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1 udy involving a very complex sample, namely, coal tar.
2 ture of polyaromatic hydrocarbons (PAH) from coal tar.
3 r component of the antipsoriatic activity of coal tar.
4 ory animals by repeatedly painting them with coal tar.
5  optimal method for source identification of coal tars.
6 from AD patients and controls, we found that coal tar activated the aryl hydrocarbon receptor (AHR),
7                                 Profiling of coal tar and crude oil by automated sequential GC-GC/MS
8                  To obtain these data, eight coal tar and crude oils were analyzed by automated seque
9  was induced by ultraviolet radiation, PUVA, coal tar, and all-trans retinoic acid; expression was si
10 istinct lithological units contaminated with coal tar at a former industrial facility.
11 ted with common PAH sources (fuel oil, soot, coal tar based skeet particles) and direct spike with a
12                                  Runoff from coal-tar-based (CT) sealcoated pavement is a source of p
13           Recent studies have concluded that coal-tar-based pavement sealants are a major source of p
14    Our results indicate that the presence of coal-tar-based pavement sealants is associated with sign
15 nd soil adjacent to parking lots sealed with coal-tar-based products.
16 nd soil adjacent to parking lots sealed with coal-tar-based products.
17 most recently, air-contaminated by PAHs from coal-tar-based sealcoat and to demonstrate potential ris
18                                              Coal-tar-based sealcoat products, widely used in the cen
19 ns in air over pavement with freshly applied coal-tar-based sealcoat, for example, were hundreds to t
20                              In AD patients, coal tar completely restored expression of major skin ba
21 s enabled the separation of three classes of coal tar compounds: (1) nonaromatic hydrocarbons; (2) un
22  dominant in situ degrader of naphthalene in coal tar-contaminated sediments.
23 yclic aromatic sulfur heterocycles (PASH) in coal tar-contaminated soil.
24 d for differential blood cell counting using coal tar dyes and mentions the eosinophil for the first
25 andard Reference Materials (SRMs) (SRM 1597, coal tar extract; SRM 1648 and SRM 1649a, air particulat
26      Treatment of SVR endothelial cells with coal tar fractions resulted in the isolation of a single
27                                              Coal tar has been directly applied to the skin, or used
28                                   The use of coal tar has caused long-term remissions in psoriasis, b
29 ces of lung tumors were observed in mice fed coal tar in their diet.
30 ironmental forensic source identification of coal tars, including the ability to predict the processe
31                                              Coal tar interfered with Th2 cytokine signaling via deph
32                              We fractionated coal tar into its components, and tested them using the
33 ds advances our understanding of why topical coal tar is an effective treatment for atopic dermatitis
34        The active antiangiogenic compound in coal tar is carbazole.
35 mining the active antipsoriatic component of coal tar is of considerable therapeutic interest.
36                                              Coal tar is one of the oldest and an effective treatment
37                       Topical application of coal tar is one of the oldest therapies for atopic derma
38 e the treatment requires hospitalization and coal tar is poorly acceptable aesthetically to patients.
39  a simpler one-parameter prediction assuming coal tar-like organic carbon performed equally well in e
40 cially at former industrial facilities where coal tar-oil was handled, e.g., wood treatment plants, h
41 ch no standards exist, sediments impacted by coal tar, or spiked with a coal tar/petroleum nonaqueous
42                        A total of 3700 kg of coal tar over 12 days in the shallow test and 860 kg ove
43 ments impacted by coal tar, or spiked with a coal tar/petroleum nonaqueous phase liquid (NAPL) were a
44 ne using pure standards, and the other using coal tar/petroleum-contaminated sediments) and agreed ve
45 , and even playgrounds, are typically 20-35% coal-tar pitch, a known human carcinogen that contains a
46                       Soil contaminated with coal tar (prebioremediation) from a former manufactured
47  pyrogenic-impacted areas sorbs similarly to coal tar, rather than octanol as typically assumed.
48                                              Coal tar restored filaggrin expression in FLG-haploinsuf
49                             For example, the coal tar S-containing compounds were pinpointed through
50 t chromatographic separation, in a reference coal-tar sample is made possible with the combination of
51   Compositional disparity within a set of 23 coal tar samples (obtained from 15 different former manu
52 s been paid to the presence of seven PAHs in coal-tar samples, namely, benz[a]anthracene, benzo[k]-fl
53 pproach for the routine analysis of numerous coal-tar samples.
54 ic, high-molecular weight PAHs (HMW-PAHs) in coal-tar samples.
55 urce-receptor modeling results indicate that coal-tar sealants remain the largest PAH source to the l
56 t jurisdiction in the U.S. to ban the use of coal-tar sealants.
57                                 We applied a coal tar sealcoat to conventional asphalt and collected
58                                              Coal tar sealcoats applied to asphalt surfaces in North
59 ingestion exposures to PAHs in settings near coal-tar-sealed pavement.
60                               The culprit in coal tar that induces cancer was finally isolated in 193
61 use and efficacy, the molecular mechanism of coal tar therapy is unknown.
62  with Th2 cytokines IL-4 and IL-13, we found coal tar to diminish spongiosis, apoptosis, and CCL26 ex
63 ected FMGP tars and a commercially available coal tar were characterized by means of fractionation, g

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