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1 ximal contrast-to-noise ratios (CNRs) in the circle of Willis.
2 light (TOF) acquisition was performed in the circle of Willis.
3 teral proximal middle cerebral artery of the circle of Willis.
4 er characterized by occlusive lesions of the circle of Willis.
5 aving or not having occlusive disease at the circle of Willis.
6 icating artery (PcomA) hypoplasticity in the circle of Willis.
7 e measure was the frequency of an incomplete circle of Willis.
8 sonance angiography was performed to examine circle of Willis anatomy and arterial spin labeled perfu
9 ble, segmental, cerebral vasoconstriction of circle of Willis arteries and normal or near-normal resu
11 ith Alzheimer's disease had grossly apparent circle of Willis atherosclerosis, a percentage that was
12 hology and lacunar infarcts, large infarcts, Circle of Willis atherosclerosis, or cerebral amyloid an
13 n revealed dolichoectasia of arteries of the circle of Willis coexisting with a fusiform aneurysm of
14 ng that all ChAT-I fibers in the MCA and the circle of Willis contain NOS, and that these fibers orig
16 l cerebral collateral circulation within the circle of Willis (CW) predisposes to ischemia and stroke
18 d severity of atherosclerotic plaques in the circle of Willis in Alzheimer's disease and multiple oth
22 hat the middle cerebral artery (MCA) and the circle of Willis received dense ChAT-immunoreactive (I)
24 he brainstem and cerebellum but can, via the Circle of Willis upon stenosis of the internal carotid a
25 Using a quantitative score of the burden of circle of Willis variants, migraine with aura subjects h
33 all 102 participants, atherosclerosis of the circle of Willis was strongly associated with lacunar an
34 tery hypoplasia, and an incomplete posterior circle of Willis, which were coupled with increased cere
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