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1 1 clone cells, but not after transfer of Th2 clone cells.
2 5) GT cells) than Th1-MoPn (505 +/- 51.6 Th1 clone cells/10(5) GT cells) (P < 0.001, as determined by
3 cient at migrating (112 +/- 35.6 labeled Th2 clone cells/10(5) GT cells) than Th1-MoPn (505 +/- 51.6
5 utation, and evolution of prions observed in cloned cells and upon crossing the species barrier remai
7 0 days after the transfer of Ag-specific Th1 clone cells, but not after transfer of Th2 clone cells.
8 abrogate the bone resorption induced by Th1 clone cells combined with gingival challenge with both A
9 nic lambs were liveborn: Three produced from cloned cells contained factor IX and neo transgenes, whe
17 cells at early and late passages including a cloned cell line (D4) expressed Pax6, a transcription fa
19 production of NO and TNF-alpha in NR8383, a cloned cell line derived from rat alveolar macrophages (
22 C-2 Me series, the CB2 Ki's obtained using a cloned cell line were 2.72/2.05 nM (E isomer) and 132/65
23 city in SH-SY5Y human neuroblastoma cells, a cloned cell line which expresses dopaminergic activity,
26 an lung cDNA inserts showed that each of the cloned cell lines contained cDNA that encoded human CD20
30 in engrailed 1/2 mutants with shorter folia, clone cell number and geometry are most similar to clone
31 sion of TCL1 also occurs in the preleukaemic clone cells of A-T patients containing the primary trans
33 inomycetemcomitans Omp29-specific CD4(+) Th1 clone cells proliferated in response to pretreatment of
37 The expression of the IGF-1 transgene in cloned cells was confirmed by reverse transcription-poly
38 ns, although both lymph node T cells and Th1 clone cells were sensitive to superantigen activity of s
39 of frizzled clones was not altered when the clone cells were simultaneously mutant for inturned, mul
40 palatal gingival sites, and Omp29-specific T clone cells were transferred into the tail veins of rats
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