1 t-induced cell differentiation compared with
co-transfection.
2 hormone receptor (TR) was over-expressed by
co-transfection.
3 Co-transfection "
add-back" experiments demonstrated that
4 Co-transfection analyses, electrophoretic mobility shift
5 as indicated by our results obtained through
co-transfection and chromatin immunoprecipitation (ChIP)
6 xc13 based on DNA binding studies as well as
co-transfection and chromatin immunoprecipitation assays
7 Co-transfection and immunoblotting experiments revealed
8 Co-transfection and knock-down experiments determined th
9 Co-transfection and microinjection studies demonstrate t
10 Mutation,
co-transfection and sex-reversal studies all point to a
11 Gel shift,
co-transfection,
and chromatin immunoprecipitation assay
12 by blot overlay, surface plasmon resonance,
co-transfection,
and co-immunoprecipitation assays.
13 gonist stimulation of [Ca(2+)]i with beta2AR
co-transfection,
approximately 53% decrease in [Ca(2+)]i
14 In the current study, we used a
co-transfection assay in lung epithelial cells to examin
15 letion mutant of the IRF-1-binding site in a
co-transfection assay of IRF-1 expression plasmid with C
16 the Gal4-Nrf2 chimeras and Gal4-Luc reporter
co-transfection assay system in HepG2 cells.
17 on of these inclusion bodies, we performed a
co-transfection assay using cultured HEK293 cells.
18 In a
co-transfection assay, basonuclin can elevate transcript
19 on of a model ARE transcript in a cell-based
co-transfection assay.
20 augmented by an LRH-1 expression vector in a
co-transfection assay.
21 tion of p53, p21, and MDM2 protein levels in
co-transfection assays and in response to bleomycin.
22 oth could activate E16 splicing in HeLa cell
co-transfection assays in a UGCAUG-dependent manner.
23 Co-transfection assays revealed for the first time that
24 Co-transfection assays showed that Erm by itself had lit
25 pithelium and in lens fibers, were tested in
co-transfection assays using cultured lens and non-lens
26 tion as judged by in vitro transcription and
co-transfection assays with an unmethylated collagen pro
27 43 SRF targets was then further validated by
co-transfection assays with SRF.
28 n vitro mapping, a phosphospecific antibody,
co-transfection assays, and untransfected bovine aortic
29 In
co-transfection assays, by overexpressing oncogenic Ha-R
30 In
co-transfection assays, KLF15 and MEF2A, a known activat
31 ors can activate transcription from MAREs in
co-transfection assays, mouse germline mutations in cnc
32 Furthermore, in
co-transfection assays, PKCnu can be trans-phosphorylate
33 In
co-transfection assays, Py large T inhibits the co-activ
34 In
co-transfection assays, the Mn-SOD promoter was transact
35 In
co-transfection assays, thrombin stimulation of a minima
36 In
co-transfection assays, using HO-1 promoter-luciferase c
37 In
co-transfection assays, wild type Pax9 activated reporte
38 ion of pJNK formation in COS-7 cell MLK7/JNK
co-transfection assays.
39 re promoter is activated by Shh in transient
co-transfection assays.
40 were also shown to be important by transient
co-transfection assays.
41 re also observed in the full length receptor
co-transfection assays.
42 sed the TSP-1 promoter activity and U19/EAF2
co-transfection blocked the p53 suppression of TSP-1 pro
43 By
co-transfections,
co-immunoprecipitations and RNAi const
44 Using
co-transfections,
co-immunoprecipitations and RNAi we di
45 Further,
co-transfection/
co-precipitation studies show the self-a
46 sion protein to Gal4-responsive promoters in
co-transfection experiments activated transcription 5-6-
47 uciferase activity measurements in transient
co-transfection experiments and electro-mobility shift a
48 d Sp2-mediated trans-activation in transient
co-transfection experiments but also revealed Sp2 to be
49 Co-transfection experiments demonstrated that dominant n
50 Co-transfection experiments demonstrated that expression
51 ppear to destabilise the wildtype protein in
co-transfection experiments in a human RPE cell line.
52 In vitro association assays and
co-transfection experiments in both MDCK and HeLa cells
53 Our
co-transfection experiments in human neuroblastoma SH-SY
54 Co-transfection experiments indicate that GRG proteins r
55 Co-transfection experiments indicated that Sp1 and Sp3 w
56 In
co-transfection experiments p73alpha, but not p73beta, a
57 Further,
co-transfection experiments revealed that the amount of
58 Co-transfection experiments showed that binding sites fo
59 Co-transfection experiments showed that KLF4 repressed t
60 ity shift analysis, promoter mutagenesis and
co-transfection experiments showed that NeuroD, a pro-ne
61 Co-transfection experiments showed that transcription fa
62 Further
co-transfection experiments showed that two of the SOX/S
63 However,
co-transfection experiments suggest that other proteins
64 atively regulated by Kruppel-like factors in
co-transfection experiments suggesting a possible mechan
65 Co-transfection experiments using a luciferase reporter
66 In
co-transfection experiments using Drosophila SL2 cells t
67 Co-transfection experiments were performed in which AQP3
68 Co-transfection experiments with a SOX9 expression plasm
69 Moreover,
co-transfection experiments with both mutant and WT tran
70 Furthermore,
co-transfection experiments with Stat3 beta demonstrate
71 Claudin-1 associated with claudin-7 in
co-transfection experiments, and claudin-7 was required
72 In
co-transfection experiments, Foxm1 directly bound to and
73 In
co-transfection experiments, Foxm1 directly bound to and
74 In
co-transfection experiments, Foxm1 protein-induced Cox-2
75 In
co-transfection experiments, Hes-1, up-regulated by over
76 In
co-transfection experiments, miR-BART20-5p inhibited T-b
77 In
co-transfection experiments, the effects of A1E proteins
78 directly transactivates the CD1 promoter in
co-transfection experiments, whereas electrophoretic mob
79 Foxf2 repressed Foxf1 promoter activity in
co-transfection experiments.
80 ption factors on SP-B promoter expression by
co-transfection experiments.
81 ignaling in SLP-76-deficient Jurkat cells in
co-transfection experiments.
82 Transient
co-transfections in Arabidopsis protoplasts showed that
83 Co-transfections in HepG2 cells showed that USF1 and USF
84 Bcl-2
co-transfection inhibited Bid-induced apoptosis but did
85 Co-transfection into cells of the p53 gene and plasmid D
86 tor function has been confirmed by transient
co-transfection into S2 cells of constructs of HIF1 subu
87 Co-transfections into COS7 cells and in vitro kinase ass
88 [3H]cholesterol/acLDL in the absence of SR-A
co-transfection),
it showed impaired cholesterol efflux
89 Using transient
co-transfections,
northern blots, antisense oligonucleot
90 Co-transfection of 293 cells with Slit and glypican-1 cD
91 , and this induction could be repressed upon
co-transfection of a c-Myc expression vector.
92 g epithelial cell line, L17, was enhanced by
co-transfection of a constitutively active MEK1.
93 and p190RhoGEF tyrosine phosphorylation, and
co-transfection of a dominant-negative inhibitor of FAK
94 Co-transfection of a JNK expression plasmid inhibited RX
95 Co-transfection of a Sp1 expression vector further incre
96 AOC1 promoter was significantly enhanced by
co-transfection of a WT1 expression construct.
97 Co-transfection of A2AR with A2BR enhanced surface expre
98 In HEK 293 cells
co-transfection of ABCA1 with either SCD1 or SCD2 inhibi
99 tyrosine phosphorylation of SH3PX1, whereas
co-transfection of ACK2 with SH3PX1 resulted in the cons
100 Co-transfection of Ambn and mutant Amelx in a eukaryotic
101 Co-transfection of an ARP-1/COUP-TFII expression vector
102 Co-transfection of an expression vector encoding wild-ty
103 Co-transfection of an hMSH2 reporter and p53 expression
104 However,
co-transfection of AP-2alpha wild-type or the dominant n
105 Co-transfection of APOBEC3A with a TRIB3 expression vect
106 ryonic kidney 293T cells following transient
co-transfection of ASIC3 and CFTR.
107 Colony survival studies revealed that
co-transfection of both mdr1b and p53 dramatically reduc
108 Co-transfection of both proteins resulted in the nuclear
109 Co-transfection of BRCA1L with BRCA1S or BRCA1S-9,10 exh
110 Furthermore,
co-transfection of c-Jun and ATF2 synergistically induce
111 Co-transfection of C/EBPalpha and NF-Y synergistically i
112 Co-transfection of C6 glioma with the combination of NR1
113 ant 5-LO inhibited in vitro activity, as did
co-transfection of cells with 5-LO plus the catalytic su
114 ditionally, repression could be augmented by
co-transfection of cells with a histone deacetylase (HDA
115 After
co-transfection of cells with ASC plus wild type pyrin,
116 Utilization of this system requires a single
co-transfection of cells with assay vectors, thereby ena
117 Co-transfection of cells with beta(5) promoter reporter
118 Co-transfection of cells with beta-catenin, Lef-1, PEA3,
119 Co-transfection of cells with both H- and L-chain cDNAs
120 Co-transfection of cells with Cas9, a gRNA specifying th
121 Co-transfection of cells with either the full-length IA-
122 Co-transfection of Chinese hamster ovary cells with HSL
123 Co-transfection of Chinese hamster ovary cells with Na(v
124 Co-transfection of Clostridium botulinum C3 toxin blocke
125 Co-transfection of CNAIP expression constructs with luci
126 Co-transfection of COS cells with truncated forms of apo
127 Co-transfection of COS-1 cells with both proMMP-2 and fu
128 Co-transfection of cultured cells with normal and mutant
129 Co-transfection of cyclin E and skp2 synergistically pro
130 This reduction was inhibited by
co-transfection of dominant negative IKKbeta as well as
131 itogen-activated protein (MAP) kinases or by
co-transfection of dominant negative MAP kinase mutant c
132 Co-transfection of dominant-negative GABPbeta1 blocks pr
133 Co-transfection of E1A with p300 abrogated p50 acetylati
134 nal activation of a promoter reporter, as is
co-transfection of E1A.
135 Moreover,
co-transfection of either full-length Mint3 or the N ter
136 Co-transfection of either MAO with parkin in HEK293 cell
137 utyrate that was significantly reduced after
co-transfection of either of two dominant-negative CREB
138 Interestingly,
co-transfection of either PITX2A or PITX2C with PITX2B r
139 Co-transfection of eNOS cDNA with constitutively active
140 Co-transfection of eNOS with Akt and stimulation of endo
141 ed beta(3)AR-promoted eNOS-Rac1 association;
co-transfection of eNOS with dominant negative Rac1 comp
142 In HCT 116 colon cancer cells,
co-transfection of FAK plasmid with p21, MDM-2, and BAX
143 Co-transfection of FEZ1 and NBR1 showed overlapping loca
144 In HepG2 cells,
co-transfection of FXR and RXR alpha is required to atta
145 ombinations except in the middle turn, where
co-transfection of hATOH1, hE2A, and hGATA3 was more eff
146 Co-transfection of hBVR with ATF-2 or c-jun promoters ca
147 ion, which can be further potentiated by the
co-transfection of HDAC7.
148 matic synergism between these two mediators;
co-transfection of HEK293 cells with Stim1 and Orai1 res
149 activated approximately 8-fold by transient
co-transfection of human granulosa cells with a Wt1 expr
150 Co-transfection of human umbilical vein endothelial cell
151 Co-transfection of HuR and RNase-L enhanced RNase-L expr
152 Co-transfection of IP10 luciferase with the active forms
153 ence nearly abolished promoter activity, and
co-transfection of junD significantly increased wild typ
154 ibited by the MEK inhibitor, PD098059, or by
co-transfection of kinase-inactive MEK1 but not by the p
155 Co-transfection of kindlin-2 with talin-H results in a s
156 Channels formed by
co-transfection of Kir6.2 and the mutant SUR1 in COS cel
157 Co-transfection of KLF-4 and HDAC expression plasmids in
158 Co-transfection of KLF4 and HDAC3 resulted in a synergis
159 Co-transfection of LGI1 with its receptors, ADAM22 or AD
160 Co-transfection of liganded RXR alpha and the known card
161 kidney (HEK) cells, rescue of SOCs required
co-transfection of low levels of both smOrai1 and smTRPC
162 Kinase assays and
co-transfection of MAP-2c and Fyn confirmed that Fyn tyr
163 Co-transfection of miR-339-5p with a BACE1 3'-UTR report
164 Significantly,
co-transfection of MITF siRNA with pre-miR-204/211 rescu
165 Importantly,
co-transfection of mutant CREB and a construct constitut
166 Moreover,
co-transfection of MyoD and Tead2 intron reporter constr
167 Co-transfection of MyoD with Id1 or Id1(NLS) increases I
168 Co-transfection of neurons with either a non-G alpha(q/1
169 the ANF and PLOD1 promoters is repressed by
co-transfection of Nkx2.5 in the C3H10T1/2 embryonic fib
170 high subcellular fluorescence observed upon
co-transfection of non-fluorescent ZnT-YC and ZnT-YN; th
171 Co-transfection of non-steroidogenic COS-1 cells with P4
172 Moreover,
co-transfection of Notch-ICD could restore suppression o
173 Co-transfection of Nox1, NOXO1, and NOXA1 reconstitutes
174 However,
co-transfection of Nrdp1 with parkin reversed the effect
175 In contrast,
co-transfection of p300 and CBP with ESE-1 enhances the
176 Transient
co-transfection of p53 family members showed that p53 an
177 down-regulation was further confirmed after
co-transfection of pAPCP plasmid with pCMV-p53 into HCT-
178 cells, the Bcd activity is increased by the
co-transfection of plasmids expressing dCBP and reduced
179 In addition,
co-transfection of plasmids expressing ISG15, Ube1L, Ubc
180 rious hGH transactivation also occurred upon
co-transfection of plasmids expressing the transactivato
181 hatase activity toward HPK1 in vivo and that
co-transfection of PP4 with HPK1 resulted in specific ki
182 Co-transfection of PPARgamma wild type vectors and treat
183 promoter IV (pIV) was assessed by transient
co-transfection of promoter-reporter constructs.
184 In HEK-293 cells
co-transfection of RGS7 but not Ggamma(2) supported expr
185 Further,
co-transfection of SC35 with PKCdelta minigene promoted
186 Co-transfection of SEF and PAX6 promoter-reporter constr
187 Ip45 knockdown was significantly reversed by
co-transfection of siHDAC6.
188 Co-transfection of Smad3, JunB, and Cbfa1/Runx2 construc
189 Co-transfection of Smad3/Smad4 and beta-catenin expressi
190 Co-transfection of SP1 cells with PHn-CC-Ex cDNA and Tia
191 Co-transfection of syntrophin gamma 2 with SCN5A in HEK2
192 Co-transfection of Tead2 and Fgfr4 promoter reporter con
193 Co-transfection of the -514 bp human reelin promoter wit
194 In addition,
co-transfection of the alternatively spliced form of TIA
195 maximal induction of FGF8.luc required both
co-transfection of the AR and the presence of androgens.
196 Co-transfection of the B cell-specific octamer transcrip
197 Co-transfection of the CAP promoter with PPARgamma and r
198 (+)/K(+)-luciferase activity 5-fold, whereas
co-transfection of the cells with the H(+)/K(+)-luc plas
199 Co-transfection of the co-activator, SRC-1, further slow
200 d-type Galpha(i1), and was not duplicated by
co-transfection of the constitutively active Galpha(s)-Q
201 Co-transfection of the FGF-BP promoter with dominant neg
202 However,
co-transfection of the kinase-dead mutant ACK2(K158R) wi
203 Co-transfection of the L420S mutant with wild-type Olfm1
204 Co-transfection of the MAO B promoter with dominant nega
205 Furthermore,
co-transfection of the methyl-CpG binding proteins, MeCP
206 tion factor with the SLC52A1 promoter; also,
co-transfection of the minimal SLC52A1 promoter with an
207 Co-transfection of the MUC5AC-luc reporter with Sp1 expr
208 Co-transfection of the mutated SRp40 attenuated betaII e
209 Indeed,
co-transfection of the OGA-3'UTR containing reporter pla
210 Co-transfection of the parietal cells with the H(+)/K(+)
211 Co-transfection of the retained constructs with transpor
212 region of Turnip crinkle virus (TCV-hp) and
co-transfection of the virus and fusion protein construc
213 n the RNAi target sequence and observed that
co-transfection of this mutant, but not wild-type Mect1-
214 Co-transfection of TIA expression vectors with a MYPT1 m
215 In COS-1 cells, the
co-transfection of TOM1L1 and Lyn, but not Syk, resulted
216 Co-transfection of tropomyosin reporters with members of
217 e endogenous level of tuberin, and transient
co-transfection of TSC1 with TSC2 resulted in higher tub
218 Co-transfection of wild-type Arc and SRF engineered to b
219 n-of-function' mutations can be corrected by
co-transfection of wild-type EPM2A cDNA, which is consis
220 Co-transfection of WT ZEBRA with aggresome-inducing muta
221 ted when high fluorescence was obtained upon
co-transfection of ZnT5-YC and ZnT6-YN, which are known
222 Co-transfections of high- and low-yielding constructs de
223 tein levels observed previously in transient
co-transfections of p53 and S100B.
224 AP2
co-transfection results in a similar repression of the C
225 Transient
co-transfection studies demonstrate that A-SREBP-1 can i
226 Co-transfection studies demonstrated that constitutively
227 Co-transfection studies in Drosophila SL2 cells indicate
228 Results from
co-transfection studies indicated superactivation of LTR
229 In
co-transfection studies PU.1 represses MOR promoter repo
230 Biochemical and
co-transfection studies revealed that TIP120B, but not t
231 Co-transfection studies show that ARP-1/COUP-TFII repres
232 Finally, results of
co-transfection studies showed that overexpression of IK
233 Co-transfection studies with individual miR mimics along
234 In
co-transfection studies, cyclin A expression stimulated
235 Using
co-transfection studies, we showed that the NRSE of the
236 In
co-transfection studies, wild-type PTPN13 inhibited Ras/
237 significantly increased EDA transcription in
co-transfection studies.
238 Using
co-transfection techniques we investigated the functiona
239 When expressed separately by
co-transfection,
the NH2-terminal portion of Scap (conta
240 We performed a series of
co-transfections using mammalian expression vectors enco
241 Co-transfections using Pax6 and c-Maf alone revealed mod
242 Specifically, a series of
co-transfections was performed using mammalian expressio
243 y using human embryonic kidney 293 cells for
co-transfection,
we determined that a substrate-trapping
244 ed with CaMKK and stimulated with forskolin,
co-transfection with 14-3-3 prevented dephosphorylation
245 Co-transfection with a beta-catenin expression vector en
246 rom the proapoptotic effects of Sox4 RNAi by
co-transfection with a construct expressing functional S
247 eNOS promoter by shear could be prevented by
co-transfection with a dominant negative I kappa Balpha.
248 ta1 by miR-29b was partially recovered after
co-transfection with a plasmid-expressing bone morphogen
249 Generation of E10(-) RNA was restored after
co-transfection with a PTM designed to exclude E10.
250 This transformation was independent of
co-transfection with activated Ha-ras.
251 Microtubule disruption or
co-transfection with alpha-synuclein A53T, a PD-associat
252 sion were observed as follows: both required
co-transfection with alpha1 and beta2 subunits for maxim
253 ulin-stimulated GLUT4 translocation, whereas
co-transfection with an inactive PI 4-kinase mutant prev
254 ERT NF-kappaB site could also be enhanced by
co-transfection with an NF-kappaB1 expression vector.
255 Co-transfection with an uncoupled mutant alpha(1B)-AR (D
256 Co-transfection with AODNs suppressed IRBP promoter acti
257 roduced high activity that was unaffected by
co-transfection with AODNs.
258 -RAP promoter activity was down-regulated by
co-transfection with C/EBP expression vectors.
259 Co-transfection with caCaMKK plus caCaMKI also stimulate
260 Co-transfection with CB2(SH3+) and gephyrin induced the
261 Co-transfection with CB2(SH3-) or CB2(SH3+) and gephyrin
262 Co-transfection with DEC1 repressed the activity of a DE
263 Further,
co-transfection with either dominant negative Egr-1 or t
264 ighly responsive to TGF-beta1, is blocked by
co-transfection with either full-length AR or AR missing
265 y after FN-f treatment that was inhibited by
co-transfection with either of two dominant negative mut
266 Co-transfection with either v-Src or a constitutively ac
267 Upon
co-transfection with ETA into COS-7 cells, Tip60 strongl
268 Co-transfection with GRIP1 or steroid receptor coactivat
269 Co-transfection with hnRNP G represses incorporation in
270 2.5-40 ng/ml and dose-dependently induced by
co-transfection with human NFATc1 in RAW264.7 cells.
271 led to decreased reporter activity, whereas
co-transfection with miR-506 inhibitors led to enhanced
272 Co-transfection with mutant ubiquitins and treatment wit
273 lioma and human embryonic kidney cells after
co-transfection with Myc epitope-tagged EAAC1 and NMDA r
274 or, bisindolylmaleimide II, and augmented by
co-transfection with PKCbetaI.
275 3a (DNMT3a) is a major target of miR-29c and
co-transfection with premiR-29c prevented DNMT3a 3'UTR v
276 Co-transfection with prolyl 4-hydroxylase generated homo
277 However, after
co-transfection with R14A+FLAG/F714G+Myc or F64A+FLAG/F7
278 s in FAM129B knockdown cells was reversed by
co-transfection with recombinant FAM129B, indicating tha
279 s the transcriptional activity of c-Jun, but
co-transfection with rLHR prevents this effect.
280 Co-transfection with shRNA vectors against HK1, HK2, and
281 ent NT2/D1-R1 cells but can be restored upon
co-transfection with specific RARs.
282 uppressed in U3A cells but can be rescued by
co-transfection with STAT1alpha but not STAT1 mutated at
283 metalloproteinase (MT4-MMP, MMP-17) because
co-transfection with the active form of MT4-MMP markedly
284 CCL4 promoter were strongly up-regulated by
co-transfection with the C/EBPbeta expression vector.
285 Co-transfection with the different AdipoR pairs yielded
286 a constitutively active MEK1 and reduced by
co-transfection with the ERK phosphatase, HVH2.
287 Neither
co-transfection with the mutant tRNA gene nor lovastatin
288 could be induced rapidly and specifically by
co-transfection with the reporter gene and various silen
289 Co-transfection with the restriction enzymes BamHI, BglI
290 In contrast,
co-transfection with three separate IRE1alpha dominant n
291 Co-transfection with two partially complementary, trunca
292 ase (ATGL), has two FoxO1-binding sites, and
co-transfection with wild type and unphosphorylated FoxO
293 8-induced apoptosis was markedly enhanced by
co-transfection with wild type Tip60 and decreased by mu
294 Co-transfections with an Nfe2l2 expression vector and a
295 ed nuclear transport function for ferritoid,
co-transfections with full-length constructs for ferrito
296 Use of inhibitor, SM7368 and
co-transfections with IkappaBalpha, RelA/p50 showed that
297 Co-transfections with MAP-2c and the extracellular signa
298 In addition,
co-transfections with mutants of Ras, Raf, and MEK showe
299 Co-transfections with the pLucRLuc:p53/Wrap53beta bidire
300 Co-transfections with wild-type VWF partially corrected