ライフサイエンスコーパス: coaptation

1 d annulus; or between TV leaflets, improving coaptation.

2 weeks, correlating to a compromised leaflet coaptation.

3 act and mitral annulus that enhanced leaflet coaptation.

4 ective tissues in the urethral wall also aid coaptation.

5 mia by reducing leaflet tethering to improve coaptation.

6 ormal valve morphology or incomplete leaflet coaptation.

7 te coaptation, and decreased the duration of coaptation.

8 was the result of a lack of complete leaflet coaptation.

9 ther posterior MVP (12) or abnormal anterior coaptation (5).

10 al leaflet closure (IMLC: apically displaced coaptation) also have restricted diastolic leaflet openi

11 cal interference with TV leaflet mobility or coaptation and is amenable to lead extraction or valve r

12 minimum number of basal chordae can improve coaptation and reduce ischemic MR.

13 to the correct length to restore MV leaflet coaptation and secured at the epicardium.

14 I+PNT, suggesting that urethral mucosal seal coaptation and tissue elasticity also contribute to cont

15 systolic regurgitant volume before complete coaptation, and decreased the duration of coaptation.

16 ameter (TAD), apical displacement of leaflet coaptation, and estimated PA systolic pressure were dete

17 lation (size), the mode of tricuspid leaflet coaptation, and tricuspid leaflet tethering-factors ofte

18 s annular dilatation, which hindered leaflet coaptation by drawing the individual scallops apart.

19 pheral innervations and control, and mucosal coaptation contribute to maintenance of continence.

20 +/- 16% vs. 42 +/- 16%; p = 0.0001) and less coaptation deficiency (1.06 +/- 0.24 for repaired and 1.

21 y (change in systolic to diastolic area) and coaptation deficiency (conjoint and reference cusp heigh

22 al anterior leaflet angle (P<0.001), greater coaptation depth and tenting area (P<0.001), larger left

23 let angle (ALAtip), posterior leaflet angle, coaptation depth, tenting area, mitral annular dimension

24 erity, annular dilation, and mode of leaflet coaptation (extent of tethering).

25 al anterior coaptation phenotype (n=10, with coaptation height >40% of the annulus similar to posteri

26 had greater leaflet displacement, thickness, coaptation height, and mitral regurgitation jet height (

27 terior leaflet projections onto the annulus, coaptation height, and mitral regurgitation jet height i

28 rea (-0.57 +/- 1.1 cm(2)) and increase in MV coaptation length (0.13 +/- 0.2 cm).

29 tenting height (P<0.01) were decreased, and coaptation length was increased (P<0.05) after TAVR.

30 he boundary zone near the annulus and at the coaptation line, with reduced strain concentration in th

31 ons that aimed for functional restoration by coaptation of all main available motor and sensory nerve

32 red to cause chronic AR by preventing proper coaptation of the 2 aortic valve cusps.

33 After coaptation of the severed nerve ends, fine wire electrod

34 1), longer lengths from papillary muscles to coaptation (P<0.0001), and more frequent chordal rupture

35 isplacement (n=50) and the abnormal anterior coaptation phenotype (n=10, with coaptation height >40%

36 g was measured from the annulus plane to the coaptation point and tethering area by tracing the leafl

37 no change in the distance between the mitral coaptation point and the septum, as measured in two plan

38 his reflects a more anterior position of the coaptation point in those who developed SAM/ LVOTO.

39 The position of the coaptation point of the mitral leaflets is dynamic and a

40 rior (PL) leaflets and the distance from the coaptation point to the septum (C-Sept) were measured be

41 8 (80%) participants with abnormal anterior coaptation progressed to posterior MVP; 17 (34%) subject

42 MR in TIC resulted from decreased leaflet coaptation secondary to annular dilatation in the septal

43 ng angle (P<0.001) despite a similar leaflet coaptation status compared with patients with left-sided

44 minimal annular dilatation distorts leaflet coaptation sufficiently to produce severe mitral regurgi

45 nted, with recent trends to preserve leaflet coaptation surfaces if possible.

46 ce implantation to improve tricuspid leaflet coaptation, thereby reducing TR.

47 gned to provide a surface for native leaflet coaptation to reduce tricuspid regurgitation (TR) by occ

48 n in the control group; furthermore, leaflet coaptation was displaced more apically (5.6+/-2.2 mm, P<

49 reate a double orifice with improved leaflet coaptation was introduced in the early 1990s.

50 ent displaying sufficient leaflet motion and coaptation with only minor paravalvular leakage in some

51 hest strain at the commissures, annulus, and coaptation zones.