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1 ughout the brain of the green treefrog (Hyla cinerea).
2 using the parameters of a Grey Heron (Ardea cinerea).
3 ion against the necrotrophic fungus Botrytis cinerea.
4 s plant host and the plant pathogen Botrytis cinerea.
5 DF1.2, and confers enhanced resistance to B. cinerea.
6 to the necrotrophic fungal pathogen Botrytis cinerea.
7 than wild-type plants to the fungus Botrytis cinerea.
8 sclerotiorum and the related fungus Botrytis cinerea.
9 us niger, Trichoderma harzianum and Botrytis cinerea.
10 lity of the ERF6-EAR transgenic plants to B. cinerea.
11 c acid is essential for the resistance to B. cinerea.
12 t the necrotrophic fungal pathogen, Botrytis cinerea.
13 the response of the transgenic plants to B. cinerea.
14 psidis, and the necrotrophic fungus Botrytis cinerea.
15 lings were infected with the fungus Botrytis cinerea.
16 by the necrotrophic fungal pathogen Botrytis cinerea.
17 ense toward the necrotrophic fungus Botrytis cinerea.
18 rks mediating the Arabidopsis response to B. cinerea.
19 mato DC3000 and the fungal pathogen Botrytis cinerea.
20 rd the necrotrophic fungal pathogen Botrytis cinerea.
21 nduced by the necrotrophic pathogen Botrytis cinerea.
22 oth exhibited decreased susceptibility to B. cinerea.
23 by the necrotrophic fungal pathogen Botrytis cinerea.
24 nd the necrotrophic fungal pathogen Botrytis cinerea.
25 enhanced susceptibility of ssi2 plants to B. cinerea.
26 are attenuated in ripe fruit infected by B. cinerea.
27 ally epistatic to rst1, for resistance to B. cinerea.
28 erexpressing line inhibited tip growth of B. cinerea.
29 bility to the necrotrophic pathogen Botrytis cinerea.
30 B1 overexpression conferred resistance to B. cinerea.
31 disease symptoms when infected with Botrytis cinerea.
32 r resistance to the fungal pathogen Botrytis cinerea.
33 to necrotrophic pathogens, such as Botrytis cinerea.
34 three displayed an enhanced resistance to B. cinerea.
35 sis resistance to the necrotrophic fungus B. cinerea.
36 abiotic stress and in the pathogenesis of B. cinerea.
37 to a necrotrophic fungal pathogen, Botrytis cinerea.
38 nd the necrotrophic fungal pathogen Botrytis cinerea.
39 more susceptible to both P. syringae and B. cinerea.
40 on against the fungal phytopathogen Botrytis cinerea.
41 exin than wild-type plants in response to B. cinerea.
42 ct in a response pathway more specific to B. cinerea.
43 by the necrotrophic fungal pathogen Botrytis cinerea.
44 to the necrotrophic fungal pathogen Botrytis cinerea.
45 stance to the necrotrophic pathogen Botrytis cinerea.
46 ysacchareae, Neisseria kochii, and Neisseria cinerea.
47 n sites of Penicillium expansum and Botrytis cinerea.
48 ty against the fungal phytopathogen Botrytis cinerea.
49 of the inclusion complexes against Botrytis cinerea.
50 ut not to the necrotrophic pathogen Botrytis cinerea.
51 e grape (Vitis vinifera) berries by Botrytis cinerea.
52 in driving time-of-day susceptibility to B. cinerea.
53 e radicals, and reduced susceptibility to B. cinerea.
54 o the necrotrophic fungal pathogen, Botrytis cinerea.
55 ant wrky33 is highly susceptible to Botrytis cinerea.
56 1 than in wild-type plants in response to B. cinerea.
57 induced resistance in Arabidopsis against B. cinerea.
58 of the most fungitoxic compounds against B. cinerea.
59 n infect and cause hypovirulence in Botrytis cinerea.
60 P13 was induced in leaves challenged with B. cinerea.
61 ses hypovirulence in Sclerotinia spp. and B. cinerea.
62 of the necrotrophic fungal pathogen Botrytis cinerea.
64 reased resistance toward gray mold (Botrytis cinerea), a pathogen responsible for major losses in agr
65 n of oxi-mC across the genome of Coprinopsis cinerea, a basidiomycete that encodes 47 TET/JBP paralog
66 ed in reduced choosiness by female Nauphoeta cinerea, a cockroach that has reproductive cycles and gi
67 However, reduced susceptibility to Botrytis cinerea, a major postharvest fungal pathogen of tomato,
68 V1/WF-1 virions into strain KY-1 of Botrytis cinerea also resulted in reductions in virulence and myc
69 to several plant pathogens (namely Botrytis cinerea, Alternaria brassicicola and Golovinomyces oront
70 economically important necrotrophic fungi B. cinerea, Alternaria brassicicola, Fusarium graminearum,
73 ptibility to the necrotrophic fungi Botrytis cinerea and Alternaria brassicicola as well as the gener
74 lity to the necrotrophic fungal pathogens B. cinerea and Alternaria brassicicola based on increased p
75 o the necrotrophic fungal pathogens Botrytis cinerea and Alternaria brassicicola concomitant with red
76 o the necrotrophic fungal pathogens Botrytis cinerea and Alternaria brassicicola in the Nossen-0 back
77 o the necrotrophic fungal pathogens Botrytis cinerea and Alternaria brassicicola, whereas HUB1 overex
81 ceptibility to the pathogenic fungi Botrytis cinerea and Alternaria brassisicola Both PAMPs and osmot
82 responses to the fungal necrotrophs Botrytis cinerea and Alternaria solani, bacterial pathogen Pseudo
83 tibility to the necrotrophic fungus Botrytis cinerea and an increased tolerance to the biotrophic Pse
84 resistance to the fungal pathogens Botrytis cinerea and Bipolaris sorokiniana but not to the bacteri
85 stance to the necrotrophic pathogen Botrytis cinerea and contributes to basal defense induced by flg2
86 -type locus of the model species Coprinopsis cinerea and encodes two tightly linked pairs of homeodom
88 s against the necrotrophic pathogen Botrytis cinerea and for the shade-triggered increased susceptibi
93 Nonpathogenic Neisseria strains (Neisseria cinerea and Neisseria flavescens) do not activate NLRP3
97 Slshn3-RNAi plants are more sensitive to B. cinerea and produce more hydrogen peroxide than wild-typ
100 rongest immunoreactivity in CA2 and fasciola cinerea and sporadic immunoreactivity in CA1; labeling i
101 diated resistance to the necrotroph Botrytis cinerea and susceptibility to the hemibiotroph Pseudomon
102 ce of a chemical cross-regulation between B. cinerea and T. arundinaceum and contributes to understan
104 to the necrotrophic foliar pathogen Botrytis cinerea and the biotrophic bacterial pathogen Xanthomona
105 sistance to the necrotrophic fungus Botrytis cinerea and the caterpillar Mamestra brassicae In additi
106 abundance of cospin in fruiting bodies of C. cinerea and the lack of trypsin-like proteases in the C.
107 to the necrotrophic fungal pathogen Botrytis cinerea and their genetic control are poorly understood.
108 tibility to the necrotrophic fungus Botrytis cinerea and to feeding by larvae of tobacco hornworm (Ma
110 e as the recipient Br-0 to the necrotroph B. cinerea and to the biotroph Hyaloperonospora arabidopsid
111 PA production in response to necrotrophic B. cinerea and virulent Pst DC3000 infection, but contribut
112 hal growth of most germinating conidia of B. cinerea and was eventually lethal to infected hyphae, si
113 In bos3, the mutant most susceptible to B. cinerea and with the highest expression of PR-1, removal
115 tibility to the necrotrophic fungus Botrytis cinerea, and increased sensitivity to salt and oxidative
116 tin matrix is the main CW target of Botrytis cinerea, and pectin methylesterification status is stron
117 mutant exhibits reduced susceptibility to B. cinerea, and the B. cinerea dcl1 dcl2 double mutant that
118 sponses against the fungal pathogen Botrytis cinerea, and thus we conclude that the regulation of sym
119 the ripening-associated genes induced by B. cinerea are LePG (for polygalacturonase) and LeExp1 (for
121 several PGs from the plant pathogen Botrytis cinerea as well as one from the saprotroph Aspergillus n
122 d the susceptibility of ripening fruit to B. cinerea, as measured by fungal biomass accumulation and
124 cutin-defective mutants for resistance to B. cinerea: att1 (for aberrant induction of type three gene
125 Trichoderma arundinaceum (Ta37) and Botrytis cinerea (B05.10) produce the sesquiterpenoids harzianum
129 activity and induced expression of Botrytis cinerea BOT genes, although their total antagonistic pot
131 olerance to the necrotrophic fungus Botrytis cinerea but susceptibility to the hemibiotrophic bacteri
132 tance to the necrotrophic fungal pathogen B. cinerea, but a negative role in the SA-dependent signali
133 to the necrotrophic fungal pathogen Botrytis cinerea, but showed normal responses to virulent and avi
134 (1) was assigned to a metabolite of Botrytis cinerea, but the spectra of several synthetic analogues
135 13 contributes to the basal resistance to B. cinerea by limiting symptom development and points out t
136 rom the necrotrophic plant pathogen Botrytis cinerea, catalyzes the multistep cyclization of farnesyl
137 Pst-AvrRpt2, Dickeya dadantii, and Botrytis cinerea Characterization of the redox status demonstrate
138 SEP4 in the plant grey mould fungus Botrytis cinerea completely blocked IFS formation and abolished t
139 sistance to the necrotrophic fungus Botrytis cinerea, consistent with substantial upregulation of the
140 ced susceptibility to B. cinerea, and the B. cinerea dcl1 dcl2 double mutant that can no longer produ
142 lants show differential susceptibility to B. cinerea depending on the time of day of inoculation.
143 cate that Sep4 plays pleiotropic roles in B. cinerea development and specifically facilities host inf
144 NA effectors are mostly produced by Botrytis cinerea Dicer-like protein 1 (Bc-DCL1) and Bc-DCL2.
147 we show that the commensal species Neisseria cinerea expresses functional fHbp on its surface and tha
149 s, which is required for full immunity to B. cinerea Finally, we present a structural model of MOS7 a
151 0 mg/L against Rhizopus stolonifer, Botrytis cinerea, Fusarium oxysporum and Colletotrichum gloeospor
153 esults in an up-regulation of most of the B. cinerea genes involved in virulence yet the presence of
154 the lack of trypsin-like proteases in the C. cinerea genome, these results suggest that cospin and it
156 hronology of the defense response against B. cinerea, highlighting the times at which signaling and m
157 th OGs and macerozyme-induced immunity to B. cinerea in Col-0, only OGs also induced immunity in gae1
159 induced by the necrotrophic fungus Botrytis cinerea, including the genes that encode the transcripti
160 coi1) and ethylene-insensitive2 (ein2) to B. cinerea, indicating that ELP2 is an important player in
161 r resistance to the fungal pathogen Botrytis cinerea, indicating that NHO1 is not limited to bacteria
163 g a four-amino acid deletion, compromises B. cinerea-induced activation of the key immunoregulatory M
164 t18:0-P appear as key players in Pst- and B. cinerea-induced cell death and reactive oxygen species a
165 pression (EAR) motif, strongly suppresses B. cinerea-induced defense gene expression, leading to hype
166 d-cultured seedling system, we found that B. cinerea-induced ethylene biosynthesis was greatly compro
168 asmonic acid (JA) and increased basal and B. cinerea-induced expression of the plant defensin PDF1.2
171 ne expression is similar in both types of B. cinerea-infected plants but is repressed in Atdpl1-1 aft
174 ssion of SlSHN3 resulted in resistance to B. cinerea infection and to X. campestris pv. vesicatoria,
176 ction transgenic plants or in response to B. cinerea infection increases ERF6 protein stability in vi
177 ated during abiotic stresses during Botrytis cinerea infection or after benzothiadiazole and methyl j
178 genic plants were more resistant to Botrytis cinerea infection than wild type, possibly as a conseque
179 direct inhibition, P. aphidis may inhibit B. cinerea infection via induced resistance in a manner ind
180 utant leaves were normally susceptible to B. cinerea infection, a double ein2 npr1 mutant was signifi
181 d more damage than wild type plants after B. cinerea infection, and pretreatment of plants with methy
182 gulated coordinately in response to Botrytis cinerea infection, but through separate signal transduct
183 exhibited reduced susceptibility to Botrytis cinerea infection, confirming AA signaling in other plan
185 in a jasmonate resistant1-1 mutant, after B. cinerea infection, suggesting that P. aphidis can bypass
202 g infection is higher and the immunity to B. cinerea is compromised in pmei10, pmei11, and pmei12 mut
204 sistance to the necrotrophic fungus Botrytis cinerea is conferred by ethylene via poorly understood m
205 We show that reduced susceptibility to B. cinerea is dependent specifically on the accumulation of
206 uggest that PA-mediated susceptibility to B. cinerea is linked to interference with the functions of
207 e against the necrotrophic pathogen Botrytis cinerea is primarily quantitative and genetically comple
208 e that oxalate production in A. niger and B. cinerea is solely dependent on the hydrolytic cleavage o
214 to the susceptibility of wrky33 plants to B. cinerea, it is insufficient for WRKY33-mediated resistan
215 and exogenous application of SA decreased B. cinerea lesion size through an NPR1-dependent mechanism
216 trait loci influencing plant response to B. cinerea, measured as expansion of necrotic lesions on le
217 d B. terrestris amplified Bd abundance on H. cinerea more so in the absence than presence of G. carol
218 , G. carolinensis reduced Bd abundance on H. cinerea more so in the presence than absence of B. terre
219 Examination of the constituents of a B. cinerea mutant that overproduces polyketides gave suffic
220 Below, we report the cloning of the Botrytis cinerea oahA gene and the demonstration that the disrupt
221 lementation we have shown that the intact B. cinerea oahA gene restores oxalate production in an Aspe
222 accumulation, and susceptibility to Botrytis cinerea, one of the most important postharvest pathogens
225 ore resistant to the phytopathogens Botrytis cinerea, Pectobacterium carotovorum, and Pseudomonas syr
226 ing influences the course of infection by B. cinerea, perhaps by changing the structure or the access
229 lturally important plant pathogens (Botrytis cinerea, Pseudomonas syringae, and Fusarium oxysporum) w
231 Macerozyme treatment or infection with B. cinerea released less soluble uronic acid, likely reflec
233 lysis revealed flg22-induced PTI to Botrytis cinerea requires BIK1, EIN2, and HUB1 but not genes invo
234 se data indicate that local resistance to B. cinerea requires ethylene-, jasmonate-, and SA-mediated
235 RELATED GENES (BRGs), which contribute to B. cinerea resistance and the suppression of disease-associ
236 YC2 fail to restore PDF1.2 expression and B. cinerea resistance in elp2, suggesting that ELP2 is requ
241 response to the necrotrophic fungus Botrytis cinerea revealed decreases in the levels of phosphatidyl
242 oes with this coating and inoculated with B. cinerea showed a significant decrease in fungal growth a
243 We suggest that reproductive aging in N. cinerea, similar to aging in general, occurs because the
246 e in necrotropic pathogen susceptibility, B. cinerea susceptibility was assessed in transgenic fruit
247 We analyzed the role of HA and Asp in the B. cinerea-T. arundinaceum interaction, including changes i
248 nic tomato lines were more susceptible to B. cinerea than the wild-type plants; however, responses to
251 tify only in infected berries proteins of B. cinerea that represent potential markers of the presence
252 ce against the necrotrophic fungus, Botrytis cinerea The induced resistance was enhanced in the P2K1
253 immunity to the necrotrophic fungus Botrytis cinerea The mos7-1 mutation, causing a four-amino acid d
256 ection with Pseudomonas syringae or Botrytis cinerea, the expression of genes regulated by both the s
257 arkedly increased plant susceptibility to B. cinerea; the effect of low R:FR was (1) independent of t
258 d by an increased susceptibility to Botrytis cinerea This process was accompanied by an overexpressio
259 cadian system of the plant pathogen Botrytis cinerea to assess if such oscillatory machinery can modu
260 copyranoside (Q3G) isolated from Echinophora cinerea to protect PC12 cells from H2O2-induced cytotoxi
262 ar dialogue between Arabidopsis cells and B. cinerea triggers major changes in host metabolism, inclu
263 aliana with the necrotrophic fungus Botrytis cinerea using millicell culture insert, that enables mol
264 ility of the ySpdSyn transgenic tomato to B. cinerea was associated with down-regulation of gene tran
265 Induction of ATG18a and autophagy by B. cinerea was compromised in the wrky33 mutant, which is h
269 tion of these pathways to defence against B. cinerea was validated through the use of multiple Arabid
270 ial impact of fHbp-containing vaccines on N. cinerea We found that immunization with Bexsero elicits
273 genic fungi, Fusarium oxysporum and Botrytis cinerea, were chosen to examine the antifungal activity
274 highly susceptible to A. brassicicola and B. cinerea, whereas T-DNA insertion alleles are embryonic l
275 sis for tadpoles of Bufo terrestris and Hyla cinerea, whereas tadpoles of B. terrestris (an obligate
277 ow the existence of a functional clock in B. cinerea, which shares similar components and circuitry w
278 to infection by the fungal pathogen Botrytis cinerea, which was associated with much stronger inducti
279 f the coprophilous basidiomycete Coprinopsis cinerea with different bacterial species and identified
280 rhythmic susceptibility of Arabidopsis to B. cinerea with the enhanced susceptibility to this pathoge
281 e indusium griseum, tenia tecta and fasciola cinerea within 5 days post-METH exposure in 70% of the m
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