ライフサイエンスコーパス: clonal anergy

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1 liferative response to specific alloantigen (clonal anergy).

2 e progression and promote the development of clonal anergy.

3 genes involved in pathways leading to T cell clonal anergy.

4 kade do not escape the ultimate induction of clonal anergy.

5 (Mls-1(a)) induces tolerance to Mls-1(a) by clonal anergy.

6 T lymphocytes that prevents the induction of clonal anergy.

7 stemic tolerance by both clonal deletion and clonal anergy.

8 factor interleukin-2 (IL-2), a state called clonal anergy.

9 contributes to defective IL-2 production in clonal anergy.

10 ls through a combination of the induction of clonal anergy and anti-inflammatory cytokines.

11 is state resulted from a combination of both clonal anergy and cytokine-mediated immunosuppression.

12 ) nondeletional T-cell tolerance mediated by clonal anergy and dependent on the structure, location,

13 hus, Ndrg1 contributes to the maintenance of clonal anergy and inhibition of T-cell-mediated inflamma

14 ral tolerance has generally been ascribed to clonal anergy and/or active immunoregulatory states.

15 ce mechanism in addition to clonal deletion, clonal anergy, and receptor editing.

16 duced T-cell hyporesponsiveness as a form of clonal anergy, and they supported an important role for

17 monstrate that T cell adaptive tolerance and clonal anergy are distinct biochemical states, possibly

18 s by tumor cells to T lymphocytes may induce clonal anergy as a mechanism of escape from immune surve

19 ligand-mediated apoptosis and the resulting clonal anergy as the mechanisms of high dose AChR alpha

20 proposed to inhibit the induction of T cell clonal anergy by either directly antagonizing negative s

21 Thus, clonal anergy can contribute to the development of Ag-sp

22 Therefore, clonal anergy can interfere with the delivery of helper

23 c cells, but may instead induce a persistent clonal anergy capable of blocking subsequent immunity.

24 ession by Th2 or TGF-beta-secreting cells or clonal anergy/deletion, depending on the Ag dose used, w

25 inhibition: bystander immune suppression and clonal anergy/deletion.

26 CTLA-4 molecules promote clonal anergy development only indirectly by limiting ce

27 Nevertheless, this induction of clonal anergy did not interfere with the capacity of nai

28 The induction of clonal anergy in a T cell inhibits IL-2 secretion becaus

29 Clonal anergy in Ag-specific CD4+ T cells is shown in th

30 progression and facilitate the induction of clonal anergy in nearby responder CD25(-)CD4(+) T cells.

31 This state was contrasted with T cell clonal anergy in which the RAS/MAPK pathway was preferen

32 ith reduced Ikaros activity are resistant to clonal anergy induced by TCR ligation in the absence of

33 ction signaling defect as the sole basis for clonal anergy induction and document the presence of a d

34 therefore explored the relationship between clonal anergy induction and the avoidance of autoimmune

35 T cell clonal anergy induction in lymphopenic nu/nu mice was fo

36 been shown to be a barrier to CD4(+) T cell clonal anergy induction.

37 Induction of T-cell clonal anergy involves serial activation of transcriptio

38 Clonal anergy is an enigmatic self-tolerance mechanism b

39 One, clonal anergy, is principally a growth arrest state, whe

40 In CD4+ T cells, clonal anergy occurs when the T-cell receptor is activat

41 Clonal anergy of autoreactive B cells is a key mechanism

42 T-cell hyporesponsiveness can be caused by clonal anergy or adaptive tolerance, but the pathophysio

43 T and B cells, perhaps, via CTLA4Ig-induced clonal anergy or deletion.

44 ory mechanisms, such as receptor editing and clonal anergy, preventing the activation of B cells expr

45 s and the development and maintenance of the clonal anergy state.

46 responses were independent of an ability of clonal anergy to block T cell clonal expansion.

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