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1 2A and alpha2C, and the 125I-labeled agonist clonidine.
2 normoxic, hypoxic, and hypoxic-treated with clonidine.
3 rawal treatment with either buprenorphine or clonidine.
4 d by the selective alpha2 adrenergic agonist clonidine.
5 dose-dependently by perineural injection of clonidine.
6 intraarterial infusions of phenylephrine and clonidine.
7 agonist phenylephrine and the alpha2-agonist clonidine.
8 increased resistance to M. avium induced by clonidine.
9 sed resistance to M. avium growth induced by clonidine.
10 Pertussis toxin blocked the effect of clonidine.
11 role in the analgesic effect of intrathecal clonidine.
12 mediates the analgesic effect of intrathecal clonidine.
13 thoroughly the neuroprotective properties of clonidine.
14 idine orally; or combined nitroglycerin plus clonidine.
15 was not reduced by peripheral or intra-BSTv clonidine.
16 dies provided data for midazolam and two for clonidine.
17 ll as the adrenergic alpha2 receptor agonist clonidine.
19 he intracerebroventricular (ICV) infusion of clonidine (0.03 microgram) caused an increase in NO conc
21 , patients were randomly assigned to receive clonidine (0.2 mg per day) or placebo just before surger
22 uring iontophoresis of phenylephrine (PE) or clonidine (0.5 mm, seven 0.1 mA pulses followed by one 0
23 interval [CI], -1.70 to -0.57), 4 trials of clonidine (-0.95; 95% CI, -1.44 to -0.47), and 2 trials
28 367 and 339, respectively; hazard ratio with clonidine, 1.08; 95% confidence interval [CI], 0.93 to 1
30 onists dexmedetomidine (25 mug/kg, i.v.) and clonidine (100 mug/kg, i.v.) inhibited shivering EMGs, B
31 nylephrine (-11 +/- 4 versus -39 +/- 4%) and clonidine (-12 +/- 4 versus -38 +/- 5%; all P < 0.005) w
32 enylephrine (-3 +/- 3 versus -28 +/- 4%) and clonidine (-15 +/- 4 versus -26 +/- 3%; all P < 0.05).
33 henylephrine (-12 +/- 8 vs. -37 +/- 8 %) and clonidine (-17 +/- 6 vs. -49 +/- 4 %) were significantly
37 tly studied and efficacious medications were clonidine (22 studies; risk ratio: 1.6, numbers needed t
43 pants choosing naltrexone therapy within the clonidine (8 [22.2%]), tramadol ER (7 [19.4%]), or bupre
47 These results suggest that pre-operative clonidine administration could be a useful supplement to
48 the present study tests the hypothesis that clonidine administration will block increased nuclear Ca
53 henylephrine (selective alpha1-agonist), and clonidine (alpha2-agonist) were determined in 10 young (
54 ether the increase in decisiveness driven by clonidine also entails changes in hippocampal search pro
58 tions in the LH of two inhibitory compounds, clonidine (an alpha-2 adrenergic agonist) and muscimol (
59 ease), phenylephrine (an alpha1 agonist) and clonidine (an alpha2 agonist) in 10 healthy men during r
64 ns had a higher spontaneous firing rate, and clonidine-an alpha2A-adrenergic agonist-inhibited LC neu
65 w, medium, and high renin, respectively, for clonidine and 83%, 82%, and 83%, respectively, for dilti
66 he effect of noradrenaline was reproduced by clonidine and antagonized by yohimbine, consistent with
68 vitro data were generally in agreement that clonidine and bFGF counteracted the effect of NMDA as wo
69 gest that tramadol ER is more effective than clonidine and comparable to buprenorphine in reducing op
72 in native sympathetic neurons in response to clonidine and guanfacine, two drugs used for treatment o
73 occurred in 329 patients (6.6%) assigned to clonidine and in 295 patients (5.9%) assigned to placebo
74 opioids as adjuncts are being challenged by clonidine and ketamine, which have more benign side effe
76 amines and diphenhydramine; second, miosis - clonidine and opioids; third, nystagmus - dextromethorph
79 is necessary for the hypotensive effects of clonidine and other sympathoinhibitory adrenergic agonis
81 Gialpha1 increased the relative efficacy of clonidine and oxymetazoline such that the two ligands no
83 In 16 other healthy subjects, the effects of clonidine and placebo on gastric emptying of solids were
84 y acting alpha2-adrenergic receptor agonist, clonidine), and hypoxia and sympathetic inhibition.Oxyha
85 honium, procaine, phencyclidine, cocaine, or clonidine), and the peptides displayed enhanced selectiv
88 sed to treat vasovagal syncope (propranolol, clonidine, and paroxetine) in a double-blind crossover s
90 f ethanol, oxotremorine, nicotine, baclofen, clonidine, and the cannabinoid receptor agonist WIN 55,2
91 Since this treatment completely antagonized clonidine antinociception (40 microg, i.c.v.), supraspin
92 2 channels in mutant mice largely eliminated clonidine antinociception and significantly decreased mo
97 a cellular substrate for the clinical use of clonidine as a treatment for hypertension as well as a r
98 likely that the use of epidural ketamine or clonidine as adjuncts to local anaesthetics will grow.
103 injection of the alpha2-adrenoceptor agonist clonidine at the site of peripheral nerve injury reduces
104 alpha(2)-adrenoceptor agonists, such as clonidine, attenuate hypoxia-induced damage to brain and
107 action, and provide the first evidence that clonidine attenuates the expression of the closely linke
108 he presence and absence of a alpha2-agonist (clonidine), beta-adrenergic receptor antagonist (propran
109 ailed to show displacement of specific [(3)H]clonidine binding to rat brain membranes for either comp
113 with oestrogen, gabapentin, paroxetine, and clonidine, but little or no benefit with other agents; a
120 deltorphin II (DELT), the alpha(2)AR agonist clonidine (CLON) or their combination to inhibit calcito
122 0.5 mg/kg BW), an alpha2-adrenergic agonist, clonidine (CLON; 0.6 mg/kg BW), or a beta-adrenergic ago
123 microinfusions (1 mul) of the alpha2 agonist clonidine (CLON; 1.32 nmol), and half received the alpha
125 nists such as oxymetazoline, cirazoline, and clonidine, confirming previous evidence that this class
126 ine and synaptic transmission was reduced by clonidine, consistent with an action at presynaptic alph
127 ulation of the release of NO in the brain by clonidine contributes to its central antihypertensive ac
128 thesized that the anxiolytic-like actions of clonidine could be mimicked by stimulation of alpha-2 re
133 s: hydrochlorothiazide, atenolol, captopril, clonidine, diltiazem (sustained release), or prazosin.
134 ous ligand(s) for imidazoline binding sites, clonidine-displacing substance (CDS), has been a major g
136 or antagonists (atropine and scopolamine) on clonidine-elicited nitrite release was more potent than
140 idazoxan (6 nmol) into the rRPa reversed the clonidine-evoked inhibition of BAT SNA and BAT thermogen
141 arinic and nicotinic receptor antagonists on clonidine-evoked nitrite (a stable product of NO) releas
142 e present study, we tested a hypothesis that clonidine-evoked NO release is dependent primarily on mu
144 dition, iontophoresis of the alpha-2 agonist clonidine, footshock administration, and LC stimulation
145 s before the clonidine) reduced responses to clonidine for both mean arterial pressure and heart rate
146 nd epidural ones, as well as the addition of clonidine for peripheral neural blockade, have been desc
147 have shown certain benefits from the use of clonidine for regional anesthesia, further investigation
149 itch was notably faster than dissociation of clonidine from alpha(2A)AR, indicating voltage-dependent
150 in quality-of-life scores (0.3 points in the clonidine group compared with -0.2 points in the placebo
152 prenorphine condition and 5% of those in the clonidine group initiated treatment with naltrexone.
153 se in hot flash frequency was greater in the clonidine group than in the placebo group after 4 weeks
155 group difference in time to relapse, but the clonidine group took longer to lapse (hazard ratio=0.67,
156 partly decoupled from opioid craving in the clonidine group, supporting the authors' hypothesized me
169 se findings, a clinical trial for the use of clonidine in the treatment of persistent vasopressor-ref
171 BB NHE3 exists in lipid rafts; (ii) EGF and clonidine increase the amount of BB NHE3; (iii) lipid ra
172 mans, the alpha2-adrenergic receptor agonist clonidine increases decisiveness in tasks that require p
173 Prolonged treatment with either DAMGO or clonidine induced a mutual cross-desensitization between
174 pha(2)-adrenergic agonist (alpha(2)-agonist) clonidine induced resistance to M. avium growth in the R
175 uble NHE3 pool, are involved in the EGF- and clonidine-induced acute increase in amount of BB NHE3; (
177 urthermore, the more pronounced morphine and clonidine-induced antinociception in male mice disappear
181 ce has indicated that a key mechanism of the clonidine-induced reversal of the effects of NMDA antago
185 ol5]enkephalin (DAMGO) or the alpha2 agonist clonidine inhibited voltage-gated Ca2+ currents in these
186 ese findings establish a causal link between clonidine inhibition of c-fos expression in brainstem an
190 lts suggest that the decisiveness seen under clonidine is due to limited exploration of potential opt
193 ion of alpha(2) adrenergic agonists, such as clonidine, is capable of alleviating neuropathic pain.
194 egy is to combine adjuvants such as opioids, clonidine, ketamine and neostigmine to potentiate the ef
199 H enhanced the alpha2-adrenoreceptor agonist clonidine-mediated inhibition (3 microM; NORM, 23 +/- 2%
200 he presence of the NO donor spermine-NO, the clonidine-mediated inhibition of forskolin- and isoprote
202 lammatory agents, anticonvulsants, ketamine, clonidine, mexiletine, antidepressants, and anxiolytics
203 cerebroventricularly applied agonists (NMDA, clonidine, muscimol, and baclofen) at several types of r
205 angiotensin-converting-enzyme inhibitors, or clonidine (n=15,044), those assigned calcium antagonists
210 the effects of the alpha2-adrenergic agonist clonidine on mean arterial pressure, renal sympathetic n
212 search processes, we compared the effect of clonidine on spatial representations in hippocampal neur
213 The present study investigated the effect of clonidine on the basal and inducible c-jun and c-fos mRN
215 aken to determine whether i.p. injections of clonidine or betaxolol affect retinal bFGF mRNA levels.
216 awal (ie, detoxification) from opioids using clonidine or buprenorphine hydrochloride is a widely use
219 re randomized 1:1 to treatment with low-dose clonidine or placebo for 9 weeks and monitored for 30 we
221 nous nitroglycerin; 0.0125, 0.025, or 0.1 mg clonidine orally; or combined nitroglycerin plus clonidi
223 (AR) transfectants, the alpha(2)-AR agonists clonidine, oxymetazoline, UK-14304, and epinephrine incr
224 tained at the end of the taper compared with clonidine participants (22 [61.1%]); tramadol ER retenti
229 trial, we characterized the effects of oral clonidine pre-treatment on intravenous diazepam/meperidi
230 s also inhibited in a dose-related manner by clonidine pretreatment (0.03-0.15 mg/kg, s.c.) and reduc
232 creased with hypoxia (137 +/- 13%; P =0.02); clonidine prevented the hypoxia induced increase (94 +/-
235 suggest that while most of topically applied clonidine reaches the retina by a systemic route one can
236 rgic (phenylephrine) and alpha 2-adrenergic (clonidine) receptor stimulation were blunted during mode
237 d opioid (morphine) and alpha(2)-adrenergic (clonidine) receptor-induced antinociception in a spinal
242 cromol; delivered ICV, 30 minutes before the clonidine) reduced responses to clonidine for both mean
243 ne, ketamine, dexmedetomidine, propofol, and clonidine) reduced the risk of delirium (relative risk,
244 nd the alpha(2)-adrenergic receptor agonist, clonidine, reduced the number of Fos-li dopaminergic neu
247 activation of adrenergic alpha2 receptors by clonidine represents a double-hit to pancreatic islets t
248 determine whether the hypotensive action of clonidine resulted from its stimulation of excess NO in
254 ising for the treatment of gastroparesis are clonidine, sildenafil, and intrapyloric botulinum toxin.
255 istration of the alpha2 adrenoceptor agonist clonidine simultaneously evokes ultrasound production an
256 e include a belladonna alkaloid preparation, clonidine, soy phyto-oestrogens, vitamin E, gabapentin,
257 P < .001) from stabilization to taper in the clonidine (stabilization mean, 0.64 [SE, .05]; taper mea
258 between 1991 and 1995 across the 3 sites for clonidine, stimulants, and antidepressants, while neurol
260 between the taper and post-taper periods for clonidine (taper mean, 13.1; post-taper mean, 3.2; P < .
264 nsitization of Ca(2+) current suppression by clonidine than guanfacine, which paralleled a more marke
265 dentified a second-line agent, perioperative clonidine, that can be used to reduce the risk of periop
266 r harmane, the alpha(2)-adrenoceptor agonist clonidine, the mu-opioid receptor agonists morphine and
268 there is little difference in the ability of clonidine to interact with or stabilize alpha(2)-AR-Gial
270 no difference in onset time.The addition of clonidine to the local anesthetic opioid mixtures seems
274 a trial was planned to compare midazolam and clonidine, two sedatives widely used within PICUs neithe
276 ign to allow separate evaluation of low-dose clonidine versus placebo and low-dose aspirin versus pla
277 son of the dynamics of voltage-dependence of clonidine- vs. norepinephrine-activated receptors uncove
278 owed that co-administration of rapamycin and clonidine was associated with an increased incidence of
281 mplete spinalization (T13) was performed and clonidine was injected to induce rhythmic activities.
284 pidural analgesia using levobupivacaine with clonidine was used in patients undergoing total hip arth
287 ly, the electrographic responses to NMDA and clonidine were potentiated, whereas the ECoG effects med
290 or responses in the leg to phenylephrine and clonidine were similar (or enhanced) postexercise vs. pr
291 esponses in the forearm to phenylephrine and clonidine were similar (or enhanced) postexercise vs. pr
293 ansmission, the effects of noradrenaline and clonidine were tested on the capsaicin-activated current
294 and apraclonidine, acted in a similar way to clonidine when applied topically but because of the numb
295 assessed if the alpha2-autoreceptor agonist clonidine (which decreases norepinephrine release) would
297 blocked the increased resistance induced by clonidine, while a Gs-protein antagonist was without eff
298 cts were identified for midazolam and 16 for clonidine with full texts obtained for 80 and 6 articles
300 after exposure to forskolin, isoproterenol, clonidine, yohimbine, pertussis toxin, and the NO donor
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