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1 ogressive, often dilated, cardiomyopathy and circulatory failure.
2 in postnatal death, most probably because of circulatory failure.
3 intubated patients with sepsis-related acute circulatory failure.
4 ns suggest that TF(-/-) embryos are dying of circulatory failure.
5 er dysfunction with vascular leakage-induced circulatory failure.
6 f Ramsay sedation scale >4]) and, in case of circulatory failure, a second set of measurements after
7 n = 90) presenting with sepsis-induced acute circulatory failure and considered for volume expansion.
8 tion of iNOS activity with L-NIL reduced the circulatory failure and liver injury, while selective in
9 -permeable radical scavenger (tempol) on the circulatory failure and MODS (kidney, liver, lung) cause
10 le(4,3-alpha)quinoxaline-1-one (ODQ), on the circulatory failure and multiple organ dysfunction syndr
11 sms by which calpain inhibitor I reduces the circulatory failure as well as the organ injury and dysf
14 f endogenous nitric oxide contributes to the circulatory failure caused by endotoxin (lipopolysacchar
15 was significant liver damage resulting from circulatory failure during cardiopulmonary arrest before
17 might participate in the pathophysiology of circulatory failure during sepsis, and represent a poten
18 e heart failure and impending respiratory or circulatory failure especially in the presence of a diag
19 y-five mechanically ventilated patients with circulatory failure for whom the decision to give fluid
21 ight ventricular assistance (and right-sided circulatory failure), hemodynamic indexes, percent trans
22 sferase and aspartate aminotransferase), and circulatory failure (hypotension) as well as the increas
25 physiological change determines whether CSF circulatory failure manifests as Alzheimer's disease (AD
30 of the acute heart failure patient, such as circulatory failure, volume overload, renal insufficienc
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