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1 ular gyrus, mid-frontal cortex, and anterior cingulate gyrus).
2 ula, and limbic lobe (anterior and posterior cingulate gyrus).
3 own the medial prefrontal cortex, toward the cingulate gyrus).
4 people by opposite activity in the anterior cingulate gyrus.
5 binding in orbitofrontal cortex and anterior cingulate gyrus.
6 primary role for dysfunction of the anterior cingulate gyrus.
7 e left fronto-temporal junction and anterior cingulate gyrus.
8 ntly lower metabolism in the dorsal anterior cingulate gyrus.
9 th glucose metabolism in the dorsal anterior cingulate gyrus.
10 ad greater activation in the right posterior cingulate gyrus.
11 lving the motor/premotor cortex and anterior cingulate gyrus.
12 ventromedial nucleus of the hypothalamus and cingulate gyrus.
13 tal and temporal regions and in the anterior cingulate gyrus.
14 phenotypic changes in a control area of the cingulate gyrus.
15 tter volume in the frontal pole and anterior cingulate gyrus.
16 us, posterior parietal cortex, and posterior cingulate gyrus.
17 o examine regional glucose metabolism in the cingulate gyrus.
18 s discussed with particular reference to the cingulate gyrus.
19 e ventral prefrontal cortex and the anterior cingulate gyrus.
20 s, cingulate gyrus, and striatum/subcallosal cingulate gyrus.
21 ed additional areas of hypometabolism in the cingulate gyrus.
22 ison group had greater increases in anterior cingulate gyrus.
23 the piriform cortex, Purkinje cells and the cingulate gyrus.
24 eft middle temporal gyrus, and the posterior cingulate gyrus.
25 l and functional alterations in the anterior cingulate gyrus.
26 n the right parietal cortex and the anterior cingulate gyrus.
27 umulation of cNFT pathology in the posterior cingulate gyrus.
28 and both NAA/Cr and NAA/mI in the posterior cingulate gyrus.
29 sponses in the hippocampus and the posterior cingulate gyrus.
30 esonance spectroscopy in the dorsal anterior cingulate gyrus.
31 ter of the basal ganglia and right posterior cingulate gyrus.
32 ative decrease in activation in the anterior cingulate gyrus.
33 11, 5-HTR1b and 5-HTR4 in the hippocampus or cingulate gyrus.
34 sorder-specific dysfunction in the posterior cingulate gyrus.
35 s), primarily in the precuneus and posterior cingulate gyrus.
36 teral prefrontal cortices, and the posterior cingulate gyrus.
37 or occipital cortex, fusiform areas, and the cingulate gyrus.
38 ingulate sulcus, connecting the SMA with the cingulate gyrus; (3) frontal 'aslant' fascicle, directly
39 activation foci bilaterally in the posterior cingulate gyrus (+/-8, -48, 26; left P < 0.001; right P
40 5), and in the contralateral dorsal anterior cingulate gyrus (ACC; r = 0.43, P < 0.05), dorsolateral
43 campus, orbitofrontal cortex (OFC), anterior cingulate gyrus (aCING), and peripeduncular nucleus of t
44 zophrenia showed significantly less anterior cingulate gyrus activation while naming the color of col
45 s associated with activation of the anterior cingulate gyrus, amygdala, orbitofrontal cortex, and dor
46 m in limbic regions (eg, perigenual anterior cingulate gyrus and amygdala) and ratings of state and t
47 gender-related structural differences in the cingulate gyrus and corresponding differences in cingula
48 ly lower glucose metabolism in the posterior cingulate gyrus and cuneus, whereas the nonhoarding OCD
49 portant revisions to the organization of the cingulate gyrus and demonstrate four structure/function
50 corpus callosum, and anterior and posterior cingulate gyrus and from automatically defined frontal,
51 n deficits were noted in the dorsal anterior cingulate gyrus and hippocampus of the depressed geriatr
52 Broca's and prefrontal areas combined, left cingulate gyrus and left superior temporal gyrus and a f
55 PTSD had increased rCBF in ventral anterior cingulate gyrus and right amygdala when generating menta
56 e occipital/ middle temporal gyri, bilateral cingulate gyrus and right sensorimotor and pre-motor reg
57 ray and white matter volumes of the anterior cingulate gyrus and superior frontal gyrus were computed
58 a K(i)(o) were observed in the left anterior cingulate gyrus and the dorsal midbrain region (P < 0.05
59 rs investigated the function of the anterior cingulate gyrus and the related neural systems that are
60 revealed that the network from the posterior cingulate gyrus and the semiology of PCE (motor manifest
61 most of the medial surface of the posterior cingulate gyrus and the ventral bank of the posterior ci
63 al cortex, supplementary motor area, anteior cingulate gyrus) and parietal (precuneus, posterior cing
65 mpus, insula, orbitofrontal cortex, anterior cingulate gyrus, and caudate in subjects with high CTQ s
66 e content in the prefrontal cortex, anterior cingulate gyrus, and hippocampus between healthy control
67 al, inferior frontal, middle frontal cortex, cingulate gyrus, and left middle frontal), with no signi
68 region (supramarginal gyri), right posterior cingulate gyrus, and left occipital lobe (lingual gyrus)
71 udate and putamen, globus pallidum, anterior cingulate gyrus, and medial temporal regions, including
72 frontal and temporal lobes, caudate nucleus, cingulate gyrus, and mediodorsal nucleus of the thalamus
74 dent functional connectivity with precuneus, cingulate gyrus, and striatum/subcallosal cingulate gyru
75 anterior and posterior cerebellum, posterior cingulate gyrus, and superior and inferior frontal gyrus
77 bilateral prefrontal cortices, the anterior cingulate gyrus, and the precuneus; and induced a more "
78 f the amygdala, the left insula and anterior cingulate gyrus, and the right subcallosal gyrus and nuc
79 tal gyrus, left superior temporal gyrus, and cingulate gyrus/anterior cingulate cortex (Brodmann area
80 Volumetric abnormalities in the anterior cingulate gyrus appear specific to the gray matter in OC
81 as 6, 7, 8, 9, 10, 11, 44, 45, 47), anterior cingulate gyrus (areas 24, 32), temporal cortex (area 21
83 activity in the orbitofrontal cortex and the cingulate gyrus associated with emotional responses when
84 derwent antemortem (1)H-MRS of the posterior cingulate gyrus at 3 tesla were included in this study.
85 ith elevated metabolic rates in the anterior cingulate gyrus at baseline are more likely to respond t
86 veral cortical areas, including the anterior cingulate gyrus (BA 24 and 25), the lateral basal fronta
87 ties of areas, including the frontal cortex, cingulate gyrus, basal ganglia, and temporal cortex.
88 edly greater activation of the left anterior cingulate gyrus, bilateral frontopolar regions, bilatera
89 ivations were observed in the right anterior cingulate gyrus (Brodmann area 24), in the intraparietal
90 tate was detected most frequently within the cingulate gyrus but it was also present in the subcortic
92 estimated for 7 structures: rostral anterior cingulate gyrus, caudal anterior cingulate gyrus, poster
93 30 identify them on the ventral bank of the cingulate gyrus (CGv), whereas standardized atlases show
94 reduced gray matter volumes in the anterior cingulate gyrus compared with individuals with BD (Montr
96 0.89 +/- 0.04, ADs 0.72 +/- 0.04; posterior cingulate gyrus: controls 1.05 +/- 0.09, ADs 0.79 +/- 0.
97 nal cortex, parahippocampal gyrus, posterior cingulate gyrus, cortex of the temporal lobes and corpus
98 frontal cortex and supplementary motor area, cingulate gyrus, cuneus and occipital gyrus, and insula
99 ) binding in several brain regions (anterior cingulate gyrus, dorsolateral prefrontal cortex, and par
100 o-parietal association cortex, precuneus and cingulate gyrus during and following seizures, similar t
101 schizophrenia fail to activate the anterior cingulate gyrus during selective attention performance.
102 insula and middle cingulate/dorsal anterior cingulate gyrus during the processing of fearful faces.
103 tivated microglia distributed throughout the cingulate gyrus, entorhinal cortex, hippocampus and dent
104 articipants included 14 consecutive cases of cingulate gyrus epilepsies confirmed by restricted magne
105 NG, AND PARTICIPANTS: We studied consecutive cingulate gyrus epilepsy cases identified retrospectivel
109 n group and age, were found in retrosplenial cingulate gyrus, found to be metabolically affected in p
110 he fragile X syndrome group was found in the cingulate gyrus, fusiform gyrus, and frontal cortex in r
111 ve (P = .04), and right posterior (P = .003) cingulate gyrus gray matter subregions compared with HCs
113 rains, the monkey frontal lobe and posterior cingulate gyrus had significantly less metabolic activit
115 ere and, to a lesser extent, in the anterior cingulate gyrus, head of the caudate nucleus and the pos
117 , caudal anterior cingulate gyrus, posterior cingulate gyrus, hippocampus, basal forebrain, amygdala,
119 brain have suggested the involvement of the cingulate gyrus in a wide variety of affective, cognitiv
121 metabolism of the amygdala, hippocampus, and cingulate gyrus in an expanded group of 17 patients with
123 e of the prefrontal cortex, hippocampus, and cingulate gyrus in major depressive disorders (MDD), but
125 oups shared underactivation in the posterior cingulate gyrus in relation to comparison subjects.
126 and superior frontal gyri, frontal pole, and cingulate gyrus in S-allele carriers compared with parti
127 ice variants in the hippocampus and anterior cingulate gyrus in suicide completers with and without a
128 ulty in localizing seizures arising from the cingulate gyrus in the absence of a magnetic resonance i
130 tion with tissue loss in the right subgenual cingulate gyrus in the VMPC, and aberrant motor behaviou
131 iddle and inferior frontal gyri and anterior cingulate gyrus, in addition to regions of the parietal
132 he right caudate nucleus and bilateral (para)cingulate gyrus increased in patients after real-rTMS wh
133 CBF increases in insular cortex and anterior cingulate gyrus; increases in anterior cingulate gyrus w
134 te gyrus) and parietal (precuneus, posterior cingulate gyrus, inferior parietal lobule (IPL), postcen
135 multiple foci in dorsal anterior and middle cingulate gyrus, insula/claustrum, amygdala/periamygdala
136 halamus, amygdala, caudate nucleus, anterior cingulate gyrus, insular cortex and orbitofrontal cortex
140 At 1-year follow-up, WM volume in the left cingulate gyrus isthmus correlated with clinical scores
141 terior cingulate WM bilaterally and the left cingulate gyrus isthmus WM, as well as the right precune
142 r, the anterior part of the cingulum and the cingulate gyrus isthmus, as well as the precuneal GM, ma
143 grey matter loss over time in the posterior cingulate gyrus, lateral and medial temporal lobe, and o
144 thalamus) and extrathalamic regions such as cingulate gyrus, lateral geniculate, temporal cortex, an
145 osteroinferior temporal lobe, left posterior cingulate gyrus, left frontal lobe expressive language r
146 re identified in the frontal lobes, anterior cingulate gyrus, left temporal lobe, and of white matter
147 results suggest that damage to the anterior cingulate gyrus may be the cause of changes in social in
149 rapy-treated on the right) and left anterior cingulate gyrus metabolism, and increases in normalized
150 ain tissues from cerebellum, midfrontal, and cingulate gyrus obtained at autopsy from 11 patients wit
151 umes of the superior frontal gyrus, anterior cingulate gyrus, orbital frontal region, hippocampus, an
153 patients had decreased rCBF in the posterior cingulate gyrus (P = .01) with extension to the medial p
154 nts had increased rCBF in the right anterior cingulate gyrus (P = .02) compared with that in control
155 entral precuneus (BA7), along with posterior cingulate gyrus (PCC, BA23, sad condition) and anteromed
156 vity for gains was reduced in CD in the left cingulate gyrus post-cocaine and in the left putamen in
157 al prefrontal cortex (DLPFC), medial frontal/cingulate gyrus, posterior cingulate cortex (PCC), and v
158 al anterior cingulate gyrus, caudal anterior cingulate gyrus, posterior cingulate gyrus, hippocampus,
159 d temporal cortices as well as the posterior cingulate gyrus, precuneus, and mesial temporal cortex.
160 in seven prefrontal subregions: the anterior cingulate, gyrus rectus, orbitofrontal cortex, precentra
161 sion in the right superior frontal gyrus and cingulate gyrus, regions associated with pain processing
162 gyri; superior parietal lobe; and posterior cingulate gyrus, resulted in a fitted accuracy of 94% an
163 ol (mI), and mI/Cr measured in the posterior cingulate gyrus reveal evidence of disease progression i
164 patients presented decreased activity in the cingulate gyrus, right PrCS and right PPC and increased
165 mentary motor cortex, anterior and posterior cingulate gyrus, right superior parietal lobe, right int
166 nificantly raised regional CBF (rCBF) in the cingulate gyrus, sensorimotor cortex, superior temporal
168 showed reduced volume of the right anterior cingulate gyrus, specifically in Brodmann's area 24'.
169 lated smaller gray matter volumes in various cingulate gyrus subregions in schizophrenia and bipolar
171 edial prefrontal cortex and rostral anterior cingulate gyrus (Talairach coordinates: x=0, y=62, z=10)
172 g network-medial prefrontal cortex, anterior cingulate gyrus, temporoparietal junction, and precuneus
174 ed greater activation in the dorsal anterior cingulate gyrus than did low-risk participants, who show
176 ule, the superior temporal gyrus, the middle cingulate gyrus, the putamen and the superior parietal l
178 including superior frontal gyrus and rostral cingulate gyrus; the ACD group had significant reduction
179 physiological connections from the posterior cingulate gyrus to parietal, temporal, mesial occipital
180 al evaluation was performed on the posterior cingulate gyrus using antibodies to synaptic vesicles, h
182 ated in patients; in contrast, the posterior cingulate gyrus was activated in patients and deactivate
183 al areas combined and fewer NODs in the left cingulate gyrus was associated with better phonological
185 ) voxel matrix) acquired axially through the cingulate gyrus was used to quantify regional brain chem
186 this follow-up study of postmortem anterior cingulate gyrus, we have found evidence of increased TDO
187 erior cingulate gyrus; increases in anterior cingulate gyrus were greater in the comparison group tha
189 ight inferior parietal cortex, and posterior cingulate gyrus when compared with control subjects.
190 in the right cerebellar cortex and the left cingulate gyrus when executing the prelearned sequence,
191 The orbitofrontal cortex and the anterior cingulate gyrus, which are regions neuroanatomically con
193 tal cortex) and inhibitory control (anterior cingulate gyrus), whose disruption results in compulsivi
194 patterns were also observed in the anterior cingulate gyrus with the proximal esophagus being repres
196 metabolic products were observed in anterior cingulate gyrus without proton decoupling in one subject
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