6 TG emulsions is modulated by lipop

239 ntained large cytoplasmic TG droplets and no chylomicron-sized particles within the secretory pathway

240 ct that GPIHBP1 binds lipoprotein lipase and chylomicrons suggest that GPIHBP1 is a key platform for

241 stinal lipid absorption and mobilization and chylomicron synthesis and secretion are highly regulated

242 ed intestinal triacylglycerol absorption and chylomicron synthesis and secretion in DGAT1-deficient (

243 s and 3 females) to investigate the kinetics chylomicron synthesis and the effect of sensory exposure

244 of Noc(-/-) mice on diets that challenge the chylomicron synthesis pathway result in significant redu

245 distal small intestine, a tissue involved in chylomicron synthesis.

246 onal roles of ACAT1 and ACAT2 in the VLDL or chylomicron synthesis/assembly process.

247 Loss of CD36 did not alter either chylomicron TG or retinyl ester uptake.

248 sed plasma TG levels associated with reduced chylomicron TG uptake into BAT and lung.

249 labeled chylomicrons in the different mice, chylomicron TG uptake was reduced by approximately 70% a

250 ic tracers showed that the difference was in chylomicron-TG extraction.

251 gher in men driven by change in postprandial chylomicron-TG level but that increase in 6-h postprandi

252 plasma NEFA and VLDL-TG pools compared with chylomicron-TG.

253 rt-term ingestion of olive oil produced more chylomicrons than did the other dietary oils, which may

254 ration of GLP-2 to men causes the release of chylomicrons that comprise previously synthesized and st

255 ge of bulky cargo, including procollagen and chylomicrons, that is sensitive to adaptor function in i

256 In chylomicrons, the area under the concentration-versus-ti

257 nism of inhibition involves the formation of chylomicrons, the essential role of the apolipoprotein a

258 Before secretion on chylomicrons, these lipids are reesterified into TG, pri

259 te mesenteric lymph flow and are absorbed in chylomicrons through mesenteric lymph.

260 The rate-limiting step in the transit of chylomicrons through the enterocyte is the exit of chylo

261 esses that mediate the transport of HDLs and chylomicrons through the lymphatic vasculature.

262 ron transport vesicles (PCTV) that transport chylomicrons to the cis-Golgi.

263 vage and that the Noc(-/-) mice have reduced chylomicron transit into the plasma following the ingest

264 the enterocyte is the generation of the pre-chylomicron transport vesicle (PCTV) from the endoplasmi

265 o bind to intestinal ER and generate the pre-chylomicron transport vesicle (PCTV).

266 uires ATP to initiate the budding of the pre-chylomicron transport vesicle from intestinal endoplasmi

267 ther ER-derived vesicles such as PTV and pre-chylomicron transport vesicle.

268 of fat in a meal can modify the absorption, chylomicron transport, and further metabolic handling of

269 entrations (P = 0.013), a more rapid rise in chylomicron triacylglycerol concentrations (P = 0.04), a

270 Chylomicron-triacylglycerol disposal was estimated from

271 Chylomicron-triacylglycerol-derived [(13)C(1)]palmitate

272 3)C]palmitate was added to the meal to label chylomicron-triacylglycerol.

273 Further, HLS patients had lowered storage of chylomicron-triacylglycerols (0.74 +/- 0.38 compared wit

274 impair postprandial disposal and storage of chylomicron-triacylglycerols.

275 between time 90 and 120 min driven by higher chylomicron-triglyceride (TG) levels.

276 However, this was minor in comparison with chylomicron-triglyceride fatty acids.

277 ed fatty acids derived from the spillover of chylomicron-triglyceride in the fasted and fed states, a

278 gh with greater fractional extraction of the chylomicron-triglyceride.

279 Metabolism of chylomicron triglycerides in visceral fat may be an impo

280 clude dietary FA that clear to the liver via chylomicron uptake, FA synthesized de novo in the liver

281 h (AUC0- t final) in lipoprotein fractions [chylomicron, very-low-density lipoprotein (VLDL), LDL, h

282 5), plasma lipoproteins were separated into chylomicrons, very-low-density-lipoproteins (VLDL) subfr

283 pholipid (PL) as lipoproteins, mostly in the chylomicron/very low density lipoprotein (VLDL) density

284 rides is the first and rate-limiting step in chylomicron/very low density lipoprotein clearance at th

285 However, this build-up of triglyceride-rich chylomicrons/very low density lipoproteins is not due to

286 receptors for other lipoproteins (e.g. LDL, chylomicrons, vitellogenin).

287 d a marked increase in triglyceride-enriched chylomicrons+VLDL.

288 In group 1, chylomicron, VLDL, IDL, and HDL cholesterol levels predi

289 n triglyceride-rich lipoproteins (containing chylomicrons, VLDL, and remnants) exhibit more complex k

290 ass spectrometry in plasma and lipoproteins (chylomicrons, VLDL, LDL, and HDL).

291 nces basolateral TG, CE, and PL secretion in chylomicron/VLDL particles.

292 the appearance of the carotenoids in plasma chylomicrons was higher after the ingestion of salads wi

293 pha-carotene, beta-carotene, and lycopene in chylomicrons was negligible.

294 for the LDL receptor in vitro, and injected chylomicrons were cleared more rapidly by the liver in t

295 Chylomicrons were isolated by ultracentrifugation, and c

296 Endogenously labeled mouse chylomicrons were produced by tamoxifen treatment of bet

297 Our data suggest that chylomicrons, which vastly exceed the size of typical CO

298 s of fasting plasma triacylglycerols in both chylomicron (x + SE: 100.3 +/- 49.5 compared with 29.2 +