ライフサイエンスコーパス: coagulative

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1 with inflammatory and acute-phase responses, coagulative activities, and transition metal ion sequest

2 se swelling of cardiomyocytes and multifocal coagulative contraction band necrosis.

3 Disse; RFA-treated liver tissue demonstrated coagulative destruction of hepatocyte organelles within

4 erplasia along with multifocal to coalescing coagulative hepatocyte necrosis.

5 ts (1984 to 1991) by grading the severity of coagulative myocyte necrosis (CMN) as absent, 0; mild-fo

6 e liver pathology characterized by extensive coagulative necrosis and an increase in hepatic IFN-gamm

7 on, and spotty necrosis, and induced diffuse coagulative necrosis and centrilobular fibrosis in some

8 ited severe liver pathology, with regions of coagulative necrosis and hepatocyte vacuolization unappa

9 tissues exhibited microvascular thrombosis, coagulative necrosis and similar levels of platelet and

10 cimens at the day of ablation revealed acute coagulative necrosis associated with intense basophilic

11 ith this parasite develop extensive areas of coagulative necrosis in the liver, and newborn larvae ar

12 Coagulative necrosis of cells was the most frequently oc

13 Within 24 hours, complete coagulative necrosis of the entire kidney occurred as a

14 interstitial by chronic inflammatory cells, coagulative necrosis of the kidney, spleen, intestine an

15 ndothelial cell damage has been described as coagulative necrosis secondary to irreversible physico-c

16 n the absence of microvascular thrombosis or coagulative necrosis that developed later.

17 ckout mice had reduced mortality and massive coagulative necrosis was observed in wild-type mice.

18 xhibited mainly microvascular thrombosis and coagulative necrosis with little evidence of interstitia

19 lymphoplasmacytic hepatitis with hepatocytic coagulative necrosis, but the hydrogenase mutants exhibi

20 ns ranged from well-organized tubercles with coagulative necrosis, epithelioid macrophages, and fibro

21 us degrees of ulceration, hemorrhage, edema, coagulative necrosis, perivascular inflammation, and vas

22 to induction of cytokine release and massive coagulative necrosis.

23 and epitheliotropism and irregular areas of coagulative necrosis.

24 infiltrates, Schwann cell proliferation, and coagulative necrosis.

25 ng the vasculature from pro-inflammatory and coagulative stresses.

26 C death after adjusting for nuclear grade or coagulative tumor necrosis.

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