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1 on employed (pharmacologic administration or cigarette smoking).
2 cardiac diseases or cardiac risks, including cigarette smoking.
3 nderstanding the brain mechanisms underlying cigarette smoking.
4  used e-cigarettes (18.9%) progressed toward cigarette smoking.
5 sk for subsequent progression to traditional cigarette smoking.
6    All analyses included fine adjustment for cigarette smoking.
7  their appeal to youth and curb the onset of cigarette smoking.
8                        The main exposure was cigarette smoking.
9 s associated with progression to traditional cigarette smoking.
10 of nicotine, the main addictive substance in cigarette smoking.
11 eir potential to undermine efforts to reduce cigarette smoking.
12 he initiation and progression of combustible cigarette smoking.
13 r controlling for age, sex, alcohol use, and cigarette smoking.
14  characterized a novel microRNA signature of cigarette smoking.
15 was largely concentrated in snus use but not cigarette smoking.
16  limit the future population-level burden of cigarette smoking.
17 de changes in gene expression in response to cigarette smoking.
18 eversible airflow obstruction usually due to cigarette smoking.
19 nant cause of lung cancer in women is active cigarette smoking.
20  configuration and with greater frequency of cigarette smoking.
21 rsons, it did not appear to be influenced by cigarette smoking.
22 ntrols matched for age, sex, handedness, and cigarette smoking.
23 lete methylation capacity with older age and cigarette smoking.
24                    Most trials examined only cigarette smoking.
25 gar and pipe smoking were stratified by ever cigarette smoking.
26  mutagen, specifically ionizing radiation or cigarette smoking.
27 d initial use of e-cigarettes and subsequent cigarette smoking.
28 cigarette smoking initiation and past 30-day cigarette smoking.
29 on pulmonary function decline as mediated by cigarette smoking.
30  whether e-cigarette use affects the risk of cigarette smoking.
31  OPSCC and a history of </= 10 pack-years of cigarette smoking.
32 otentially offer a lower risk alternative to cigarette smoking.
33  previous genome-wide association studies of cigarette smoking.
34 mprecise estimates, with limited control for cigarette smoking.
35 ciated with increased exercise and decreased cigarette smoking.
36 mmon mental disorders (1.77, 1.08-2.90), and cigarette smoking (2.01, 1.29-3.14).
37 e following RFs were considered: (1) current cigarette smoking, (2) dyslipidemia, (3) diabetes mellit
38 conditions: (1) nicotine patch+denicotinized cigarette smoking, (2) nicotine patch+abstinence from sm
39                             She discontinued cigarette smoking 20 years ago and has an occasional gla
40 Further, employees had a lower prevalence of cigarette smoking (9.7 vs. 17.3% in 2010, P < 0.001) com
41 emale sex (around 2-fold increased odds) and cigarette smoking (about 5-fold reduced odds) predicted
42 ent cigarette smoking initiation and current cigarette smoking, accounting for established sociodemog
43                                              Cigarette smoking adds an estimated $100 billion in annu
44 hese data suggest that, after accounting for cigarette smoking, adherence to a set of healthy behavio
45              Our risk model included current cigarette smoking (adjusted odds ratio (OR) = 3.3, 95% c
46 3; 95% CI, 1.43-9.76, respectively), current cigarette smoking (adjusted odds ratios: 2.48; 95% CI, 1
47 0.56, respectively), and higher intensity of cigarette smoking (adjusted proportional odds ratios: 2.
48 o be associated with young age at diagnosis, cigarette smoking, advanced stage at presentation, and a
49 rst aim of this study was to clarify whether cigarette smoking affects tear secretion, goblet cell de
50 of this study provide further evidences that cigarette smoking affects the regenerative potentials of
51              Parity, oral contraceptive use, cigarette smoking, age at menarche, and diabetes were as
52  ratio [aHR], 2.4; 95% CI, 1.5-3.9), current cigarette smoking (aHR, 1.9; 95% CI, 1.1-3.4), and syste
53 nce use has declined over the last 30 years, cigarette smoking, alcohol and illicit drug use, sexual
54 ed non-significant when adjusted for current cigarette smoking, alcohol consumption, and illicit drug
55 ndex, education, family history of diabetes, cigarette smoking, alcohol drinking, physical activity,
56     Besides accelerating adult FEV1 decline, cigarette smoking also modifies how early-life exposures
57     These observations provide evidence that cigarette smoking alters the DNA methylation patterning
58 ng tendency, parental cigarette smoking, and cigarette smoking among friends.
59 -environment interactions were observed with cigarette smoking and a SNP in ADIPOR1 in African Americ
60  the association between biomarker-confirmed cigarette smoking and acute respiratory distress syndrom
61 n which information on important confounders-cigarette smoking and alcohol consumption-was lacking.
62 , being unmarried, living in the rural area, cigarette smoking and alcohol drinking were associated w
63  have been formally established as caused by cigarette smoking and are included in official estimates
64 may be a part of the biological link between cigarette smoking and atherosclerosis.
65                                              Cigarette smoking and attention-deficit/hyperactivity di
66 essed the relationship between self-reported cigarette smoking and biomarker levels, the validity of
67 l airway collapse [ECAC]) is associated with cigarette smoking and chronic obstructive pulmonary dise
68 ideal treatment medication would reduce both cigarette smoking and cocaine abuse.
69   An ideal pharmacotherapy would reduce both cigarette smoking and cocaine abuse.
70 gnificantly modifies the association between cigarette smoking and COPD.
71 of snus independently predicted the onset of cigarette smoking and current cigarette smoking at follo
72 ehensively determine the association between cigarette smoking and DNA methylation, we conducted a me
73          We examined the association between cigarette smoking and drinking outcomes in the COMBINE (
74                     Frequency of combustible cigarette smoking and e-cigarette use within the past 30
75  been achieved in reducing the prevalence of cigarette smoking and exposure to secondhand smoke.
76 ding assessment of possible joint effects of cigarette smoking and fine particulate matter (particula
77 e pulmonary disease (COPD) is linked to both cigarette smoking and genetic determinants.
78               The negative health effects of cigarette smoking and HIV infection are synergistic.
79         We investigated associations between cigarette smoking and incidence of basal cell carcinoma
80                      The association between cigarette smoking and inflammation is well known.
81 f DNA methylation in the association between cigarette smoking and inflammation.
82 ation in monocytes, a cell type sensitive to cigarette smoking and involved in atherogenesis, may be
83           Evidence of an association between cigarette smoking and latent tuberculosis infection (LTB
84 ty of exposure to 15 case-control studies of cigarette smoking and lung cancer (1985-2009).
85 fication of the association of pack-years of cigarette smoking and lung cancer by intensity of cigare
86 Application of our method in this example of cigarette smoking and lung cancer demonstrated that redu
87 study criteria, including no current tobacco cigarette smoking and no known health problems or prescr
88 s these offer enticing clues that, even amid cigarette smoking and other commonalities in the etiolog
89    DR was associated with diabetes duration, cigarette smoking and peripheral neuropathy.
90 y have implications for predicting long-term cigarette smoking and problems with other substance abus
91           Except for the association between cigarette smoking and provoked VTE, which is potentially
92 outcome measures were weighted prevalence of cigarette smoking and quit ratio (ratio of former smoker
93 e sought to describe a microRNA signature of cigarette smoking and relate it to smoking-associated cl
94 irway inflammation and oxidative stress from cigarette smoking and related carcinogens.
95 y was to investigate the association between cigarette smoking and smoking cessation and the prevalen
96 s are crucial for reducing the prevalence of cigarette smoking and smoking-related illnesses.
97 essment of interactions with patients' heavy cigarette smoking and the relationship of this mechanism
98 tion persists beyond adolescence by studying cigarette smoking and the use of snus (Swedish oral mois
99                                              Cigarette smoking and tooth loss are seldom considered c
100 n the frequency and intensity of combustible cigarette smoking and vaping.
101                           Educational level, cigarettes smoking and physical exercise were not signif
102 rom smoking, (3) placebo patch+denicotinized cigarette smoking, and (4) placebo patch+abstinence from
103 entry, adjusting for age, education, income, cigarette smoking, and alcohol consumption.
104  level, sensation-seeking tendency, parental cigarette smoking, and cigarette smoking among friends.
105 fficients for age, sex, waist circumference, cigarette smoking, and family history of colorectal canc
106 s: older age, white race, diabetes mellitus, cigarette smoking, and hypertension.
107 teristics with regard to HLA shared epitope, cigarette smoking, and inflammation (cytokines).
108  hernia, advanced age, male sex, white race, cigarette smoking, and obesity with an intra-abdominal b
109 reviously treated TB, co-infection with HIV, cigarette smoking, and overcrowding were risk factors as
110 ears with at least a 30 pack-year history of cigarette smoking, and, if a former smoker, had quit wit
111 olesterol, WBC count, and history of current cigarette smoking; and change in CRVE is independently a
112   Nevertheless, there is limited research on cigarette smoking as a potential predictor of differenti
113 with ever use of e-cigarettes or past 30-day cigarette smoking associated with past 30-day e-cigarett
114 s, and the pooled odds ratio for past 30-day cigarette smoking at follow-up was 4.28 (95% CI, 2.52-7.
115      The pooled probabilities of past 30-day cigarette smoking at follow-up were 21.5% for baseline p
116 e smoking initiation, current (past 30 days) cigarette smoking at follow-up, and the intensity of cig
117 snus use were associated with high-intensity cigarette smoking at follow-up.
118 e smoking at follow-up, and the intensity of cigarette smoking at follow-up.
119 d the onset of cigarette smoking and current cigarette smoking at follow-up.
120 e past 30-day e-cigarette users, past 30-day cigarette smoking at follow-up.
121                       Our data indicate that cigarette smoking before first cancer diagnosis increase
122 cutive functions, plays an important role in cigarette smoking behavior.
123 versive conditioning during sleep will alter cigarette-smoking behavior during ensuing wakefulness.
124 ditioning during sleep significantly reduced cigarette-smoking behavior in a sleep stage-dependent ma
125          We also included data from India on cigarette smoking, bidi smoking, chewing tobacco, and se
126 use of nonsteroidal anti-inflammatory drugs, cigarette smoking, body mass index, and history of polyp
127 ncentrations correlated with age, sex, race, cigarette smoking, body mass index, inheritance of the e
128 practice.A 64-year-old man with a history of cigarette smoking but no significant comorbidities prese
129 as associated with a decreased prevalence of cigarette smoking, but not with lower prevalence of obes
130 on of the excess odds ratio per pack-year of cigarette smoking by time since smoking cessation among
131 ihypertensive medication, diabetes mellitus, cigarette smoking, cardiovascular disease history, atria
132                                              Cigarette smoking causes emphysema, a fatal disease invo
133  the association between e-cigarette use and cigarette smoking cessation among adult cigarette smoker
134                    The effect of duration of cigarette smoking cessation on colorectal cancer risk by
135                     The primary endpoint was cigarette smoking cessation.
136            Members had a lower prevalence of cigarette smoking compared to nonmembers (overall year-a
137 A) have a substantially higher prevalence of cigarette smoking compared to the general population.
138 d with changes in CBF from the denicotinized cigarette smoking conditions to the abstinent conditions
139                       The risk of death from cigarette smoking continues to increase among women and
140                                              Cigarette smoking contributes to the development of dest
141 ciation between baseline e-cigarette use and cigarette smoking, controlling for sex, age, race/ethnic
142 c obstructive pulmonary disease (COPD) where cigarette smoking (CS) is a risk factor for disease deve
143                                              Cigarette smoking damages the extracellular matrix in a
144                               Progression to cigarette smoking, defined using 3 specific states along
145 , use of antihypertensive treatment, current cigarette smoking, diabetes status, body mass index, C-r
146 sociation between 5 modifiable risk factors (cigarette smoking, diabetes, elevated low-density lipopr
147 ere older than 50 years of age, a history of cigarette smoking did not appear to influence the associ
148                                      Tobacco cigarette smoking did not significantly influence relaps
149 that the timing of dopaminergic responses to cigarette smoking differs between men and women.
150     Taken together, our results suggest that cigarette smoking disrupts endogenous proresolving pathw
151                                              Cigarette smoking during pregnancy is a major public hea
152                                     Maternal cigarette smoking during pregnancy remains one of the mo
153             Age, male sex, alcohol drinking, cigarette smoking, elevated alanine aminotransferase, se
154 cy at age 40 years associated with change in cigarette smoking exposure constituted another measure o
155 in substance use (drug use, alcohol use, and cigarette smoking) following surgery to compensate for a
156                  Smokers were abstinent from cigarette smoking for 12 hours for all sessions.
157                                              Cigarette smoking has a broad impact on genome-wide meth
158                                              Cigarette smoking has been associated with higher risk o
159  pulmonary disease (COPD) and persists after cigarette smoking has stopped.
160 e (a composite of drug use, alcohol use, and cigarette smoking, hereafter referred to as composite su
161 hree to six) were obtained for cannabis use, cigarette smoking, high-risk alcohol use, depression and
162 sociated with higher odds of ever or current cigarette smoking, higher odds of established smoking, h
163    National Health Interview Surveys yielded cigarette smoking histories for the US adult population
164 atus (surrogate of human papillomavirus) and cigarette smoking history (pack-years) randomly assigned
165 disease, two diagnostic breathing tests, and cigarette smoking history.
166 haviors, particularly through differences in cigarette smoking; however, the association with fatal C
167 on of modifiable risk factors: dyslipidemia, cigarette smoking, hypertension, obesity, and hyperglyce
168 treatment, including precipitous declines in cigarette smoking, improvements in hypertension treatmen
169                                              Cigarette smoking in asthma is associated with a cortico
170 ion into lung lining fluid was diminished by cigarette smoking in humans and mice.
171 roduced nicotine pharmacokinetics resembling cigarette smoking in humans.
172                                 A history of cigarette smoking in patients with new-onset adult asthm
173 is associated with the highest prevalence of cigarette smoking in the United States, and is also a ti
174                       The disease risks from cigarette smoking increased in the United States over mo
175 tes mellitus, and radiation therapy (but not cigarette smoking) increased the risk for anastomotic le
176                                              Cigarette smoking increases complication risk in surgica
177 These combined negative effects suggest that cigarette smoking increases interleukin-18 levels throug
178                                              Cigarette smoking increases overall mortality, but it is
179                                              Cigarette smoking increases risk for multiple diseases.
180                                              Cigarette smoking increases the risk of developing IPF s
181                                              Cigarette smoking-induced changes in alveolar macrophage
182                         We hypothesized that cigarette smoking induces extensive transcriptomic chang
183  snus use were independently associated with cigarette smoking initiation (adjusted odds ratios: 2.56
184 ine snus use were associated with subsequent cigarette smoking initiation and current cigarette smoki
185  associated with greater risk for subsequent cigarette smoking initiation and past 30-day cigarette s
186 ngitudinal studies reporting odds ratios for cigarette smoking initiation associated with ever use of
187      Among baseline never cigarette smokers, cigarette smoking initiation between baseline and follow
188 moking, the pooled odds ratio for subsequent cigarette smoking initiation was 3.62 (95% CI, 2.42-5.41
189                  The pooled probabilities of cigarette smoking initiation were 30.4% for baseline eve
190        Among baseline non-cigarette smokers, cigarette smoking initiation, current (past 30 days) cig
191                                              Cigarette smoking is a chronic relapsing brain disorder,
192             These data indicate that current cigarette smoking is a clinically important marker for i
193                                              Cigarette smoking is a leading cause of preventable mort
194                                              Cigarette smoking is a leading modifiable cause of death
195                                              Cigarette smoking is a major factor for the development
196                                              Cigarette smoking is a major public health danger.
197                                              Cigarette smoking is a major risk factor for acquisition
198                                              Cigarette smoking is a major risk factor for chronic obs
199                                              Cigarette smoking is a major risk factor in the developm
200                                              Cigarette smoking is a risk factor for many perioperativ
201                                              Cigarette smoking is a risk factor for type 2 diabetes.
202                                              Cigarette smoking is a significant risk factor for Alzhe
203                                        While cigarette smoking is a strong risk factor for invasive p
204                Prenatal exposure to maternal cigarette smoking is a well-established risk factor for
205                                     Maternal cigarette smoking is a well-established risk factor for
206                                              Cigarette smoking is an established environmental risk f
207                                              Cigarette smoking is associated with a decreased inciden
208                                              Cigarette smoking is associated with cognitive decline a
209 his study adds to the existing evidence that cigarette smoking is associated with higher all-cause an
210                                      Purpose Cigarette smoking is associated with increased incidence
211                                              Cigarette smoking is associated with metabolite abnormal
212                                      Current cigarette smoking is associated with reduced acute respo
213                                              Cigarette smoking is associated with structural changes
214                                              Cigarette smoking is linked to important aspects of tube
215                                Intriguingly, cigarette smoking is most strongly and robustly associat
216 rs; and prohibiting ENDS use in places where cigarette smoking is prohibited.
217                                              Cigarette smoking is significantly associated with new-o
218       Despite well-established evidence that cigarette smoking is the largest modifiable risk factor
219                                              Cigarette smoking is the leading cause of preventable de
220                                              Cigarette smoking is the leading preventable cause of de
221                                              Cigarette smoking is the main risk factor for COPD, and
222 he association between psychotic illness and cigarette smoking is well known, the reasons are unclear
223 tes to the lung destruction that accompanies cigarette smoking; it simultaneously inhibits lung tumor
224     We show herein, for the first time, that cigarette smoking leads to an increase in platelet-activ
225                                              Cigarette smoking leads to upregulation of brain nicotin
226                                              Cigarette smoking leads to upregulation of nicotinic ace
227 d as critical neural substrates perpetuating cigarette smoking, little is known about their circuit-l
228 diometabolic risk factors, including current cigarette smoking, low fruit and vegetable intake, low p
229           Study 2: to isolate the effects of cigarette smoking, marijuana intoxication, withdrawal, a
230  brain activation, genetic variation, and/or cigarette smoking may account for this mixed response pr
231                    Our findings suggest that cigarette smoking may increase Alzheimer's disease onset
232           These findings suggest that active cigarette smoking may increase susceptibility to acute r
233 se pathways that are activated by asthma and cigarette smoking may optimize therapeutic responses.
234                Prenatal exposure to maternal cigarette smoking may promote obesity by enhancing dieta
235  of whom were exposed prenatally to maternal cigarette smoking (mean [SD], 11.1 [6.8] cigarettes/d).
236                                              Cigarette smoking measured both by history and biomarker
237 eral epidemiological studies have shown that cigarette smoking might alter the incidence of Alzheimer
238                                              Cigarette smoking more than doubled HSIL risk.
239                            One woman had AC. Cigarette smoking more than doubled HSIL risk.
240             Six lifestyle factors, including cigarette smoking, obesity, no regular use of nonsteroid
241 nd meta-analysis was to assess the impact of cigarette smoking on clinical outcomes following periodo
242 ative effect of self-reported tooth loss and cigarette smoking on COPD among United States adults age
243 vides potential insights into the effects of cigarette smoking on gene expression in whole blood and
244 dearth of studies regarding the influence of cigarette smoking on periodontal inflammatory conditions
245  55-65 years, with at least 20 pack-years of cigarette smoking or current smokers.
246 proximate steady-state concentration between cigarette smoking or vaping, or during nicotine patch us
247  [OR] = 6.31; 95% CI, 5.39-7.39) and current cigarette smoking (OR = 5.96; 95% CI, 5.67-6.27).
248 s (OR = 7.42; 95% CI, 5.63-9.79) and current cigarette smoking (OR = 7.88; 95% CI, 6.01-10.32).
249 d understanding of causal mechanisms linking cigarette smoking, oral health, and COPD, particularly t
250 cific associations were observed for current cigarette smoking (P = 0.05 among adenomas and P < 0.001
251 tification of comorbid conditions related to cigarette smoking, particularly cardiovascular diseases,
252  atherosclerosis through pathways other than cigarette smoking per se.
253 lesterol, and glucose) and health behaviors (cigarette smoking, physical activity, diet, and body mas
254 evalence of the five major HRBs-alcohol use; cigarette smoking, physical inactivity, unhealthy diet,
255                  Two-sided z tests comparing cigarette smoking prevalence revealed no significant dif
256 roviders should counsel their patients about cigarette smoking, preventive dental care, and COPD risk
257                                              Cigarette smoking promotes body weight reduction in huma
258 vy drinking, treatment attendance, drug use, cigarette smoking, psychiatric symptoms, and HIV-risk be
259  in the late luteal phase, whereas aging and cigarette smoking reduced (11)C-vorozole uptake.
260                We tested the hypothesis that cigarette smoking-relevant nicotine inhalation during pr
261      Substantial progress has been made, but cigarette smoking remains one of the most pressing globa
262                                              Cigarette smoking remains the leading avoidable cause of
263 rovide some mechanistic explanations whereby cigarette smoking renders a patient susceptible to tuber
264 s coexpressed with the microRNA signature of cigarette smoking revealed enrichment for immune-related
265                                      Passive cigarette smoking showed very little, if any, increased
266                                              Cigarette smoking, specifically current smoking, remains
267                          After adjusting for cigarette smoking status, participants with high (>3 mg/
268     Associations were strongest for parental cigarette smoking status; compared to children of non-sm
269  tooth loss (0, 1 to 5, 6 to 31, or all) and cigarettes smoking status (never, former, or current) wi
270 sychosocial, and behavioral risk factors for cigarette smoking, the pooled odds ratio for subsequent
271 and the private sector to reduce the toll of cigarette smoking through reduced initiation and increas
272 uding diabetes mellitus, hyperlipidemia, and cigarette smoking to cases.
273 andenburg, Germany, who had complete data on cigarette smoking, tooth loss, and covariates.
274 S) of complex behavioural phenotypes such as cigarette smoking typically employ self-report phenotype
275 es have high rates of marijuana relapse, and cigarette smoking versus recent abstinence does not dire
276 he second leading cause of lung cancer after cigarette smoking via inhalation; however, exposure thro
277                    The report concluded that cigarette smoking was a cause of lung cancer in men and
278                                   Conclusion Cigarette smoking was associated with a reduction in sur
279                                              Cigarette smoking was associated with higher prevalence
280                                 In contrast, cigarette smoking was associated with increased IgM anti
281                                              Cigarette smoking was associated with lower concentratio
282                      The question of whether cigarette smoking was associated with lung cancer was ce
283 t finding from VTE subtype analyses was that cigarette smoking was associated with provoked but not u
284                                   History of cigarette smoking was available and allowed classificati
285 ignificant gene-environment interaction with cigarette smoking was confirmed.
286  lung cancer risk between hyperkeratosis and cigarette smoking was identified, which suggests that pa
287 udy (2012-2014), a study of 332 adults whose cigarette smoking was measured in a naturalistic environ
288     A Cox model, adjusting for pack-years of cigarette smoking, was used to calculate hazard ratios a
289 okers, both pharmacologic administration and cigarette smoking were associated with activity decrease
290 d 95% confidence intervals for pack-years of cigarette smoking were estimated by logistic regression,
291            Individual-level data on parental cigarette smoking were obtained from 12 case-control stu
292  hypertension, high blood sugar, and regular cigarette smoking were rare.
293 ver time (a measure similar to pack-years of cigarette smoking), were calculated using predicted BMIs
294 non-cigarette smokers had subsequently tried cigarette smoking, were current (past 30 days) cigarette
295 in general that is not driven exclusively by cigarette smoking, which is the main risk factor for chr
296                       Although reductions in cigarette smoking will achieve the greatest impact on lu
297 amine the association of current or previous cigarette smoking with clinical and inflammatory variabl
298 y population, we examined the association of cigarette smoking with DNA methylation using single nucl
299 pidemiologic evidence clearly links maternal cigarette smoking with FGR, insight into the molecular m
300 ociations of SNPs, age, body mass index, and cigarette smoking with the rate of progression and relat

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