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1 tasis and has long been used as a marker for clathrin mediated endocytosis.
2 l inhibited LGR5 internalization by blocking clathrin-mediated endocytosis.
3 trieved directly from the plasma membrane by clathrin-mediated endocytosis.
4 asma membrane to control Arf6 activation and clathrin-mediated endocytosis.
5 usion at the plasma membrane and enhance its clathrin-mediated endocytosis.
6 tion of the canonical eukaryotic pathway for clathrin-mediated endocytosis.
7 rrow-derived DCs was facilitated by TLR2 via clathrin-mediated endocytosis.
8 endophilin is a temporally regulated step in clathrin-mediated endocytosis.
9 Endophilin is a key protein involved in clathrin-mediated endocytosis.
10 -binding protein involved in a late stage of clathrin-mediated endocytosis.
11 (+) efflux and M1 protein internalization by clathrin-mediated endocytosis.
12 ytosis; moreover, it is 200-fold faster than clathrin-mediated endocytosis.
13 n depletion was observed to severely inhibit clathrin-mediated endocytosis.
14 minal results in removal of transporters via clathrin-mediated endocytosis.
15 s to assemble, because it is internalized by clathrin-mediated endocytosis.
16 eved from the presynaptic plasma membrane by clathrin-mediated endocytosis.
17 vels that internalizes lipoprotein cargo via clathrin-mediated endocytosis.
18 role for PtdIns(4,5)P2 in the regulation of clathrin-mediated endocytosis.
19 ration, dynamin assembly, and fission during clathrin-mediated endocytosis.
20 erent ligands, including HA and heparin, via clathrin-mediated endocytosis.
21 B1 surprisingly colocalized with the zone of clathrin-mediated endocytosis.
22 AP-2 is the core-organizing element in clathrin-mediated endocytosis.
23 ese proteins is amphiphysin 1, implicated in clathrin-mediated endocytosis.
24 polyhedrovirus (AcMNPV), enter host cells by clathrin-mediated endocytosis.
25 1 or 3 in either bulk endosome formation or clathrin-mediated endocytosis.
26 s heparan sulfate proteoglycans, and engages clathrin-mediated endocytosis.
27 iphysin/Rvs (BAR) domains play a key role in clathrin-mediated endocytosis.
28 e abundance of CFTR is in part controlled by clathrin-mediated endocytosis.
29 he HIV-1 replication cycle is independent of clathrin-mediated endocytosis.
30 n by N-BAR domain-containing proteins during clathrin-mediated endocytosis.
31 embrane, binds clathrin, and plays a role in clathrin-mediated endocytosis.
32 bunit of the AP-2 complex and is involved in clathrin-mediated endocytosis.
33 ntegrin redistribution is thought to require clathrin-mediated endocytosis.
34 te [PI(4,5)P(2)] plays a fundamental role in clathrin-mediated endocytosis.
35 ty-dependent bulk endocytosis (ADBE) but not clathrin-mediated endocytosis.
36 daptor and suggest a role for SAM domains in clathrin-mediated endocytosis.
37 sis, indicating that RRV enters into RFs via clathrin-mediated endocytosis.
38 TPase that catalyses membrane fission during clathrin-mediated endocytosis.
39 icate that RRV entry into RFs is mediated by clathrin-mediated endocytosis.
40 repulsion, which required internalization by clathrin-mediated endocytosis.
41 n of chemokines was reduced by inhibitors of clathrin-mediated endocytosis.
42 pression of myosin VI has no major impact on clathrin-mediated endocytosis.
43 that HPV16 is dependent on caveolin-1 after clathrin-mediated endocytosis.
44 GTPase in promoting membrane scission during clathrin-mediated endocytosis.
45 complex must be tightly regulated to promote clathrin-mediated endocytosis.
46 w that cell-surface EpoR is internalized via clathrin-mediated endocytosis.
47 n in cortical tension may spatially regulate clathrin-mediated endocytosis.
48 carried out by adaptor protein-2 (AP-2) via clathrin-mediated endocytosis.
49 e apical membrane of PTECs, which determines clathrin-mediated endocytosis.
50 ion of ankyrin-B (AnkB) in coupling GLUT4 to clathrin-mediated endocytosis.
51 tion of tissues through mechanoregulation of clathrin-mediated endocytosis.
52 rus G rapidly recycles from the membrane via clathrin-mediated endocytosis.
53 ted enhancement is impaired by inhibitors of clathrin-mediated endocytosis.
54 efine alternative mechanisms that facilitate clathrin-mediated endocytosis.
55 Fcho proteins during the earliest stages of clathrin-mediated endocytosis.
56 homology domain (ENTH) is a major player in clathrin-mediated endocytosis.
57 dophilin has been assigned as a component of clathrin-mediated endocytosis.
58 nase signalling pathways, but independent of clathrin-mediated endocytosis.
59 tis C virus (HCV) enters its target cell via clathrin-mediated endocytosis.
60 wth via its role as major adaptor module for clathrin-mediated endocytosis.
61 c cargoes, demonstrating its pivotal role in clathrin-mediated endocytosis.
64 icle recycling via interconnected actions in clathrin-mediated endocytosis and actin dynamics in neur
67 This protein complex is internalized via clathrin-mediated endocytosis and degraded in lysosomes,
70 ct rotational behaviors of nanocargos during clathrin-mediated endocytosis and intracellular transpor
73 enveloped RNA viruses that infect cells via clathrin-mediated endocytosis and low-pH-triggered fusio
76 pressed PI4P 5-kinase PIP5K6 is required for clathrin-mediated endocytosis and polar tip growth in po
78 ABAR trafficking, this pathway requires both clathrin-mediated endocytosis and protein kinase C to re
79 e a variety of cellular functions, including clathrin-mediated endocytosis and receptor signaling.
80 in Lowe syndrome patient fibroblasts impacts clathrin-mediated endocytosis and results in an endocyti
83 ic adaptors function redundantly to regulate clathrin-mediated endocytosis and to recruit components
84 ate of transferrin receptors internalized by clathrin-mediated endocytosis and, more broadly, a mecha
86 ced interferon signaling or an inhibition of clathrin-mediated endocytosis, and PKD inhibitors do not
87 t transferrin receptor (TfR), enter cells by clathrin-mediated endocytosis, and traffic with that rec
88 y and the nontranscriptional effect of SA on clathrin-mediated endocytosis are independent mechanisms
90 ssue of the JCI, Soda et al. have identified clathrin-mediated endocytosis as a central mechanism by
91 cellular internalization pathways identified clathrin-mediated endocytosis as the main route for eHEV
92 ith putative functions in general aspects of clathrin-mediated endocytosis as well as in the internal
93 mplex AP-2 (CLAP), an essential component of clathrin-mediated endocytosis, as binding partners of ot
94 xin-9 (SNX9) and dynamins, key components of clathrin-mediated endocytosis, as binding partners of XL
95 cyon on NRG1 cleavage and shedding depend on clathrin-mediated endocytosis, beta-secretase 1, and int
98 pinocytosis induced by Shh is independent of clathrin-mediated endocytosis but dependent on dynamin,
99 ized with transferrin, which enters cells by clathrin-mediated endocytosis, but not with cholera toxi
101 sorting proteins that are indispensable for clathrin-mediated endocytosis, but their precise functio
102 Both types of particles were internalized by clathrin-mediated endocytosis, but virions and ISVPs exh
103 lar mechanisms of cellular processes such as clathrin-mediated endocytosis, but, for quantitative mic
104 adaptors, stimulate ubiquitin-dependent and clathrin-mediated endocytosis by interacting with both a
106 ate receptor (GluR) abundance at synapses by clathrin-mediated endocytosis can control synaptic stren
107 measure three different endocytic pathways: clathrin-mediated endocytosis, caveolae-mediated endocyt
108 ize nutrients and cell surface receptors via clathrin-mediated endocytosis, cells assemble at least 5
109 f endocytosis (early endosome antigen 1) and clathrin-mediated endocytosis (clathrin heavy chain) dur
110 y homologous to PICALM and also functions in clathrin-mediated endocytosis, clathrin coat assembly pr
112 lectron microscopy indicated the presence of clathrin-mediated endocytosis (CME) and bulk endocytosis
113 niae invasion of HL-1 cells occurred through clathrin-mediated endocytosis (CME) and independently of
119 ve constructed here for proteins involved in clathrin-mediated endocytosis (CME) exhibits distinctive
120 hondrial uncoupler, is a potent inhibitor of clathrin-mediated endocytosis (CME) in different systems
121 axin-1, and superoxide dismutase-1) inhibits clathrin-mediated endocytosis (CME) in mammalian cells b
122 disputed the need for F-actin and Arp2/3 in Clathrin-Mediated Endocytosis (CME) in multicellular org
123 dings on the relative importance of actin in clathrin-mediated endocytosis (CME) in yeast versus mamm
128 e receptors at the postsynaptic membrane via clathrin-mediated endocytosis (CME) is a key mechanism f
131 amin 1 and 3 double knock-out neurons, where clathrin-mediated endocytosis (CME) is dramatically impa
146 echanisms by which epithelial cells regulate clathrin-mediated endocytosis (CME) of transferrin are p
147 We show that cargo internalized either via clathrin-mediated endocytosis (CME) or independently of
151 ceptor complexes) are taken into the cell by clathrin-mediated endocytosis (CME) utilizing a core mac
152 upling was less efficient but not abolished; clathrin-mediated endocytosis (CME) was severely impaire
153 tion is important for vesicle fission during clathrin-mediated endocytosis (CME), and it has been pro
155 disassembly factor, Auxilin2, which inhibits clathrin-mediated endocytosis (CME), impaired the AtPep1
156 demonstrated that Ca(2)(+) is important for clathrin-mediated endocytosis (CME), the mechanistic rol
166 Myosin 1E (Myo1E) is recruited to sites of clathrin-mediated endocytosis coincident with a burst of
168 thrin revealed the dynamics of EGF-activated clathrin-mediated endocytosis during internalization.
169 known mechanism for endocytosis in yeast is clathrin-mediated endocytosis, even though clathrin-inde
172 adhesion molecule A (JAM-A), virions undergo clathrin-mediated endocytosis followed by proteolytic di
173 rcoma-associated herpesvirus (KSHV) utilizes clathrin-mediated endocytosis for its infectious entry i
174 d the functional requirement of dynamin- and clathrin-mediated endocytosis for orthobunyavirus entry
176 s and RNAi specific for macropinocytosis and clathrin-mediated endocytosis had no effect on RVFV infe
178 LM (CALM) gene, whose product is involved in clathrin-mediated endocytosis, has been identified in tw
179 Given the diversity of proteins regulated by clathrin-mediated endocytosis, how this process may dist
180 otein that has been implicated in regulating clathrin-mediated endocytosis; however, a role for mAbp1
181 otassium channel TASK-1 are internalized via clathrin-mediated endocytosis in a cooperative manner.
182 The large GTPase dynamin plays a key role in clathrin-mediated endocytosis in animal cells, although
185 polyphosphoinositide phosphatase involved in clathrin-mediated endocytosis in conventional synapses.
188 protein that has been shown to play roles in clathrin-mediated endocytosis in HeLa cells and podocyte
191 ressed and catalyzes membrane fission during clathrin-mediated endocytosis in nonneuronal cells.
194 These findings emphasize the importance of clathrin-mediated endocytosis in regulating CTLA-4 traff
195 ified >50 proteins that assemble at sites of clathrin-mediated endocytosis in structures called actin
197 tide reduced run-down, suggesting a role for clathrin-mediated endocytosis in the regulation of the s
200 , dynamin and emphasize similarities between clathrin-mediated endocytosis in yeast and higher eukary
201 ved proteins appear sequentially at sites of clathrin-mediated endocytosis in yeast and mammals.
202 epsin is believed to play important roles in clathrin-mediated endocytosis, including generation of t
203 ncreased transferrin uptake occurred through clathrin-mediated endocytosis, indicating that nanocompo
204 nscytosis was significantly inhibited by the clathrin-mediated endocytosis inhibitor Pitstop 2 or siR
205 approximately 90 nm) makes it unlikely that clathrin-mediated endocytosis internalizes as a unit the
213 en these sorting motifs are mutated, or when clathrin-mediated endocytosis is inhibited, MHC-II-Ii co
214 f the structural changes taking place during clathrin-mediated endocytosis is largely based on electr
216 The role of clathrin light chain (CLC) in clathrin-mediated endocytosis is not completely understo
217 ip between constitutive and ligand-triggered clathrin-mediated endocytosis is only poorly characteriz
219 on mediated by NEDD4-2 or NEDD4-1 leading to clathrin-mediated endocytosis is the common mode of regu
223 protein that participates in early stages of clathrin-mediated endocytosis, is downregulated as well
226 of endocytosis have been proposed including clathrin-mediated endocytosis, kiss-and-run endocytosis,
228 s as an uncoating factor and that defects in clathrin-mediated endocytosis likely contribute to patho
229 s) adapter proteins couple components of the clathrin-mediated endocytosis machinery with regulators
230 ev-binding protein (Hrb), a component of the clathrin-mediated endocytosis machinery, as a critical m
231 ytic membranes, including vesicles formed by clathrin-mediated endocytosis, macropinosomes, and Rab5
232 own to play an important role in the control clathrin mediated endocytosis of EGFR and other cell sur
234 1), Ca(2+), or protein kinase C (PKC) impair clathrin-mediated endocytosis of EGFR, the formation of
236 , and APSD levels were blunted by inhibiting clathrin-mediated endocytosis of GluA2 subunits with the
237 f-concept genetic evidence that blocking the clathrin-mediated endocytosis of LGR5 could be used to p
238 protein (ARH) is well known for its role in clathrin-mediated endocytosis of low-density lipoprotein
240 n-2 sigma subunit (AP2sigma2) is pivotal for clathrin-mediated endocytosis of plasma membrane constit
241 or adjusting excitatory synaptic strength is clathrin-mediated endocytosis of postsynaptic glutamate
243 ctin-1 at the extracellular surface prevents clathrin-mediated endocytosis of ROMK1 and leads to accu
248 4-(N)-GemC18-SLNs entered tumor cells due to clathrin-mediated endocytosis of the 4-(N)-GemC18-SLNs i
251 hese manipulations was without effect on the clathrin-mediated endocytosis of transferrin receptor (T
253 ) and their subsequent reformation either by clathrin-mediated endocytosis or budding from bulk endos
254 e at the plasma membrane and not involved in clathrin-mediated endocytosis or endosomal recycling, as
258 Independently, AP2M, a core component in clathrin-mediated endocytosis, plays a role in the forma
259 eletal dynamics, phosphoinositide signaling, clathrin-mediated endocytosis, polarized blebbing, and e
261 Consistent with its role in participating in clathrin-mediated endocytosis, Rab14 localizes in nonlip
264 that PICALM, an adaptor protein involved in clathrin-mediated endocytosis, regulates APP internaliza
265 ension of BMPRII-LF accounted for its faster clathrin-mediated endocytosis relative to BMPRII-SF, acc
268 characterize a molecularly distinct mode of clathrin-mediated endocytosis requiring ligand ubiquityl
269 and the implications of the epsin's role in clathrin-mediated endocytosis resulting from the interpl
270 vesicles in primary cardiomyocytes, in which clathrin-mediated endocytosis seems to be the pre-domina
271 s of branched actin networks at the sites of clathrin-mediated endocytosis sheds light on the role of
272 Syp1p, SGIP1-alpha arrives early at sites of clathrin-mediated endocytosis, suggesting that Syp1p/Ede
274 to enter host cells through exploitation of clathrin-mediated endocytosis, the entry pathway for fil
276 pe are known to route surface receptors into clathrin-mediated endocytosis through interaction with t
278 of cell migration, filopodia formation, and clathrin-mediated endocytosis to show how a holistic vie
279 nociceptive neuronal excitability, the AP-2 clathrin-mediated endocytosis trafficking mechanism may
280 caveolar pathway in HPV16 infection because clathrin-mediated endocytosis typically does not lead to
282 te that TbetaRI is targeted for constitutive clathrin-mediated endocytosis via a di-leucine (Leu(180)
283 ined by confocal microscopy, indicating that clathrin-mediated endocytosis was not involved in THY-1-
286 architecture of the protein machinery during clathrin-mediated endocytosis was visualized using a new
289 c actin filaments are a crucial component of clathrin-mediated endocytosis when endocytic proteins ca
291 athrin-coated vesicles (CCVs), is pivotal in clathrin-mediated endocytosis, which internalizes plasma
292 e in a number of essential pathways, such as clathrin-mediated endocytosis, which involve dramatic me
293 on of transferrin, a process that depends on clathrin-mediated endocytosis, while its ablation by CRI
296 myosin VI knock-out) gives rise to defective clathrin-mediated endocytosis with shallow clathrin-coat
297 While TfR1 constitutively traffics through clathrin-mediated endocytosis, with or without ligand, t
300 f the three major internalization motifs for clathrin-mediated endocytosis (YXXPhi, [DE]XXXL[LI], or
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