ライフサイエンスコーパス: cisplatinum

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1 ell growth and increase cytotoxic actions of cisplatinum.

2 drug resistance, as shown for MMC, araC and cisplatinum.

3 s treated with cyclophosphamide or VP-16 and cisplatinum.

4 A cross-linking agents, nitrogen mustard and cisplatinum.

5 ge, including UV, gamma-IR, camptothecin, or cisplatinum.

6 damage caused by the chemotherapeutic agent, cisplatinum.

7 verse classes of DNA-damaging agents such as cisplatinum, 1-beta-D-arabinofuranosylcytosine, UV light

8 osphamide (CY, 84 mg/kg), VP-16 (24 mg/kg) + cisplatinum (2.4 mg/kg), carboplatinum (50 mg/kg), chlor

9 00 mg/d IV over 15 minutes daily for 5 days; cisplatinum 25 mg/m2/d IV CI over 24 hours, days 1 to 4;

10 mg/kg) intravenously on day 1, and cycle B = cisplatinum (4 mg/kg) intravenously on day 1 and etoposi

11 Chemoradiation consisted of: cisplatinum, 5-fluorouracil, and radiation to a median d

12 activate p38 MAP kinase after treatment with cisplatinum and 1-beta-D-arabinofuranosylcytosine but no

13 Phase III comparison with cisplatinum and fluorouracil chemotherapy is warranted.

14 a1(-/-) mutants are 5-fold more sensitive to cisplatinum compared with wild-type cells.

15 ctional roles of p53 in cellular response to cisplatinum (CP).

16 Introduction of UV-damaged or cisplatinum-damaged cytomegalovirus-driven beta-galactos

17 Similar results were obtained with cisplatinum, etoposide, and camptothecin.

18 Ara-C = cytosine arabinoside, and Platinum = cisplatinum), in a group of patients with Hodgkin's dise

19 dogenous MDM2 splicing in response to UV and cisplatinum-induced DNA damage.

20 Furthermore, the inhibitory effect of cisplatinum on cell proliferation was enhanced markedly

21 Concurrent cisplatinum-or carboplatin-based chemotherapy and 60 to

22 The combination of macitentan and cisplatinum resulted in the potentiation of the cytotoxi

23 d prolonged in vitro survival in response to cisplatinum treatment and showed decreased chemotherapy-

24 sensitizes these cells to UV-B radiation and cisplatinum treatment-induced apoptosis.

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