ライフサイエンスコーパス: clinical form

1 ss, with type 2 (USH2) being the most common clinical form.

2 same geographic location, regardless of the clinical form.

3 The lymphocutaneous clinical form (54%) was the most frequent.

4 eptide, and there was no association between clinical forms and positive results of serological assay

5 major variable shared among these different clinical forms; and epidemiologic data, studies in anima

6 Pre-clinical and clinical use of beta-lap (clinical form, ARQ501 or 761) is hampered by poor pharma

7 P may present as acute, subacute, or chronic clinical forms but with frequent overlap of these variou

8 this study, we aimed to: 1) determine if the clinical forms can be differentiated at the transcriptio

9 ry bowel disease (IBD) presents as two major clinical forms, Crohn's disease (CD) and ulcerative coli

10 y susceptibility genes for both of its major clinical forms, Crohn's disease and ulcerative colitis.

11 Two distinct clinical forms have been described as the following phen

12 y prevalent in modern society and is, in its clinical form, insufficient sleep syndrome, one of the m

13 art disease and those with the indeterminate clinical form of Chagas disease, respectively.

14 ing further evidence that CAM-DR is a viable clinical form of drug resistance.

15 fects, HPP causes dental defects, and a mild clinical form of HPP, odontohypophosphatasia, features o

16 The most prevalent clinical form of PAP is autoimmune PAP (aPAP) whereby Ig

17 orrelates with and accurately classifies the clinical form of the disease.

18 Several clinical forms of alpha-thalassaemia and beta-thalassaem

19 asthma (IIA) has been used to denote various clinical forms of asthma related to irritant exposure at

20 ecular basis for the embryonic and perinatal clinical forms of biliary atresia is largely undefined.

21 associated with the indeterminate or cardiac clinical forms of Chagas disease and whether IL-17 expre

22 in the prognosis of digestive pathologies of clinical forms of Chagas disease.

23 re intracellular parasites that cause unique clinical forms of cutaneous leishmaniasis.

24 xpression profiles may be useful in defining clinical forms of disease and providing insights into th

25 progression is not uniform; it may relate to clinical forms of disease and/or staging of liver pathol

26 n CD8(+) T cells from CCPs with asymptomatic clinical forms of disease.

27 lationship between these disorders and other clinical forms of frontotemporal lobar degeneration.

28 ts in ganglioside catabolism causing various clinical forms of GM1- and GM2-gangliosidoses have long

29 ementia, blindness) and different protracted clinical forms of GM1- and GM2-gangliosidoses.

30 era of 16 patients presenting with different clinical forms of histoplasmosis were monitored at regul

31 01, have the strongest associations with all clinical forms of IIM in white patients.

32 ciated herpesvirus (KSHV) is linked with all clinical forms of Kaposi sarcoma and several lymphoproli

33 ntified gammaherpesvirus associated with all clinical forms of Kaposi's sarcoma (KS), body-cavity-bas

34 s (KSHV) is consistently associated with all clinical forms of KS, and in vitro infection of dermal m

35 on mechanism of protection against different clinical forms of leishmaniasis.

36 ite Leishmania amazonensis can cause diverse clinical forms of leishmaniasis.

37 era from infected individuals with different clinical forms of schistosomiasis recognized SmCT-SOD bu

38 ociations were analyzed separately for three clinical forms of spastic cerebral palsy (hemiplegia, di

39 ritical role in the development of different clinical forms of tegumentary leishmaniasis.

40 gate whether leprosy patients with different clinical forms of the disease can be categorized based o

41 biologic correlates with the genetic and/or clinical forms of this disease.

42 M. tuberculosis infection involves different clinical forms of tuberculosis, such as pulmonary tuberc

43 31 people with MSA of the three different clinical forms (parkinsonian, cerebellar, and mixed), 19

44 Tetanus can present with unusual clinical forms; therefore, the diagnosis and management

45 brainstem is preferentially involved in both clinical forms, though there are no clinical signs of br

46 the lysosome give rise to juvenile and adult clinical forms with a wide range of clinical symptomatol