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1 depression diagnosis maximally affecting the cingulum.
2 and an ordering transition at the chimpanzee cingulum.
3 r longitudinal fasciculi and in the anterior cingulum.
4 arning deficits had hypoplasia or atresia of cingulum.
5 gulum; three patients had aberrant fibers of cingulum.
6 ronal structure and function in the anterior cingulum.
7 ith seizure burden in the bilateral PWMB and cingulum.
8 impaired axonal integrity of the hippocampal cingulum.
9 perior and posterior corona radiata, and the cingulum.
10 nferior fronto-occipital fasciculi and right cingulum.
11 fornix bilaterally and right dorsal-anterior cingulum.
12 SA in the orbitofrontal cortex and posterior cingulum.
13 uced mean and axial diffusivity in the right cingulum.
14 bundles, particularly in the left subgenual cingulum.
15 ations were not found in the parahippocampal cingulum, a comparison temporal association pathway.
16 and microstructure of connections within the cingulum, a major white matter tract and conduit of proj
17 reduced microarchitectural integrity of the cingulum, a white matter fiber that connects the entorhi
18 sults demonstrate that temporary blockade of cingulum activity at the time of nerve section reduces a
19 microglial activation in corpus callosum and cingulum along with severe astrogliosis and scar formati
21 es in HS-TLE correlated with parahippocampal cingulum and anterior commissure atrophy, indicating an
22 calculating the DTI indices of the bilateral cingulum and cingulum hippocampal part for each subject.
23 y groups had bilateral atrophy of the dorsal cingulum and corpus callosum fibers, which we interpret
25 se error-corrected), mainly in the uncinate, cingulum and corpus callosum, whereas responders were in
28 Higher CRF was associated with higher FA in cingulum and lower MD in hippocampus and entorhinal cort
29 rostrum of the corpus callosum to enter the cingulum and merged with fibres of the lateral pathway w
31 wed decreased activation particularly in the cingulum and thalamus during both the encoding and recal
33 mentioned regions and, in addition, with the cingulum and the left hippocampus (cluster level, P < .0
34 hanges were regionally specific to the right cingulum and the right superior and inferior longitudina
35 and long segments of the arcuate fasciculus, cingulum and uncinate--predominantly in the left hemisph
36 er changes, which were most prominent in the cingulum and which correlated with disease severity.
37 st-injury, NFC was observed primarily in the cingulum, and appeared as swollen axons and terminal bul
40 the callosal genu, thalamus, right posterior cingulum, and fornix crus (seven studies; largest cluste
41 oups in subdivisions of the corpus callosum, cingulum, and fornix were measured as indicators of trai
43 m in the striatum, as well as in the cuneus, cingulum, and parietal lobe, in all SCA17 patients and p
44 splenium of the corpus callosum, the fornix, cingulum, and superior and inferior longitudinal fascicu
45 normalities observed in the corpus callosum, cingulum, and temporal lobe likely constitute the neural
46 he splenium of the corpus callosum, the left cingulum, and the anterior part of the left arcuate fasc
48 nferior longitudinal, superior longitudinal, cingulum, and uncinate) tracts were quantified using tra
49 o FA values in the corpus callosum and right cingulum; and eye-hand coordination sub-scores to FA val
50 synchronization in the thalamus; cerebellum; cingulum; and insular, prefrontal, and parieto-occipital
51 the cerebellum; basal ganglia; hippocampus; cingulum; and temporo-occipital, insular, frontal, and p
52 ing, because characteristics of the anterior cingulum are shown to be related to impulse, attention,
54 CD200R(+) /Iba1(+) ameboid microglia in the cingulum at P1-P5 were the only CD200R(+) cells in the n
55 n contrast, developmental differences in the cingulum at the level of the parahippocampal region were
56 that a temporary anesthetic blockade of the cingulum at the time of nerve section delayed the onset
57 s Test score by radial diffusivity along the cingulum (beta = -4.3 x 10(4); P < .01), and T2* in the
58 tracts, particularly in the corpus callosum, cingulum, bilateral superior and inferior longitudinal f
62 greater fractional anisotropy in the dorsal cingulum bundle and better performance on measures of re
63 e tracts (crossing between anterior parts of cingulum bundle and body of corpus callosum), which show
64 ildhood to early adulthood, higher FA of the cingulum bundle and inferior frontooccipital fasciculus
68 of a local anesthetic (bupivacaine) into the cingulum bundle at the time of nerve section could reduc
69 esults indicate that higher FA of the dorsal cingulum bundle may be an acquired feature of persistent
72 right superior longitudinal fasciculi, right cingulum bundle projecting to the hippocampus, left infe
73 a specific fiber tract, the corpus callosum/cingulum bundle that conveys reciprocal connections betw
75 sterior, and parahippocampal portions of the cingulum bundle were reconstructed separately using dete
76 g impairments and in the hippocampal part of cingulum bundle which accounted for long-term memory bin
77 culus) and to the cingulate cortex (via left cingulum bundle), whereas behaviorally salient but nonbe
78 d microstructural integrity of the posterior cingulum bundle, as measured by diffusion tensor imaging
79 rations in white matter (in corpus callosum, cingulum bundle, corona radiata, and superior fronto-occ
80 pared to matched controls in the left dorsal cingulum bundle, splenium of the corpus callosum, right
88 he middle cerebellar peduncles (P<0.001), in cingulum bundles (P=0.002), and in the right orbitofront
89 ted positively with FA in the right and left cingulum bundles (r = .342, p = .052; r = .477, p = .005
91 had 3% to 5% lower FA in the right and left cingulum bundles than FH- individuals (p = .012, p = .05
92 associated with reduced FA in the bilateral cingulum bundles, particularly in the left subgenual cin
95 as associated with decreased FA in the genu, cingulum cingulate gyri, centrum semiovale, inferior lon
97 sor imaging abnormalities of the cerebellum, cingulum, corpus callosum, internal capsule, thalamus, b
98 le, the NTRK1-T effect was replicated in the cingulum, corpus callosum, superior and inferior longitu
99 eta = -4.3 x 10(4); P < .01), and T2* in the cingulum cortical projection at 25% depth (beta = -1.7;
100 ities in parahippocampal gyrus and posterior cingulum, extending laterally into adjacent temporo-pari
102 erior corona radiata, cortico-spinal tracts, cingulum fibre bundles, external capsule, forceps minor
104 actions for FA were found in the hippocampal cingulum, fornix, and stria terminalis, posterior corona
105 coordination sub-scores to FA values in the cingulum, fornix, anterior commissure, corpus callosum a
106 have differential effects on the hippocampal cingulum, fornix, stria terminalis, posterior corona rad
110 uperior longitudinal fasciculi and the right cingulum-hippocampal projection compared with their coun
111 d significantly lower FA values in the right cingulum-hippocampal projection than their counterparts
112 ded cerebral cortex, lateral septal nucleus, cingulum, hippocampus, thalamus, amygdala, and vestibula
113 which allows us to optimize the location of cingulum in a global sense based on the diffusion measur
117 ociated with lower FA in the right posterior cingulum, left callosal splenium, right inferior fronto-
121 lesions in limbic system structures (eg, the cingulum) on the right than did subjects who did not mee
122 cts, such as the callosal genu and splenium, cingulum, optic radiations, and the superior longitudina
123 , and was correlated with FIQ and PIQ in the cingulum, optic radiations, superior fronto-occipital fa
125 TSD patients had increasing FA of the dorsal cingulum over time, and at reassessment these FA values
126 ns in the microarchitecture of the posterior cingulum (PC), a white matter tract proximal to the hipp
127 odifications of the activity of the anterior cingulum, PCC and/or precuneus, left dorsolateral PFC, a
129 ntervention group had an increased FC in the cingulum, precuneus, and bilateral parietal cortex and a
130 with alterations in diffusivity in the left cingulum (r = -0.66, P = .01) and superior longitudinal
132 fornix crus [AD, beta = 0.02 (P = .046)] and cingulum [RD, beta = -0.01 (P = .02); MD, beta = -0.01 (
134 osed method provides an approach to localize cingulum robustly, which is a very important feature for
135 tensors' information throughout the tract of cingulum simultaneously, which is quite different from t
136 he greatest change within right paracallosal cingulum (sporadic bvFTD, FA: -6.7%/yr, p < 0.001; MD: 3
137 nate fasciculus, superior cingulum, temporal cingulum, superior longitudinal fasciculus, arcuate fasc
138 lowing tracts: uncinate fasciculus, superior cingulum, temporal cingulum, superior longitudinal fasci
139 rs was examined by applying DiI to the right cingulum; the labeled fibers ran throughout the CC and r
140 patients had defects within fornices and/or cingulum; three patients had aberrant fibers of cingulum
141 tural connectivity produced by damage to the cingulum tract explained the compensatory increases in f
142 related to the level of injury to the dorsal cingulum tract, which connects medial frontal and pariet
143 0.002) cingulum tracts, bilateral descending cingulum tracts (P < 0.001) and left uncinate tracts (P
144 descending (left P < 0.001; right P = 0.003) cingulum tracts, and uncinate tracts (left P < 0.001; ri
145 terior (P = 0.003) and posterior (P = 0.002) cingulum tracts, bilateral descending cingulum tracts (P
146 fic fibre tracts (including corpus callosum, cingulum, uncinate and corticospinal tracts) as well as
147 Fractional anisotropy (FA) was computed for cingulum, uncinate and superior longitudinal fasciculi.
148 racts in emotional regulation circuitry (ie, cingulum, uncinate fasciculus, and forceps minor) and (1
149 oduce an approach to extract the skeleton of cingulum using active contour model, which allows us to
151 crostructural variation in the left anterior cingulum was closely related to interindividual control
152 aled that fractional anisotropy of the right cingulum was inversely correlated with AD PRSs (p = .009
153 usivity in the parahippocampal region of the cingulum was negatively associated with verbal memory de
154 ty in the left parahippocampal region of the cingulum was negatively associated with visuospatial mem
155 d by fractional anisotropy) of the posterior cingulum was observed in participants with PTSD (p<0.05)
157 ffusivity (MD) within the right paracallosal cingulum were greatest (FA: -6.8%/yr, p < 0.001; MD: 2.9
158 temporal pole, amygdala, insula, and dorsal cingulum, were aberrant in FXS and iAUT as compared with
159 white matter integrity in the left posterior cingulum, which may contribute to cognitive impairment d
161 uggest that abnormal uncinate fasciculus and cingulum WM structure may underlie emotional, but not be
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