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1 n of their endophytic bacteria (Enterobacter cloacae).
2 of M. morganii, C. freundii, E. coli, and E. cloacae).
3 injection of prepupae with live Enterobacter cloacae.
4 ical to plasmid pNDM-HF727 from Enterobacter cloacae.
5 segment (D-limited mice), with Enterobacter cloacae.
6 -1,3 dextran (DEX) expressed on Enterobacter cloacae.
7 r spp., P. aeruginosa, K. pneumoniae, and E. cloacae.
8 revealed that the organism was Enterobacter cloacae.
9 or treatment of infections with Enterobacter cloacae.
10 ri but 97.0% similar to that of Enterobacter cloacae.
11 eding, restored the immunity to Enterobacter cloacae.
12 o case patients yielded S. marcescens and E. cloacae.
13 most laboratories recognized as Enterobacter cloacae.
14 re Pseudomonas aeruginosa (22), Enterobacter cloacae (21), Acinetobacter spp. (13), Enterobacter aero
15 Klebsiella pneumoniae (14.2%), Enterobacter cloacae (9.1%), Acinetobacter spp. (6.2%), Serratia marc
16 nterpretive criteria, excluding Enterobacter cloacae (98.3% S) and E. faecalis (86.0% S), and MIC(90)
17 hia coli, Klebsiella pneumonia, Enterobacter cloacae, Acinetobacter baumannii, and methicillin-resist
19 ortant enzymes CTX-M-15, KPC-2, Enterobacter cloacae AmpC, Pseudomonas aeruginosa AmpC, OXA-10, and O
21 this study involving clinical isolates of E. cloacae and E. aerogenes, susceptibility testing methods
23 vivo, we colonized gnotobiotic pigs with E. cloacae and inoculated pigs with 2.74 x 10(4) genome cop
24 A case of ventriculitis due to Enterobacter cloacae and Pseudomonas fulva following placement of an
26 , approximately 10,000 nM), and Enterobacter cloacae and Serratia marcescens were highly resistant (I
27 la pneumoniae, one (<1%) of 890 Enterobacter cloacae, and one (1%) of 162 Enterobacter aerogenes.
31 s were chemically conjugated to Enterobacter cloacae beta-lactamase (bL), and their abilities to effe
33 genes revealed that one-third (15/45) of E. cloacae bloodstream isolates produced SHV-type extended-
35 o effectively blocked adhesion of pili or E. cloacae, but no effect was observed with nonspecific ant
38 s, reduced HuNoV shedding was observed in E. cloacae colonized pigs, characterized by significantly s
40 terminant was highly conserved within the E. cloacae complex and mediated resistance to up to 600 muM
42 3 nonduplicate Enterobacter isolates (102 E. cloacae complex, 41 E. aerogenes) were tested, including
43 a coli, Enterobacter aerogenes, Enterobacter cloacae complex, Klebsiella pneumoniae, or Klebsiella ox
44 vast majority of clinical isolates of the E. cloacae complex, predominantly in (sub)species that freq
46 cus aureus, and aac(3)-VIa from Enterobacter cloacae (conferring resistance to kanamycin, spectinomyc
47 ates of Serratia marcescens and Enterobacter cloacae, demonstrating the presence of in-frame translat
48 freundii, Clostridium species, Enterobacter cloacae, Enterococcus faecalis, Klebsiella oxytoca, Kleb
49 nterobacteria Escherichia coli, Enterobacter cloacae, Erwinia herbicola, and Salmonella typhimurium.
50 r pathogenic bacteria including Enterobacter cloacae, Escherichia coli J96, Pseudomonas aeruginosa, p
51 important Gram-negative species-Enterobacter cloacae, Escherichia coli, Klebsiella pneumoniae, and Ps
53 ystallographic structure of the Enterobacter cloacae GC1 extended-spectrum class C beta-lactamase, in
55 the nitroreductase enzyme from Enterobacter cloacae has been determined for the oxidized form in sep
57 tly, enteric commensal bacteria Enterobacter cloacae has been recognized as a helper in HuNoV infecti
59 raocular infection secondary to Enterobacter cloacae infection is a devastating disease which, despit
63 H2O2 in the leaves of plants 3h after the E. cloacae inoculation, according to a mechanism involving
66 tematic collection of multidrug-resistant E. cloacae isolated between 2001 and 2011 from bloodstream
70 3 Escherichia coli isolates, 5 Enterobacter cloacae isolates, 2 S. marcescens isolates, 1 Proteus mi
74 ructures of the D305A mutant of Enterobacter cloacae MurA and the D313A mutant of Escherichia coli Ar
75 icted the Cys-115-PEP adduct of Enterobacter cloacae MurA in various reaction states by X-ray crystal
76 Cys115 of Escherichia coli and Enterobacter cloacae MurA is the active site nucleophile alkylated by
77 ined to be 8.3, by titration of Enterobacter cloacae MurA with the alkylating agent iodoacetamide as
78 tructure of the C115S mutant of Enterobacter cloacae MurA, which was crystallized in the presence of
81 ichia coli (n = 129, 30.0%) and Enterobacter cloacae (n = 62, 14.4%) were the main Enterobacteriaceae
82 avin mononucleotide cofactor of Enterobacter cloacae nitroreductase (NR), determined under a variety
84 insensitive nitroreductase from Enterobacter cloacae (NR) catalyzes two-electron reduction of nitroar
87 icroarray analysis of Aag2 challenged with E.cloacae or infected with Dengue virus revealed high tran
88 Unlike wild-type (WT) forms, such as the E. cloacae P99 and Citrobacter freundii enzymes, the ES GC1
90 cephalosporin hydrolysis by the Enterobacter cloacae P99 cephalosporinase (beta-lactam hydrolase, EC
91 tomyces sp. R61, a PBP, and the Enterobacter cloacae P99 cephalosporinase, a class C beta-lactamase.
94 h the class C beta-lactamase of Enterobacter cloacae P99 in two ways, by acylation and by phosphylati
95 drolysis by beta-lactamase from Enterobacter cloacae P99 indicated kcat values of 476 +/- 170 and 248
96 The class C beta-lactamase of Enterobacter cloacae P99 is closely similar in structure to the DD-pe
97 The class C beta-lactamase of Enterobacter cloacae P99 is competitively inhibited by low concentrat
98 lass C serine beta-lactamase of Enterobacter cloacae P99 is irreversibly inhibited by O-aryloxycarbon
99 r the class C beta-lactamase of Enterobacter cloacae P99 suggest an acyl-transfer transition state fo
101 al class A (TEM-2) and class C (Enterobacter cloacae P99) beta-lactamases in a time-dependent fashion
102 y the class C beta-lactamase of Enterobacter cloacae P99, have been studied in order to more firmly e
103 etected Escherichia coli CMY-2, Enterobacter cloacae P99, Klebsiella pneumoniae ACT-1, and the AmpC b
108 negative bacteria, specifically Enterobacter cloacae, Pseudomonas aeruginosa, and Pantoea agglomerans
111 pergillosis (20%, 3 of 15), and Enterobacter cloacae, Serratia marcescens, Pneumocystis carinii pneum
112 O157:H7, Klebsiella pneumoniae, Enterobacter cloacae, Shigella dysenteriae, Salmonella enterica, S. p
114 ane-bound selenate reductase of Enterobacter cloacae SLD1a-1 is purified in low yield and has relativ
116 such as Klebsiella pneumoniae, Enterobacter cloacae, Stenotrophomonas maltophilia, and the Burkholde
118 mase from a clinical isolate of Enterobacter cloacae strain GC1 with improved hydrolytic activity for
119 Shiga toxin 1 (Stx1)-producing Enterobacter cloacae strain, M12X01451, from a human clinical specime
120 erogenes and 13,954 isolates of Enterobacter cloacae tested using a Vitek system; for the same specie
121 iose-H(+) symporter (MelY) from Enterobacter cloacae that had enhanced fermentation on 1% maltose Mac
123 es of endophthalmitis caused by Enterobacter cloacae: two in patients with acute postoperative endoph
124 The bacterial isolate was identified as E. cloacae using a polyphasic approach that included phenot
125 than the totals were noted with Enterobacter cloacae versus ampicillin-sulbactam, aztreonam, ticarcil
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