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1 tic potential of CN-105 in a murine model of closed head injury.
2 ese lesions evolve following relatively mild closed head injury.
3  volunteers and 20 patients within 6 days of closed head injury.
4 was performed on anesthetized mice following closed head injury.
5 e rates of depressive illness 50 years after closed head injury.
6 very of a fallen patient who has sustained a closed head injury.
7       MR images were obtained 3 months after closed-head injury.
8 trations peaked at 30 to 42 hrs after severe closed-head injury.
9 e, injury severity, and outcome after severe closed-head injury.
10 lity, symptoms, and objective findings after closed-head injury.
11 es C for 24 hrs) on neurologic outcome after closed-head injury.
12 ds to establish the presence and severity of closed head injuries.
13  and normothermia in 82 patients with severe closed head injuries (a score of 3 to 7 on the Glasgow C
14 tonic saline administration to children with closed head injury appears to be a promising therapy for
15 fficult to reproduce TAI in animal models of closed head injury, but in vitro stretch injury models r
16  this study, based on advantages of both the closed head injury (CHI) and controlled cortical impact
17                This review will examine mild closed head injury (CHI) and the current evidence on hea
18 Edition (ICD-9) codes denoting open (OHI) or closed head injury (CHI) in isolation or in combination
19 al models have been developed to mimic human closed head injury (CHI).
20 ynaptic proteins in an experimental model of closed head injury (CHI).
21                                       Severe closed head injury in mice induced abundant MAC depositi
22                                              Closed-head injury-induced lesions in the right putamen
23    Our results suggest that mild-to-moderate closed head injury may increase PD risk decades later.
24                                      Using a closed-head injury model of TBI in mice, we showed by MR
25                             In patients with closed head injury, neither the administration of erythr
26        Clinical evaluation of patients after closed-head injury, particularly mild head trauma, must
27           Standards for management of severe closed head injury should help to establish a foundation
28 nsequences of a clinically relevant model of closed head injury, the lateral fluid percussion injury
29  induced by a weight drop model to produce a closed head injury to mice and the effect of inhaled nit
30                                              Closed head injury to the developing rat brain causes an
31                                       Severe closed head injury was induced in rats by the impact of
32 core of < or = 7) adult patients with severe closed-head injury were studied during the prospective,
33 o 65 years of age with coma after sustaining closed head injuries who were randomly assigned to be tr
34 ythropoietin, n = 102; placebo, n = 98) with closed head injury who were unable to follow commands an

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