ライフサイエンスコーパス: clinical deterioration

1 ictors of waitlist mortality (or removal for clinical deterioration).

2 history of 2 weeks of symptom worsening, or clinical deterioration).

3 , consciousness level, and other evidence of clinical deterioration.

4 of all ages and frequently leads to a rapid clinical deterioration.

5 , persistent bacillary load, and progressive clinical deterioration.

6 s never an option because of the rapidity of clinical deterioration.

7 tional imaging in the presence of continuing clinical deterioration.

8 cute problem, and it did not seem to precede clinical deterioration.

9 individuals at the greatest risk of serious clinical deterioration.

10 focused on prespecified measures of nonfatal clinical deterioration.

11 elin is limited, contributing to progressive clinical deterioration.

12 nal critical care resources to patients with clinical deterioration.

13 on of patients removed from the waitlist for clinical deterioration.

14 ers, 2206 patients were removed for death or clinical deterioration.

15 te a protocol designed to detect evidence of clinical deterioration.

16 y escalation of immunosuppression to prevent clinical deterioration.

17 with appropriate surveillance for unexpected clinical deterioration.

18 illacea did not appear to be associated with clinical deterioration.

19 es and more than 8 hours before the onset of clinical deterioration.

20 e are merely plausible surrogates for future clinical deterioration.

21 n case of persistent ST-segment elevation or clinical deterioration.

22 thema nodosum leprosum) that result in major clinical deterioration.

23 Services interventions to reduce this early clinical deterioration.

24 er unsuspected CHF in patients with COPD and clinical deterioration.

25 line in exercise capacity may be a marker of clinical deterioration.

26 s of age, followed by steady or intermittent clinical deterioration.

27 as hemodynamic aberrancies may cause severe clinical deterioration.

28 at risk for worsening disease in advance of clinical deterioration.

29 By 48 hrs, 36 (68%) patients had manifested clinical deterioration.

30 ion with a specific p38 MAPK inhibitor halts clinical deterioration.

31 ncreasing body temperature may be related to clinical deterioration.

32 ; this shift heralds CD4+ cell depletion and clinical deterioration.

33 relationship between insulin deficiency and clinical deterioration.

34 Conversely, of true removals for clinical deterioration, 154 of 612 (25.2%) were misclass

35 oved from the waitlist due to death (331) or clinical deterioration (2194) since 2002.

36 nd half as likely to be removed for death or clinical deterioration (9.2% vs 18.2%, P = 0.046).

37 left ventricular dysfunction were at risk of clinical deterioration after digoxin withdrawal despite

38 tprocedure blush reduction are predictors of clinical deterioration after embolization.

39 Of the 12 patients, three experienced acute clinical deterioration after embolization.

40 ) higher rate of death or removal because of clinical deterioration and a 42% (95% CI, 10-85%) higher

41 of pulmonary hypertension is associated with clinical deterioration and a substantially increased mor

42 ogressors), and five were selected for rapid clinical deterioration and CD4 count decline (rapid prog

43 and right heart failure have a high risk of clinical deterioration and death during or soon after en

44 one day prior to infection results in rapid clinical deterioration and death of mice within 6 days p

45 drugs, and is among the events that predict clinical deterioration and death.

46 left ventricular function and can result in clinical deterioration and even death.

47 Responders were patients who experienced no clinical deterioration and had improvement or stabilizat

48 in bone marrow-derived cells caused delayed clinical deterioration and hemorrhagic conversion of the

49 Occurrence of AF in PH indicates clinical deterioration and more advanced disease.

50 Dexamethasone therapy prevented clinical deterioration and reduced the need for blood tr

51 Recurrent clinical deterioration and repeat medical emergency team

52 of patients (n = 337) experienced recurrent clinical deterioration and repeat medical emergency team

53 Patients more likely to experience recurrent clinical deterioration and repeat medical emergency team

54 pse, relapse or study discontinuation due to clinical deterioration, and acute exacerbation.

55 nsitivity, diarrhea and vomiting, skin rash, clinical deterioration, and patient's wishes in one pati

56 ory of 2 weeks of clinical deterioration, or clinical deterioration as judged by the clinician.

57 strated improvement or stabilization without clinical deterioration, as compared with 65 of 146 in th

58 of symptomatic improvement and lower risk of clinical deterioration, as evaluated by changes in the N

59 ear the most striking finding was that while clinical deterioration continued in the placebo-treated

60 IM) versus virus persistence and progressive clinical deterioration (CVB3-PERS).

61 failure by day 8 after enrolment, defined as clinical deterioration, development of a serious adverse

62 failure by day 8 after enrolment, defined as clinical deterioration, development of a serious adverse

63 Of the 122 enrolled, four were excluded (clinical deterioration, diagnosis of cystic fibrosis, pr

64 le to identify patients at risk of recurrent clinical deterioration following medical emergency team

65 of 3,200 patients with and without recurrent clinical deterioration following medical emergency team

66 se in probability that a patient will have a clinical deterioration from sepsis within 24 h.

67 at our institution after rapidly progressive clinical deterioration from this infection, with necroti

68 ity of heart failure and reduces the risk of clinical deterioration, hospitalization, and other serio

69 We present an example of severe clinical deterioration in a patient with HCM due to supe

70 f impedance cardiography (ICG) in predicting clinical deterioration in ambulatory patients with heart

71 tablishment of chronic infection and ensuing clinical deterioration in CF.

72 he risk of virus persistence and progressive clinical deterioration in CVB3 cardiomyopathy.

73 Clinical deterioration in human immunodeficiency virus t

74 Antiglutamatergic treatment reduced clinical deterioration in moderate-to-severe Alzheimer's

75 , in the form of LTP, is crucial to contrast clinical deterioration in MS.

76 odulatory therapy, resulting in irreversible clinical deterioration in patients with MMP.

77 onsidered in the differential diagnosis of a clinical deterioration in posttransplant patients treate

78 own about the effect of drugs on the risk of clinical deterioration in surviving patients.

79 Failure to detect clinical deterioration in the hospital is common and ass

80 We looked at evidence of clinical deterioration in the Metoprolol CR/XL Randomize

81 This suggests that clinical deterioration in these patients may occur by a

82 The introduction of a two-tier response to clinical deterioration increased ICU admissions triggere

83 me of early treatment failure defined as [1] clinical deterioration indicated by development of shock

84 -hippocampal markers to predict the earliest clinical deterioration is less clear.

85 coccal meningitis (CM) frequently experience clinical deterioration, known as cryptococcosis-associat

86 infections may be accompanied by (transient) clinical deterioration, known as paradoxical reaction.

87 disease process characterized by progressive clinical deterioration leading rapidly to cardiac death

88 ed at 20 hours and at discharge and included clinical deterioration, need for craniotomy, and death.

89 In patients with PH and AF, parameters of clinical deterioration (NYHA/WHO functional class, 6-min

90 a step-wise fashion up to 30 cm H2O or until clinical deterioration occurred.

91 ant parameter in the simulation of long-term clinical deterioration of bond integrity.

92 HF) device diagnostic information to predict clinical deterioration of HF in patients with systolic l

93 however, and that it may play a role in the clinical deterioration of patients with congestive heart

94 Is late clinical deterioration often or invariably tied to the i

95 curacy of different techniques for detecting clinical deterioration on the wards in a large, multicen

96 on Rating Scale with a history of 2 weeks of clinical deterioration, or clinical deterioration as jud

97 ed iloprost, due to lower airway reactivity, clinical deterioration, or death.

98 iting for ReTx with fewer removed for death, clinical deterioration, or improvement.

99 of patients who died, and an apparent acute clinical deterioration preceded death in 47% of these pa

100 eart failure is characterized by progressive clinical deterioration reflected in frequent hospital ad

101 e 68 listed patients in whom the rapidity of clinical deterioration, reflected in increasing APACHE I

102 rt failure outpatients at increased risk for clinical deterioration remains a critical challenge, wit

103 Once stability was achieved, clinical deterioration requiring intensive care, coronar

104 Clinical deteriorations result in increases to the LAS;

105 Further work-up after clinical deterioration revealed an increase in the lesio

106 Clinical deterioration, side effects, and poor complianc

107 e learning methods more accurately predicted clinical deterioration than logistic regression.

108 ion but did not significantly reduce cardiac clinical deterioration through 5 years of follow-up.

109 more likely than patients without recurrent clinical deterioration to be subsequently admitted to IC

110 Secondary outcomes included clinical deterioration (transfer to ICU, initiation of v

111 every 2 weeks until disease progression and clinical deterioration, unacceptable toxicity, or other

112 After clinical deterioration using meropenem monotherapy, trea

113 truly removed from the waitlist for death or clinical deterioration were misclassified as "other." Th

114 Patients with recurrent clinical deterioration were more likely than patients wi

115 physiologic signatures may be present before clinical deterioration, when treatment might be more eff

116 f cumulative treatment failure at day 14 was clinical deterioration within 48 hours of enrollment (33

117 variables, and found that memantine reduced clinical deterioration without significant adverse effec