ライフサイエンスコーパス: classical conditioning

1 ition rate in rabbit (Orcytolagus cuniculus) classical conditioning.

2 were also defective in learning the eyeblink classical conditioning.

3 r-level cognitive processes in some forms of classical conditioning.

4 light termination, and thus plays a role in classical conditioning.

5 l structures involved in, and necessary for, classical conditioning.

6 he foregut, however, were found to attenuate classical conditioning.

7 bes two neurophysiological correlates of the classical conditioning.

8 well described by a neural network model of classical conditioning.

9 g of conditioned responses (CRs) in eyeblink classical conditioning.

10 h the ryanodine receptor, may be involved in classical conditioning.

11 smaller in cells from animals that received classical conditioning.

12 xcitability were still present 1 month after classical conditioning.

13 s the CS-preceding-US pairing requirement of classical conditioning.

14 nase A during paired stimulation to initiate classical conditioning.

15 d the unconditioned stimulus is essential in classical conditioning.

16 ventral tegmental area while mice engaged in classical conditioning.

17 ability in B photoreceptors after multitrial classical conditioning.

18 B to medial type A synaptic connection after classical conditioning.

19 ontribute to long-term plasticity underlying classical conditioning.

20 incrementally learned associations, such as classical conditioning.

21 Hermissenda express differential effects of classical conditioning.

22 n training also produced RF plasticity, like classical conditioning.

23 tage model of AMPAR synaptic delivery during classical conditioning.

24 g AMPARs using an in vitro model of eyeblink classical conditioning.

25 ed and demonstrate that this is regulated by classical conditioning.

26 ed cortical plasticity and reduced eye blink classical conditioning.

27 ed paradigm, where infants undergo olfactory classical conditioning.

28 c nerve terminals using an in vitro model of classical conditioning.

29 in presynaptic structural plasticity during classical conditioning.

30 mimicked the CS in the single-cell analog of classical conditioning.

31 of associative learning in instrumental and classical conditioning.

32 bsence of the conditioned stimulus following classical conditioning.

33 y comparison subjects underwent differential classical conditioning.

34 cript in naive brain that is suppressed upon classical conditioning.

35 ing of ICSS thresholds, effects resistant to classical conditioning.

36 nt areas of the insect brain after olfactory classical conditioning.

37 edictive representations are learned through classical conditioning.

38 ditioned stimulus (US) pathway of odor-shock classical conditioning.

39 tributing to habituation, sensitization, and classical conditioning, also undergoes operant condition

40 asticity of human auditory cortex induced by classical conditioning and contingency reversal.

41 o underwent 2 associative forms of learning: classical conditioning and differential conditioning.

42 Beta-adrenergic involvement in classical conditioning and extinction was investigated.

43 beta-adrenergic involvement in classical conditioning and extinction was investigated.

44 oteinase is regulated in the early stages of classical conditioning and functions in the conversion o

45 compare cerebellum-dependent delay eyeblink classical conditioning and hippocampus-dependent context

46 f perceptual and motor operations, including classical conditioning and implicit learning.

47 ative memory for an auditory stimulus during classical conditioning and its accompanying specific rec

48 ine receptor in both a cellular correlate of classical conditioning and light adaptation.

49 te the behavioral and neural consequences of classical conditioning and may have important consequenc

50 ody (MB) is critically involved in olfactory classical conditioning, and cAMP signaling molecules are

51 itive abilities, including immediate memory, classical conditioning, and spatial learning.

52 ere cAMP signaling is required for olfactory classical conditioning, are dispensable for the regulati

53 Current theories of classical conditioning assume that learning depends on t

54 tudying the molecular and cellular basis for classical conditioning, based on its ability to associat

55 Operant and classical conditioning both produce an increased respons

56 tly reduced an associative form of learning (classical conditioning) but had no effect on two nonasso

57 lasticity of human auditory responses due to classical conditioning, but go further in revealing dist

58 ey functional magnetic resonance imaging and classical conditioning by pairing a visual shape (condit

59 Since the formal description of classical conditioning by Pavlov, lesion studies in anim

60 ing eyeblinks and shows that robust eyeblink classical conditioning can be readily established in the

61 rcement learning-based theoretical models of classical conditioning (crudely, prediction learning) ar

62 day or more) impairs acquisition of eyeblink classical conditioning (EBC), a cerebellar-dependent Pav

63 Acquisition and retention of eyeblink classical conditioning (EBCC) was examined in 30 healthy

64 campal damage are impaired at delay eyeblink classical conditioning (EBCC).

65 um are critically involved in trace eyeblink classical conditioning (EBCC).

66 is a straightforward adaptation of Pavlov's classical conditioning experiment, that we used to measu

67 Olfactory classical conditioning experiments have previously demon

68 e that observed in interval timing tasks and classical conditioning experiments.

69 Classical conditioning features prominently in many etio

70 eeking behavior and that the LT trained in a classical conditioning format transferred to an operant

71 Computational models of classical conditioning have made significant contributio

72 rovide an alternative cellular mechanism for classical conditioning-Hebbian long-term potentiation (L

73 The authors examined 400 ms delay eyeblink classical conditioning in 20 participants with Fragile X

74 Using classical conditioning in adult mice of either sex, we s

75 tion, we used a neural correlate of eyeblink classical conditioning in an isolated brainstem from the

76 Thus, classical conditioning in Aplysia appears to be mediated

77 A cellular analog of classical conditioning in Aplysia was used to determine

78 enhancement of sensorimotor synapses during classical conditioning in Aplysia-like LTP of sensorimot

79 or-dependent LTP appears to be necessary for classical conditioning in Aplysia.

80 forms of learning such as sensitization and classical conditioning in Aplysia.

81 postsynaptic, possibly Hebbian, mechanism in classical conditioning in Aplysia.

82 reviously to be critical for nondifferential classical conditioning in Aplysia.

83 Light duration and intensity influence classical conditioning in Hermissenda through their effe

84 cells and interneurons that are involved in classical conditioning in Hermissenda.

85 ircuitry supporting delay and trace eyeblink classical conditioning in humans and laboratory animals

86 possible to study the functional anatomy of classical conditioning in humans.

87 neurobiology and individual determinants of classical conditioning in humans.

88 to an acute stressful experience facilitates classical conditioning in male rats but impairs conditio

89 Thus, classical conditioning in mammals activates MAPK, which

90 ewed interest in the possibility of studying classical conditioning in monkeys.

91 esult from less conscious processes, such as classical conditioning in the case of immune, hormonal,

92 ine receptor (D1R) agonist failed to support classical conditioning in the cellular analog, whereas D

93 (Sepia officinalis) were used to demonstrate classical conditioning in this species and to determine

94 Trace conditioning is a form of classical conditioning in which a time gap separates the

95 aversion (CTA) learning is a robust form of classical conditioning in which animals rapidly associat

96 Trace conditioning, a form of classical conditioning in which the presentation of the

97 -based memory traces that underlie olfactory classical conditioning in young and aged Drosophila.

98 The mimicking of classical conditioning, including acquisition, extinctio

99 mulates empirical data from many variants of classical conditioning, including delay and trace paradi

100 Olfactory classical conditioning increases the odor responsiveness

101 Eyeblink classical conditioning is a relatively simple form of as

102 mory formation after single-trial appetitive classical conditioning is dependent on an intact NO-cGMP

103 ensorimotor synapses in a cellular analog of classical conditioning is disrupted by infusing the Ca(2

104 Classical conditioning is thought to play a key role in

105 of awareness during the acquisition stage of classical conditioning is unknown.

106 Interest in classical conditioning is usually focused on anticipator

107 Classical conditioning is widely used to study motivatio

108 During olfactory classical conditioning, large subsets of dopaminergic ne

109 Classical conditioning models elegantly describe how ani

110 ecific reflex modification (CRM) occurs when classical conditioning modifies responding to an uncondi

111 xamined how cAMP manipulations and olfactory classical conditioning modulate olfactory responses in t

112 se data represent the first demonstration of classical conditioning of a steroid hormone response to

113 It is concluded that in vitro classical conditioning of an abducens nerve eye-blink re

114 Classical conditioning of Aplysia's siphon-withdrawal re

115 The cerebellum is necessary for classical conditioning of discrete behavioral responses

116 ic form of associative learning and memory - classical conditioning of discrete responses learned wit

117 Using classical conditioning of eye blink and other discrete r

118 Classical conditioning of eyeblink responses has been on

119 w learning occurs in cerebellar circuits for classical conditioning of eyeblinks are meeting this cha

120 These results suggest that appetitive classical conditioning of feeding resulted in the pairin

121 likely that reinforcement during appetitive classical conditioning of feeding was mediated by affere

122 ere we test these competing hypotheses using classical conditioning of perceptually similar odours in

123 Exposure to familiar stimuli facilitates classical conditioning of physiological responses, inclu

124 experiments involving the cellular analog of classical conditioning of siphon withdrawal.

125 Classical conditioning of the eye-blink response, perhap

126 cessory inferior olive in infant rats during classical conditioning of the eye-blink response.

127 r interpositus nucleus in infant rats during classical conditioning of the eye-blink response.

128 Classical conditioning of the eyeblink or nictitating me

129 etention of a simple form of motor learning, classical conditioning of the eyeblink reflex, depends o

130 test nor the learning of motor timing during classical conditioning of the eyeblink reflex.

131 d its associated circuitry are essential for classical conditioning of the eyeblink response and othe

132 Classical conditioning of the eyeblink response is a for

133 possible modulatory role of motor cortex in classical conditioning of the eyeblink response was exam

134 In delay classical conditioning of the eyeblink response, the cer

135 anges in cerebellar AMPA receptors following classical conditioning of the eyeblink-nictitating membr

136 the crucial involvement of the cerebellum in classical conditioning of the eyeblink/nictitating membr

137 Behavioral and physiological data about classical conditioning of the eyelid response and motor

138 cific facilitation), which may contribute to classical conditioning of the gill and siphon withdrawal

139 Studies of sensitization and classical conditioning of the gill-withdrawal reflex in

140 Differential classical conditioning of the gill-withdrawal response (

141 act preparation to test whether differential classical conditioning of the GWR also depends on activa

142 Classical conditioning of the mollusc, Hermissenda crass

143 Classical conditioning of the nictitating membrane-eye b

144 Here we apply classical conditioning of the proboscis extension respon

145 ntribution of various cellular mechanisms to classical conditioning of the reflex with a siphon tap c

146 and motor neurons contributes importantly to classical conditioning of the reflex.

147 ransduction pathways have been implicated in classical conditioning of this preparation, although the

148 To understand the effect of classical conditioning on the output of type B photorece

149 ined after rabbits were given either paired (classical conditioning) or explicitly unpaired (control)

150 A classical conditioning paradigm (P3-P4) showed that the

151 tioning was measured using delayed eye blink classical conditioning paradigm and results were compare

152 Here, we developed a classical conditioning paradigm in which 6- to 8-d-old l

153 tion tests, subjects were trained in a delay classical conditioning paradigm using a tone conditioned

154 A forelimb-withdrawal classical conditioning paradigm was used in awake cats (

155 of sucrose and quinine paired with cues in a classical conditioning paradigm while the electrophysiol

156 ic stimulation of motor cortex and eye blink classical conditioning paradigm, to test whether dystoni

157 Rabbits were trained in a delay classical conditioning paradigm, using a tone as the con

158 imaging (fMRI) in an olfactory version of a classical conditioning paradigm, whereby neutral faces w

159 ociate a juice reward with a visual cue in a classical conditioning paradigm.

160 In classical conditioning paradigms, conditioned stimuli tr

161 Classical conditioning paradigms, such as trace conditio

162 ple, by intracortical microstimulation or by classical conditioning paradigms.

163 uence requirement observed in most mammalian classical conditioning paradigms.

164 portant roles in the description of numerous classical conditioning paradigms.

165 le using electrochemical technology during a classical conditioning procedure.

166 In contrast, classical conditioning procedures in which the CS and US

167 ry preferences through honeybee colonies via classical conditioning procedures.

168 previously neutral environmental stimuli via classical conditioning processes.

169 as verbal information or distinct stimuli in classical conditioning, provide signals that activate pl

170 Drosophila antennal lobe, we show here that classical conditioning rapidly alters the neural code re

171 In a prenatal model of classical conditioning, rat fetuses received presentatio

172 he role of the cerebellar cortex in eyeblink classical conditioning remains unclear.

173 irming that orientation discrimination under classical conditioning requires primary visual cortex (V

174 Classical conditioning resulted in synaptic facilitation

175 rabbits during and after a single trace fear classical conditioning session.

176 tly predicts that hippocampal involvement in classical conditioning should be critical not only when

177 signals can be exploited by visual cortex as classical conditioning stimuli, enabling the perceptual

178 ene's function to specific pathways used for classical conditioning, such as conditioned stimulus (CS

179 We find in a classical conditioning task that individual mouse GPh ne

180 hippocampal function simulate performance in classical conditioning tasks using the error backpropaga

181 During classical conditioning tasks, conditioned stimuli appear

182 al tegmental area (VTA) while mice performed classical conditioning tasks.

183 uts to dopamine neurons while mice performed classical conditioning tasks.

184 ards even when choice is not required, as in classical conditioning tasks.

185 Classical conditioning that involves mnemonic processing

186 d and non-affected side and normal eye blink classical conditioning that was not different from healt

187 Six hours after classical conditioning the levels of both autophosphoryl

188 Classical conditioning the rabbit nictitating membrane i

189 Classical conditioning, the simplest form of associative

190 thermore, in contrast to adult somatosensory classical conditioning, these data suggest that the cort

191 In classical conditioning to an auditory conditioned stimul

192 etic resonance imaging (fMRI) with olfactory classical conditioning to differentiate the neural respo

193 nt article applies a neural network model of classical conditioning to investigate the possible cause

194 Results implicate specific brain regions in classical conditioning to meth and demonstrate the impor

195 aging of dendritic spines with auditory-cued classical conditioning to test if the formation of a fea

196 this study, neurophysiological correlates of classical conditioning training were identified and char

197 ole of tTLL in an in vitro model of eyeblink classical conditioning using an isolated brainstem prepa

198 ornis is an invertebrate model used to study classical conditioning using light as the conditioned st

199 n of the reinforcement pathway that supports classical conditioning was analyzed in additional behavi

200 Trace eyeblink classical conditioning was assessed in patients with bil

201 Here, a single-cell analog of classical conditioning was developed.

202 Memory for classical conditioning was retained for at least 24 hr.

203 angry faces, one of which, through previous classical conditioning, was associated with a burst of w

204 Using olfactory classical conditioning, we observe that both scFv antibo

205 During classical conditioning, we observed opposite dynamics in

206 that produce reinstatement in other forms of classical conditioning, we observed spontaneous recovery

207 in a pattern-based imaging paradigm of human classical conditioning, we were able to identify dissoci

208 Moreover, correlates of classical conditioning were specific to stimulation of A

209 ng Maintenance Days 5 and 10, rats underwent classical conditioning, whereby passive cocaine infusion

210 facilitation (PA-LTF), a cellular analog of classical conditioning, which was expressed at Aplysia s

211 diately after, and 1 hr after one session of classical conditioning with a tone CS and a corneal airp

212 he timing of neurotransmitter release during classical conditioning with the use of two-photon micros