ライフサイエンスコーパス: clinical marker

1 us must be considered when CTpr is used as a clinical marker.

2 ated hemoglobin (HbA1c) remains the standard clinical marker.

3 disease and opens up possibilities for novel clinical markers.

4 stenosis was seen with increasing numbers of clinical markers.

5 tocol, which uses death certificate data and clinical markers.

6 nd differentially expressed genes in WAT and clinical markers.

7 of spinal cord atrophy and correlations with clinical markers.

8 he rich information contained in established clinical markers.

9 through the combination of both genetic and clinical markers.

10 Current clinical markers allow for more individualized risk stra

11 6.5-68.3), far higher than either genetic or clinical markers alone.

12 These findings validate HPV persistence as a clinical marker and endpoint.

13 Olfactory impairment may be an important clinical marker and predictor of these conditions and ma

14 of 0.086 (P = 0.02) that was independent of clinical markers and adjuvant therapy.

15 can be identified early by a combination of clinical markers and etiologic factors.

16 Its relationship with standard clinical markers and long-term adverse events in chronic

17 The ALFSG index, a combination of clinical markers and measurements of the apoptosis bioma

18 of AKI through the combined use of available clinical markers and novel inflammatory, injury, and rep

19 er methylation was inversely correlated with clinical markers and symptoms associated with PTSD.

20 lements, vitamins, hormones, selenoproteins, clinical markers, and perfluorinated compounds (PFCs), w

21 The diagnosis is difficult because clinical markers are often lacking, and symptoms usually

22 Periodontopathic clinical markers are poorly understood in the pediatric

23 Risk factors and clinical markers associated with ScGVHD remain largely u

24 Patients with primary care also had better clinical markers at diagnosis.

25 anisms that can be predicted by using simple clinical markers at the time of the first severe wheezin

26 true clinical significance of this important clinical marker can be clearly established.

27 positive individuals show a deterioration in clinical markers consistent with the prodrome of PD.

28 Spirometry is routinely used as a clinical marker for assessing pulmonary allograft functi

29 breast cancer development, and as a primary clinical marker for breast cancer prognosis, has been we

30 Serum CPK may be a useful clinical marker for choline deficiency in humans.

31 We found no definitive clinical marker for differentiating class 1 and class 2

32 HBV e-antigen (HBeAg), a clinical marker for disease severity, is a soluble varia

33 termine the utility of this measurement as a clinical marker for future coronary events and long-term

34 ese findings suggest that PR status may be a clinical marker for genetic subgroups of meningioma and

35 As a clinical marker for heart failure, BNP is limited by con

36 Gamma-H2AX may be a useful clinical marker for monitoring the efficacy of aminoflav

37 nduced cutaneous tissue injury, represents a clinical marker for non-melanoma skin cancer (NMSC) risk

38 aging and has the potential to be used as a clinical marker for progression from mild cognitive impa

39 linked with mixed manic states and may be a clinical marker for recurrent dysphoric mania.

40 suicidal self-injury prior to treatment is a clinical marker for subsequent suicide attempts and shou

41 rmore, proepithelin may prove to be a useful clinical marker for the diagnosis of prostate tumors.

42 autoantibodies represent a highly promising clinical marker for the identification of SS.

43 We carried out a study to identify the clinical markers for patients at high risk of developing

44 tiators of metastasis and potentially useful clinical markers for patients with cancer.

45 illebrand disease (VWD) in the Molecular and Clinical Markers for the Diagnosis and Management of Typ

46 xonomy of cancers and finding biological and clinical markers from microarray experiments is problema

47 these molecular markers, along with standard clinical markers, has been developed and internally vali

48 The expression levels of the standard clinical markers HER2, estrogen receptor (ER), progester

49 DNA methylation is potentially an important clinical marker in cancer molecular diagnostics.

50 rodromal AD, and may be a potentially useful clinical marker in identifying MCI patients who are most

51 d, the five AAs were not as good as existing clinical markers in differentiating diabetic patients fr

52 global imaging metrics outperformed baseline clinical markers in predicting outcome, including SUVtot

53 Clinical markers in the peripheral blood guide the treat

54 Main Outcomes and Measures: Assessment of clinical markers included the following standardized sca

55 uced mortality and blocks the development of clinical markers including hypotension and hypothermia.

56 e identification rates to selected objective clinical markers, including positive blood cultures, vas

57 after TOF repair and were related to adverse clinical markers, including ventricular dysfunction, exe

58 lity/anger during MDEs is a highly prevalent clinical marker of a more severe, chronic, and complex d

59 degraded by plasmin, is the most widely used clinical marker of activated blood coagulation.

60 of conditioned threat responses is a robust clinical marker of anxiety disorders.

61 ultimodal genomics data with a commonly used clinical marker of cardiovascular disease.

62 As a powerful clinical marker of circulatory collapse, the fast Fourie

63 commend any of these biomarkers as a routine clinical marker of CVD in children.

64 twisting rate (UTR) has been introduced as a clinical marker of diastolic function.

65 rculating MMP-9 level could be utilized as a clinical marker of disease activity.

66 ther validate spreading depolarizations as a clinical marker of early brain injury and establish a cl

67 Endothelial dysfunction is an established clinical marker of early coronary artery disease and has

68 make CRG-L2 an excellent candidate as a new clinical marker of HCC.

69 tively assessed plasma BNP as a quantitative clinical marker of heart failure severity in 107 consecu

70 re hypothesized as a requisite mechanism and clinical marker of infarct development.

71 The WBC count is a clinical marker of inflammation and a strong predictor o

72 physiologic reserve that may be an important clinical marker of longer surgical LOS.

73 Lower brainstem dysfunction, a clinical marker of neurologic injury not captured by the

74 ALT levels may be a useful clinical marker of reexposure.

75 We provide evidence that the clinical marker of resistance (delayed parasite clearanc

76 Since there is no reliable clinical marker of that response we recommend an indomet

77 Since there is no reliable clinical marker of that response, we recommend an indome

78 Thus, in obese patients, clinical markers of a prothrombotic state may indicate a

79 hma, baseline blood eosinophil levels and/or clinical markers of asthma severity predict response to

80 Clinical markers of calcium and bone metabolism were mea

81 or mean differences were computed regarding clinical markers of cardiovascular risk.

82 xtraction fraction, cerebral blood flow, and clinical markers of cerebrovascular impairment in adults

83 Two of the clinical markers of disease activity measured in this st

84 Radiological and clinical markers of disease activity were significantly

85 The data were assessed with respect to clinical markers of disease severity (Risk Adjusted Cong

86 polymorphisms in the TNF-alpha promoter and clinical markers of disease severity in CBD.

87 n Crohn disease, but not with histologic and clinical markers of inflammation.

88 pheral or central neurologic function or any clinical markers of neurologic function in older people

89 f an association of vitamin B-12 status with clinical markers of neurologic function.

90 f VEGFR1 and VEGF-B correlate with edema and clinical markers of NP disease and therefore represent p

91 e cross-linking in proteins and peptides are clinical markers of oxidative stress, aging, and neurode

92 me was mortality and secondary outcomes were clinical markers of patient recovery, for example, multi

93 t of the correlation between the presence of clinical markers of periodontitis, such as pockets and c

94 resents the best data for long-term outcome, clinical markers of prognosis, and the results of random

95 ed, and their relationships with established clinical markers of progression were examined.

96 x could be incorporated with the traditional clinical markers of risk to refine patient prognosis.

97 onflicts among gene expression, genetic, and clinical markers of risk.

98 In this population, clinical markers of severe RA were present in a majority

99 M patients and correlated exercise data with clinical markers of severity, computed tomography scans,

100 95[+/-2.3]%, P = 0.0002) and correlated with clinical markers of steroid responsiveness.

101 Clinical markers of surgical stress that were collected

102 MMPs correlated with clinical markers of tissue damage, further implicating d

103 ghly correlated with genomic alterations and clinical markers of tumor behavior.

104 Premenstrual exacerbation may be a clinical marker predicting a more symptomatic and relaps

105 Clinical markers, protein, and GeneChip analysis were us

106 ospitalizations with corresponding objective clinical markers remained stable or decreased.

107 chocardiographic scores with biochemical and clinical markers showed that only posttreatment 5-HIAA l

108 umors analyzed, much more widely than common clinical markers such as cell proliferation-related gene

109 ese demonstrated the increased expression of clinical markers such as Ki-67, prostate specific antige

110 ical perspective because these syndromes are clinical markers suggesting specific underlying patholog

111 he NRSF splice variant represents a specific clinical marker that could prove useful in detection of

112 ativity before transplantation is a valuable clinical marker that identifies patients at risk for dev

113 automatically measured TID ratio is a useful clinical marker that is sensitive and highly specific fo

114 l echocardiography is a common and important clinical marker that is strongly associated with an incr

115 White blood cell count (WBC) is an important clinical marker that varies among different ethnic group

116 nal imaging and hemodynamic, functional, and clinical markers, three-dimensional cardiac motion impro

117 ence of BCL-6 gene mutations may be a useful clinical marker to determine whether reduction in immuno

118 opathology of the disease and is a potential clinical marker to help in treatment decisions after a f

119 together, activin is a novel candidate as a clinical marker to identify those acute pancreatitis pat

120 herefore, IP 5-HT levels may prove a helpful clinical marker to predict postoperative LD and clinical

121 ametric MRI, whole-mount histopathology, and clinical markers to evaluate the potential utility of (1

122 Assessment was performed for clinical markers using standardized scales for hyposmia,

123 Simple clinical markers, weighted according to prognostic impac

124 Data sets of clinical markers were simulated by use of differential e