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1 Ab1 showed anticoagulant activity by global coagulation tests.
2 tivity, but demonstrate inhibition of global coagulation tests.
3 gulation and other analytical limitations of coagulation tests.
4 f normal cFIX antigen levels, which improved coagulation tests.
5 The method could extend and improve current coagulation testing.
6 etry panels, parathyroid hormone assays, and coagulation testing.
7 bin 16 g/dl), with normal liver function and coagulation testing.
9 anding of TSOA pharmacology, their effect on coagulation tests and, hence, a correct interpretation o
10 nificant coagulopathy (as defined by routine coagulation tests) and is used to justify preprocedure u
11 itory effect on PL-restricted in vitro blood coagulation tests, and are comprised mainly of Ab agains
12 val, expression levels, in vitro and in vivo coagulation tests, and histopathology for up to 16 month
13 s been reinforced by the fact that screening coagulation tests (APTT, prothrombin time--INR) are ofte
17 with TBI have abnormalities on conventional coagulation tests at admission to the emergency departme
18 lation has been difficult using conventional coagulation tests, but thrombocytopenia, fibrin polymeri
19 , although some of the more widely available coagulation tests can provide information that is potent
27 ma transfusion in either correcting abnormal coagulation tests or reducing perceived risk of hemorrha
28 1% (10 of 123) of patients with any abnormal coagulation test results and 9.7% (85 of 877) of patient
30 clinicians in the setting of mildly abnormal coagulation test results, and there is no evidence that
32 ancer range from asymptomatic basic abnormal coagulation tests to massive clinical thromboembolism, w
37 dministered to address abnormal preoperative coagulation tests, with the hope to mitigate bleeding co
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