ライフサイエンスコーパス: clathrin

1 ffolding proteins, such as beta-arrestin and clathrin.

2 ical Golgi coat proteins coatomer (COPI) and clathrin.

3 r of clathrin coats after the recruitment of clathrin.

4 A directly impacts recruitment of EndoB2 and clathrin.

5 showing that AP-2 does not co-localize with clathrin.

6 Endocytosis mediated by clathrin, a cellular process by which cells internalize

7 Clathrin, a cytosolic protein composed of heavy and ligh

8 Clathrin accumulated in the cytoplasmic virus assembly c

9 dentified the medium subunit (micro1) of the clathrin adaptor AP-1 as a top hit.

10 cilitates BUBR1-dependent recruitment of the clathrin adaptor AP2 to IR.

11 binding and recruits beta-arrestins and the clathrin adaptor AP2 to trigger PC1 internalization.

12 membrane-facing in the open form of the AP2 clathrin adaptor complex.

13 sm to diversify the functional repertoire of clathrin adaptor complexes.

14 Here we report that Ent5, an endosomal clathrin adaptor in Saccharomyces cerevisiae, regulates

15 ntracellular loop of PAR4 and found that the clathrin adaptor protein complex-2 (AP-2) is important f

16 ion factor-binding protein 3), a multidomain clathrin adaptor protein that sorts cargo proteins at th

17 ) and LL, that are important for binding the clathrin adaptor proteins AP-1 and AP-2in vitro Surprisi

18 AP-1 is a clathrin adaptor recruited to the trans-Golgi Network wh

19 res an ExxxLL motif predicted to recruit the clathrin adaptor, Adaptor protein 2 (AP2).

20 ain structure that serves as an unusual AP-1 clathrin adaptor-dependent Golgi export signal in one Ki

21 are structurally conserved between COPI and clathrin/adaptor proteins.

22 phosphatase 1 regulatory subunit 9b) and the clathrin adaptors AP-1 and AP-2.

23 Clathrin adaptors display diversity in both the type and

24 To function, clathrin requires clathrin adaptors that link it to transmembrane protein

25 important roles in recruitment of two major clathrin adaptors, Gga (Golgi-localized, gamma-adaptin e

26 plex and endosomal compartments and recruits clathrin adaptors.

27 mblies on membranes that display five unique clathrin adaptors.

28 vaccinia virus transiently recruits AP-2 and clathrin after fusion with the plasma membrane.

29 this by developing coarse-grained models for clathrin and AP2, employing a Monte Carlo click interact

30 describe the functional interactions between clathrin and AP2.

31 ally affects the dynamics and recruitment of clathrin and associated adaptors, and leads to reduction

32 n and curvature-generating proteins, such as clathrin and BAR domain proteins.

33 ANK functionally interacts with clathrin and clathrin associated adaptor protein (AP) co

34 ell surface endogenously expressed CB1Rs was clathrin and dynamin dependent and could be modeled as a

35 Inhibitors of clathrin and dynamin prevented CLR endocytosis and activ

36 cytic and endocytic vesicles, such as Vamp2, Clathrin and Dynamin, are sequestered in unreleased CGs,

37 are CME hotspots, and that key CME proteins, clathrin and dynamin, show a strong preference towards p

38 th DFz2-containing vesicles derived from the clathrin and dynamin-dependent endocytic pathway, which

39 In particular, clathrin and dynamin-dependent endocytosis has been well

40 y modulated the interaction of betaarrs with clathrin and ERK MAP kinase.

41 s assemble into a BBSome that interacts with clathrin and is localized to membranes of the flagellar

42 se and protein kinase C activities, and both clathrin and lipid rafts.

43 ifespan of endosomal structures that contain clathrin and other adaptors, suggesting a defect in coat

44 Clathrin and other proteins assemble into small invagina

45 sembly at the plasma membrane, intracellular clathrin and phosphatidylinositol-3,4-bisphosphate predi

46 AnkB binds directly to GLUT4 and clathrin and promotes their association in adipocytes.

47 Our findings provide evidence that clathrin and SYP121 functions are important for the coor

48 ting as limiting scaffold protein organizing clathrin and TACC3 complex crosslinking K-fibers.

49 r process in eukaryotic cells which involves clathrin and/or adaptor proteins, lipid kinases, phospha

50 oporin p62, DEP-domain containing protein 5, clathrin, and dynamin-1 were different between groups, s

51 Clathrin- and actin-mediated endocytosis is essential in

52 differentially sensitive to perturbation of clathrin- and caveolin-mediated endocytosis.

53 evin2, we show that CGs are endocytosed in a clathrin- and dynamin-dependent manner.

54 Our data indicate that clathrin/AP-mediated cycling of ANK between the TGN, end

55 Together, these findings uncover AP-2 and clathrin as players in Slack channel regulation.

56 ates to the process of adaptor clustering as clathrin assembles around a growing pit remains unclear.

57 e the formation kinetics and distribution of clathrin assemblies on membranes that display five uniqu

58 Coordinated clathrin assembly provides the earliest spatial cue for

59 etween equilibrium and kinetic parameters of clathrin assembly to the eventual adaptor distribution i

60 ated pits (CCPs), in which adaptors nucleate clathrin assembly.

61 lathrin clustered but instead by the rate of clathrin assembly.

62 ANK functionally interacts with clathrin and clathrin associated adaptor protein (AP) complexes as lo

63 endrite through direct interactions with the clathrin-associated adaptor protein complexes (APs) in C

64 as affinity handles, we identified candidate clathrin-associated proteins (CAPs) in Trypanosoma cruzi

65 hieve three-dimensional particle tracking of clathrin-associated structures with velocities up to 4.5

66 y report the phosphoinositide composition of clathrin-associated structures, and the use of these sen

67 Disabled-2 (Dab2) is a widely expressed clathrin binding endocytic adaptor protein and known for

68 These findings support a model in which Ent5 clathrin binding performs a mechanistic role in coat mat

69 involve the recruitment and stabilization of clathrin-binding adaptor proteins and the clathrin coat.

70 Quantitative clathrin-binding analyses revealed no significant altera

71 type and number of evolutionarily conserved clathrin-binding boxes.

72 beta subunit (beta2) of higher fungi lacks a clathrin-binding domain, and experiments showing that AP

73 lyses revealed no significant alterations in clathrin-binding efficiency, suggesting that the inabili

74 A clathrin-binding protein fragment ("hook") is inducibly

75 This fast endocytosis is independent of clathrin but mediated by dynamin and actin.

76 STIM1/ORAI1 colocalizes with clathrin, but not with caveolin, at the apical membrane

77 fs in the C-terminus of A36 recruit AP-2 and clathrin by interacting directly with the Epsin15 homolo

78 In HeLa cells depleted of clathrin by siRNA, activated PAR4 failed to internalize.

79 zation in cells that could be independent of clathrin, caveolin, actin, and lipid phase separation.

80 s recently discovered, HPV entry occurs by a clathrin-, caveolin-, and dynamin-independent endocytosi

81 diated endocytosis (CME) or independently of clathrin (CIE) remains segregated in the ERC, likely on

82 lls assemble at least 50 proteins, including clathrin, clathrin-interacting proteins, actin filaments

83 mount of clathrin recruited or the degree of clathrin clustered but instead by the rate of clathrin a

84 lasses containing 1) anterograde cargoes and clathrin clusters or 2) retrograde cargoes and coatomer

85 Clathrin coat assembly on membranes requires cytosolic a

86 tection and tracking of fluorescently tagged clathrin coat components within cultured cells.

87 Inducible overexpression of the Arabidopsis clathrin coat disassembly factor, Auxilin2, which inhibi

88 Spatiotemporal heterogeneity of clathrin coat dynamics is also observed during morpholog

89 cant spatiotemporal alterations in endocytic clathrin coat dynamics.

90 as key membrane determinants for assembly of clathrin coat proteins that drive formation of clathrin-

91 of clathrin-binding adaptor proteins and the clathrin coat.

92 the cell surface and link them to the PM and clathrin coat.

93 chain of botulinum toxin C, nor by block of clathrin-coat maturation.

94 cytic defects and a striking accumulation of clathrin-coated intermediates, strongly implicating Sac

95 or protein 2 (AP2) complexes, which initiate clathrin-coated pit (CCP) assembly, are activated by con

96 , exhibit increased rates of CME and altered clathrin-coated pit dynamics.

97 importance of molecular events required for clathrin-coated pit initiation.

98 activity of neutral sphingomyelinase but not clathrin-coated pit maturation.

99 When and where a clathrin-coated pit will form and what cargo it will con

100 ing structures at the plasma membrane termed clathrin-coated pits (CCPs) that mediate vesicle formati

101 CME is initiated by the formation of clathrin-coated pits (CCPs), in which adaptors nucleate

102 hat ADAM17 is constitutively internalised by clathrin-coated pits and that physiological stimulators

103 st partially defined by the cytoskeleton and clathrin-coated pits, in which receptors and G proteins

104 At clathrin-coated pits, PI(3)P is produced by the INPP4A h

105 Then as the cargoes being enclosed in clathrin-coated pits, they slow down the active rotation

106 centration upon Wnt-induced recruitment into clathrin-coated pits.

107 targets the proteins for internalization via clathrin-coated pits.

108 f endocytosis following MC4R localization to clathrin-coated sites and exclusion of the receptor from

109 The earliest stages of clathrin-coated structure (CCS) assembly involve the rec

110 ar organisms on formation and dissolution of clathrin-coated structures (CCSs) have not been directly

111 Dynamics of endocytic clathrin-coated structures can be remarkably divergent a

112 hages resulted in the binding of mu1A of the clathrin-coated vesicle AP-1 complex.

113 The precise sequence of events promoting clathrin-coated vesicle assembly is still debated.

114 athways include effector endocytosis through clathrin-coated vesicle trafficking, defense signaling t

115 as sorting signals for packaging cargo into clathrin-coated vesicles (CCVs), and also facilitate dow

116 ty, and leads to presynaptic accumulation of clathrin-coated vesicles (CCVs)-all without decreasing G

117 strate that REEP6 is detected in a subset of Clathrin-coated vesicles and interacts with the t-SNARE,

118 localizes to the trans-Golgi network (TGN), clathrin-coated vesicles and the plasma membrane.

119 l of cargo proteins to incorporate them into clathrin-coated vesicles for trafficking.

120 Clathrin-coated vesicles form by rapid assembly of discr

121 EEP6 in trafficking of cargo via a subset of Clathrin-coated vesicles to selected membrane sites in r

122 are important components for the cleavage of clathrin-coated vesicles, phagosomes, and mitochondria.

123 athrin coat proteins that drive formation of clathrin-coated vesicles.

124 embrane, which triggers the formation of new clathrin-coated vesicles.

125 embly of Grb2/Sos1 complexes associated with clathrin-coated vesicles.

126 ortant mediators of cargo protein sorting in clathrin-coated vesicles.

127 t solute uptake occurs in both caveolae- and clathrin-coated vesicles.

128 omyces cerevisiae, regulates the behavior of clathrin coats after the recruitment of clathrin.

129 teins are found to regulate the formation of clathrin coats under certain conditions, but can also su

130 fficking systems, including COPI, COPII, and clathrin complexes.

131 microscopy to be modified and to have higher clathrin content than those of cells not exposed to elev

132 Instead, we find that clathrin contributes to the regularity of vesicle scissi

133 ant as tools to investigate the effects that clathrin defects have on secretion pathways and plant gr

134 The endocytosis was clathrin dependent and partially dependent on beta-arres

135 mechanism of internalization was shown to be clathrin dependent.

136 level, we observed a striking impairment of clathrin-dependent and -independent endocytosis in proxi

137 We examined the selective regulation of clathrin-dependent EGFR signaling and endocytosis.

138 ed outcome of neuronal injury with disrupted clathrin-dependent endocytosis and impaired receptor des

139 namin is a GTPase that plays a vital role in clathrin-dependent endocytosis and other vesicular traff

140 link between SYP121-dependent secretion and clathrin-dependent endocytosis at the plasma membrane.

141 phatase-activating protein that functions in clathrin-dependent endocytosis, and beta-1,3-glucoronylt

142 pling of cell surface GLP-1R activation with clathrin-dependent endocytosis, the SNXs were found to c

143 Activated GPCRs undergo clathrin-dependent endocytosis.

144 mutant triggered NaV1.7 internalization in a clathrin-dependent manner involving the E3 ubiquitin lig

145 DKK1 induced internalization of CKAP4 in a clathrin-dependent manner, further supporting CKAP4 as a

146 endosomal acidification but is distinct from clathrin-dependent or macropinocytic uptake pathways.

147 We show that loss of Ent5 disrupts clathrin-dependent traffic and prolongs the lifespan of

148 and the GTPase dynamin but does not require clathrin-dependent uptake.

149 ence of ECM, prolactin is internalised via a clathrin-dependent, but caveolin-independent, route.

150 found to be reduced approximately twofold in clathrin-depleted cells as a whole and approximately fiv

151 Secretion assays revealed that clathrin depletion causes a near-complete block in secre

152 There is growing evidence that clathrin does not determine the membrane morphology of t

153 on of ClaH corroborated the observation that clathrin does not play an important role in endocytosis

154 i evoke a slower mode of endocytosis that is clathrin, dynamin, and actin dependent.

155 VEGFR2, by interference with the function of clathrin, dynamin, or Rab5, increases dramatically the c

156 We identify five (clathrin, dynamin1, AP2, sorting nexins [SNX] SNX27, and

157 are necessary and sufficient for functional clathrin engagement.

158 dherin is cleaved by calpain upon entry into clathrin-enriched domains.

159 icle recycling by promoting the uncoating of clathrin following synaptic vesicle uptake.

160 During endocytosis, clathrin forms a cage-like coat around the membrane and

161 Our results show that clathrin function at the PM is required to induce plant

162 ly, our data demonstrate the multiplicity of clathrin functions in cortical pattern formation and pro

163 Taken together, our data indicate that clathrin has a function in DCV biogenesis beyond its est

164 We examined clathrin heavy chain (ClaH-GFP) which localized to three

165 protein zeta/delta, annexin A1/A3/A4/A5/A6, clathrin heavy chain 1, glyceraldehyde-3-phosphate dehyd

166 We found that LdRab5b interacts with clathrin heavy chain and hemoglobin receptor.

167 We investigate how the deletion of clathrin heavy chain impairs the dynamics and the morpho

168 However, LdRab5a failed to interact with the clathrin heavy chain, and interaction with hemoglobin re

169 ith an inducible short hairpin RNA targeting clathrin heavy chain, resulting in approximately 85% pro

170 , which has a stomatal function defect, as a clathrin heavy chain1 (CHC1) mutant allele and show that

171 to distinct domains on late Golgi, and these clathrin "hubs" dispersed in synchrony after the late Go

172 This investigation focuses on clathrin in Aspergillus nidulans, with the aim of unders

173 To investigate the role of clathrin in DCV biogenesis, we stably transduced PC12 ce

174 electron microscopy, we address the role of clathrin in this process.

175 Here we demonstrate that clathrin-independent dynamin 2-mediated caveolar uptake

176 The molecular underpinnings of this clathrin-independent endocytic pathway are largely unkno

177 uman papillomaviruses enter host cells via a clathrin-independent endocytic pathway involving tetrasp

178 zation of Ptr2 through a recently identified clathrin-independent endocytic pathway that requires the

179 n signaling, is PI3K/Akt dependent, and is a clathrin-independent endocytic process, we determined wh

180 th integrins and is a major cargo protein of clathrin-independent endocytic vesicles.

181 show that tau assemblies enter cells through clathrin-independent endocytosis and escape from damaged

182 n as caveolae that participate in signaling, clathrin-independent endocytosis and mechanotransduction

183 at SNX9 is required for RhoGTPase-dependent, clathrin-independent endocytosis, and in this capacity,

184 adapted to utilize THY-1, a cargo protein of clathrin-independent endocytotic vesicles, to facilitate

185 spergillus nidulans, we show that AP-2 has a clathrin-independent essential role in polarity maintena

186 d, in the course of evolution, a specialized clathrin-independent function necessary for fungal polar

187 rom the plasma membrane to the nucleus via a clathrin-independent mechanism.

188 wing engulfment into a membrane vesicle by a clathrin-independent process.

189 Although the clathrin inhibitor PitStop2 did not impact the ability o

190 le at least 50 proteins, including clathrin, clathrin-interacting proteins, actin filaments, and acti

191 of these Fabs selectively disrupted betaarr-clathrin interaction, and when expressed as an intrabody

192 Clathrin is a ubiquitous protein that mediates membrane

193 are altered in chc mutants, indicating that clathrin is important for stomatal regulation.

194 Our results show that clathrin is not required for elongating or shaping the e

195 We find that the direct binding of Ent5 with clathrin is required for its role in coat behavior and c

196 To establish clathrin light chain (CLC) function in vivo, we engineer

197 Using clathrin light chain (TcCLC) and EpsinR (TcEpsinR) as af

198 Two clathrin light chain isoforms, CLCa and CLCb, are integr

199 isms, as well as Retinitis Pigmentosa Type 2-Clathrin Light Chain, a membrane protein with a novel do

200 Sodium channel and clathrin linker 1 (SCLT1) mutations were associated with

201 nalization of GPCRs via interaction with the clathrin machinery and mediate signalling via 'non-class

202 tasis and has long been used as a marker for clathrin mediated endocytosis.

203 ggers down-regulation of Kv1.3 by inducing a clathrin-mediated endocytic event that targets the chann

204 The robustness of processes like clathrin-mediated endocytosis (CME) across a diverse ran

205 niae invasion of HL-1 cells occurred through clathrin-mediated endocytosis (CME) and independently of

206 Cell surface receptor uptake via clathrin-mediated endocytosis (CME) and subsequent intra

207 Clathrin-mediated endocytosis (CME) constitutes the majo

208 The critical initiation phase of clathrin-mediated endocytosis (CME) determines where and

209 Current understanding of clathrin-mediated endocytosis (CME) dynamics is based on

210 hondrial uncoupler, is a potent inhibitor of clathrin-mediated endocytosis (CME) in different systems

211 Clathrin-mediated endocytosis (CME) involves nanoscale b

212 Clathrin-mediated endocytosis (CME) is a fundamental pro

213 e receptors at the postsynaptic membrane via clathrin-mediated endocytosis (CME) is a key mechanism f

214 Clathrin-mediated endocytosis (CME) is a major internali

215 Clathrin-mediated endocytosis (CME) is the major route o

216 Clathrin-mediated endocytosis (CME) is used to internali

217 Clathrin-mediated endocytosis (CME) manages the sorting

218 We show that cargo internalized either via clathrin-mediated endocytosis (CME) or independently of

219 During clathrin-mediated endocytosis (CME), endocytic-site matu

220 disassembly factor, Auxilin2, which inhibits clathrin-mediated endocytosis (CME), impaired the AtPep1

221 receptors are internalized and regulated by clathrin-mediated endocytosis (CME).

222 ase dynamin mediates membrane fission during clathrin-mediated endocytosis (CME).

223 This protein complex is internalized via clathrin-mediated endocytosis and degraded in lysosomes,

224 ct rotational behaviors of nanocargos during clathrin-mediated endocytosis and intracellular transpor

225 enveloped RNA viruses that infect cells via clathrin-mediated endocytosis and low-pH-triggered fusio

226 Thus, XLalphas restricts clathrin-mediated endocytosis and plays a critical role

227 pressed PI4P 5-kinase PIP5K6 is required for clathrin-mediated endocytosis and polar tip growth in po

228 Dynamin plays a crucial role in clathrin-mediated endocytosis and synaptic transmission

229 cellular internalization pathways identified clathrin-mediated endocytosis as the main route for eHEV

230 thrin revealed the dynamics of EGF-activated clathrin-mediated endocytosis during internalization.

231 The conditional use of actin during clathrin-mediated endocytosis in mammalian cells suggest

232 SA inhibited clathrin-mediated endocytosis in pollen tubes associated

233 Clathrin-mediated endocytosis is a major regulator of ce

234 Our data suggest that clathrin-mediated endocytosis is increased in PAPC-expre

235 Here we show that clathrin-mediated endocytosis is required for ECM-depend

236 1), Ca(2+), or protein kinase C (PKC) impair clathrin-mediated endocytosis of EGFR, the formation of

237 f-concept genetic evidence that blocking the clathrin-mediated endocytosis of LGR5 could be used to p

238 Clathrin-mediated endocytosis of transferrin (Tf) and it

239 hese manipulations was without effect on the clathrin-mediated endocytosis of transferrin receptor (T

240 uptake is dependent on macropinocytosis and clathrin-mediated endocytosis pathways.

241 ension of BMPRII-LF accounted for its faster clathrin-mediated endocytosis relative to BMPRII-SF, acc

242 In yeast, clathrin-mediated endocytosis requires a pulse of polyme

243 nociceptive neuronal excitability, the AP-2 clathrin-mediated endocytosis trafficking mechanism may

244 ined by confocal microscopy, indicating that clathrin-mediated endocytosis was not involved in THY-1-

245 architecture of the protein machinery during clathrin-mediated endocytosis was visualized using a new

246 nd these receptors are responsible for bulk, clathrin-mediated endocytosis within the cell.

247 ced interferon signaling or an inhibition of clathrin-mediated endocytosis, and PKD inhibitors do not

248 xin-9 (SNX9) and dynamins, key components of clathrin-mediated endocytosis, as binding partners of XL

249 ize nutrients and cell surface receptors via clathrin-mediated endocytosis, cells assemble at least 5

250 Consistent with clathrin-mediated endocytosis, expression of a dynamin d

251 Given the diversity of proteins regulated by clathrin-mediated endocytosis, how this process may dist

252 protein that participates in early stages of clathrin-mediated endocytosis, is downregulated as well

253 eletal dynamics, phosphoinositide signaling, clathrin-mediated endocytosis, polarized blebbing, and e

254 The second step involves clathrin-mediated endocytosis, which functions outside o

255 on of transferrin, a process that depends on clathrin-mediated endocytosis, while its ablation by CRI

256 l inhibited LGR5 internalization by blocking clathrin-mediated endocytosis.

257 e apical membrane of PTECs, which determines clathrin-mediated endocytosis.

258 ion of ankyrin-B (AnkB) in coupling GLUT4 to clathrin-mediated endocytosis.

259 tion of tissues through mechanoregulation of clathrin-mediated endocytosis.

260 ted enhancement is impaired by inhibitors of clathrin-mediated endocytosis.

261 efine alternative mechanisms that facilitate clathrin-mediated endocytosis.

262 rus G rapidly recycles from the membrane via clathrin-mediated endocytosis.

263 Fcho proteins during the earliest stages of clathrin-mediated endocytosis.

264 homology domain (ENTH) is a major player in clathrin-mediated endocytosis.

265 (+) efflux and M1 protein internalization by clathrin-mediated endocytosis.

266 carried out by adaptor protein-2 (AP-2) via clathrin-mediated endocytosis.

267 B1 surprisingly colocalized with the zone of clathrin-mediated endocytosis.

268 mparable uptake kinetics and a predominantly clathrin-mediated endocytotic mechanism, irrespective of

269 We also show that clathrin-mediated internalization of TNFR1 C-terminal fr

270 inase-dependent and blocked by inhibitors of clathrin-mediated internalization; and EGFR activity was

271 2-BUBR1 interaction and prevents spontaneous clathrin-mediated IR endocytosis.

272 ata provide the first systematic analysis of clathrin-mediated trafficking in T. cruzi, allowing comp

273 In African trypanosomes clathrin-mediated trafficking is responsible for endocyt

274 on of enlarged EspG/TfR/Rab11 positive, EEA1/Clathrin negative stalled recycling structures.

275 lypicans requires dynamin- and Rab5, but not clathrin or active BMP signalling.

276 iated pathway, rather than inhibitors of the clathrin or caveolar routes.

277 r, it was neither saturable nor dependent on clathrin or receptor binding.

278 ce, showed persistent formation of endocytic clathrin pits and vesicles during mitosis.

279 over the entire cell surface and found that clathrin pits form at a lower rate during late mitosis.

280 lustering is determined not by the amount of clathrin recruited or the degree of clathrin clustered b

281 rated that this interaction is essential for clathrin recruitment to the DRG membrane, Slack channel

282 To function, clathrin requires clathrin adaptors that link it to tran

283 of epidermal growth factor (EGF) ligand and clathrin revealed the dynamics of EGF-activated clathrin

284 ebrates demonstrates that CLCs contribute to clathrin's role in vivo by influencing cargo selectivity

285 ediate membrane cargo trafficking, including Clathrin, Sec13-Sec31, and alphabeta'epsilon-COP.

286 nt reduction in synaptojanin1 recruitment to clathrin structures, indicating broad control of CCP ass

287 R and TfR were recruited to largely distinct clathrin structures.

288 This instance of clathrin subunit deletion in vertebrates demonstrates th

289 ind to AP-2 complex components as well as to clathrin, suggesting roles in endocytosis and vesicle re

290 These findings suggest that clathrin supports specific functions in multiple cell ty

291 n pharmacological inhibition of dynamin-2 or clathrin terminal domain (TD) ligand association, these

292 taarr1 complex can efficiently interact with clathrin terminal domain and ERK2 MAPK in vitro.

293 and other trypanosomes and also suggest that clathrin trafficking in at least some life stages of T.

294 The assembly of clathrin triskelia into polyhedral cages during endocyto

295 B1 vesicles also contained caveolin, whereas clathrin was almost undetectable on those vesicles.

296 Although clathrin was essential for growth, ClaH did not colocali

297 Surprisingly, the onset of clathrin waves, but not individual endocytic events, req

298 In the lipid-stressed cells, MC4R and clathrin were redistributed to the plasma membrane where

299 to lysosomes, we tested the role of GGA1 and clathrin, which mediate sorting in the canonical Golgi-t

300 chanical theory for the assembly behavior of clathrin, yielding good agreement with our simulations a