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1 on employed (pharmacologic administration or cigarette smoking).
2 cardiac diseases or cardiac risks, including cigarette smoking.
3 nderstanding the brain mechanisms underlying cigarette smoking.
4 used e-cigarettes (18.9%) progressed toward cigarette smoking.
5 sk for subsequent progression to traditional cigarette smoking.
6 All analyses included fine adjustment for cigarette smoking.
7 their appeal to youth and curb the onset of cigarette smoking.
8 The main exposure was cigarette smoking.
9 s associated with progression to traditional cigarette smoking.
10 of nicotine, the main addictive substance in cigarette smoking.
11 eir potential to undermine efforts to reduce cigarette smoking.
12 he initiation and progression of combustible cigarette smoking.
13 r controlling for age, sex, alcohol use, and cigarette smoking.
14 characterized a novel microRNA signature of cigarette smoking.
15 was largely concentrated in snus use but not cigarette smoking.
16 limit the future population-level burden of cigarette smoking.
17 de changes in gene expression in response to cigarette smoking.
18 eversible airflow obstruction usually due to cigarette smoking.
19 nant cause of lung cancer in women is active cigarette smoking.
20 configuration and with greater frequency of cigarette smoking.
21 rsons, it did not appear to be influenced by cigarette smoking.
22 ntrols matched for age, sex, handedness, and cigarette smoking.
23 lete methylation capacity with older age and cigarette smoking.
24 Most trials examined only cigarette smoking.
25 gar and pipe smoking were stratified by ever cigarette smoking.
26 mutagen, specifically ionizing radiation or cigarette smoking.
27 d initial use of e-cigarettes and subsequent cigarette smoking.
28 cigarette smoking initiation and past 30-day cigarette smoking.
29 on pulmonary function decline as mediated by cigarette smoking.
30 whether e-cigarette use affects the risk of cigarette smoking.
31 OPSCC and a history of </= 10 pack-years of cigarette smoking.
32 otentially offer a lower risk alternative to cigarette smoking.
33 previous genome-wide association studies of cigarette smoking.
34 mprecise estimates, with limited control for cigarette smoking.
35 ciated with increased exercise and decreased cigarette smoking.
37 e following RFs were considered: (1) current cigarette smoking, (2) dyslipidemia, (3) diabetes mellit
38 conditions: (1) nicotine patch+denicotinized cigarette smoking, (2) nicotine patch+abstinence from sm
40 Further, employees had a lower prevalence of cigarette smoking (9.7 vs. 17.3% in 2010, P < 0.001) com
41 emale sex (around 2-fold increased odds) and cigarette smoking (about 5-fold reduced odds) predicted
42 ent cigarette smoking initiation and current cigarette smoking, accounting for established sociodemog
44 hese data suggest that, after accounting for cigarette smoking, adherence to a set of healthy behavio
46 3; 95% CI, 1.43-9.76, respectively), current cigarette smoking (adjusted odds ratios: 2.48; 95% CI, 1
47 0.56, respectively), and higher intensity of cigarette smoking (adjusted proportional odds ratios: 2.
48 o be associated with young age at diagnosis, cigarette smoking, advanced stage at presentation, and a
49 rst aim of this study was to clarify whether cigarette smoking affects tear secretion, goblet cell de
50 of this study provide further evidences that cigarette smoking affects the regenerative potentials of
52 ratio [aHR], 2.4; 95% CI, 1.5-3.9), current cigarette smoking (aHR, 1.9; 95% CI, 1.1-3.4), and syste
53 nce use has declined over the last 30 years, cigarette smoking, alcohol and illicit drug use, sexual
54 ed non-significant when adjusted for current cigarette smoking, alcohol consumption, and illicit drug
55 ndex, education, family history of diabetes, cigarette smoking, alcohol drinking, physical activity,
56 Besides accelerating adult FEV1 decline, cigarette smoking also modifies how early-life exposures
59 -environment interactions were observed with cigarette smoking and a SNP in ADIPOR1 in African Americ
60 the association between biomarker-confirmed cigarette smoking and acute respiratory distress syndrom
61 n which information on important confounders-cigarette smoking and alcohol consumption-was lacking.
62 , being unmarried, living in the rural area, cigarette smoking and alcohol drinking were associated w
63 have been formally established as caused by cigarette smoking and are included in official estimates
66 essed the relationship between self-reported cigarette smoking and biomarker levels, the validity of
67 l airway collapse [ECAC]) is associated with cigarette smoking and chronic obstructive pulmonary dise
71 of snus independently predicted the onset of cigarette smoking and current cigarette smoking at follo
72 ehensively determine the association between cigarette smoking and DNA methylation, we conducted a me
76 ding assessment of possible joint effects of cigarette smoking and fine particulate matter (particula
82 ation in monocytes, a cell type sensitive to cigarette smoking and involved in atherogenesis, may be
85 fication of the association of pack-years of cigarette smoking and lung cancer by intensity of cigare
86 Application of our method in this example of cigarette smoking and lung cancer demonstrated that redu
87 study criteria, including no current tobacco cigarette smoking and no known health problems or prescr
88 s these offer enticing clues that, even amid cigarette smoking and other commonalities in the etiolog
90 y have implications for predicting long-term cigarette smoking and problems with other substance abus
92 outcome measures were weighted prevalence of cigarette smoking and quit ratio (ratio of former smoker
93 e sought to describe a microRNA signature of cigarette smoking and relate it to smoking-associated cl
95 y was to investigate the association between cigarette smoking and smoking cessation and the prevalen
97 essment of interactions with patients' heavy cigarette smoking and the relationship of this mechanism
98 tion persists beyond adolescence by studying cigarette smoking and the use of snus (Swedish oral mois
102 rom smoking, (3) placebo patch+denicotinized cigarette smoking, and (4) placebo patch+abstinence from
104 level, sensation-seeking tendency, parental cigarette smoking, and cigarette smoking among friends.
105 fficients for age, sex, waist circumference, cigarette smoking, and family history of colorectal canc
108 hernia, advanced age, male sex, white race, cigarette smoking, and obesity with an intra-abdominal b
109 reviously treated TB, co-infection with HIV, cigarette smoking, and overcrowding were risk factors as
110 ears with at least a 30 pack-year history of cigarette smoking, and, if a former smoker, had quit wit
111 olesterol, WBC count, and history of current cigarette smoking; and change in CRVE is independently a
112 Nevertheless, there is limited research on cigarette smoking as a potential predictor of differenti
113 with ever use of e-cigarettes or past 30-day cigarette smoking associated with past 30-day e-cigarett
114 s, and the pooled odds ratio for past 30-day cigarette smoking at follow-up was 4.28 (95% CI, 2.52-7.
115 The pooled probabilities of past 30-day cigarette smoking at follow-up were 21.5% for baseline p
116 e smoking initiation, current (past 30 days) cigarette smoking at follow-up, and the intensity of cig
123 versive conditioning during sleep will alter cigarette-smoking behavior during ensuing wakefulness.
124 ditioning during sleep significantly reduced cigarette-smoking behavior in a sleep stage-dependent ma
126 use of nonsteroidal anti-inflammatory drugs, cigarette smoking, body mass index, and history of polyp
127 ncentrations correlated with age, sex, race, cigarette smoking, body mass index, inheritance of the e
128 practice.A 64-year-old man with a history of cigarette smoking but no significant comorbidities prese
129 as associated with a decreased prevalence of cigarette smoking, but not with lower prevalence of obes
130 on of the excess odds ratio per pack-year of cigarette smoking by time since smoking cessation among
131 ihypertensive medication, diabetes mellitus, cigarette smoking, cardiovascular disease history, atria
133 the association between e-cigarette use and cigarette smoking cessation among adult cigarette smoker
137 A) have a substantially higher prevalence of cigarette smoking compared to the general population.
138 d with changes in CBF from the denicotinized cigarette smoking conditions to the abstinent conditions
141 ciation between baseline e-cigarette use and cigarette smoking, controlling for sex, age, race/ethnic
142 c obstructive pulmonary disease (COPD) where cigarette smoking (CS) is a risk factor for disease deve
145 , use of antihypertensive treatment, current cigarette smoking, diabetes status, body mass index, C-r
146 sociation between 5 modifiable risk factors (cigarette smoking, diabetes, elevated low-density lipopr
147 ere older than 50 years of age, a history of cigarette smoking did not appear to influence the associ
150 Taken together, our results suggest that cigarette smoking disrupts endogenous proresolving pathw
154 cy at age 40 years associated with change in cigarette smoking exposure constituted another measure o
155 in substance use (drug use, alcohol use, and cigarette smoking) following surgery to compensate for a
160 e (a composite of drug use, alcohol use, and cigarette smoking, hereafter referred to as composite su
161 hree to six) were obtained for cannabis use, cigarette smoking, high-risk alcohol use, depression and
162 sociated with higher odds of ever or current cigarette smoking, higher odds of established smoking, h
163 National Health Interview Surveys yielded cigarette smoking histories for the US adult population
164 atus (surrogate of human papillomavirus) and cigarette smoking history (pack-years) randomly assigned
166 haviors, particularly through differences in cigarette smoking; however, the association with fatal C
167 on of modifiable risk factors: dyslipidemia, cigarette smoking, hypertension, obesity, and hyperglyce
168 treatment, including precipitous declines in cigarette smoking, improvements in hypertension treatmen
173 is associated with the highest prevalence of cigarette smoking in the United States, and is also a ti
175 tes mellitus, and radiation therapy (but not cigarette smoking) increased the risk for anastomotic le
177 These combined negative effects suggest that cigarette smoking increases interleukin-18 levels throug
183 snus use were independently associated with cigarette smoking initiation (adjusted odds ratios: 2.56
184 ine snus use were associated with subsequent cigarette smoking initiation and current cigarette smoki
185 associated with greater risk for subsequent cigarette smoking initiation and past 30-day cigarette s
186 ngitudinal studies reporting odds ratios for cigarette smoking initiation associated with ever use of
187 Among baseline never cigarette smokers, cigarette smoking initiation between baseline and follow
188 moking, the pooled odds ratio for subsequent cigarette smoking initiation was 3.62 (95% CI, 2.42-5.41
209 his study adds to the existing evidence that cigarette smoking is associated with higher all-cause an
222 he association between psychotic illness and cigarette smoking is well known, the reasons are unclear
223 tes to the lung destruction that accompanies cigarette smoking; it simultaneously inhibits lung tumor
224 We show herein, for the first time, that cigarette smoking leads to an increase in platelet-activ
227 d as critical neural substrates perpetuating cigarette smoking, little is known about their circuit-l
228 diometabolic risk factors, including current cigarette smoking, low fruit and vegetable intake, low p
230 brain activation, genetic variation, and/or cigarette smoking may account for this mixed response pr
233 se pathways that are activated by asthma and cigarette smoking may optimize therapeutic responses.
235 of whom were exposed prenatally to maternal cigarette smoking (mean [SD], 11.1 [6.8] cigarettes/d).
237 eral epidemiological studies have shown that cigarette smoking might alter the incidence of Alzheimer
241 nd meta-analysis was to assess the impact of cigarette smoking on clinical outcomes following periodo
242 ative effect of self-reported tooth loss and cigarette smoking on COPD among United States adults age
243 vides potential insights into the effects of cigarette smoking on gene expression in whole blood and
244 dearth of studies regarding the influence of cigarette smoking on periodontal inflammatory conditions
246 proximate steady-state concentration between cigarette smoking or vaping, or during nicotine patch us
249 d understanding of causal mechanisms linking cigarette smoking, oral health, and COPD, particularly t
250 cific associations were observed for current cigarette smoking (P = 0.05 among adenomas and P < 0.001
251 tification of comorbid conditions related to cigarette smoking, particularly cardiovascular diseases,
253 lesterol, and glucose) and health behaviors (cigarette smoking, physical activity, diet, and body mas
254 evalence of the five major HRBs-alcohol use; cigarette smoking, physical inactivity, unhealthy diet,
256 roviders should counsel their patients about cigarette smoking, preventive dental care, and COPD risk
258 vy drinking, treatment attendance, drug use, cigarette smoking, psychiatric symptoms, and HIV-risk be
261 Substantial progress has been made, but cigarette smoking remains one of the most pressing globa
263 rovide some mechanistic explanations whereby cigarette smoking renders a patient susceptible to tuber
264 s coexpressed with the microRNA signature of cigarette smoking revealed enrichment for immune-related
268 Associations were strongest for parental cigarette smoking status; compared to children of non-sm
269 tooth loss (0, 1 to 5, 6 to 31, or all) and cigarettes smoking status (never, former, or current) wi
270 sychosocial, and behavioral risk factors for cigarette smoking, the pooled odds ratio for subsequent
271 and the private sector to reduce the toll of cigarette smoking through reduced initiation and increas
274 S) of complex behavioural phenotypes such as cigarette smoking typically employ self-report phenotype
275 es have high rates of marijuana relapse, and cigarette smoking versus recent abstinence does not dire
276 he second leading cause of lung cancer after cigarette smoking via inhalation; however, exposure thro
283 t finding from VTE subtype analyses was that cigarette smoking was associated with provoked but not u
286 lung cancer risk between hyperkeratosis and cigarette smoking was identified, which suggests that pa
287 udy (2012-2014), a study of 332 adults whose cigarette smoking was measured in a naturalistic environ
288 A Cox model, adjusting for pack-years of cigarette smoking, was used to calculate hazard ratios a
289 okers, both pharmacologic administration and cigarette smoking were associated with activity decrease
290 d 95% confidence intervals for pack-years of cigarette smoking were estimated by logistic regression,
293 ver time (a measure similar to pack-years of cigarette smoking), were calculated using predicted BMIs
294 non-cigarette smokers had subsequently tried cigarette smoking, were current (past 30 days) cigarette
295 in general that is not driven exclusively by cigarette smoking, which is the main risk factor for chr
297 amine the association of current or previous cigarette smoking with clinical and inflammatory variabl
298 y population, we examined the association of cigarette smoking with DNA methylation using single nucl
299 pidemiologic evidence clearly links maternal cigarette smoking with FGR, insight into the molecular m
300 ociations of SNPs, age, body mass index, and cigarette smoking with the rate of progression and relat
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