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1 established treatment option in migraine and cluster headache.
2 be similarly effective in both migraine and cluster headache.
3 ested a pivotal role for the hypothalamus in cluster headache.
4 tter as the key area for the basic defect in cluster headache.
5 of cervical hypersensitivity in migraine and cluster headache.
6 vel avenue for the treatment of migraine and cluster headache.
7 the structure of the brains of patients with cluster headache.
8 ent as has been reported in acute attacks of cluster headache.
9 the primum movens in the pathophysiology of cluster headache.
10 che attacks in nine patients who had chronic cluster headache.
11 in persons suffering from both migraine and cluster headache.
12 the primum movens in the pathophysiology of cluster headache.
13 or factor in headache attacks in migraine or cluster headache.
14 cerebral blood flow (rCBF) in patients with cluster headache.
15 l injection and tearing (SUNCT syndrome) and cluster headache.
16 hysiology of migraine and to a lesser extent cluster headache.
17 in the same pattern previously described in cluster headache.
18 to similarly affected patients with chronic cluster headache.
19 ut was present in two of three patients with cluster headache.
20 junctival injection and tearing (SUNCT; 5%), cluster headache (4%), hemicrania continua (1%) and prim
22 ontext of the differential diagnosis between cluster headache and paroxysmal hemicrania leads to a co
23 The trigeminal-autonomic cephalgias include cluster headache and paroxysmal hemicranias, in which he
26 a model to explain the pain in migraine and cluster headache, and has been used to characterize the
27 aptic activity, during nitroglycerin-induced cluster headache attacks in nine patients who had chroni
28 etter than placebo in the acute treatment of cluster headache but not in the acute management of migr
31 therapeutic rationale to plant migraine and cluster headache firmly in the brain as neurological pro
39 ncluding migraine, tension-type headache and cluster headache, is the currently accepted view that th
41 e human retina in acute migraine (n = 8) and cluster headache (n = 5) using fluorescein or indocyanin
44 eminal autonomic cephalalgias, which include cluster headache, paroxysmal hemicrania, short-lasting u
45 ur at night or during sleep, with a focus on cluster headaches, paroxysmal hemicrania, short-lasting
46 that the current view of the neurobiology of cluster headache requires complete revision and that thi
48 From a physiological viewpoint, therefore, cluster headache should be described as a neurovascular
49 patients with medically intractable chronic cluster headache were implanted in the suboccipital regi
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