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1 e intraocular pressure via relaxation of the ciliary muscle.
2 annular electrode operating as an artificial ciliary muscle.
3 through receptor-mediated mechanisms in the ciliary muscle.
4 as observed in the ciliary processes and the ciliary muscle.
5 djacent sclera and could be traced up to the ciliary muscle.
6 n that TIGR protein also is expressed in the ciliary muscle.
7 the isolated human outflow system devoid of ciliary muscle.
8 decreases the amount of TIGR protein in the ciliary muscle.
9 alter the amount of TIGR protein within the ciliary muscle.
10 rved in ciliary muscle fibers throughout the ciliary muscle.
11 ular anterior segments, which lack an intact ciliary muscle.
12 outflow tissues in the absence of an intact ciliary muscle.
13 ial preparations of isolated human or monkey ciliary muscles.
14 sting the shape of its lens with the help of ciliary muscles.
15 as decreased and structurally altered in the ciliary muscle (46 +/- 5.6%) and trabecular meshwork (37
16 olvement occurred in the ciliary body and/or ciliary muscle (7 eyes [58%]), iris (6 eyes [50%]), and
17 ermine the cellular distribution of MMP-1 in ciliary muscle, additional sections were double-immunost
20 ceptors do not mediate the relaxation of the ciliary muscle and reduction of intraocular pressure in
23 sses through extracellular spaces within the ciliary muscle and then through the suprachoroidal space
28 ected in the anterior corneal stroma, in the ciliary muscle, beneath the anterior border of the iris,
29 y (OD) along two line segments overlying the ciliary muscle, by using a high-resolution imaging densi
30 expression in preconfluent and postconfluent ciliary muscle cell cultures by immunocytochemistry.
31 hypothesis that increased MMP production by ciliary muscle cells has a role in increasing uveosclera
33 undertaken to determine whether treatment of ciliary muscle cells with the prostaglandin (PG) analogu
38 n cell membranes isolated from rhesus monkey ciliary muscle, ciliary processes, trabecular meshwork,
39 WDR36 gene expression in lens, iris, sclera, ciliary muscles, ciliary body, trabecular meshwork, reti
42 Age-related changes in lens elasticity and ciliary muscle contractility can affect how ocular param
43 on HCSM may be relevant to the regulation of ciliary muscle contraction and aqueous humor outflow.
44 g to the findings, authors suggest that both ciliary muscle contraction and ciliary body edema may pl
49 se changes in uveoscleral outflow across the ciliary muscle could cause elevation of intraocular pres
51 and William Wallace had all zeroed in on the ciliary muscle, describing its anatomy in varying detail
53 ngs quantify the movements of the zonule and ciliary muscle during accommodation, and identify their
54 ation and support the view that reduction of ciliary muscle ECM may contribute to increased uveoscler
55 data support the possibility that changes in ciliary muscle ECM may contribute to increased uveoscler
58 MT2 and CMT3), but paradoxically, the apical ciliary muscle fibers are thicker in hyperopia (CMTMAX a
59 ata indicated that in children the posterior ciliary muscle fibers are thicker in myopia (CMT2 and CM
60 IGR protein immunoreactivity was observed in ciliary muscle fibers throughout the ciliary muscle.
62 the ciliary muscle to increase spaces among ciliary muscle fibers, thereby reducing hydraulic resist
65 rmation may be altered in glaucomatous human ciliary muscle (g-HCM) cells compared with normal (n)-HC
67 owman and Brucke wrong about the role of the ciliary muscle in accommodation, and for different reaso
69 meshwork, Schlemm's canal, scleral spur, and ciliary muscle indicates a structural or functional role
70 ong calponin immunoreactivity was present in ciliary muscle, iris dilator and sphincter muscles, and
71 ensity was approximately fivefold greater in ciliary muscle, iris root, and sclera than in trabecular
73 d distribution of calponin proteins in human ciliary muscle, iris, and other anterior segment tissues
74 trong MMP-1 immunoreactivity was observed in ciliary muscle, iris, sclera, corneal endothelium, and c
76 in the extracellular matrix (ECM) of monkey ciliary muscle is reduced during anterior segment inflam
77 use uveoscleral outflow, which traverses the ciliary muscle, is increased by prostaglandins (PGs), th
83 due to direct action of phenylephrine on the ciliary muscle or to secondary optical factors associate
84 e POAG regions compared with normal regions--ciliary muscle (P < 0.001), ciliary stroma (P < 0.001),
88 eshwork, Schlemm's canal, ciliary processes, ciliary muscle, retina, choroid, cultured RPE cells, and
91 crease in the diameter of the unaccommodated ciliary muscle ring was highly correlated with advancing
95 decreased (-0.105 mm/D, P < 0.001), and the ciliary muscle thickened anteriorly (+0.013 to +0.026 mm
96 e used to determine the relationship between ciliary muscle thickness in various regions of the muscl
97 r diopter of accommodation in LED, CMRD, and ciliary muscle thickness were not related to subject age
99 tomographer was used to measure cycloplegic ciliary muscle thicknesses at 1 mm (CMT1), 2 mm (CMT2),
101 initiate the alteration of collagens in the ciliary muscle to increase spaces among ciliary muscle f
102 ic pathway activating the iris sphincter and ciliary muscle to mediate pupillary constriction and len
104 wo line segments overlying the immunostained ciliary muscle using two-dimensional imaging densitometr
106 The distribution of TIGR protein in the ciliary muscle was determined by confocal scanning laser
110 es extracellular matrix remodeling of ocular ciliary muscle, with a resultant increase in drainage of
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