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1 rhosis who were HCV mono-infected or HIV/HCV co-infected.
2 ); worldwide more than 15 million people are co-infected.
3 DR tuberculosis who were tested for HIV were co-infected.
5 brosis in 8% of all patients (31% in HIV/HCV co-infected and 3% in HIV mono-infected individuals).
6 gorithms and treatment regimens for both HIV co-infected and hepatitis C mono-infected patients are s
10 pe 1 to 4 are virtually identical in the HIV co-infected as in the HCV monoinfected, regardless of wh
12 antigen-specific antibody-producing cells in co-infected C3H mice compared to B6 mice as early as 2 w
13 doplasmic reticulum (ER) and PM fractions of co-infected cells by the anti-FLAG antibody demonstrates
17 ys of co-infection, and after 4 weeks 95% of co-infected fleas contained an average of 103 antibiotic
18 were hepatitis B-co-infected or hepatitis C-co-infected, had new AIDS-defining conditions within 30
20 spore-trapping experiment demonstrates that co-infected hosts shed more transmission propagules than
23 miR-122 and miR-200a are greater in HIV/HCV co-infected individuals compared to HIV-1 mono-infected
24 red with individuals infected with only HIV, co-infected individuals had a higher risk of death from
25 d/or AIDS-related mortality was higher among co-infected individuals than those with only HIV infecti
26 an achieve similar response rates in HCV/HIV-co-infected individuals with no adverse effect on HIV RN
27 g no primary data, samples of HCV or HIV-HCV co-infected individuals, or samples relying on self-repo
37 e excluded patients if they were hepatitis B-co-infected or hepatitis C-co-infected, had new AIDS-def
39 XDR tuberculosis has been transmitted to HIV co-infected patients and is associated with high mortali
40 ations predict virologic response in HIV/HCV co-infected patients and to examine variables associated
41 itis C virus (HCV) mono-infected and HIV/HCV co-infected patients are in need of safe, effective, all
42 ths related to HIV in the United States, and co-infected patients bear a significant proportion of th
43 mmunodeficiency virus type 1 (HIV-1) and HBV-co-infected patients because it had been reported that E
44 Compared with HCV-monoinfected patients, co-infected patients had a higher rate of hepatic decomp
48 ofile and SVR12 rates of 96.7% among HIV/HCV co-infected patients participating in an Italian compass
51 or without ribavirin (arms B1-3) and HIV/HCV co-infected patients to 12 weeks of therapy with or with
56 usly untreated HCV-mono-infected and HIV/HCV-co-infected patients without cirrhosis achieved SVR12 ra
57 the context of human immunodeficiency virus co-infected patients, has threatened global TB control.
58 the standard treatment for HCV infection for co-infected patients, who have the greatest risk for liv
72 of hepatic decompensation was greater among co-infected than monoinfected patients (7.4% vs. 4.8% at
74 n, we find the highest disease prevalence in co-infected treatments both at the host genotype and pop
76 DNA barcode sequence by allelic exchange and co-infected with a wild-type reference to calculate the
79 ecies in North America, could potentially be co-infected with avian and human IAVs, facilitating the
80 heterophil to lymphocyte ratios and were all co-infected with avian malaria, consistent with evidence
83 uncontrolled, pilot study enrolling patients co-infected with HCV genotype 1 and HIV, administration
84 en resulted in high SVR rates among patients co-infected with HCV genotype 1 and HIV-1 whether treate
86 e following transplantation than in patients co-infected with hepatitis C (37 +/- 9 vs. 70 +/- 33 U/L
87 frequently found in transplantation patients co-infected with hepatitis C although it appears to have
90 s6-Cdc25C immunoprecipitated from sf-9 cells co-infected with His6-Prk and His6-Cdc25C baculoviruses,
92 n American, and 13% were Caucasian; 27% were co-infected with HIV and 67% had no or mild fibrosis.
93 eron-free regimens are approved for patients co-infected with HIV and genotype-2 or genotype-3 hepati
97 therapy became widely available, individuals co-infected with HIV and HCV have had a higher risk of d
100 deline-recommended treatment for individuals co-infected with HIV and hepatitis B, this regimen might
101 ve been incompletely examined among patients co-infected with HIV and hepatitis C virus (HCV) in the
106 tuberculosis and high mortality among those co-infected with HIV-1 necessitates new therapeutic appr
108 cine efficacy against CIN2+, where all cases co-infected with HPV-16/18 were removed, vaccine efficac
110 epatitis C virus (HCV) infection in patients co-infected with human immunodeficiency virus (HIV).
113 rcine alveolar macrophages (PAM) cells model co-infected with PRRSV/PCV2 with modification in vitro,
114 stem and immunoprecipitation of insect cells co-infected with RAD51 and RAD52 recombinant viruses, an
117 We enrolled patients (aged >/=18 years) co-infected with stable HIV and chronic HCV genotypes 1-
119 iretroviral-induced liver injury in patients co-infected with the human immunodeficiency virus and in
121 This restriction is overcome when cells are co-infected with vaccinia virus (VACV), a vertebrate DNA
123 (and other body fluids) in 66 HIV- and HHV-8-co-infected women without KS so that we could examine pr
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