ライフサイエンスコーパス: cigarette smoker

1 R, n = 9) or poor (PR, n = 7; including five cigarette smokers).

2 function independent of changes in lipids in cigarette smokers.

3 users and was appreciably less than that of cigarette smokers.

4 Participants were 220 cigarette smokers.

5 n are efficacious in producing abstinence in cigarette smokers.

6 s and in 17% of bronchial brushes from heavy cigarette smokers.

7 bolytic therapy for myocardial infarction in cigarette smokers.

8 dults with invasive pneumococcal disease are cigarette smokers.

9 The participants were 199 cigarette smokers.

10 tients had pulmonary contusion, and 59% were cigarette smokers.

11 effects of intravenous nicotine in 16 active cigarette smokers.

12 mokers, while 88.2% of the HIV subjects were cigarette smokers.

13 xcretion of both isoprostanes is elevated in cigarette smokers.

14 PD probands were found only in current or ex-cigarette smokers.

15 enty-seven percent came from households with cigarette smokers.

16 has been found in type-II alcoholism and in cigarette smokers.

17 that MAO A would be reduced in the brain of cigarette smokers.

18 owing evaluation of associations among never cigarette smokers.

19 sferrin differed in upper and lower lobes of cigarette smokers.

20 aspirin and antioxidant vitamins in chronic cigarette smokers.

21 g never cigarette smokers but not among ever cigarette smokers.

22 pregulate beta2* nAChRs more than nonmenthol cigarette smokers.

23 similar in menthol compared with nonmenthol cigarette smokers.

24 hewing tobacco separately for never and ever cigarette smokers.

25 s) cigarette smokers, or were high-intensity cigarette smokers.

26 g to susceptibility to infectious disease in cigarette smokers.

27 els of testosterone within the same range as cigarette smokers.

28 d cutpoint recommended for identifying adult cigarette smokers.

29 investigated dopamine synthesis capacity in cigarette smokers.

30 nd pipe smoking were not elevated among ever cigarette smokers.

31 to contribute to the nicotine dependence of cigarette smokers.

32 on profiles found in alveolar macrophages of cigarette smokers.

33 lso shown that these reflexes deteriorate in cigarette smokers.

34 rodigestive reflexes on airway protection in cigarette smokers.

35 emity PAD identified from medical records as cigarette smokers.

36 luated in two prospective cohorts of Chinese cigarette smokers.

37 at high concentrations in the bloodstream of cigarette smokers.

38 on to the increased atherothrombotic risk of cigarette smokers.

39 r genotype increases risk particularly among cigarette smokers.

40 and thereby investigate vascular disease in cigarette smokers.

41 ene influence the pathogenesis of COPD among cigarette smokers.

42 ere periodontitis (25.7%) compared to former cigarette smokers (20.2%), and non-smokers (13.1%).

43 42.4% (95% CI, 39.7% to 45.1%) were current cigarette smokers, 20.3% (CI, 18.6% to 22.1%) were forme

44 At admission, 75% were current and 8% former cigarette smokers, 3% were current cigar or pipe smokers

45 Fourteen cigarette smokers (31+/-4 pack years) and 14 age- and se

46 Among the 27 619 current cigarette smokers, 4224 died during follow-up, with 1130

47 to be men (94% versus 67%, P<0.001), current cigarette smokers (68% versus 32%, P<0.001), and obese (

48 be male (52.4% vs. 36.2%; P = 0.0003), to be cigarette smokers (68.5% vs. 55.3%; P = 0.003), and to h

49 Tobacco cigarette smokers (75%), who were not abstaining from ci

50 ely to be male (87% vs 67%, P=0.01), current cigarette smokers (84% vs 32%, P<0.001), younger (44+/-8

51 ge, 44 years), predominantly male (80%), and cigarette smokers (95%).

52 myeloid dendritic cells (mDCs) derived from cigarette smokers activate autoreactive Th1 and Th17 cel

53 ing of an AIDS diagnosis was being a current cigarette smoker (adjusted odds ratio = 2.57, 95% confid

54 Among cigarette smokers, African Americans and Native Hawaiian

55 ietary intake of n-3 fatty acids may protect cigarette smokers against COPD.

56 Heavy cigarette smokers also had an increased risk of primary

57 nhibition, to the degree observed in chronic cigarette smokers, also increases low-dose nicotine self

58 rtality for menthol compared with nonmenthol cigarette smokers among 65 600 participants in the South

59 with venous occlusion plethysmography in 12 cigarette smokers and 12 age- and gender-matched nonsmok

60 adult participants (11 moderately dependent cigarette smokers and 13 nonsmokers).

61 Twenty normocholesterolemic cigarette smokers and 20 matched healthy control subject

62 ment and root preparation in 53 patients (15 cigarette smokers and 38 non-smokers).

63 of this compound are elevated in urine from cigarette smokers and in humans with hypercholesterolemi

64 us coeruleus (LC) were measured in long-term cigarette smokers and in nonsmokers.

65 These results indicate that cigarette smokers and nonsmokers exposed to cigarette sm

66 tor (nAChR) agonists augment cognition among cigarette smokers and nonsmokers, yet the systems-level

67 um endotoxin concentrations are increased in cigarette smokers and that endotoxin concentrations are

68 ticipants aged 16 to 26 years who were never cigarette smokers and were attitudinally nonsusceptible

69 or gum disease, 18.9% reported being current cigarette smokers, and 6.3% reported having COPD.

70 death rates from prostate cancer in current cigarette smokers, and inconsistent findings in incidenc

71 ylase (HDAC) activity is reduced in cells of cigarette smokers, and low concentrations of theophyllin

72 The lungs of cigarette smokers are known to contain increased concent

73 Cigarette smokers are more susceptible to periodontal di

74 Associations were observed among never cigarette smokers but not among ever cigarette smokers.

75 ches substantially increase quit rates among cigarette smokers, but their safety in patients with myo

76 se contributes to endothelial dysfunction in cigarette smokers by administering the inhibitor allopur

77 Among baseline never cigarette smokers, cigarette smoking initiation between

78 Among baseline non-cigarette smokers, cigarette smoking initiation, current

79 Among cigarette smokers, combined use of varenicline and bupro

80 Urinary IPF2alpha-I is elevated in cigarette smokers compared with controls (1525 +/- 180 v

81 5% confidence interval (CI): 1.09, 1.81) for cigarette smokers compared with never smokers.

82 ain responses to external stimuli in chronic cigarette smokers compared with nonsmokers, only a few s

83 by weight reduction, if indicated), and, for cigarette smokers, counseling aimed at cessation.

84 and self-perceived oral symptoms (OSs) among cigarette smokers (CSs) (group 1), individuals exclusive

85 mmation between water-pipe smokers (WPs) and cigarette smokers (CSs).

86 een craving and regional metabolism in heavy cigarette smokers exposed to cigarette-related cues.

87 Many cigarette smokers express a desire to quit smoking, but

88 alth effects, first detectable in adolescent cigarette smokers, extend into adulthood.

89 Recidivism rates for cigarette smokers following treatment often exceed 80%.

90 for screening high-risk individuals (such as cigarette smokers) for cancer.

91 as validated by comparing blood levels among cigarette smokers from the National Health and Nutrition

92 We found that heavy cigarette smokers (>60 pack-years) have significantly mo

93 Current, former, and non- cigarette smokers had 5.1, 3.9, and 2.8 missing teeth, r

94 Moderate cigarette smokers had a significantly higher risk for mo

95 Cigarette smokers had considerably more cough, phlegm, a

96 Cigarette smokers had significantly higher breath ethane

97 1, 2013), we determined whether baseline non-cigarette smokers had subsequently tried cigarette smoki

98 Current cigarette smokers had the highest prevalence of moderate

99 Pancreatic carcinomas from cigarette smokers harbor more mutations than do carcinom

100 uced risk for Parkinson's disease (PD) among cigarette smokers has been observed consistently during

101 ies suggest that children raised in homes of cigarette smokers have a higher incidence of asthma than

102 Neutrophils in peripheral blood of cigarette smokers have been shown to be primed and more

103 Cigarette smokers have enhanced oxidative stress from ci

104 Cigarette smokers have higher rates of in vivo and in vi

105 ide treatment reduces cue-induced craving in cigarette smokers; however, the mechanism by which bupro

106 8), healthy nonsmokers (NS; n = 8), or heavy cigarette smokers (HS; n = 8).

107 s (SES) of the mother, and the presence of a cigarette smoker in the household.

108 The risk among cigarette smokers increased with more years of exposure

109 pendent vasodilation in normocholesterolemic cigarette smokers independent of changes in lipids.

110 and cigarette smoking cessation among adult cigarette smokers, irrespective of their motivation for

111 we hypothesized that the delayed healing in cigarette smokers is caused by the afflicted regenerativ

112 e specimens could provide a marker to screen cigarette smokers long before any symptoms of bladder ca

113 Some cigarette smokers may not be ready to quit immediately b

114 ce imaging data from 20 moderately dependent cigarette smokers (mean age = 25 years; no history of ne

115 relapse were assessed in daily marijuana and cigarette smokers (n = 15) under two within-subject, cou

116 concentration of ELF GSH in HIV-seropositive cigarette smokers (n = 30) compared with nonsmokers (n =

117 F) and serum from control subjects (n = 10), cigarette smokers (n = 8), and individuals with CBD (n =

118 Postmortem LCs were obtained from long-term cigarette smokers (n=7) and from nonsmokers (n = 9), all

119 These data suggest that in abstinent cigarette smokers, nicotine augments initial responses t

120 Among cigarette smokers not willing or able to quit within the

121 was strongly associated with HNC among never cigarette smokers (odds ratio (OR) = 1.71, 95% confidenc

122 es, were far more likely to relapse than non-cigarette smokers (odds ratio: 19, p < .01).

123 n cross-sectional baseline analyses, current cigarette smokers of either sex had significantly more m

124 Restricting analysis to cigarette smokers only provided increased evidence for l

125 an increased risk of lung cancer among male cigarette smokers or asbestos-exposed persons taking bet

126 d IPD incidence rates of men who were either cigarette smokers or nonsmokers at the time of enrollmen

127 g was weakly associated with HNC among never cigarette smokers (OR = 1.20, 95% CI: 0.81, 1.77), analy

128 death were more likely to have been current cigarette smokers (OR = 1.3, 95% CI = 1.0 to 1.8).

129 y and OSCC risk among those who were current cigarette smokers (OR, 4.2; CI, 2.4-7.1) was stronger th

130 garette smoking, were current (past 30 days) cigarette smokers, or were high-intensity cigarette smok

131 maintenance of strong nicotine dependence in cigarette smokers posit (i) a rapid brain nicotine accum

132 ess tobacco and HNC were observed among ever cigarette smokers, possibly reflecting residual confound

133 ated reflexive pharyngeal swallow in chronic cigarette smokers predispose them to risks of aspiration

134 A total of 17 cigarette smokers rated the pleasantness of cigarette pi

135 atigue in n = 18 healthy, briefly abstinent, cigarette smokers scanned repeatedly in a placebo-contro

136 Current cigarette smokers should also be given sufficient inform

137 Therefore, cigarette smokers should be: 1) encouraged to abstain fr

138 A total of 606 cigarette smokers started open-label nicotine patch trea

139 Cigarette smokers tend to drink more alcohol than their

140 l aspects of nicotine addiction, and menthol cigarette smokers tend to upregulate beta2* nAChRs more

141 ary 8-iso-PGF2alpha was 7.1-fold higher in a cigarette smoker than respective control subjects.

142 of lower acoustic neuroma risk among current cigarette smokers than among never smokers.

143 that the risk of tooth loss is greater among cigarette smokers than among non-smokers.

144 cancer (1.4 [1.2-1.7]) that was stronger for cigarette smokers than for never smokers (p for interact

145 capacity was elevated in a larger sample of cigarette smokers that included females.

146 tional imaging to examine neural activity in cigarette smokers, the most prevalent substance-dependen

147 Among never cigarette smokers, the odds ratio for ever cigar smoking

148 Among cigarette smokers, there was some evidence of a dose-res

149 ogistic regression models among baseline non-cigarette smokers to assess whether baseline water pipe

150 Nine were former cigarette smokers, two were current smokers, and one rep

151 d with smoking and nicotine dependence among cigarette smokers, using 1990-1991 data from the Coronar

152 Eighty-one tobacco-dependent cigarette smokers (volunteer sample) underwent positron

153 tional magnetic resonance imaging in chronic cigarette smokers, we measured neural and behavioral res

154 For biologic and behavioral reasons, cigarette smokers weigh less than nonsmokers.

155 Compared with never-smokers, current cigarette smokers were 1.7 times as likely to have root

156 crease in binding to MAO-B was observed when cigarette smokers were compared to nonsmoking subjects.

157 In study 1, 18 healthy volunteer cigarette smokers were exposed to three conditions in a

158 Five hundred six adult (>/=18 years) cigarette smokers were randomly assigned and 315 (62%) c

159 AGE-apolipoprotein B and serum AGE levels in cigarette smokers were significantly higher than those i

160 Current cigarette smokers were those who had smoked > or =100 ci

161 e likely to be female, and less likely to be cigarette smokers when compared with other patients with

162 treatment trial, the authors identified 222 cigarette smokers who failed to show a reduction of more

163 In 2011, current cigarette smokers who had ever used e-cigarettes were mo

164 The 1510 participants were cigarette smokers who were not willing or able to quit s

165 Among baseline non-past 30-day cigarette smokers who were past 30-day e-cigarette users