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1 as an initiating event in the development of clear-cell renal carcinoma.
2 l in individual patients with organ-confined clear-cell renal carcinoma.
3 hypermethylation, is also common in sporadic clear cell renal carcinomas.
4 he development of the malignant phenotype of clear cell renal carcinomas.
5 ) disease and is implicated in most sporadic clear cell renal carcinomas.
6 e at risk of developing multiple independent clear cell renal carcinomas.
7 n Hippel-Lindau disease and in most sporadic clear cell renal carcinomas.
8 nic anhydrase-IX, which is over-expressed in clear-cell renal carcinomas.
9 evelopment of sporadic hemangioblastomas and clear-cell renal carcinomas.
10 ion of vascular endothelial growth factor by clear-cell renal carcinomas.
11 ectrum of malignant neoplasms: conventional (clear cell) renal carcinoma (11 of 11 cases), transition
12 ET with (124)I-cG250 can identify accurately clear-cell renal carcinoma; a negative scan is highly pr
17 s inactivated in both sporadic and inherited clear cell renal carcinoma associated with VHL disease.
20 is an archetypical tumor-initiating event in clear cell renal carcinoma (ccRCC) that leads to the act
27 HL) disease tumor suppressor gene in a human clear cell renal carcinoma cell line, UOK 121, that cont
29 nducible factor (HIF)-2alpha in VHL-positive clear cell renal carcinoma cells phenocopied loss of VHL
31 rt a case of a 56-year-old male patient with clear cell renal carcinoma confirmed on a histopathologi
32 Lindau gene inactivation is observed in most clear cell renal carcinoma, driving the malignant phenot
33 the loss of the VHL tumor suppressor gene in clear cell renal carcinoma for potential clinical benefi
34 e inactivated in VHL disease and in sporadic clear-cell renal carcinomas, has recently been shown to
35 se, small mitochondria have been observed in clear cell renal carcinomas known to have frequent VHL a
36 al-cord hemangioblastomas, retinal angiomas, clear-cell renal carcinoma, neuroendocrine tumors and cy
40 Introduction of wild type VHL transgene into clear cell renal carcinoma restored low level expression
41 e are also found in the majority of sporadic clear cell renal carcinoma, the most common malignant ne
42 dy chimeric G250 ((124)I-cG250) PET predicts clear-cell renal carcinoma, the most common and aggressi
43 1a in seven cases and pT1b in one case (five clear cell renal carcinoma, two chromophobe type, and on
45 ntral nervous system, pheochromocytomas, and clear cell renal carcinoma, which result from somatic in
46 X antigen, has shown the potential to target clear cell renal carcinoma with high sensitivity and spe
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