ライフサイエンスコーパス: cinnamaldehyde

1 ced by formalin, acetic acid, capsaicin, and cinnamaldehyde.

2 mmatory capacity was primarily attributed to cinnamaldehyde.

3 aldehydes, a hindered aromatic aldehyde, and cinnamaldehyde.

4 ctrophilic compounds such as mustard oil and cinnamaldehyde.

5 ingredients such as allyl isothiocyanate and cinnamaldehyde.

6 in a chemospecific anti-aldol reaction with cinnamaldehyde.

7 duct of decarboxylation from such compounds, cinnamaldehydes.

8 for by elevated levels of benzaldehydes and cinnamaldehydes.

9 Four treatments (control, 0.1% cinnamaldehyde, 0.75% calcium chloride and combination o

10 ve 4-chloro-N-methyl-N-nitrosoaniline (76%), cinnamaldehyde (55%), 3-phenyl-5-hydroxyisoxazoline (26%

11 ensitive, lung-specific neurons responded to cinnamaldehyde, a TRPA1 agonist, with increases in intra

12 of 1,2,4-trimethoxybenzene, indole, and (E)-cinnamaldehyde, all blossom components, is highly attrac

13 products [e.g., allyl isothiocyanate (ITC), cinnamaldehyde, allicin, and gingerol].

14 TRPA1 activators, including cinnamaldehyde, allyl isothiocyanate (AITC), and 4-hydro

15 The cinnamaldehyde alone and a combination of cinnamaldehyde

16 Cinnamaldehyde also induced TRPA1-like inward currents (

17 included octanal, octanoic acid, octanol and cinnamaldehyde among others (all at 30microM).

18 75% calcium chloride and combination of 0.1% cinnamaldehyde and 0.75% calcium chloride) were used to

19 lcohol dehydrogenase subfamily that includes cinnamaldehyde and benzaldehyde dehydrogenases.

20 he cinnamaldehyde alone and a combination of cinnamaldehyde and calcium chloride treatments yielded b

21 and evaluated in the DA reaction between (E)-cinnamaldehyde and cyclopentadiene.

22 3-phenylpropionate, methyl 2-phenylacetate, cinnamaldehyde and methyl cinnamate were produced during

23 tal irritants and pungent chemicals, such as cinnamaldehyde and mustard oil.

24 king interactions between the phenyl ring of cinnamaldehyde and phenylated SAMs allowed tuning of rea

25 ontrolling the liquid phase hydrogenation of cinnamaldehyde and related benzylic aldehydes over Pt na

26 The TRPA1 agonists allyl isothiocyanate and cinnamaldehyde and the TRPV4 agonist GSK1016790A caused

27 tion of the alpha,beta-unsaturated aldehyde (cinnamaldehyde) and the catalyst.

28 h three reference substrates ketoisophorone, cinnamaldehyde, and (S)-carvone.

29 rent structures, including carvone, eugenol, cinnamaldehyde, and acetophenone.

30 emicals such as allyl isothiocyanate (AITC), cinnamaldehyde, and allicin, produce nociceptive sensati

31 ing nonenolizable aliphatic aldehydes, trans-cinnamaldehyde, and beta-substituted styrenes were also

32 ymethylfurfural, anisaldehyde, benzaldehyde, cinnamaldehyde, and phenylaldehyde are commonly generate

33 The cinnamaldehyde- and crotonaldehyde-derived phosphonates

34 (hydroxycinnamoyl-SCoAs) to cinnamaldehydes; cinnamaldehydes are then reduced to cinnamyl alcohols by

35 lic plant compounds, such as mustard oil and cinnamaldehyde, are TRPA1 agonists, it is unknown whethe

36 trophilic compounds allyl isothiocyanate and cinnamaldehyde as well as heat.

37 ilic reactivity of iminium ions derived from cinnamaldehyde by a factor of 14.

38 rations of proanthocyanidins (PAC) and trans-cinnamaldehyde (CA).

39 th most other common oral irritants, such as cinnamaldehyde, capsaicin, and alcohol, which irritate m

40 d cinnamic acids (hydroxycinnamoyl-SCoAs) to cinnamaldehydes; cinnamaldehydes are then reduced to cin

41 ed that 2,3-butanediol, hexanal, hexanol and cinnamaldehyde contributed the most to classification of

42 However, no additional cinnamaldehyde coupling products could be detected in th

43 tives of two such compounds, mustard oil and cinnamaldehyde, covalently bind mouse TRPA1.

44 Wadsworth-Emmons reaction, were treated with cinnamaldehyde derivatives, acetic acid, and borohydride

45 ivo vagal innervated mouse lung preparation, cinnamaldehyde evoked action potential discharge in mous

46 We report here that peptidyl cinnamaldehydes function as reversible, slow-binding inh

47 Among the eight tested compounds, cinnamaldehyde had the greatest anti-neuroinflammatory c

48 ly are salicylic acid, tannic acid and trans-cinnamaldehyde have been identified.

49 anistic insight from kinetic mapping reveals cinnamaldehyde hydrogenation is structure-insensitive ov

50 to selectively orient the reactant molecule cinnamaldehyde in a configuration associated with hydrog

51 ns are capable of forming E,E,E-trienes from cinnamaldehydes in good yield.

52 in, sinapic acid, cinnamic acid, eugenol and cinnamaldehyde) in multilevel ratios.

53 Cinnamaldehyde inhalation in vivo mimicked capsaicin in

54 nnamaldehyde, with ozone having no effect on cinnamaldehyde-insensitive fibres.

55 ic analysis of an isolated Au(iii)-activated cinnamaldehyde intermediate.

56 rocess evolves through a [3 + 2] 1,3-DC when cinnamaldehyde is used in the presence of an azomethine

57 Saturating activation by cinnamaldehyde or mustard oil occluded potentiation but

58 They add to cinnamaldehyde or paraformaldehyde, for example, to prod

59 Treatment with allyl isothiocyanate, cinnamaldehyde, or GSK1016790A caused an increase in ATP

60 Products from radical coupling of cinnamaldehydes, particularly sinapaldehyde, which were

61 uinone and the electrophilic TRPA1 activator cinnamaldehyde produced antinociception that was lost in

62 ty in enhancing C = O hydrogenation (through cinnamaldehyde reorientation), a general phenomenon exte

63 ipeptide Gly-Glu-Glu to the para position of cinnamaldehyde resulted in an inhibitor (Cinn-GEE) of su

64 urons that express TRPA1, a mustard oil- and cinnamaldehyde-sensitive channel, and that these respons

65 al Diabrotica to structural analogues of (E)-cinnamaldehyde, the major attractant for Diabrotica unde

66 Cinnamaldehyde, the most specific TRPA1 activator, excit

67 yed to install a pyridyl to the alkene trans-cinnamaldehyde while Ag(I) ions are used in a second ste

68 The reaction of cinnamaldehyde with iodonium ylide 1a catalyzed by (5S)-

69 eatment of the tosylhydrazone sodium salt of cinnamaldehyde with transition metal catalysts.

70 ansient receptor potential (TRP) A1 agonist, cinnamaldehyde, with ozone having no effect on cinnamald