ライフサイエンスコーパス: coagulation factor V

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1 P=2.4x10(-14)) was 79.7 kb downstream of F5, coagulation factor V.

2 A point mutation in coagulation factor V (A1691G) is associated with increas

3 ins of intermediate abundance were selected, coagulation factor V, adiponectin, C-reactive protein (C

4 eptides were 17.0, 25.4, 24.2, and 14.0% for coagulation factor V, adiponectin, CRP, and thyroxine bi

5 ing) were 9.2, 110, 120, and 246 pmol/mL for coagulation factor V, adiponectin, CRP, and thyroxine bi

6 man genetic disorder, combined deficiency of coagulation factor V and factor VIII.

7 a type II (CDAII) and combined deficiency of coagulation factors V and VIII (F5F8D) are the 2 known h

8 lopathy characterized by inactivation of the coagulation factors V and VIII and a derepression of the

9 ng disorder associated with plasma levels of coagulation factors V and VIII approximately 5% to 30% o

10 The discoidin C2 domains of coagulation factors V and VIII are known to interact wit

11 ve bleeding disorder, combined deficiency of coagulation factors V and VIII.

12 cific subset of secreted proteins, including coagulation factors V and VIII.

13 MAD4, INPP5D, and IRAK3; and a disruption of coagulation factors V and VIII.

14 The activation of coagulation factors V and X by Russell's viper venom (RV

15 eral substrates, namely mucin, pepsin, human coagulation factor V, and erythroid spectrin.

16 Blood coagulation factor V circulates as a procofactor with li

17 These genes include human coagulation factor V (F5), Weel protein tyrosine kinase

18 A polymorphism in coagulation factor V, factor V Leiden (FVL), is the majo

19 Affected individuals have normal levels of coagulation factor V (FV) activity, but demonstrate inhi

20 Coagulation factor V (FV) circulates as an inactive proc

21 c donor splice site in a patient with severe coagulation factor V (FV) deficiency and life-threatenin

22 ion, which endocytoses fluorescently labeled coagulation factor V (FV) from the media into alpha-gran

23 Coagulation factor V (FV) is a central regulator of the

24 Activation of blood coagulation factor V (FV) is a key reaction of hemostasi

25 binase assembly by directly interacting with coagulation factor V (FV), which has been activated by F

26 ed that Gal8 may also interact with platelet coagulation factor V (FV).

27 The coagulation factors V (FV) and VIII (FVIII) are importan

28 The C domains of coagulation factors V (FV) and VIII (FVIII) are structur

29 protein 2) cargo receptor complex transports coagulation factors V (FV) and VIII (FVIII) from the end

30 s soluble coreceptor MCFD2, LMAN1 transports coagulation factors V (FV) and VIII (FVIII).

31 -derived prothrombin activator homologous to coagulation factors V (FV) and Xa (FXa).

32 A genomic target, a region in the human coagulation Factor V gene (226-bp), was subsequently dir

33 ed a higher risk of severe preeclampsia with coagulation factor V gene (proaccelerin, labile factor)

34 gene, the Coagulation Factor II gene and the Coagulation Factor V gene.

35 Coagulation factor V is a critical cofactor for the acti

36 In an essential step of blood coagulation, factor V is proteolytically processed by th

37 nt use of HRT and the presence or absence of coagulation factor V Leiden and prothrombin 20210 G-->A

38 Deficiency of blood coagulation factor V or tissue factor causes the death o

39 of the method to a tryptic digest of bovine coagulation factor V resulted in identification of sulfa

40 single point mutation in the gene coding for coagulation factor V results in a form of factor Va that

41 h tracks with reduced plasma levels of blood coagulation factors V, VII, VIII, IX, X, and XII.

42 two Npn-1 CUB domains and the amino-terminal coagulation factor V/VIII domain (CF V/VIII) are essenti

43 collapse; antibody (YW107.4.87) binds to the coagulation factor V/VIII domains (b1b2) of NRP1 and blo

44 1691A Leiden mutation in the gene coding for coagulation factor V was determined in the PHS group usi

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