ライフサイエンスコーパス: clonic seizure

1 our institution, she had a generalized tonic-clonic seizure.

2 to first seizure of any type and first tonic-clonic seizure.

3 urring during, or immediately after, a tonic-clonic seizure.

4 f eclampsia, as defined by a witnessed tonic-clonic seizure.

5 approximately 30 suspected generalized tonic-clonic seizures.

6 rred immediately following generalized tonic-clonic seizures.

7 d with the tonic phases of generalized tonic-clonic seizures.

8 rogressively severe myoclonus and rare tonic-clonic seizures.

9 le childhood epilepsy with generalized tonic-clonic seizures.

10 ly involved in secondarily generalized tonic-clonic seizures.

11 ions and for impaired consciousness in tonic-clonic seizures.

12 seizures, required ASIC1a to interrupt tonic-clonic seizures.

13 ced threshold to electrically evoked minimal clonic seizures.

14 used by kainic acid, in the absence of tonic-clonic seizures.

15 havioral endpoints of epileptogenesis, tonic-clonic seizures.

16 t week 3 to 4 of life from generalized tonic-clonic seizures.

17 nique capacity to initiate generalized tonic-clonic seizures.

18 at a mean age of 42 days from massive tonic-clonic seizures.

19 notype consisting of 3/sec absence and tonic-clonic seizures.

20 seizure (1.3 [1.1 to 1.6]), and first tonic-clonic seizure (1.5 [1.2 to 1.8]).

21 10 suppressed minimal clonic seizures (6 Hz mouse model) without typical rotar

22 th new-onset seizure was a generalized tonic-clonic seizure (72 [33%]).

23 myoclonus (all patients), generalized tonic-clonic seizures (all patients) and complex partial seizu

24 prefrontal cortex are associated with tonic-clonic seizures and a normal survival rate.

25 res, and 1 had 4 witnessed generalized tonic-clonic seizures and approximately 30 suspected generaliz

26 ers manifesting with action myoclonus, tonic-clonic seizures and ataxia.

27 Moreover, incidence of tonic clonic seizures and hindlimb extension were reduced in w

28 epilepsy with early onset, generalized tonic-clonic seizures and juvenile lethality by 3 weeks of age

29 hildhood absence epilepsy, generalized tonic clonic seizures and the epileptic encephalopathy, Dravet

30 frequent myoclonic jerks, generalized tonic-clonic seizures and, less commonly, absences.

31 its acoustically triggered generalized tonic-clonic seizures, and often times the EEG, recorded with

32 died prematurely following generalized tonic-clonic seizures, and they were equally susceptible to th

33 Generalized tonic-clonic seizures are among the most dramatic physiologica

34 mes in which generalized myoclonic and tonic-clonic seizures are expressed.

35 Generalised tonic-clonic seizures are the greatest risk factor for SUDEP;

36 s of GABA metabolism, and one in which tonic-clonic seizures associate with increased central nervous

37 n to sound stimuli-induced generalized tonic-clonic seizures (audiogenic reflex seizures) and is a va

38 , neurological insult, total number of tonic-clonic seizures before randomisation, seizure type and t

39 lete and reversible blockade of only the F&F clonic seizure behavior was induced by AP7 (1 and 7.5 nm

40 caused low seizure threshold, sporadic tonic-clonic seizures, brain enlargement and ectopic neurons i

41 age between oral automatisms and generalized clonic seizures, by measuring changes in mRNA expression

42 g the networks involved in generalized tonic-clonic seizures can provide insights into mechanisms of

43 ed early onset spontaneous generalized tonic-clonic seizures concurrent with a significant reduction

44 Tonic-clonic seizures developed spontaneously after 5 months o

45 Only 17 of these had a tonic-clonic seizure during follow-up.

46 e with EEG abnormality were at risk of tonic-clonic seizures during follow-up compared with 16% of th

47 ining three had infrequent generalized tonic-clonic seizures during nocturnal sleep.

48 ted with afebrile focal or generalized tonic-clonic seizures during the first to second year of life;

49 y (JAE), and epilepsy with generalized tonic clonic seizures (EGTCS).

50 t paradigm, pentylenetetrazole-induced tonic-clonic seizures, exclude the possibility that the geneti

51 Fewer generalized clonic seizures failed to reliably produce changes in se

52 on phase of the experiment, only generalized clonic seizures ("forebrain seizures") were expressed.

53 (JME), one predisposing to generalized tonic-clonic seizures (GTCS) and a second to myoclonic seizure

54 zed with the algorithm: 25 generalized tonic-clonic seizures (GTCSs) from 11 patients, and 19 episode

55 ltered if (1) a minimum of eight generalized clonic seizures had been expressed, and (2) a minimum of

56 ncreased latencies to forelimb clonus, tonic clonic seizures, hindlimb extension, and death compared

57 le childhood epilepsy with generalized tonic-clonic seizures (ICEGTC), which bears similarity to SMEI

58 ic hyperactivity immediately following tonic-clonic seizures in DS mice, which leads to lethal bradyc

59 astic behaviors and eventually induces tonic-clonic seizures in rodents.

60 seizures are at low risk of subsequent tonic-clonic seizures in the period of time to which therapeut

61 fects of treatment on the frequency of tonic-clonic seizures in those patients with such pre-randomis

62 pothalamic brain sites following generalized clonic seizures induced by kainic acid.

63 greater latency and lower incidence of tonic-clonic seizures induced by pentylenetetrazol (PTZ; 70 mg

64 SPECT from 59 secondarily generalized tonic-clonic seizures obtained during epilepsy surgery evaluat

65 (JME), epilepsy with only generalized tonic-clonic seizures occurring either randomly during the day

66 e absence epilepsy, five patients with tonic-clonic seizures on awakening and 30 control subjects had

67 epsy and two out of five patients with tonic-clonic seizures on awakening, but in none of the 30 cont

68 le absence epilepsy, and epilepsy with tonic clonic seizures only under the sole heading of idiopathi

69 can provide notification of an ongoing tonic-clonic seizure, or peace of mind in the absence of notif

70 Because generalised tonic-clonic seizures precede most cases of SUDEP, patients mu

71 Daily flurothyl-induced generalized clonic seizures resulted in a progressive decrease in bo

72 rter latencies to onset of generalized tonic-clonic seizures than controls.

73 used in the MESS study are greater for tonic-clonic seizures than they are for partial seizures.

74 h the remaining 7-12% comprising generalized clonic seizures that transition into brainstem seizures.

75 icate that the higher the frequency of tonic-clonic seizures, the higher the risk of SUDEP; furthermo

76 ere was an increase in the flurothyl-induced clonic seizure threshold (anticonvulsant effect) in the

77 ere was an increase in the flurothyl-induced clonic seizure threshold after an E injection alone whil

78 ction alone while there was a reduced tonic--clonic seizure threshold in the presence of both hormone

79 ine, and he continued to have frequent tonic-clonic seizures throughout his life.

80 in is unusually sensitive to recurrent tonic-clonic seizures upon routine handling and to seizures in

81 ctrical stimulation until the first forelimb clonic seizure was induced.

82 othyl and the threshold for clonic and tonic-clonic seizures was determined.

83 holds for flurothyl-induced clonic and tonic-clonic seizures were determined.

84 es conferring a low threshold to ECT minimal clonic seizures were mapped to the telomeric region of m

85 Tonic-clonic seizures were observed in HAGS animals, while LAG

86 of aged apoE4 TR mice had more severe tonic-clonic seizures which occasionally progressed to tonic e

87 ice) exhibited spontaneous generalized tonic-clonic seizures (which occurred in the absence of cardia

88 All had generalized tonic-clonic seizures with onset associated with fever, consis

89 normal early development, generalized tonic-clonic seizures with onset at 5-10 years of age, and pro

90 zures recorded, the majority are generalized clonic seizures, with the remaining 7-12% comprising gen

91 e of the normoglycemic rats, developed tonic-clonic seizures within 12h after ischemia.