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1 tic potential of CN-105 in a murine model of closed head injury.
2 ese lesions evolve following relatively mild closed head injury.
3 volunteers and 20 patients within 6 days of closed head injury.
4 was performed on anesthetized mice following closed head injury.
5 e rates of depressive illness 50 years after closed head injury.
6 very of a fallen patient who has sustained a closed head injury.
7 MR images were obtained 3 months after closed-head injury.
8 trations peaked at 30 to 42 hrs after severe closed-head injury.
9 e, injury severity, and outcome after severe closed-head injury.
10 lity, symptoms, and objective findings after closed-head injury.
11 es C for 24 hrs) on neurologic outcome after closed-head injury.
12 ds to establish the presence and severity of closed head injuries.
13 and normothermia in 82 patients with severe closed head injuries (a score of 3 to 7 on the Glasgow C
14 tonic saline administration to children with closed head injury appears to be a promising therapy for
15 fficult to reproduce TAI in animal models of closed head injury, but in vitro stretch injury models r
16 this study, based on advantages of both the closed head injury (CHI) and controlled cortical impact
18 Edition (ICD-9) codes denoting open (OHI) or closed head injury (CHI) in isolation or in combination
28 nsequences of a clinically relevant model of closed head injury, the lateral fluid percussion injury
29 induced by a weight drop model to produce a closed head injury to mice and the effect of inhaled nit
32 core of < or = 7) adult patients with severe closed-head injury were studied during the prospective,
33 o 65 years of age with coma after sustaining closed head injuries who were randomly assigned to be tr
34 ythropoietin, n = 102; placebo, n = 98) with closed head injury who were unable to follow commands an
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