ライフサイエンスコーパス: clonus

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1 ivation, hyperreflexia, and stimulus-induced clonus.

2 Burst firing also occurred during F and F clonus.

3 us emerges immediately following generalized clonus.

4 y reduces the delay to onset of first facial clonus.

5 nset to the occurrence of the first forelimb clonus.

6 mb and hindlimb tonic rigidity and posttonic clonus; (6) low-amplitude EEG during postictal depressio

7 neuronal firing was silent during post-tonic clonus, a behavior seen in GEPR-9s only after AGS repeti

8 Post-tonic clonus and an increased duration of post-ictal behaviora

9 ractivity, teeth chattering, low posture and clonus and potentiated splayed postural effects.

10 or/trembling, head weaving, splayed posture, clonus and wet dog shakes.

11 luded latencies to focal clonus, generalized clonus, and maximal seizure.

12 firing patterns appeared during generalized clonus before and after AGS kindling.

13 l/side)] into PRh did not affect generalized clonus before or after AGS kindling.

14 During F&F clonus, burst firing, an indicator of increased excitabi

15 AGS in GEPR-3s culminate in generalized clonus, but daily repetition of AGS (AGS kindling) resul

16 et to myoclonic twitch and face and forelimb clonus by 2- to 3-fold.

17 cies and decreased the incidence of forelimb clonus compared to T to knockout mice, which were not di

18 ed prior to the appearance of the post-tonic clonus component of repetitive AGS.

19 tion of the study a significant reduction in clonus duration (53.90 +/- 3.27 vs. 40.09 +/- 4.14) stil

20 dala [89.60 +/- 5.55 vs. 48.67 +/- 15.8] and clonus duration (71.2 +/- 5.94 vs. 29.40 +/- 8.87; Mean

21 to clonus was significantly prolonged while clonus duration was reduced indicating a less severe sei

22 in their daily lives, and several noted less clonus, easier stepping, and/or other improvements.

23 ditional behavior, facial and forelimb (F&F) clonus emerges immediately following generalized clonus.

24 izure parameters included latencies to focal clonus, generalized clonus, and maximal seizure.

25 responsible for the generation of post-tonic clonus in GEPR-9s.

26 or cPRF neurons in generation of generalized clonus in unkindled GEPR-3s, which is increased by AGS k

27 included in the spastic syndrome, including clonus, increased flexor reflex activity, and flexor spa

28 F&F clonus is thought to be driven from forebrain structures

29 F clonus, which was unexpected since F and F clonus is thought to originate primarily in forebrain st

30 zure behavior, facial and forelimb (F and F) clonus, not seen prior to kindling.

31 constant rate via the lateral tail vein and clonus onset was recorded in the presence and absence of

32 ng and falling, severe forelimb and hindlimb clonus, opisthotonos and explosive running.

33 ration and induces an additional generalized clonus phase [post-tonic clonus (PTC)], which involves e

34 sly unexpressed seizure behavior-generalized clonus (post-tonic clonus, PTC).

35 itional generalized clonus phase [post-tonic clonus (PTC)], which involves expansion of the localized

36 zure behavior-generalized clonus (post-tonic clonus, PTC).

37 ures are wet-rat-shakes, facial and forelimb clonus, rearing and spike-and-waves in the EEG.

38 at her baseline mental status, although some clonus remained.

39 ildtype mice increased latencies to forelimb clonus, tonic clonic seizures, hindlimb extension, and d

40 Additionally, delay to clonus was significantly prolonged while clonus duration

41 neuronal firing occurred during generalized clonus, which changed to burst firing after AGS kindling

42 that cPRF neurons may be involved in F and F clonus, which was unexpected since F and F clonus is tho

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