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1 ufficiency, single-ventricle physiology, and coagulation disorder.
2 T) is associated with risk of thrombosis and coagulation disorders.
3 may have detrimental effects associated with coagulation disorders.
4 ce in patients with F10 deficiency and other coagulation disorders.
5 rections and decisions in surgery and innate coagulation disorders.
6 r in characterization and treatment of blood coagulation disorders.
8 Lonomia obliqua are often associated with a coagulation disorder and hemorrhagic syndrome in humans.
9 ing 220 plasma samples of patients suffering coagulation disorders and 80 plasma samples of non-patie
11 rogens, 11 (20%) had a myeloproliferative or coagulation disorder, and in 7 (13%) the cause remained
12 ire ebolavirus (ZEBOV) leads to hypotension, coagulation disorders, and an impaired immune response a
14 , before the application of gene therapy for coagulation disorders becomes widespread, several obstac
15 nd had progressive cholestasis and jaundice, coagulation disorders, bilateral ureterostomies, repetit
17 casts new light on our understanding of the coagulation disorder found in P. falciparum infection.
19 This rational approach, applicable to other coagulation disorders, helps with interpreting the poor
21 n in its clinical application to the complex coagulation disorders of acute and chronic liver disease
23 factor (TF) is thought to play a role in the coagulation disorders that characterize filoviral infect
24 cently identified a class of diseases--blood coagulation disorders--that were associated with a 14-fo
25 used in the ICU have the potential to cause coagulation disorders, the exact incidence will vary bas
26 tory mediators and markers of autoimmune and coagulation disorders with cerebral palsy (CP), examinin
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