ライフサイエンスコーパス: ciliary motility

1 nd are left-biased at the LRO in response to ciliary motility.

2 3 and that RS3 and the CSC are important for ciliary motility.

3 ositioned to integrate signals that regulate ciliary motility.

4 ivity of specific dynein isoforms to control ciliary motility.

5 (FAP221) complex is essential for control of ciliary motility.

6 begun to elucidate the mechanisms underlying ciliary motility.

7 alus, and defined a unique role for hydin in ciliary motility.

8 oregulated gene (PACRG), a protein linked to ciliary motility.

9 romolecular complexes that are essential for ciliary motility.

10 associated with the protofilament ribbon and ciliary motility, also positively regulates lifespan.

11 e from the underlying epithelia, to maximize ciliary motility and clearance of bacteria.

12 ctional ion signaling compartment connecting ciliary motility and flow to molecular LR signaling.

13 is caused by a central pair defect impairing ciliary motility and fluid transport in the brain.

14 Thus, Ofd1 is required for ciliary motility and function in zebrafish, supporting d

15 long which motor proteins transmit force for ciliary motility and intraflagellar transport.

16 es that have a range of functions, including ciliary motility and mucous secretion, and contain enzym

17 ubpopulations of cells which retained active ciliary motility and others which demonstrated specializ

18 broken at the left-right organizer (LRO) by ciliary motility and the resultant directional flow of e

19 y dyskinesia (PCD) has relied on analysis of ciliary motility and ultrastructure; however, these test

20 n factor that controls centriole docking and ciliary motility, and airways fail to become fully cilia

21 terms of the assembly and stability of DMTs, ciliary motility, and other microtubule systems.

22 inducing gene product whose mutation impairs ciliary motility, and polycystin-2, whose ablation is as

23 Nevertheless, neither cilia nor ciliary motility are absolutely required for otolith tet

24 onal control of primary cilium formation and ciliary motility are beginning to be understood, but lit

25 n mechanism, axonemal integrity and possibly ciliary motility are required for signal amplification o

26 o TTLL6 paralogs caused severe deficiency in ciliary motility associated with abnormal waveform and r

27 sh likewise disrupts dynein arm assembly and ciliary motility, causing primary ciliary dyskinesia phe

28 a, and, consequently, this process underlies ciliary motility, cilium-based signaling, and ciliopathi

29 x1c1 in zebrafish also caused laterality and ciliary motility defects.

30 5, DRC4/GAS8) have been linked to defects in ciliary motility in humans and lead to a ciliopathy know

31 roscopy to investigate the role of cilia and ciliary motility in otolith formation.

32 of mutant forms leads to profound defects in ciliary motility, including the failure of the hydrodyna

33 However, how ciliary motility is perceived and transduced into asymme

34 yses begin to reveal the mechanisms by which ciliary motility is regulated.

35 to generate sensory potentials and regulate ciliary motility, is normally localized in the cilia, pr

36 and Ncx4a morphants and that the defects in ciliary motility, KV fluid flow and placement of interna

37 In mutants with defective cilia (iguana) or ciliary motility (lrrc50), otoliths are frequently ectop

38 utant mice with impaired ciliogenesis and/or ciliary motility of the node.

39 y only one or two cilia per cell, which lack ciliary motility-related proteins (DNAH5; CCDC39) as see

40 constituent of airway lining fluid, enhances ciliary motility, relaxes airway smooth muscle, inhibits

41 al, and biophysical studies, some aspects of ciliary motility remain elusive, such as the regulation

42 Furthermore, our work suggests additional ciliary motility-signaling connections, since EFHC1 (EF-

43 Asymmetric ICOs occur with onset of LRO ciliary motility, thus representing the earliest known L

44 at the LRO that connect ciliary sensation of ciliary motility to downstream left-right signaling.

45 e (MT) biology, the sliding MT hypothesis of ciliary motility was born.

46 , a cation channel, are required for sensing ciliary motility, yet their function and the molecular m