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1 the LAD is better than that to the right or circumflex.
2 stenoses (left anterior descending, 9; left circumflex, 2; mean, 59 +/- 23% diameter stenosis) under
3 giography (left anterior descending, 4; left circumflex, 2; right coronary artery, 4; and right coron
6 the detection of > 50% stenosis in the left circumflex (74% and 96%; 50% and 100%; 63% and 91%, resp
7 e fed a high-cholesterol diet underwent left circumflex ameroid constrictor placement to induce chron
12 nterval, 1.00 to 1.64; P=0.046) for the left circumflex, and 1.32 (95% confidence interval, 1.03 to 1
13 ose in distal left anterior descending, left circumflex, and right coronary arteries (0.80+/-0.09 ver
14 and regional (left anterior descending, left circumflex, and right coronary arteries) MBF and flow re
17 detection of left anterior descending, left circumflex, and right coronary artery stenosis, sensitiv
18 matically for left anterior descending, left circumflex, and right coronary artery territories as an
19 t size in the left anterior descending, left circumflex, and right coronary artery territories, where
20 y territories-left anterior descending, left circumflex, and right-as well as left ventricular (LV) v
27 eft main stem, left anterior descending, and circumflex arteries of 20 subjects after a normal corona
29 loon occlusion of the anterior descending or circumflex arteries, each separated by 5 min of reperfus
30 fusion away from the anterior descending and circumflex arteries, suggesting a role for the coronary
31 anterior descending arteries and 86% of left circumflex arteries, whereas calcified nodules within th
34 onary artery (16+/-5 percent, P=0.01) or the circumflex artery (23+/-6 percent, P=0.06), although the
35 subjected to either chronic occlusion of the circumflex artery (group I, no perfusion defect) or acut
36 ior descending artery (LAD) (n = 27) or left circumflex artery (LCx) (n = 29) were evaluated with qua
38 tion of right coronary artery (RCA) and left circumflex artery (LCX) lesions (0.84 +/- 0.08 vs. 0.70
39 he 6 critical-stenosis dogs, the LAD-to-left circumflex artery (LCx) microsphere flow ratio was 0.22+
41 left anterior descending artery (LAD), left circumflex artery (LCX), and right coronary artery (RCA)
43 erior descending coronary artery [20%], left circumflex artery [12%] and right coronary artery [15%])
44 /- 1%), 123I-IPPA defect magnitude (LAD/left circumflex artery [LCX] count ratios) decreased from 0.6
45 All pigs underwent ameroid placement on the circumflex artery and 3 weeks later received surgical FG
46 (left anterior descending artery [LAD] left circumflex artery and posterior descending artery [PDA])
48 collateral channels were induced in the left circumflex artery bed of 12 chronically instrumented dog
49 nt microsphere-derived MBF ratio in LAD/left circumflex artery beds demonstrated close correlation wi
50 m elastance at end systole), cardiac output, circumflex artery blood flow, and myocardial mechanical
52 /- 4 mm Hg, whereas pressure in the occluded circumflex artery decreased from 61 +/- 4 to 55 +/- 4 mm
53 e past month or who required grafting of the circumflex artery distal to the first obtuse marginal br
57 ion (VF) was evaluated using a 2-minute left circumflex artery occlusion during the last minute of an
58 Urinary 8-epi PGF2 alpha was unchanged after circumflex artery occlusion in a canine model of coronar
59 ervention, and early reperfusion therapy for circumflex artery occlusion should be considered when no
60 CFI differences during either IMA with left circumflex artery occlusion were inconsistently positive
65 nth after the surgery, occlusion of the left circumflex artery regularly produced ventricular fibrill
69 ardial flow reserve were reduced in the left circumflex artery territory (both P<0.001), and hibernat
70 ment of an ameroid constrictor into the left circumflex artery to induce chronic myocardial ischemia.
72 In 10 dogs, low coronary blood flow in the circumflex artery was delivered with a roller pump throu
74 selected for coronary angiography, the left circumflex artery was the culprit vessel in 34.6% of non
75 artery, 10 right coronary artery, and 5 left circumflex artery) retrieved by using directional corona
77 1-hour reperfusion of a major branch of the circumflex artery, 201Tl and either tetrofosmin or sesta
78 a 1-hr reperfusion of a major branch of the circumflex artery, 201Tl and Q12 were injected intraveno
79 descending artery, 90% and 70% for the left circumflex artery, and 74% and 79% for the right coronar
80 chloride staining demonstrated that the left circumflex artery, and not the LAD, group had atrial inf
96 was accompanied by a decrease in flow in the circumflex bed (from 1.31 to +/- 0.14 to 1.09 +/- 0.15 m
97 D), then in the right coronary artery (RCA), circumflex branch (LCx) and the left main coronary arter
100 r descending branch, 115.9 mm +/- 19.7; left circumflex branch, 97.2 mm +/- 12.5; and right coronary
102 left anterior descending coronary (LAD) and circumflex (CFX) arteries (CBF(LAD+CFX)) and coronary si
104 of normal left anterior descending and left circumflex coronary arteries dose dependently reduced th
105 orkshire pigs with chronically occluded left circumflex coronary arteries were randomly assigned to r
106 id, and distal left anterior descending, and circumflex coronary arteries were targeted with a single
107 y (PTCA) of the left anterior descending and circumflex coronary arteries with standard clinical angi
108 h injury to the left anterior descending and circumflex coronary arteries with standard percutaneous
110 dial artery and study SVG were the right and circumflex coronary arteries, which had >70% proximal st
113 descending coronary artery, 96% for the left circumflex coronary artery (22/23), and 100% for the rig
119 ssive ameroid occlusion of the proximal left circumflex coronary artery (LCx); after 2 months, animal
120 urrent applied to the intimal surface of the circumflex coronary artery 30 minutes after oral CVS-112
121 .01; P < 0.01) defect count ratios (LAD/left circumflex coronary artery [LCx]) differentiated between
122 ry [LAD]: n = 13, r = 0.89, p < 0.0001; left circumflex coronary artery [LCx]: n = 11, r = 0.7, p < 0
124 ng coronary artery, 44% and 90% for the left circumflex coronary artery and 75% and 77% for the right
125 ted to ameroid-induced occlusion of the left circumflex coronary artery and randomized to bFGF (1.74
126 ted to ameroid-induced occlusion of the left circumflex coronary artery and randomized to bFGF 1.74 m
127 Doppler flow probe implanted around the left circumflex coronary artery and with catheters in left ve
128 ong the left anterior descending and/or left circumflex coronary artery by intracoronary ultrasound.
129 provoked by transient occlusion of the left circumflex coronary artery during submaximal exercise.
131 eft anterior descending coronary artery/left circumflex coronary artery flow 0.53+/-0.16 in the contr
132 ry bypass grafts to a stenosed branch of the circumflex coronary artery have an excellent patency rat
133 bypass grafts anastomosed to a branch of the circumflex coronary artery have significantly better pat
136 t a rate of 2 ng/kg per minute into the left circumflex coronary artery in normal dogs (n = 5) and in
137 can be safely infused into the right or the circumflex coronary artery in the presence of a temporar
138 m the infarction zone eight weeks after left circumflex coronary artery ligation in pigs, demonstrati
143 n chest) before and continuously during left circumflex coronary artery occlusion to induce acute IMR
145 occurrence of TLR for restenosis of the left circumflex coronary artery ostium (LCX-ISR) (HR, 2.51; 9
146 dogs with left anterior descending and left circumflex coronary artery stenoses that reduced hyperem
147 ories: the left anterior descending and left circumflex coronary artery territories and the right cor
148 anterior descending coronary artery and left circumflex coronary artery territories, whereas AC + SC
151 dogs, a Doppler guidewire was placed in the circumflex coronary artery to measure coronary flow velo
152 with electrically induced thrombosis of the circumflex coronary artery treated with TPA revealed tha
153 d coronary sinus (CS) as well as CS and left circumflex coronary artery using cardiac computed tomogr
154 hypothesis, a 2-minute occlusion of the left circumflex coronary artery was made during the last minu
155 either the left anterior descending or left circumflex coronary artery was occluded for 90 minutes (
158 e segments of LAD and three segments of left circumflex coronary artery) increased by 19.3% (3.21 +/-
159 cava, a flow probe around the proximal left circumflex coronary artery, and catheters in the left at
160 either the left anterior descending or left circumflex coronary artery, and the S-VF DFT was determi
161 h an ameroid constrictor applied to the left circumflex coronary artery, in each pig, peak beta-galac
162 When ryanodine was infused directly into the circumflex coronary artery, it did not affect LV global
163 neous placement of a copper stent in the mid circumflex coronary artery, resulting in an intense infl
176 rnal diameter and wall thickness, and a left circumflex coronary blood flow velocity transducer.
179 the right coronary artery (20.4 +/- 3.0) and circumflex counts (22.2 +/- 4.1, P < .001 for either ver
185 lse-negative studies were single-vessel left circumflex disease, increased wall thickness, small cham
186 viewed our experience in revascularizing the circumflex distribution with off-pump techniques via lef
188 tein and that the different properties of E1(circumflex)E4 contribute to different processes in both
190 These data support the hypothesis that E1(circumflex)E4 is a multifunctional protein and that the
192 nt of organotypic raft cultures harboring E1(circumflex)E4 mutant HPV16 genomes there were alteration
193 To identify the role(s) of the viral E1(circumflex)E4 protein in the HPV life cycle, we characte
194 id in basal cells, in which we also found E1(circumflex)E4 protein to be expressed at low levels.
195 arious mutations in E4 indicated that the E1(circumflex)E4 protein-encoding requirements for these va
198 resting left anterior descending artery/left circumflex flow ratio compared with placebo (P<0.03) and
199 ry was occluded, whereas in group 2 (n = 9), circumflex flow was decreased by 30% before dobutamine (
200 osphere-derived LAD flow, normalized to left circumflex flow, correspondingly increased between day 0
203 nsional marker coordinates before and during circumflex ischemia, and tightening of the Paneth suture
207 nosine infusion via a surgically placed left circumflex (LCx) catheter (n=11) or via a right atrial c
208 sis of the left anterior descending (LAD) or circumflex (LCx) coronary arteries during adenosine vaso
209 odels of MI were generated: 1) proximal left circumflex (LCx) coronary artery occlusion involving the
211 ated left anterior descending (LAD) and left circumflex (LCx) coronary artery vasodilatation without
212 betes and left anterior descending (LAD) and circumflex (LCx) coronary calcium scores, independent fr
216 ronically instrumented dogs with either left circumflex (LCx) infusion of adenosine or partial LCx oc
218 ss was significantly greater within the left circumflex (LCX) ostium compared to the parent vessel (P
219 MCE acoustic intensity in the LAD and left circumflex (LCx) regions were fit to the following: y=A(
221 for the LAD risk area and the adjacent left circumflex (LCx) territory, and peak background-subtract
222 m ostium of CS to the intersection with left circumflex (LCX), and anatomical relation of LCX and CS
223 nding to the left anterior descending (LAD), circumflex (LCX), and right coronary (RCA) territories.
224 left anterior descending (LAD, n = 5), left circumflex (LCx, n = 5), and carotid (n = 5) arteries ef
225 eatment was ineffective in the HICHOL group (circumflex/left anterior descending blood flow ratios: 1
226 7% in saphenous vein graft lesions, 42.4% in circumflex lesions, 42.3% in left anterior descending le
227 tudied 10 sheep with ischemic MR produced by circumflex ligation with inferior infarction, 6 acutely
229 surgery or catheter-based intervention, the circumflex marginal vessels may be approached by thoraco
230 r dysfunction was then induced by repetitive circumflex microembolization until LV ejection fraction
232 oxide (C(15)O) after partially occluding the circumflex (n = 3) or the left anterior descending (n =
233 in the left anterior descending (n = 3), the circumflex (n = 3), or the right coronary artery (n = 2)
234 the left anterior descending (n = 11), left circumflex (n = 7) or right (n = 1) coronary artery.
235 scending system and the second to either the circumflex (n=2926) or right coronary artery (n=685) sys
240 versus right dominance among those with left circumflex or left main culprit lesions was 1.25 (95% CI
241 escending segments were reassigned to either circumflex or right coronary (12% and 11%, respectively)
242 atients, was less common with disease of the circumflex or right coronary arteries than with disease
243 r in patients who received the second ITA to circumflex or right coronary artery and remained similar
244 tery disease, it may be placed to either the circumflex or right coronary artery system with similar
245 allic coil by standard methods in the right, circumflex, or left anterior descending coronary artery.
247 left anterior descending (P=0.038) and left circumflex (P=0.009) regions persisted, which indicated
248 ary circulation (left anterior descending or circumflex) (p = 0.02, p < 0.0001), pulsatile flow (i.e.
250 n both the left anterior descending and left circumflex plus stent implantation in the right coronary
252 ative circumflex artery occluded, aortic and circumflex pressures and microsphere flows were measured
253 antly lower than groups 2 and 3, whereas the circumflex region cGMP in group 4 was significantly incr
254 gion, (18)F-FDG uptake was lower than in the circumflex region in group 1 (0.14 +/- 0.03 micro mol/mi
255 e region was significantly lower than in the circumflex region in group 1 but was similar to that in
256 /g; P < 0.05) but was similar to that in the circumflex region in group 2 (0.20 +/- 0.03 micro mol/mi
260 ed as lower Ecc in the RCA (P<0.01) and left circumflex regions (P<0.05) measured in the subendocardi
262 itory (39% of reassigned segments), standard circumflex segments were reassigned to the left anterior
263 r descending artery are located opposite the circumflex takeoff, spare the flow divider and maintain
265 oronary segments were reassigned to the left circumflex territory (39% of reassigned segments), stand
266 artery was randomized to either the right or circumflex territory and the study SVG was used for the
267 en the 2 techniques were most notable in the circumflex territory, where fixed defects were observed
268 group showed endothelial dysfunction in the circumflex territory, which was normalized by L-arginine
270 P<0.0001) and was more accurate for the left circumflex than for the left anterior descending coronar
273 ts underwent off-pump bypass grafting of the circumflex vessels via thoracotomy from December 1995 to
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