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1 angiogenesis associated with mobilization of circulating endothelial progenitor cells.
2 2 mRNA transcripts in ischemic tissue and in circulating endothelial progenitor cells.
4 d a strong correlation between the number of circulating endothelial progenitor cells and the subject
5 in and plasticity of cardiac fibroblasts and circulating endothelial progenitor cells, and consider w
6 al inhibition of ADAM17, modulates postnatal circulating endothelial progenitor cell (CEPC) numbers v
10 s of endothelial cells, decreased numbers of circulating endothelial progenitor cells, decreased capi
11 electin on endothelial cells, thus tethering circulating endothelial progenitor cells (EPC) and facil
12 Estradiol (E2) modulates the kinetics of circulating endothelial progenitor cells (EPCs) and favo
15 e microfluidic platform capable of capturing circulating endothelial progenitor cells (EPCs) by under
17 ently, evidence has accumulated to show that circulating endothelial progenitor cells (EPCs) contribu
18 h coronary atherosclerosis have increases in circulating endothelial progenitor cells (EPCs) expressi
19 There is considerable interest in exploiting circulating endothelial progenitor cells (EPCs) for ther
20 mice, hyperoxia enhances the mobilization of circulating endothelial progenitor cells (EPCs) from the
25 logy has been stimulated by the concept that circulating endothelial progenitor cells (EPCs) may play
27 hematopoietic stem cell (HSC) production of circulating endothelial progenitor cells (EPCs) to revas
29 peculated that PAH patients are deficient in circulating endothelial progenitor cells (EPCs), potenti
30 Diabetes is associated with a deficit of circulating endothelial progenitor cells (EPCs), which h
32 is but also involves vasculogenesis and that circulating endothelial progenitor cells from bone marro
36 ith rapid tumor progression have 2-fold more circulating endothelial progenitor cells in peripheral b
37 ntration of sFlt1 with concomitant decreased circulating endothelial progenitor cell levels along wit
39 CatK deficiency is associated with decreased circulating endothelial progenitor cells-like CD31(+)/c-
40 hanced the proliferative capacity of ECs and circulating endothelial progenitor cell numbers after va
43 We hypothesize that increased numbers of circulating endothelial progenitor cells will be associa
44 e of this study was to determine the role of circulating endothelial progenitor cells with osteoblast
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