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1 udy involving a very complex sample, namely, coal tar.
2 ture of polyaromatic hydrocarbons (PAH) from coal tar.
3 r component of the antipsoriatic activity of coal tar.
4 ory animals by repeatedly painting them with coal tar.
5 optimal method for source identification of coal tars.
6 from AD patients and controls, we found that coal tar activated the aryl hydrocarbon receptor (AHR),
9 was induced by ultraviolet radiation, PUVA, coal tar, and all-trans retinoic acid; expression was si
11 ted with common PAH sources (fuel oil, soot, coal tar based skeet particles) and direct spike with a
14 Our results indicate that the presence of coal-tar-based pavement sealants is associated with sign
17 most recently, air-contaminated by PAHs from coal-tar-based sealcoat and to demonstrate potential ris
19 ns in air over pavement with freshly applied coal-tar-based sealcoat, for example, were hundreds to t
21 s enabled the separation of three classes of coal tar compounds: (1) nonaromatic hydrocarbons; (2) un
24 d for differential blood cell counting using coal tar dyes and mentions the eosinophil for the first
25 andard Reference Materials (SRMs) (SRM 1597, coal tar extract; SRM 1648 and SRM 1649a, air particulat
30 ironmental forensic source identification of coal tars, including the ability to predict the processe
33 ds advances our understanding of why topical coal tar is an effective treatment for atopic dermatitis
38 e the treatment requires hospitalization and coal tar is poorly acceptable aesthetically to patients.
39 a simpler one-parameter prediction assuming coal tar-like organic carbon performed equally well in e
40 cially at former industrial facilities where coal tar-oil was handled, e.g., wood treatment plants, h
41 ch no standards exist, sediments impacted by coal tar, or spiked with a coal tar/petroleum nonaqueous
43 ments impacted by coal tar, or spiked with a coal tar/petroleum nonaqueous phase liquid (NAPL) were a
44 ne using pure standards, and the other using coal tar/petroleum-contaminated sediments) and agreed ve
45 , and even playgrounds, are typically 20-35% coal-tar pitch, a known human carcinogen that contains a
50 t chromatographic separation, in a reference coal-tar sample is made possible with the combination of
51 Compositional disparity within a set of 23 coal tar samples (obtained from 15 different former manu
52 s been paid to the presence of seven PAHs in coal-tar samples, namely, benz[a]anthracene, benzo[k]-fl
55 urce-receptor modeling results indicate that coal-tar sealants remain the largest PAH source to the l
62 with Th2 cytokines IL-4 and IL-13, we found coal tar to diminish spongiosis, apoptosis, and CCL26 ex
63 ected FMGP tars and a commercially available coal tar were characterized by means of fractionation, g
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