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1 decarboxylase (PvlArgDC) formed by the self-cleavage of a proenzyme into a 5-kDa subunit and a 12-kD
4 and P), have been developed for heterolytic cleavage of H2 into a proton and a hydride, akin to frus
6 poptosis is characterized by endonucleolytic cleavage of chromosomal DNA into an oligonucleosomal lad
9 L3 is necessary for Ca2+- and Mg2+-dependent cleavage of DNA into both oligonucleosomal and high mole
10 of various alpha-keto acids, as well as the cleavage of glycine into CO(2) and NH(3), with concomita
11 e current study was designed to test whether cleavage of CPAF into CPAFn and CPAFc is a physiological
13 We will argue that in meningococcal sepsis, cleavage of albumin into fragments by protease(s) occurs
14 U(L)28, U(L)32, and U(L)33, is required for cleavage of concatameric DNA into genomic lengths and fo
18 cofactors, which promoted factor I-mediated cleavage of C3b into iC3b as well as decay-accelerating
22 teine protease, apopain, as was indicated by cleavage of the proenzyme into its proteolytically activ
24 of SATB1 by disrupting its dimerization and cleavage of genomic DNA into loop domains to ensure rapi
25 rom the incubation with CMP, indicating that cleavage of pro-FD into mature FD by MASP-1 occurred on
27 required for DNA repair, and no irreversible cleavage of S-adenosylmethionine into methionine and 5'-
28 d enhanced activation of caspase-3 activity, cleavage of PARP into Mr 89,000 and 28,000 fragments, an
30 iruses by inducing nuclear lysis followed by cleavage of host cells into numerous anucleate vesicles
31 ant biochemical hallmark of apoptosis is the cleavage of chromatin into oligonucleosomal fragments.
32 n appears to cause apoptosis as evidenced by cleavage of DNA into oligonucleosomal-sized fragments, f
33 enzyme A (CoA)-dependent and MgATP-dependent cleavage of citrate into oxaloacetate and acetyl-CoA, re
34 oduction of insulin from proinsulin involves cleavage of intact proinsulin into proinsulin conversion
36 hese findings indicate that the differential cleavage of Notch into S2 and S3 products regulated by A
39 r H acts as a cofactor for factor I-mediated cleavage of C3b into the inactive form iC3b and thus pre
40 asmic reticulum into the Golgi apparatus, in cleavage of gPrEnv into the two envelope subunits (the s
43 otein precursor, is responsible for the self-cleavage of the precursor into two mature products, P150
44 not restrict viral gene expression; however, cleavage of viral DNA into unit-length genomes as well a
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