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1 and EWS/FLI1 fusion proteins associated with Clear Cell Sarcoma and Ewing's Sarcoma, respectively, we
2 otein in maintaining tumor cell viability of Clear Cell sarcoma and indicate that intracellular antib
3 a, salivary-duct cancer, ovarian cancer, and clear-cell sarcoma and among patients with colorectal ca
4 al sarcomas, round-cell/myxoid liposarcomas, clear-cell sarcomas and gastrointestinal stromal tumors
6 chromosomal translocation t(12;22) found in Clear Cell sarcoma (CCS) fuses the genes for Ewing's sar
10 81 inhibited the growth of a patient-derived clear cell sarcoma cell line whose oncogenic potential i
11 ions in primary melanocytes and melanoma and clear cell sarcoma cells through hypoxia inducible facto
13 in children, including renal-cell carcinoma, clear-cell sarcoma, (congenital) mesoblastic nephroma, r
15 of chemotherapy (including doxorubicin) for clear cell sarcoma improves recurrence-free survival.
16 or stages III to IV/FH WT or stages I to IV/clear cell sarcoma of the kidney (high-risk [HR] group)
17 combination chemotherapy for LR or HR WT or clear cell sarcoma of the kidney have equivalent 2-year
18 terminus of BCOR in 23 of 27 (85%) pediatric clear cell sarcomas of the kidney (CCSK) from two indepe
19 ), stages III to IV FH WT, or stages I to IV clear-cell sarcoma of the kidney (high-risk[HR]) were ra
20 al translocation associated with soft tissue clear cell sarcoma results in a chimeric protein EWS-ATF
21 ignant melanoma of soft parts or soft tissue clear cell sarcoma which shares t(12;22) chromosome tran
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