ライフサイエンスコーパス: co-inhibitory

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1 lasmodium control and that crosstalk between co-inhibitory and co-stimulatory pathways in pathogen-sp

2 Modulation of co-inhibitory and co-stimulatory receptors of the immune

3 , a member of the B7 family of costimulatory/co-inhibitory ligands expressed by both malignant cells

4 imately 50% of Tc17 cells also expressed the co-inhibitory molecule CTLA-4, and only a minority (<20%

5 dent upon sustained T cell expression of the co-inhibitory molecule PD-1.

6 the first genetic evidence that BTN2A2 is a co-inhibitory molecule that modulates T cell-mediated im

7 Interestingly, the co-inhibitory molecules cytotoxic T lymphocyte antigen-4

8 Co-inhibitory molecules play a major role in modulating

9 PD-L1(+) DC, MDSC, TAM and Treg, as well as co-inhibitory molecules-double-positive, severely exhaus

10 lves a complex network of co-stimulatory and co-inhibitory molecules.

11 While therapies targeting the co-inhibitory or immune checkpoint receptors PD-1 and CT

12 d side effects, suggesting the importance of co-inhibitory pathway for both prevention of autoimmunit

13 ptor and its ligands, PD-L1 and PD-L2, are a co-inhibitory pathway that contributes to the negative r

14 will focus on the role of co-stimulatory and co-inhibitory pathways in two systemic (systemic lupus e

15 which is a large array of co-stimulatory and co-inhibitory pathways that modulate the host response.

16 these might be increased by blocking T-cell co-inhibitory pathways, such as preventing interaction b

17 ogated following coordinate blockade of PD-1 co-inhibitory pathways, which are also upregulated durin

18 incident with selective upregulation of CD85 co-inhibitory pathways.

19 12 application with systemic blockade of the co-inhibitory receptor CTLA-4 on T cells led to tumor er

20 Mice lacking the co-inhibitory receptor cytotoxic T lymphocyte antigen-4

21 tting of chronic diseases where constitutive co-inhibitory receptor expression on T cells dampens eff

22 une restoration potential of blockade of the co-inhibitory receptor programmed death 1 (PD-1) during

23 tes various immune responses by engaging the co-inhibitory receptor programmed death-1.

24 e binding affinities of the PD-1-PD-L1/PD-L2 co-inhibitory receptor system, and discovered an unexpec

25 The next wave of co-inhibitory receptor targets that are being explored i

26 effects of programmed cell death 1 (PD1), a co-inhibitory receptor that impairs T-cell function and

27 B and T lymphocyte attenuator (BTLA) is a co-inhibitory receptor that interacts with herpesvirus e

28 Tim-3 is a co-inhibitory receptor that is expressed on IFN-g-produc

29 d by the constellation of co-stimulatory and co-inhibitory receptors expressed on the cell surface.

30 at occur in cancer, highlighting the role of co-inhibitory receptors in contributing to this process

31 However, all co-inhibitory receptors share an ability to oppose activ

32 f CD57 and loss of CD28, with an increase in co-inhibitory receptors such as PD-1 and Tim-3.

33 This has provided impetus to identify other co-inhibitory receptors that could be exploited to enhan

34 -domain molecule 3 (Tim-3), which are potent co-inhibitory receptors, and their persistent expression

35 Co-inhibitory receptors, such as CTLA-4 and PD-1, have a

36 s exist to limit Akt activation by different co-inhibitory receptors.

37 s but potentially between co-stimulatory and co-inhibitory receptors.

38 on of multiple additional co-stimulatory and co-inhibitory receptors.

39 prominent sites of synapsis initiation, and CO-inhibitory role(s) that limit CO number.

40 the recent advances in our understanding of co-inhibitory signaling and its potential clinical appli

41 In addition to blocking co-inhibitory signals in secondary lymphoid organs, cyto

42 Here, we identified a critical role for the co-inhibitory SLAM family member 2B4 (CD244) in attenuat

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