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1  activated by neurotransmitter released from climbing fibers ().
2 lutamate transporter 2 (VGluT2, to visualize climbing fibers).
3 llar cortex each receive input from a single climbing fiber.
4 l day (P) 21, most are contacted by a single climbing fiber.
5 inje cells which receive input from the same climbing fiber.
6 l Purkinje cells receive input from multiple climbing fibers.
7 grade tracers in the inferior olive to label climbing fibers.
8 ber filopodia and synaptic competition among climbing fibers.
9  spines in synaptic contact with parallel or climbing fibers.
10  class of precerebellar neurons that project climbing fibers.
11 in the context of the background activity of climbing fibers.
12 ides can be activated synchronously by their climbing fibers.
13 ns of variability driven by the parallel and climbing fibers.
14 ibers to Purkinje cells with the help of the climbing fibers.
15  structural plasticity by using, as a model, climbing fibers.
16  modulation of CSs and SSs depends on intact climbing fibers.
17  glutamate molecules in response to a single climbing fiber action potential.
18                                The region of climbing fibers activated by a localized peripheral stim
19                In addition, we observed that climbing fibers activated by periocular airpuffs also re
20   Here, we show that glutamate released from climbing fibers activates ionotropic and metabotropic re
21                                              Climbing fiber activation appears to reduce PKC expressi
22 tivity and changes in cerebellar output, and climbing fiber activation did not induce VOR-decrease le
23 hat result in synchronous (to within 1 msec) climbing fiber activation of Purkinje cells (complex spi
24 he present results indicate that synchronous climbing fiber activation of the left and right crus IIA
25 nely tuned timing between parallel fiber and climbing fiber activation.
26 re was no trial-by-trial correlation between climbing fiber activity and changes in cerebellar output
27 iated with the conditioned stimulus (CS) and climbing fiber activity associated with unconditioned st
28 an be insulated from ongoing fluctuations in climbing fiber activity by assuming that changes in pf*S
29                                   Cerebellar climbing fiber activity encodes performance errors durin
30 es the timing of both evoked and spontaneous climbing fiber activity in these cerebellar regions with
31 dy was to test the hypothesis that increased climbing fiber activity induced by ibogaine mediates exc
32                              This pattern of climbing fiber activity is markedly similar to the respo
33                During VOR-increase training, climbing fiber activity on one trial predicted changes i
34      The effect of visual stimulation on the climbing fiber activity was studied in 19 floccular Purk
35                        By evoking changes in climbing fiber activity, movement errors are thought to
36 sy fiber synapse is controlled by nucleus or climbing fiber activity, the circuit is unable to retain
37 ity, or there is a single mechanism tuned to climbing-fiber activity that follows activity in vestibu
38 n of the cerebellar cortex (particularly the climbing fiber afferents and their sole source, the infe
39 ng electrical stimulation of mossy fiber and climbing fiber afferents as CS and US, while alternating
40 three zones receiving their olivo-cerebellar climbing fiber afferents from parts of, respectively, th
41 e postsynaptic response to the activation of climbing fiber afferents, believed to be fundamental to
42 kinje cells may phasically control their own climbing fiber afferents.
43                                Activation of climbing fibers alone failed to induce the long-term dec
44                                      At both climbing fiber and inhibitory synapses onto PCs, we foun
45    Developmental and experimental studies of climbing fiber and mossy fiber connectivity in the cereb
46                   Conjunctive stimulation of climbing fiber and parallel fiber inputs results in long
47 nje cell (PC) synapse induced by conjunctive climbing fiber and PF stimulation in vivo.
48         IO neurons excite Purkinje cells via climbing fibers and depress their parallel fiber inputs.
49                      Multiple innervation by climbing fibers and enhanced parallel fiber synaptic cur
50  or anatomical evidence for synapses between climbing fibers and Golgi cells.
51  and the cellular regions of termination for climbing fibers and parallel fibers are well separated.
52 tiple climbing fiber innervation, we labeled climbing fibers and performed reconstructions of immunof
53 al control over the induction of learning by climbing fibers and Purkinje cells can expand the learni
54 g training, joint control of learning by the climbing fibers and Purkinje cells may expand the learni
55                                 We show that climbing fibers and some other cerebellar afferent fiber
56                 The interactions between the climbing fibers and the Purkinje cells were examined on
57 uantify the interactions between the olivary climbing fibers and the Purkinje cells when the cerebell
58 sion patterns that suggest a contribution of climbing fibers and their collaterals.
59     A delayed arborization of mutant olivary climbing fibers and their defective translocation from t
60 ligin isoforms differentially contributed to climbing-fiber and basket/stellate-cell synapse function
61 ow stimulus frequencies, and a comparison of climbing-fiber and simple-spike signals contained the in
62    Learning could be guided by comparison of climbing-fiber and vestibular signals at all stimulus fr
63                                 In contrast, climbing fibers are anticalretinin and anti-NR1 immunopo
64                                 We show that climbing fibers are distributed in narrow sagittal strip
65 rebellum at the time of, and at sites where, climbing fibers are eliminated.
66 rvation inhibits PKC expression and (2) that climbing fibers are essential for the observed changes i
67 lts indicate that instructive signals in the climbing fibers are not necessary for cerebellum-depende
68 y gated in vivo, even under conditions where climbing fibers are robustly activated by performance er
69                                              Climbing fibers are well suited to studies of recovery f
70                                      Because climbing fibers arise exclusively from the contralateral
71 ve used the innervation of Purkinje cells by climbing fibers as a model system to explore potential f
72 ron, Hashimoto et al. show that, by the time climbing fibers ascend the dendrites, the winner and los
73       CFRs reflect the discharge of a single climbing fiber at multiple synaptic sites on the proxima
74    Purkinje cells are innervated by multiple climbing fibers at birth but undergo an activity-depende
75                                  In mammals, climbing fiber axons compete for sole innervation at eac
76                  The inferior olive projects climbing fiber axons to cerebellar Purkinje neurons, whe
77           In contrast, CART was expressed in climbing fiber bands in all four transverse zones of the
78  We found that in mice CART was expressed in climbing fiber bands that generally corresponded to the
79 nje cells in anesthetized pigmented rabbits, climbing fiber burst patterns were investigated by deter
80 eform changes with the number of spikes in a climbing fiber burst, which depends on the phase of oliv
81  channel blockers and is mimicked by a brief climbing fiber burst.
82 sibility that signals are present within the climbing fiber bursts.
83  distal climbing-fiber synapses and weakened climbing-fiber but not parallel-fiber synapses, consiste
84 at did not elicit instructive signals in the climbing fibers, but nevertheless induced robust and con
85 al instructive signals carried by cerebellar climbing fibers, but with a stronger influence of the ba
86                                        Thus, climbing fibers can, in addition to modulation of their
87 ving excitatory synaptic input from a single climbing fiber (CF) and from approximately 200,000 paral
88 Here, we show that release of glutamate from climbing fiber (CF) axons produces AMPA receptor current
89 ns, but, regardless of activation frequency, climbing fiber (CF) coactivation provides an instructive
90 eatures include morphological alterations in climbing fiber (CF) innervation of Purkinje cells (PCs).
91              It is well established that the climbing fiber (CF) input to a cerebellar Purkinje cell
92                                              Climbing fiber (CF) input to the cerebellum is thought t
93 -term amplification of synaptic responses to climbing fiber (CF) or PF stimulation and enhance the am
94  of cerebellar function, coactivation of the climbing fiber (CF) synapse, which provides massive, inv
95 incident activity of parallel fiber (PF) and climbing fiber (CF) synapses causes a long-lasting decre
96                                              Climbing fiber (CF) synapses have a high probability of
97 sticity expressed at parallel fiber (PF) and climbing fiber (CF) synapses.
98 ays a predominant role in terminating DSE at climbing fiber (CF) to PC synapses, while both neuronal
99                    We find that the powerful climbing fiber (CF) to Purkinje cell synapse regulates t
100  receptors are postsynaptically expressed at climbing fiber (CF) to Purkinje cell synapses in mice, r
101                                              Climbing fiber (CF)-evoked calcium transients play a key
102 t glutamate transporter currents recorded at climbing fiber (CF)-PC synapses are absent in mice lacki
103  Purkinje cells (PCs), and prevented loss of climbing fiber (CF)-PC synapses in comparison to vehicle
104 ted the hypotheses that there are functional climbing fiber (CF)-Purkinje cell (PC) and parallel fibe
105                    Powerful synapses between climbing fibers (CF) and Purkinje cells are crucial to c
106  GluR1 was expressed postsynaptically at the climbing fibers (CF) synapse at early ages during Purkin
107                        Communication between climbing fibers (CFs) and molecular layer interneurons (
108 ons receive two major excitatory inputs, the climbing fibers (CFs) and parallel fibers (PFs).
109 f the C1q family of proteins, is provided by climbing fibers (CFs) and serves as a crucial anterograd
110 not detectable: both were mono-innervated by climbing fibers (CFs) extending along their well-develop
111 rve competitive elimination of supernumerary climbing fibers (CFs) in the cerebellum of live mouse pu
112                                   Cerebellar climbing fibers (CFs) provide powerful excitatory input
113  to cerebellar Purkinje cells (PCs) and from climbing fibers (CFs) to PCs is functional.
114 quired for bursting, activation of AMPARs by climbing fibers (CFs) was sufficient to trigger bursts.
115  main input synapses from parallel (PFs) and climbing fibers (CFs).
116 s that receive input from distinct groups of climbing fibers (CFs); however, the physiological proper
117 med two-photon in vivo imaging of cerebellar climbing fibers (CFs; the terminal arbor of olivocerebel
118 atory response, demonstrating the ability of climbing fiber collaterals to significantly excite CN ne
119      Our results challenge the view that the climbing fiber conveys an all-or-none signal to the cere
120                                     Although climbing fiber-dependent plasticity at pf*Pkj synapses a
121                                              Climbing fiber discharge [complex spikes (CSs)] increase
122 racking task in the monkey (Macaca mulatta), climbing fiber discharge dynamically controls the inform
123 ing in the granule cell population, allowing climbing-fiber-driven Purkinje cell learning at arbitrar
124 oded, we recorded the activity of individual climbing fibers during cerebellum-dependent eyeblink con
125 ells was found to inhibit parallel fiber and climbing fiber EPSCs for tens of seconds.
126 mbing fiber synapse, which in turn decreases climbing fiber-evoked dendritic calcium signals.
127 results in long-term potentiation (LTP) of a climbing fiber-evoked glutamate transporter current reco
128  kinetics in patches with the time course of climbing fiber-evoked responses indicates that the gluta
129 ng olivary microlesions reflects the loss of climbing fiber-evoked stellate cell discharge.
130 ayer interneurons, these results reveal that climbing fibers exert control over inhibition at both th
131 zation and observed its effects on mossy and climbing fiber extension and growth cone size in vitro.
132  fibers to Purkinje cells' synapse guided by climbing fibers, feedforward inhibition of Purkinje cell
133 ltering the excitatory input provided by the climbing fiber from the inferior olive, which evokes a p
134         PCs integrate two excitatory inputs, climbing fibers from inferior olive and parallel fibers
135 ound the following topographic relationship: climbing fibers from the caudal lateral mcIO were locate
136 t and right vermal lobule IX, which receives climbing fibers from the dorsomedial cell column.
137 ssy fibers and a teaching signal through the climbing fibers from the inferior olive.
138 ated in lateral P2+ and P2- ZII stripes, and climbing fibers from the middle lateral mcIO were locate
139 e located in P1+ and medial P1- ZII stripes; climbing fibers from the rostral lateral mcIO were locat
140 rograde inhibition of synapses received from climbing fibers, granule cell parallel fibers, and inhib
141                    Our findings identify the climbing fiber-->Purkinje neuron synapse as an important
142 th types of response were shown to be mainly climbing fiber in origin and therefore evoked by transmi
143 ng pathways, respectively, that terminate as climbing fibers in the "hindlimb-receiving" parts of the
144 s limits their ability to forward signals to climbing fibers in the cerebellar cortex.
145  activity-dependent pruning of supernumerary climbing fibers in the cerebellum.
146 ike the bank-gyrus subdivisions, most of the climbing fibers in the sulcus do not innervate the super
147 njunctive stimulation of parallel fibers and climbing fibers induced a long-term decrease (at least 1
148    This study tested the hypotheses (1) that climbing fiber innervation inhibits PKC expression and (
149    Together, the results suggest that single climbing fiber innervation of Purkinje cells is critical
150 ms, including the neuromuscular junction and climbing fiber innervation of Purkinje cells, are models
151 viously, we demonstrated similar deficits in climbing fiber innervation when analyzed on PN14.
152 naptic Purkinje cell responses, monosynaptic climbing fiber innervation, and cerebellar-dependent beh
153 hological correlate of differential multiple climbing fiber innervation, we labeled climbing fibers a
154 firing provide for a new hypothesis in which climbing fiber input adjusts the encoding of SS informat
155 n not only during complex spikes elicited by climbing fiber input but also with direct electrical sti
156 ts suggest a novel function of CSs, in which climbing fiber input dynamically controls the state of P
157                                              Climbing fiber input produces complex spike synchrony ac
158                             Stimulation of a climbing fiber input to cerebellar Purkinje cells was sh
159                                          The climbing fiber input to Purkinje cells acts as a teachin
160                   Loss of the inferior olive-climbing fiber input to the cerebellar cortex after trea
161                                          The climbing fiber input to the cerebellar cortex is thought
162                                          The climbing fiber input to the cerebellum from the inferior
163                                              Climbing fiber input to the cerebellum is believed to se
164         In the present study we examined the climbing fiber input to the medial half of folium IXcd,
165 neous dendritic calcium transients linked to climbing fiber input were observed in multiple neighbori
166  that shows paired pulse facilitation and to climbing fiber input with a large all-or-none AMPA-media
167 h the parallel fiber is coactivated with the climbing fiber input, but how order sensitivity is achie
168  Purkinje cell synapses under control of the climbing fiber input, which provides an error signal as
169 esence of cerebellar microzones organized by climbing fiber input.
170 in simple spike firing can be independent of climbing fiber input.
171 trikingly, such stimulations trigger delayed climbing-fiber input signals in the stimulated Purkinje
172  Theories of cerebellar learning assert that climbing fiber inputs control plasticity at synapses ont
173                                              Climbing fiber inputs to Purkinje cells are thought to b
174 nsory (SI) cerebral cortex and the timing of climbing fiber inputs to the lateral hemispheres of the
175                                              Climbing-fiber inputs appear to play a fast and primary,
176 ely depressed in Purkinje cells that receive climbing-fiber inputs from the instruction.
177 tion of pursuit and therefore do not receive climbing-fiber inputs related to the instruction, simple
178  to a large calcium release transient if the climbing fiber is activated up to 100 ms before or up to
179 ndicates that PF-PC LTD under control of the climbing fibers is not required for general motor adapta
180 ly robust putative error signals in the same climbing fibers: learned increases and decreases in the
181                  Moreover, ethanol inhibited climbing fiber long-term depression, a form of synaptic
182 er-related pattern formation, elimination of climbing fibers, long-term potentiation, long-term depre
183 ggest that interneurons driven by vestibular climbing fibers may determine SS modulation.
184 rning, MCS and ZCS cells developed increased climbing fiber (MCS) or parallel fiber (ZCS) input durin
185 dritic spines, we find that pairing IP3 with climbing fiber-mediated calcium entry leads to a large c
186 y learned movement will generate a series of climbing fiber-mediated corrections.
187  oriented modules made up of Purkinje cells, climbing fibers, molecular layer interneurons, and cereb
188 rally (mossy fiber neurons) or intramurally (climbing fiber neurons).
189 re misspecified to dI4 interneurons, and the climbing-fiber neurons (inferior olive nucleus) are comp
190                                   Vestibular climbing fibers not only evoke low-frequency CFRs, but a
191 , with accompanying later enlargement of the climbing fiber nucleus and reductions in mossy fiber nuc
192 n these experiments we demonstrated that the climbing fibers of all four areas, i.e., the left and ri
193 ns caused by cerebellar afferents (e.g., the climbing fibers of the inferior olive) is a critical fac
194 g, we analyzed excitatory synapses formed by climbing fibers on Purkinje cells in cerebellum and inhi
195 kinje cell would typically be activated by a climbing fiber only once.
196           Following glutamate spillover from climbing fibers or application of CNQX, evoked GABA rele
197 d, instructive signals carried by either the climbing fibers or Purkinje cell simple spikes may be su
198 vironment along translational axes and their climbing fibers originate in the lateral half of the med
199 nferior olivary nuclei from which vestibular climbing fibers originate; the beta-nucleus and dorsomed
200 ibers emanating from the pontine nucleus and climbing fibers originating from the inferior olive) can
201  and timing in the cerebellar cortex via the climbing fiber pathway, but direct characterization of t
202 calcium spikes, which was modulated by a non-climbing fiber pathway.
203 ebellar theories assume that mossy fiber and climbing fiber pathways carry information from different
204 ecording techniques that the mossy fiber and climbing fiber pathways converge in cerebellar cortex.
205 in access to the cerebellum is via ascending climbing fiber pathways.
206 ty of fluo-4 dextran by measuring Ca(res) at climbing fiber presynaptic terminals.
207 ral to Math1 yet within Wnt1(+) territory, a climbing fiber primordium dominated by Ngn1-expressing c
208 e involved in eye movement control via their climbing fiber projection to the cerebellar flocculus, P
209 s control limb and digit movements via their climbing fiber projection to the lateral cerebellar hemi
210 ial relationship between the mossy fiber and climbing fiber projections to crus IIa in the lateral he
211 g a crucial time in postnatal development of climbing fiber-Purkinje cell connectivity suggest a role
212 bdivisions; however, the basic properties of climbing fiber-Purkinje cell EPSCs such as kinetics, amp
213 , the establishment of the proper pattern of climbing fiber-Purkinje cell innervation, and induction
214               Here we report that LTD at the climbing fiber-Purkinje cell synapse in rat cerebellum w
215 eport that tetanic stimulation of cerebellar climbing fiber-Purkinje cell synapses results in long-te
216 the result of glutamate diffusing out of the climbing fiber-Purkinje cell synaptic clefts ().
217                      At the same time, adult climbing fibers react by sprouting new branches through
218 eptor-mediated quantal events resulting from climbing fiber release are observed in Bergmann glial ce
219 t, we propose that exocytosis can occur from climbing fiber release sites located directly across fro
220 f cortical input results in a lengthening of climbing fiber response latency to peripheral stimuli.
221 stimulation of SI results in a shortening of climbing fiber response latency.
222 urkinje cells whose low-frequency interburst climbing fiber response was modulated by movement about
223        The earliest learning occurs when one climbing-fiber response to a learning instruction causes
224   Purkinje cells have two action potentials: Climbing fiber responses (CFRs) and simple spikes (SSs).
225                                              Climbing fiber responses in cerebellar Purkinje cells ar
226 all stimulus frequencies tested, but only if climbing fiber responses were compared with the vestibul
227 re activated by voltage waveforms taken from climbing fiber responses, suggesting that they help shap
228 inje cells showed characteristic all-or-none climbing fiber responses.
229                   Repetitive stimulations of climbing fibers resulted in a NMDAR-dependent reduction
230                                              Climbing fibers run in the horizontal plane and terminat
231 nodulus, while leaving intact the vestibular climbing fiber signal from the contralateral inferior ol
232  postcomplex spike pause, a component of the climbing fiber signal in Purkinje neurons, and show that
233 us of the inferior olive reduced a modulated climbing fiber signal to the contralateral uvula-nodulus
234                                We found that climbing fibers signaled both the unexpected delivery an
235                      This reduced vestibular climbing fiber signaling to the contralateral folia 8-10
236 transmission in these pathways controls when climbing fiber signals can modify cerebellar activity.
237 cy of vestibularly modulated mossy fiber and climbing fiber signals in evoking CFRs and SSs in Purkin
238                     It is suggested that the climbing fiber signals may act as a molecular 'filter' o
239 al correlation of metabotropic responses and climbing fiber signals produces persistent phosphorylati
240 icated that the comparison of vestibular and climbing-fiber signals across the 100 msec delay must be
241 igh-frequency burst (approximately 500/s) of climbing fiber spikes.
242              Complex spikes were elicited by climbing fiber stimulation, and their somatic waveforms
243 otentials remained just above two spikes per climbing fiber stimulation, but the instantaneous freque
244 own that Golgi cell activity is regulated by climbing fiber stimulation, yet there is little function
245 f excitation and inhibition following single climbing fiber stimulation.
246 f the Bergmann glial AMPA response evoked by climbing fiber stimulation.
247  neurons depends on coincidence detection of climbing fiber stimulus evoking extracellular calcium fl
248 naptic reuptake of glutamate released at the climbing fiber synapse (4).
249 tes suggests that postsynaptic uptake at the climbing fiber synapse removes at least 22 percent of re
250                  Depression dominated at the climbing fiber synapse, facilitation was prominent at th
251 decreasing the probability of release at the climbing fiber synapse, which in turn decreases climbing
252 nexpectedly essential for maintaining normal climbing-fiber synapse numbers.
253 2 occurs at both parallel fiber synapses and climbing fiber synapses early in development but is rest
254 delta receptors are transiently expressed in climbing fiber synapses in the second postnatal week.
255 synapses; however, PSD-93 also is present at climbing fiber synapses of the adult rat, where delta2 i
256 phorabphilin is specifically enriched in the climbing fiber synapses of the cerebellar cortex.
257 ted the glutamate concentration transient at climbing fiber synapses on Purkinje cells by measuring t
258 leading to sustained release of glutamate at climbing fiber synapses on Purkinje cells.
259 and AMPA glutamate receptors at parallel and climbing fiber synapses on the developing Purkinje cells
260 st, delta receptors were found to be high at climbing fiber synapses only at P10 and P14.
261 the inferior olive (IO) that in turn produce climbing fiber synapses onto Purkinje cells.
262                In cerebellar Purkinje cells, climbing fiber synapses provide an instructive signal fo
263 postnatal ages (P2 and P5) and was higher in climbing fiber synapses than in parallel fiber synapses
264 ence on the associative activation of PF and climbing fiber synapses.
265 ebellar Purkinje cells caused loss of distal climbing-fiber synapses and weakened climbing-fiber but
266 airments in their distribution and function (climbing-fiber synapses) to large decreases in synapse n
267  antiexcitotoxic cellular response to strong climbing fiber synaptic activation, as occurs following
268 hysiology and function of the inferior olive/climbing fiber system and are interpreted to provide add
269 e primary role of the inferior olive and the climbing fiber system in timing is the encoding of tempo
270 ngs elucidate an unappreciated aspect of the climbing fiber teaching signal, and are consistent with
271 ffect of varying the number of pulses in the climbing fiber teaching signal.
272 e of glutamate simultaneously at hundreds of climbing fiber terminals distributed widely over the sur
273 set, suggesting that glutamate released from climbing fiber terminals escapes synaptic clefts and rea
274 rmine whether AT2 receptors are expressed in climbing fiber terminals which arise to the molecular la
275                                              Climbing fibers terminate only on smooth dendrites near
276                        Within the rat AZ/PZ, climbing fibers terminated selectively within the dendri
277 ls in the sulcus were innervated by multiple climbing fibers than in the gyrus or bank subdivisions;
278  stronger influence of the background on the climbing fibers than on learning.
279 f the inferior olivary nucleus (IO) form the climbing fibers that excite Purkinje cells of the cerebe
280 ts of inferior olivary nuclei, the source of climbing fibers that innervate Purkinje cells.
281 s: complex spikes (CSs) are evoked by single climbing fibers that originate from the contralateral in
282 ivate the inferior olive which gives rise to climbing fibers that project directly onto Purkinje cell
283  afferent mossy fibers and tertiary afferent climbing fibers that project to the uvula-nodulus (folia
284                                              Climbing fibers, the projections from the inferior olive
285                                           At climbing fiber to Purkinje cell (PC) synapses in cerebel
286 ses in rat brain slices at 34 degrees C: the climbing fiber to Purkinje cell synapse, the parallel fi
287  dynamics of recovery from depression at the climbing fiber to Purkinje cell synapse, where marked pr
288 determine the mechanism of depression at the climbing fiber to Purkinje cell synapse.
289 rise to these characteristic features at the climbing fiber to Purkinje cell synapse.
290 are transmitted by mossy/parallel fibers and climbing fibers to cerebellar Purkinje cells that acquir
291         Our data suggest that the ability of climbing fibers to induce plasticity can be dynamically
292 s in inferior olivary (IO) neurons that send climbing fibers to innervate cerebellar Purkinje cells f
293 he next trial, and optogenetic activation of climbing fibers to mimic their encoding of performance e
294 rior olivary neurons and transported through climbing fibers to the molecular layer of the cerebellar
295                                           At climbing fiber-to-Purkinje cell synapses, the number of
296                                              Climbing fibers typically fire approximately once a seco
297 ia cells encase synapses between presynaptic climbing fiber varicosities and postsynaptic Purkinje ce
298 llins as neurexin ligands for the excitatory climbing-fiber versus parallel-fiber synapses.
299                                  Patterns of climbing-fiber, vestibular, and Purkinje cell simple-spi
300 in paired-pulse depression at the cerebellar climbing fiber, where glial ensheathment of synapses is

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