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1 uding those that regulate disassembly of the clathrin coat.
2 o the correct spatiotemporal assembly of the clathrin coat.
3 the cell surface and link them to the PM and clathrin coat.
4 of clathrin-binding adaptor proteins and the clathrin coat.
5 ished for clathrin-AP-1/AP-2 coats, to a non-clathrin coat.
6 organization of the inner AP-1 layer of the clathrin coat.
7 regulation to ensure the correct assembly of clathrin coats.
8 on structural elements with COPI, COPII, and clathrin coats.
9 pe and results in loss of ARH from endocytic clathrin coats.
12 h a perturbation of the coupling between the clathrin coat and the actin cytoskeleton, which we confi
14 oated vesicle, or with an in vitro assembled clathrin coat, and recruits Hsc70 to its specific heavy-
15 yotes, and many of the proteins required for clathrin coat assembly and disassembly have orthologs in
17 functions in clathrin-mediated endocytosis, clathrin coat assembly protein AP180, was quantified as
18 ribosylation factor 1 (ARF1), essential for clathrin coat assembly, Golgi architecture, and vesicula
21 s was not accompanied by the accumulation of clathrin coated buds on their surface and this process p
22 ular plasma membrane invaginations capped by clathrin-coated buds, occurs selectively at inhibitory s
23 ch in metazoans links endocytic cargo to the clathrin coat, but had no assigned function in yeast, wa
26 d the genes encoding a set of early arriving clathrin-coat constituents, FCHO1 and FCHO2, in HeLa cel
27 Inducible overexpression of the Arabidopsis clathrin coat disassembly factor, Auxilin2, which inhibi
29 te and facilitates compensatory endocytosis, clathrin-coat disassembly, and vesicle reavailability at
33 e fission, which resulted in accumulation of clathrin-coated endocytic intermediates on the plasma me
34 ce also indicates that Ulk1/2 mediates a non-clathrin-coated endocytosis in sensory growth cones.
35 h Vps class C/HOPS subunits incorporate into clathrin-coated endosomal domains and carriers in mammal
38 -active processes-vacuolar acidification and clathrin-coat formation--as modulators of sertraline's a
39 nistic models for disassembly of nonneuronal clathrin coats has been limited by the absence of a func
40 of the heat shock response or disassembly of clathrin coats, however, where binding of a short hydrop
41 at about 11 A resolution, the structure of a clathrin coat (in the D6-barrel form) with specifically
43 cytic defects and a striking accumulation of clathrin-coated intermediates, strongly implicating Sac
44 ecreased dynamin recruitment to the necks of clathrin-coated invaginations resulting in impaired vesi
45 tor protein 4 (AP-4) is a component of a non-clathrin coat involved in protein sorting at the trans-G
48 or protein 2 (AP2) complexes, which initiate clathrin-coated pit (CCP) assembly, are activated by con
51 This corresponded to a decreased rate of clathrin-coated pit (CCP) initiation and increased lifet
52 DEO ABSTRACT: Some endocytic cargoes control clathrin-coated pit (CCP) maturation, but it is not know
54 raphy to investigate the massive increase in clathrin-coated pit abundance that is selectively observ
57 synthesis and its metabolic processing, the clathrin-coated pit endocytosis pathway, and the ubiquit
64 after a 4 h chase and became undetectable if clathrin-coated pit-mediated trafficking was blocked wit
65 Classical CME proceeds via the formation of clathrin-coated pits (CCPs) at the plasma membrane, whic
67 ns (EAPs) mediate assembly and maturation of clathrin-coated pits (CCPs) into cargo-containing vesicl
69 es of the observed heterogeneous dynamics of clathrin-coated pits (CCPs) might be the different cargo
70 rgo receptors, either recruiting cargos into clathrin-coated pits (CCPs) or initiating clathrin-coat
71 ntrol its fate by regulating the dynamics of clathrin-coated pits (CCPs) that mediate their internali
72 ing structures at the plasma membrane termed clathrin-coated pits (CCPs) that mediate vesicle formati
73 substantial increase in the ratio of "open" clathrin-coated pits (CCPs) to "necked"/"closed" CCVs an
74 composition of the lifetime distributions of clathrin-coated pits (CCPs) to measure independent aspec
75 P(2) levels and is concentrated at endocytic clathrin-coated pits (CCPs) via interactions with the sc
77 cargos are known to accumulate into maturing clathrin-coated pits (CCPs), whether and how cargo recru
81 which the GAK was disrupted showed a lack of clathrin-coated pits and a complete block in clathrin-me
82 that results in an accumulation of arrested clathrin-coated pits and a greatly reduced synaptic vesi
83 e clathrin-mediated endocytosis with shallow clathrin-coated pits and a strong reduction in the inter
85 ls through receptor-mediated endocytosis via clathrin-coated pits and caveolae, that actin filaments
86 tic adaptor, which is highly concentrated at clathrin-coated pits and coordinates acquisition of bila
89 hat ADAM17 is constitutively internalised by clathrin-coated pits and that physiological stimulators
90 per-resolution images of living cells, using clathrin-coated pits and the transferrin cargo as model
91 ies showed FcRY-mediated internalization via clathrin-coated pits and transport involving early and r
93 eptor signaling, in cells in which endocytic clathrin-coated pits are frozen at a deeply invaginated
96 catalyzes the scission of deeply invaginated clathrin-coated pits at the plasma membrane, but the mec
100 ns can be marked as cargo for inclusion into clathrin-coated pits by common internalization signals (
102 ntry that is low-pH dependent occurs through clathrin-coated pits in a manner similar to wild-type vi
104 is generally believed that the formation of clathrin-coated pits in epithelial cells occurs randomly
105 y ligands, MORs are rapidly internalized via clathrin-coated pits in heterologous cells and dissociat
109 ocalization of the SNX9.dynamin-2 complex to clathrin-coated pits is blocked by interactions with the
110 d capsids laterally diffused into assembling clathrin-coated pits less than 30 s after attachment.
112 rin receptors are delivered selectively from clathrin-coated pits on the plasma membrane into a speci
113 e ESCRT-0 complex accumulates at a subset of clathrin-coated pits on the surface of human cells.
114 ocal activity, and axonal boutons containing clathrin-coated pits showed a more pronounced decrease i
115 erized, and it remains controversial whether clathrin-coated pits specialize to internalize particula
116 indings reveal a link between progression of clathrin-coated pits to endocytic vesicles and an activa
117 , these findings indicate that BRAG2 acts at clathrin-coated pits to promote integrin internalization
118 D1A, which encodes a protein associated with clathrin-coated pits where cell-surface receptors reside
119 toplasmic domain of E-selectin or disrupting clathrin-coated pits with hypertonic medium blocked inte
121 PS15, a protein required for the assembly of clathrin-coated pits, and DN PAK-1, an obligate mediator
122 Triad3A associates with Arc, localizes to clathrin-coated pits, and is associated with endocytic s
123 oenvironments, three-dimensional tracking of clathrin-coated pits, and long-term imaging spanning >10
124 Lp(a) internalization was also dependent on clathrin-coated pits, and Lp(a) was targeted for lysosom
125 ysically interacts with AP-2, is enriched on clathrin-coated pits, and requires clathrin but not RAB-
126 1 and inducing GLUT1 internalization through clathrin-coated pits, as well as indirectly, by reducing
128 onstruct of EPS15, an essential component of clathrin-coated pits, blocked the entry of RRV into RFs.
129 sion by forming a collar around the necks of clathrin-coated pits, but the specific structural intera
130 thrin interactor, is recruited to late-stage clathrin-coated pits, clinical manifestations have been
131 st partially defined by the cytoskeleton and clathrin-coated pits, in which receptors and G proteins
132 )P(3) biosensors, disappearance of endocytic clathrin-coated pits, nearly complete inhibition of KCNQ
134 n thought to coordinate cargo selection into clathrin-coated pits, results in a significant impairmen
136 endocytosis of ligand-receptor complexes via clathrin-coated pits, trafficking of the internalized li
137 ntrast to INPP5B visits late stage endocytic clathrin-coated pits, was earlier shown to contain anoth
138 ed mechanism for 7TMR internalization is via clathrin-coated pits, where clathrin and adaptor protein
139 ns in a complex with the GTPase dynamin-2 at clathrin-coated pits, where it provokes fission of vesic
140 accumulate at the base of arrested endocytic clathrin-coated pits, where they support the growth of d
141 -coated vesicles and an increase in U-shaped clathrin-coated pits, which may result from sequestratio
142 ntly, ligand-bound EGFR is incorporated into clathrin-coated pits--membrane structures containing cla
155 rin-mediated endocytosis and associates with clathrin-coated pits/vesicles at the plasma membrane.
157 as key membrane determinants for assembly of clathrin coat proteins that drive formation of clathrin-
158 f endocytosis following MC4R localization to clathrin-coated sites and exclusion of the receptor from
160 ar organisms on formation and dissolution of clathrin-coated structures (CCSs) have not been directly
161 we found that actin patches associated with clathrin-coated structures (CCSs) in cultured mouse cell
163 oth the large insert isoform of myosin VI on clathrin-coated structures and the no-insert isoform on
164 Further, the steady-state morphology of clathrin-coated structures appears to be a manifestation
169 ficially dimerized construct of myosin VI on clathrin-coated structures suggests that wild type myosi
170 nse core vesicles) and endocytic structures (clathrin-coated structures) and the proteins associated
171 lathrin machinery, localizes to cell surface clathrin-coated structures, and is enriched in placental
174 teins are found to regulate the formation of clathrin coats under certain conditions, but can also su
176 (FCHo1/2) were required for plasma membrane clathrin-coated vesicle (CCV) budding and marked sites o
177 and then compared the protein composition of clathrin-coated vesicle (CCV) fractions from control and
186 P-2 cooperate to increase the probability of clathrin-coated vesicle formation and to control the num
187 rate clone 15) is well known for its role in clathrin-coated vesicle formation at the plasma membrane
188 of adaptor protein-1 (AP1), responsible for clathrin-coated vesicle formation at the trans-Golgi, wa
190 dicate that Eps15 plays an important role in clathrin-coated vesicle formation not only at the plasma
195 roteins from HeLa cells and identified known clathrin-coated vesicle proteins with >90% accuracy.
196 iling is a universal method for defining the clathrin-coated vesicle proteome and may be adapted for
198 Thus, our results reveal the importance of clathrin-coated vesicle trafficking in C. burnetii infec
199 athways include effector endocytosis through clathrin-coated vesicle trafficking, defense signaling t
202 e, auxilin, associates with a freshly budded clathrin-coated vesicle, or with an in vitro assembled c
206 ( approximately 300-nm diameter) and typical clathrin-coated vesicles ( approximately 90 nm) makes it
208 tests on subcellular fractions enriched for clathrin-coated vesicles (CCVs) indicated that pip5k1 an
211 as sorting signals for packaging cargo into clathrin-coated vesicles (CCVs), and also facilitate dow
212 ptor protein-2 (AP2), a central component of clathrin-coated vesicles (CCVs), is pivotal in clathrin-
213 on of the entire population of intracellular clathrin-coated vesicles (CCVs), suggesting a more globa
214 its of the adaptor protein (AP) complexes of clathrin-coated vesicles (CCVs), together with an FKBP a
215 ty, and leads to presynaptic accumulation of clathrin-coated vesicles (CCVs)-all without decreasing G
220 region that is required for localization to clathrin-coated vesicles and contains a putative pleckst
221 tic function, including accumulation of free clathrin-coated vesicles and delayed vesicle reavailabil
223 strate that REEP6 is detected in a subset of Clathrin-coated vesicles and interacts with the t-SNARE,
224 closely related JC virus can enter cells in clathrin-coated vesicles and subsequently traffic to cav
226 ns were found to be associated with isolated clathrin-coated vesicles and to colocalize with clathrin
227 es strong evidence that, as in animal cells, clathrin-coated vesicles are a major means of internalis
230 ex-2 (AP-2) is required for the formation of clathrin-coated vesicles at the plasma membrane (PM).
231 or coat protein involved in the formation of clathrin-coated vesicles at the trans-Golgi network.
233 one, either auxilin or GAK, not only uncoats clathrin-coated vesicles but also acts as a chaperone du
238 sis occurs in plants, but the involvement of clathrin-coated vesicles has been unclear; a new study p
241 ile suggests the involvement of caveolae and clathrin-coated vesicles in the transcytotic process.
245 al triskelia, suggesting that disassembly of clathrin-coated vesicles may proceed through a partially
248 We propose that AP180 directs Vamp7B into clathrin-coated vesicles on contractile vacuoles, creati
249 le vacuoles offer a valuable system to study clathrin-coated vesicles on internal organelles within e
250 capture of the hydrolase-MPR complexes into clathrin-coated vesicles or transport carriers (TCs) des
252 n to localization on the plasma membrane and clathrin-coated vesicles that originated from the plasma
253 ls is to link ubiquitinated Notch ligands to Clathrin-coated vesicles through other Clathrin adapter
254 2 may stimulate Gap1 incorporation into AP-1/clathrin-coated vesicles to promote Gap1 trafficking fro
255 EEP6 in trafficking of cargo via a subset of Clathrin-coated vesicles to selected membrane sites in r
256 SNAREs, required for the fusion of endocytic clathrin-coated vesicles with endosomes and also for sub
257 f clathrin-coated profiles (in this case, of clathrin-coated vesicles) is observed at inhibitory syna
258 he plasma membrane, which invaginate to form clathrin-coated vesicles, a process that is well underst
259 o the adapter complex of the "inner" coat in clathrin-coated vesicles, and a heterotrimeric B-subcomp
260 ological structures, including mitochondria, clathrin-coated vesicles, and the actin cytoskeleton, in
261 kely determine the functional specificity of clathrin-coated vesicles, and together they control a mu
262 ed removal of receptors from the membrane in clathrin-coated vesicles, but it remains unclear how cla
263 e endophilins results in the accumulation of clathrin-coated vesicles, but not of clathrin-coated pit
264 uce ATP7B incorporation into AP-1-containing clathrin-coated vesicles, caused loss of TGN localizatio
266 rin heavy chain (CHC), the main component of clathrin-coated vesicles, is well characterized for its
267 etails governing the sorting of a SNARE into clathrin-coated vesicles, namely the direct recognition
268 ted structures, and is enriched in placental clathrin-coated vesicles, new possibilities for Ced-6/Gu
269 are important components for the cleavage of clathrin-coated vesicles, phagosomes, and mitochondria.
272 and the late secretory route are mediated by clathrin-coated vesicles, while the COat Protein I and I
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